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Objective: To report a rare case of intralabyrinthine schwannoma extending to the tympanic cavity and undergoing surgical management. Case report: A 56-year-old man presented with left deafness, who had been treated since 1990 for left otitis media and hearing loss with tympanic perforations. In 2005, he was suspected of neurogenic tumor in a histopathologic examination of granulomatous tissues on the typmpanic perforations and referred to us for diagnosis and management. On presentation, the patient was deaf in the left ear and had left tympanic perforation with yellowish-white tumor lesion in the left perforated area. The tumor had an apparent origin in the cochlear to vestibule, which was separate from the facial nerve, destroyed the promontory bone, and protruded into the tympanic cavity. The patient underwent a partial resection of the tumor which extended to the tympanic cavity and closure of tympanic perforation. Histopathologic examination of the tumor was consistent with schwannoma. A subsequent follow-up has not shown increase of the tumor. Conclusions: Reports of intralabyrinthine schwannoma have increased in recent years. The disease is uncommon, and the case reported herein is extremely rare with extension of the tumor to the tympanic cavity. Indication for the surgical treatment of intralabyrinthine schwannoma should be determined in terms of postoperative complications. [ABSTRACT FROM AUTHOR]

Conclusions . The clinical and surgical findings of this study indicated advanced cholesteatoma in many patients with facial paralysis. The outcome of facial paralysis was good. Poor outcomes were observed in cases with petrosal cholesteatoma and in those who underwent surgery ≥2 months after the onset of paralysis. Objective . To investigate clinical features of cholesteatoma associated with facial paralysis. Material and methods . Sixteen patients with facial paralysis due to middle ear cholesteatoma were reviewed. After removal of the cholesteatoma lesion, a limited area of the fallopian canal, that in which facial nerve edema or redness was evident, was opened. Incision of the epineural sheath for nerve decompression was not performed. Results . Initial paralysis was incomplete in 11 patients (69%). The onset of paralysis was sudden in 12 patients (75%). Labyrinthine fistulae ( n =9; 56%) and bone destruction in the cranial fossa ( n =10; 63%) were frequently observed. Six patients (38%) were totally deaf due to labyrinthitis. The outcome of facial paralysis was good in 13 patients (81%). Patients who underwent surgery ≥2 months after the onset of paralysis frequently had a poor outcome. Paralysis was not improved in two cases with petrosal cholesteatoma. [ABSTRACT FROM AUTHOR]