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In order to study the degradation of herbicide----Quinclorac using microorganism, separate and screen the degrading bacteria CSUFTM62 from soil, this paper researched the degradation effect of bacterial strain on the Quinclorac under different situations, including different increments, different temperatures, different concentrations of pesticide, different pH, and different additive amount of nutrients. The results show that the optimal degradation temperature is 30 ° C; the optimum pH is 7; the degradation rate of Quinclorac reaches its maximum when the concentration is 50mg / L. Adding yeast extract could increase the amount of strain growth but never affect its degradation effects on Quinclorac; combined with the morphological characteristics, the strains CSUFTM62 is identified as the Stenotrophomonas maltophilia by measuring the 16S sequence.

In order to explore the relevant molecular genetic mechanisms of photosynthetic rate (PR) and chlorophyll content (CC) in rice (Oryza sativa L.), we conducted a series of related experiments using a population of recombinant inbred lines (Zhenshan97B x IRAT109). We found a significant correlation between CC and PR (R= 0.19**) in well-watered conditions, but no significant correlation during water stress (r= 0.08). We detected 13 main quantitative trait loci (QTLs) located on chromosomes 1, 2, 3, 4, 5, 6, and 10, which were associated with CC, including six QTLs located on chromosomes 1, 2, 3, 4, and 5 during water stress, and seven QTLs located on chromosomes 2, 3, 4, 6, and 10 in well-watered conditions. These QTLs explained 47.39% of phenotypic variation during water stress and 56.19% in well-watered conditions. We detected four main QTLs associated with PR; three of them (qPR2, qPR10, qPR11) were located on chromosomes 2, 10, and 11 during water stress, and one (qPR10) was located on chromosome 10 in well-watered conditions. These QTLs explained 34.37% and 18.41% of the phenotypic variation in water stress and well-watered conditions, respectively. In total, CC was largely controlled by main QTLs, and PR was mainly controlled by epistatic QTL pairs.