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2. Canonical and noncanonical functions of complement in systemic lupus erythematosus.

3. Factor H–Related Protein 1 Drives Disease Susceptibility and Prognosis in C3 Glomerulopathy

4. Homodimeric Minimal Factor H: In Vivo Tracking and Extended Dosing Studies in Factor H Deficient Mice.

5. Plasma Lectin Pathway Complement Proteins in Patients With COVID-19 and Renal Disease.

6. Gain-of-function factor H-related 5 protein impairs glomerular complement regulation resulting in kidney damage.

7. Temporal changes in complement activation in haemodialysis patients with COVID-19 as a predictor of disease progression.

8. Complement Factor H Modulates Splenic B Cell Development and Limits Autoantibody Production.

9. Complement factor H protects mice from ischemic acute kidney injury but is not critical for controlling complement activation by glomerular IgM.

10. Update on C3 glomerulopathy.

11. Dense Deposit Disease and C3 Glomerulopathy.

12. Recent insights into C3 glomerulopathy.

13. Ultraviolet-Radiation-Induced Keratinocyte Apoptosis in C1q-Deficient Mice.

14. Complement factor H-deficient mice develop spontaneous hepatic tumors.

15. Conversion of the Liver into a Biofactory for DNaseI Using Adeno-Associated Virus Vector Gene Transfer Reduces Neutrophil Extracellular Traps in a Model of Systemic Lupus Erythematosus.

16. Adeno-Associated Virus Vector Gene Delivery Elevates Factor I Levels and Downregulates the Complement Alternative Pathway In Vivo.

17. Hyperfunctional complement C3 promotes C5-dependent atypical hemolytic uremic syndrome in mice.

18. Binding of factor H to tubular epithelial cells limits interstitial complement activation in ischemic injury.

19. Treatment with human complement factor H rapidly reverses renal complement deposition in factor H-deficient mice.

20. Complement in human diseases: Lessons from complement deficiencies

21. Crry deficiency in complement sufficient mice: C3 consumption occurs without associated renal injury

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