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1. Hypothalamic and hindbrain NPY, AGRP and NE increase consummatory feeding responses.

2. Basomedial hypothalamic injections of neuropeptide Y conjugated to saporin selectively disrupt hypothalamic controls of food intake.

3. Glucoprivation increases expression of neuropeptide Y mRNA in hindbrain neurons that innervate the hypothalamus.

4. Leptin receptor-expressing neurons in ventromedial nucleus of the hypothalamus contribute to weight loss caused by fourth ventricle leptin infusions.

5. Leptin-sensitive neurons in the arcuate nucleus integrate activity and temperature circadian rhythms and anticipatory responses to food restriction.

6. Leptin-sensitive neurons in the arcuate nuclei contribute to endogenous feeding rhythms.

7. Localized glucoprivation of hindbrain sites elicits corticosterone and glucagon secretion.

8. Hypotensive hypovolemia and hypoglycemia activate different hindbrain catecholamine neurons with projections to the hypothalamus.

9. Hindbrain catecholamine neurons mediate consummatory responses to glucoprivation

10. Repeated 2-deoxy-D-glucose-induced glucoprivation attenuates Fos expression and glucoregulatory responses during subsequent glucoprivation.

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