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Your search keyword '"Kim, Ji-Soo"' showing total 5 results
Start Over Author "Kim, Ji-Soo" Topic magnetic resonance imaging Topic nystagmus
5 results on '"Kim, Ji-Soo"'

1. Isolated labyrinthine infarction as a harbinger of anterior inferior cerebellar artery territory infarction with normal diffusion-weighted brain MRI

Author

Kim, Ji Soo, Cho, Kyung-Hee, and Lee, Hyung

Subjects
*INFARCTION, *INNER ear diseases, *MAGNETIC resonance imaging, *CEREBROVASCULAR disease, *DEAFNESS, *VERTIGO, *NYSTAGMUS
Abstract

Abstract: Objectives: This paper aims to determine the frequency of isolated labyrinthine infarction as an initial manifestation of anterior inferior cerebellar artery (AICA) territory infarction and discuss its clinical implication. Methods: We studied 54 consecutive patients with AICA infarction diagnosed by brain MRI from the acute stroke registry. Results: We identified 4 patients (7.4%) with AICA territory infarction who initially presented with vertigo and hearing loss mimicking acute labyrinthitis and subsequently suffered from delayed neurological deficits. All patients had normal brain MRIs including diffusion-weighted images at the time of isolated audiovestibular loss, but all subsequently exhibited acute infarct(s) in the AICA territory on follow-up brain MRIs when they developed additional neurological deficits. Conclusion: Labyrinthine infarction may be a warning sign of impending pontocerebellar infarction in the AICA territory. Our results indicate that even a classical peripheral presentation, e.g., acute hearing loss, and acute vertigo with unidirectional nystagmus, may be a manifestation of ischemic stroke in the posterior circulation. [Copyright &y& Elsevier]

Published
2009
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3. Evolution of abnormal eye movements in Wernicke's encephalopathy: Correlation with serial MRI findings

Author

Kim, Kitae, Shin, Dong Hoon, Lee, Yeong-Bae, Park, Kee-Hyung, Park, Hyeon-Mi, Shin, Dong-Jin, and Kim, Ji-Soo

Subjects
*EYE movement disorders, *WERNICKE'S encephalopathy, *MAGNETIC resonance imaging, *STEM cell transplantation, *ACUTE myeloid leukemia, *NYSTAGMUS
Abstract

Abstract: A 33-year-old woman with Wernicke''s encephalopathy (WE) due to poor oral intake after allogeneic stem cell transplantation for acute myeloid leukemia showed a sequential development of bilateral gaze-evoked nystagmus (GEN), rightward gaze palsy, and upbeat nystagmus. Initial MRIs obtained when she had GEN only showed a lesion involving the medullary tegmentum, and follow-up MRIs revealed additional lesions in the pontine and midbrain tegmentum along with development of rightward gaze palsy, and finally bilateral medial thalamus lesions in association with upbeat nystagmus. The evolution of abnormal ocular motor findings and serial MRI changes in our patient with WE provide imaging evidence on relative vulnerability of the neural structures, and on the progression of lesions and ocular motor findings in thiamine deficiency. [Copyright &y& Elsevier]

Published
2012
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4. Pure upbeat nystagmus in association with bilateral internuclear ophthalmoplegia

Author

Choi, Jae-Hwan, Jung, Na-Yeon, Kim, Min-Ji, Choi, Kwang-Dong, Kim, Ji Soo, and Jung, Dae-Soo

Subjects
*NYSTAGMUS, *EYE paralysis, *MAGNETIC resonance imaging, *BRAIN imaging, *PONS Varolii, *EYE movement disorders
Abstract

Abstract: A 66-year-old man developed primary position upbeat nystagmus and bilateral internuclear ophthalmoplegia (INO). Video-oculography showed primary position upbeat nystagmus with exponentially decreasing slow phases, which disappeared in darkness. Brain MRI disclosed enhancing lesions involving bilateral dorsomedial pons extending from the middle to upper portion. Upbeat nystagmus in association with bilateral INO may be attributed by the damage of the cell groups of the paramedian tracts (PMT), the projections from the interstitial nucleus of Cajal (INC) to PMT, or the connections between INC and the nucleus of Roller. [Copyright &y& Elsevier]

Published
2012
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5. Vestibular paroxysmia: Diagnostic criteria

Author

Dominik Straumann, Michael Strupp, Ji Soo Kim, Thomas Brandt, Jose A. Lopez-Escamez, Joanna C. Jen, John P. Carey, Alexandre Bisdorff, [Strupp, Michael] Univ Munich, Univ Hosp Munich, Dept Neurol, Munich, Germany, [Brandt, Thomas] Univ Munich, Univ Hosp Munich, Dept Neurol, Munich, Germany, [Strupp, Michael] Univ Munich, Univ Hosp Munich, German Ctr Vertigo & Balance Disorders, Munich, Germany, [Brandt, Thomas] Univ Munich, Univ Hosp Munich, German Ctr Vertigo & Balance Disorders, Munich, Germany, [Lopez-Escamez, Jose A.] Univ Granada, Otol & Neurotol Grp CTS495, Dept Genom Med,PTS, Ctr Genom & Oncol Res,Pfizer,Junta Andalucia GENy, E-18071 Granada, Spain, [Lopez-Escamez, Jose A.] Univ Hosp Granada, Dept Otolaryngol, Granada, Spain, [Kim, Ji-Soo] Seoul Natl Univ, Bundang Hosp, Coll Med, Dept Neurol, Seoul, South Korea, [Straumann, Dominik] Univ Zurich, Univ Zurich Hosp, Dept Neurol, Zurich, Switzerland, [Jen, Joanna C.] Univ Calif Los Angeles, Dept Neurol & Neurobiol, Los Angeles, CA USA, [Carey, John] Johns Hopkins Univ, Sch Med, Dept Otorhinolaryngol, Baltimore, MD USA, [Bisdorff, Alexandre] Ctr Hosp Emile Mayrisch, Dept Neurol, Esch, Luxembourg, and Federal Ministry of Education and Research

Subjects
Male, Disabling positional vertigo, Aura, Nystagmus, 0302 clinical medicine, attacks, Vertigo, Prevalence, Benign Paroxysmal Positional Vertigo, 030223 otorhinolaryngology, Stroke, Excitation, Vestibular system, Superior canal dehiscence, 8th cranial nerve, biology, Episodic ataxia type-2, General Neuroscience, ICVD, Vestibulocochlear Nerve, Magnetic Resonance Imaging, Cerebellopontine angle, Sensory Systems, Carbamazepine, Vestibular Diseases, Anesthesia, Head Movements, Female, medicine.symptom, Otologic Surgical Procedures, Benign paroxysmal positional vertigo, neurovascular compression, Oxcarbazepine, Asymptomatic, Diagnosis, Differential, 03 medical and health sciences, otorhinolaryngologic diseases, medicine, Humans, Medical history, Vascular compression, dizziness, business.industry, Recurrent attacks, Vestibular Function Tests, medicine.disease, biology.organism_classification, Otorhinolaryngology, Symptoms, Neurology (clinical), business, Microvascular decompression, 030217 neurology & neurosurgery
Abstract

This paper describes the diagnostic criteria for vestibular paroxysmia (VP) as defined by the Classification Committee of the Barany Society. The diagnosis of VP is mainly based on the patient history and requires: A) at least ten attacks of spontaneous spinning or non-spinning vertigo; B) duration less than 1 minute; C) stereotyped phenomenology in a particular patient; D) response to a treatment with carbamazepine/oxcarbazepine; and F) not better accounted for by another diagnosis. Probable VP is defined as follows: A) at least five attacks of spinning or non-spinning vertigo; B) duration less than 5 minutes; C) spontaneous occurrence or provoked by certain head-movements; D) stereotyped phenomenology in a particular patient; E) not better accounted for by another diagnosis.Ephaptic discharges in the proximal part of the 8th cranial nerve, which is covered by oligodendrocytes, are the assumed mechanism. Important differential diagnoses are Meniere's disease, vestibular migraine, benign paroxysmal positional vertigo, epileptic vestibular aura, paroxysmal brainstem attacks (in multiple sclerosis or after brainstem stroke), superior canal dehiscence syndrome, perilymph fistula, transient ischemic attacks and panic attacks. Current areas of uncertainty in the diagnosis of VP are: a) MRI findings of vascular compression which are not diagnostic of the disease or predictive for the affected side because they are also observed in about 30% of healthy asymptomatic subjects; and b) response to treatment with carbamazepine/ oxcarbazepine supports the diagnosis but there are so far no randomized controlled trials for treatment of VP.

Published
2016

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