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36 results on '"Lambert E"'

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1. A 17-Mer self-peptide of acetylcholine receptor binds to B cell MHC class II, activates helper T cells, and stimulates autoantibody production and electrophysiologic signs of myasthenia gravis.

2. Recombinant human acetylcholine receptor alpha-subunit induces chronic experimental autoimmune myasthenia gravis.

3. Pathological mechanisms in experimental autoimmune myasthenia gravis. II. Passive transfer of experimental autoimmune myasthenia gravis in rats with anti-acetylcholine recepotr antibodies.

4. Genetic control of autoimmunity to acetylcholine receptors: role of Ia molecules.

5. Study of long-term anticholinesterase therapy. Effects on neuromuscular transmission and on motor end-plate fine structure.

6. Myasthenia gravis in two cats.

7. Congenital canine myasthenia gravis: II. Acetylcholine receptor metabolism.

8. Selected IgG rapidly induces Lambert-Eaton myasthenic syndrome in mice: complement independence and EMG abnormalities.

9. Synthetic peptide of human acetylcholine receptor alpha-subunit sequence 125-147 (methionine 144), a more potent autoantigen than its norleucine 144 analog.

10. Structure-function correlations in myasthenia gravis and a new myasthenic syndrome.

11. End-plate potentials in experimental autoimmune myasthenia gravis in rats.

12. Region of peptide 125-147 of acetylcholine receptor alpha subunit is exposed at neuromuscular junction and induces experimental autoimmune myasthenia gravis, T-cell immunity, and modulating autoantibodies.

13. Congenital canine myasthenia gravis: I. Deficient junctional acetylcholine receptors.

14. Experimental autoimmune myasthenia gravis: a sequential and quantitative study of the neuromuscular junction ultrastructure and electrophysiologic correlations.

15. Recently recognized congenital myasthenic syndromes: (a) end-plate acetylcholine (ACh) esterase deficiency (b) putative abnormality of the ACh induced ion channel (c) putative defect of ACh resynthesis or mobilization - clinical features, ultrastructure and cytochemistry.

16. Passively transferred experimental autoimmune myasthenia gravis. Sequential and quantitative study of the motor end-plate fine structure and ultrastructural localization of immune complexes (IgG and C3), and of the acetylcholine receptor.

17. Ultrastructural localization of the terminal and lytic ninth complement component (C9) at the motor end-plate in myasthenia gravis.

18. Synaptic vesicle abnormality in familial infantile myasthenia.

19. A new myasthenic syndrome with end-plate acetylcholinesterase deficiency, small nerve terminals, and reduced acetylcholine release.

20. Ultrastructural localization of immune complexes (IgG and C3) at the end-plate in experimental autoimmune myasthenia gravis.

21. Alternate-day prednisone: preliminary report of a double-blind controlled study.

23. Acetylcholine release in diaphragm of rats with chronic experimental autoimmune myasthenia gravis.

24. Immune complexes (IgG and C3) at the motor end-plate in myasthenia gravis: ultrastructural and light microscopic localization and electrophysiologic correlations.

26. Ultrastructural localization of the acetylcholine receptor in myasthenia gravis and in its experimental autoimmune model.

27. Myasthenogenicity of human acetylcholine receptor synthetic alpha-subunit peptide 125-147 does not require intramolecular disulfide cyclization.

28. Congenital myasthenia gravis in 13 smooth fox terriers.

29. Myasthenia gravis induced by monoclonal antibodies to acetylcholine receptors.

30. Content of acetylcholine receptor and antibodies bound to receptor in myasthenia gravis, experimental autoimmune myasthenia gravis, and Eaton-Lambert syndrome.

32. SYMPOSIUM ON MUSCLE DISEASE.

34. Small cell carcinoma of lung. Observations on four patients including one with a myasthenic syndrome.

35. Histometric study of neuromuscular junction ultrastructure. I. Myasthenia gravis.

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