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1. NOX3-TARGETED THERAPIES FOR INNER EAR PATHOLOGIES.

2. NOX1 is responsible for cell death through STAT3 activation in hyperoxia and is associated with the pathogenesis of acute respiratory distress syndrome.

3. Hyperinflammation of chronic granulomatous disease is abolished by NOX2 reconstitution in macrophages and dendritic cells.

4. A key role for NOX4 in epithelial cell death during development of lung fibrosis.

6. Reduced expression of the NADPH oxidase NOX4 is a hallmark of adipocyte differentiation.

7. Insulin-induced vascular endothelial growth factor expression is mediated by the NADPH oxidase NOX3.

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