1. Demographic characteristics and delayed neurological sequelae risk factors in carbon monoxide poisoning.
- Author
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Sarı Doğan F, Güneysel Ö, Gökdağ E, Güneş M, and Sümen SG
- Subjects
- Adolescent, Adult, Aged, Aged, 80 and over, Attention, Carbon Monoxide Poisoning metabolism, Carbon Monoxide Poisoning psychology, Carbon Monoxide Poisoning therapy, Child, Child, Preschool, Cognitive Dysfunction epidemiology, Cognitive Dysfunction etiology, Cognitive Dysfunction physiopathology, Cognitive Dysfunction psychology, Confusion epidemiology, Confusion etiology, Confusion physiopathology, Confusion psychology, Female, Hospitalization, Humans, Hyperbaric Oxygenation statistics & numerical data, Hyperphagia epidemiology, Hyperphagia etiology, Hyperphagia physiopathology, Hyperphagia psychology, Infant, Length of Stay, Male, Memory Disorders epidemiology, Memory Disorders etiology, Memory Disorders physiopathology, Memory Disorders psychology, Middle Aged, Muscle Rigidity epidemiology, Muscle Rigidity etiology, Muscle Rigidity physiopathology, Muscle Rigidity psychology, Nervous System Diseases etiology, Nervous System Diseases physiopathology, Nervous System Diseases psychology, Neurologic Examination, Physical Examination, Postural Balance, Risk Factors, Sensation Disorders epidemiology, Sensation Disorders etiology, Sensation Disorders physiopathology, Sensation Disorders psychology, Time Factors, Tremor epidemiology, Tremor etiology, Tremor physiopathology, Tremor psychology, Urinary Incontinence epidemiology, Urinary Incontinence etiology, Urinary Incontinence physiopathology, Urinary Incontinence psychology, Young Adult, Carbon Monoxide Poisoning physiopathology, Carboxyhemoglobin metabolism, Nervous System Diseases epidemiology
- Abstract
Aim: Carbon monoxide (CO) is a colorless, odorless gas and tasteless. CO poisoning (COP) is one of the most frequently encountered inhalation poisonings. The most common cause of morbidity in COP is delayed neurological sequelae (DNS). DNS is the occurrence of neuropsychiatric findings within 2-240 days after discharge of patients with COP and there are no definitive diagnostic criteria. The aim of our study is; to determine the risk factors and incidence of DNS., Method: Our study is a retrospective, observational study. Patients with the diagnosis of COP in the emergency department between 2015 and 2016 were included in the study. Patients age, gender, findings in the initial physical examination (PE) and neurological examination (NE), blood carboxyhemoglobin (COHb) level, relation between hyperbaric oxygen (HBO) treatment and DNS were assessed., Results: Total of 72 patients were included in the study. Mean age was 33.43 ± 20.89. It was determined that pathological findings in the initial NE are a significant predictive factor for DNS (Odds ratio 18.600, p:0.004). Significant relation between NE and HBO treatment was present (p:00.1). There was no statistically significant relationship between initial COHb level and receiving HBO treatment (p:0.9). Median COHb level of patients with DNS was 30 (min:10, max: 43), median COHb level of patients without DNS was 25 (min:10, max:44) and there was no statistically significant relationship between the two groups according to COHb levels (p:0.7)., Conclusion: Pathological findings in the initial neurological examination had a predictive value for delayed neurological sequelae in patients with carbon monoxide poisoning., Competing Interests: Declaration of competing interest Authors have no conflict of interests., (Copyright © 2019 Elsevier Inc. All rights reserved.)
- Published
- 2020
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