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1. A rare haplotype of the RET proto-oncogene is a risk-modifying allele in hirschsprung disease.

2. Segregation at three loci explains familial and population risk in Hirschsprung disease.

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3. MEN2A-RET-induced cellular transformation by activation of STAT3.

4. A single-nucleotide polymorphic variant of the RET proto-oncogene is underrepresented in sporadic Hirschsprung disease.

5. Incidence of RET mutations in patients with Hirschsprung's disease.

6. Exon structure and flanking intronic sequences of the human RET proto-oncogene.

7. Inclusion of malignant fibrous histiocytoma in the tumour spectrum associated with hereditary non-polyposis colorectal cancer