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Jews have lived in Switzerland since ancient times. This article discusses recent anti-Semitic incidents and concerns in Geneva and traces the complex history, over a number of centuries, of restrictions and progressive emancipation of Jews in Switzerland. [ABSTRACT FROM AUTHOR]

In recent years, scientific methods of bio- and geoarchaeology have become increasingly important for archaeological research. Political changes since the 1990s have reshaped the archaeological community. At the same time environmental topics have gained importance in modern society, but the debate lacks an historical understanding. Regarding medieval rural archaeology, we need to ask how this influences our archaeological research on medieval settlements, and how ecological approaches fit into the self-concept of medieval archaeology as a primarily historical discipline. Based mainly on a background in German medieval archaeology, this article calls attention to more complex ecological research questions. Medieval village formation and the late medieval crisis are taken as examples to sketch some hypotheses and research questions. The perspective of a village ecosystem helps bring together economic aspects, human ecology and environmental history. There are several implications for archaeological theory as well as for archaeological practice. Traditional approaches from landscape archaeology are insufficient to understand the changes within village ecosystems. We need to consider social aspects and subjective recognition of the environment by past humans as a crucial part of human-nature interaction. Use of the perspective of village ecosystems as a theoretical background offers a way to examine individual historical case studies with close attention to human agency. Thinking in terms of human ecology and environmental history raises awareness of some interrelations that are crucial to understanding past societies and cultural change. [ABSTRACT FROM AUTHOR]

We are currently facing a global pandemic of obesity and Type 2 diabetes. In some settings, the population prevalence of Type 2 diabetes is 50%, and half of those affected will die from diabetes-related complications. Eight centuries ago, an epidemic of bubonic plague swept across Europe, killing at least half of its victims. We here draw comparisons between these two pandemics, proposing close analogies between the 'BlackDeath' of the 14th century and the modern-day equivalent of Type 2 diabetes. Both diseases can be considered in terms of an aetiological agent, a reservoir, a vector and a predisposing toxic environment; populations can be considered as highly susceptible to the transmissable agents of Type 2 diabetes in the setting of calorie excess, inadequate food labelling, poorly regulated advertising and sedentary lifestyles. As for tackling a pandemic of a contagious microbial pathogen,we believe that breaking the cycle of transmission in the diabetes epidemic must be underpinned by political will and prompt, decisive legislation backed by the medical community. Far from fearing that such measures edge us towards a 'nanny state', we believe individuals should expect a responsible government to safeguard them from the toxicmilieu that puts them at risk of obesity and its complications, and that communities and populations have the right to have their health protected. [ABSTRACT FROM AUTHOR]

This work presents, for the first time, a series of detailed space-time maps of Black Death mortality and infected area propagation throughout the fourteenth century AD Europe. The maps integrate a variety of interdisciplinary knowledge bases about the devastating epidemic and provide researchers and the interested public with an informative description of the Black Death dynamics (temporal evolution, local and global geographical patterns, etc.). Concerning the relation of the Black Death with bubonic plague, our space-time findings provide considerable support to the view that Black Death was a different kind of epidemic than bubonic plague and, a the same time, they generate no damaging hard evidence against the new proposals concerning the Black Death etiology. [ABSTRACT FROM AUTHOR]

The second plague pandemic started in Europe with the Black Death in 1346 and lasted until the 19th century. Based on ancient DNA studies, there is a scientific disagreement over whether the bacterium, Yersinia pestis , came into Europe once (Hypothesis 1) or repeatedly over the following four centuries (Hypothesis 2). Here, we synthesize the most updated phylogeny together with historical, archeological, evolutionary, and ecological information. On the basis of this holistic view, we conclude that Hypothesis 2 is the most plausible. We also suggest that Y. pestis lineages might have developed attenuated virulence during transmission, which can explain the convergent evolutionary signals, including pla decay, that appeared at the end of the pandemics., Competing Interests: The authors declare no competing interest., (Copyright © 2021 the Author(s). Published by PNAS.)

Plague, caused by the bacterium Yersinia pestis , can spread through human populations by multiple transmission pathways. Today, most human plague cases are bubonic, caused by spillover of infected fleas from rodent epizootics, or pneumonic, caused by inhalation of infectious droplets. However, little is known about the historical spread of plague in Europe during the Second Pandemic (14-19th centuries), including the Black Death, which led to high mortality and recurrent epidemics for hundreds of years. Several studies have suggested that human ectoparasite vectors, such as human fleas ( Pulex irritans ) or body lice ( Pediculus humanus humanus ), caused the rapidly spreading epidemics. Here, we describe a compartmental model for plague transmission by a human ectoparasite vector. Using Bayesian inference, we found that this model fits mortality curves from nine outbreaks in Europe better than models for pneumonic or rodent transmission. Our results support that human ectoparasites were primary vectors for plague during the Second Pandemic, including the Black Death (1346-1353), ultimately challenging the assumption that plague in Europe was predominantly spread by rats., Competing Interests: The authors declare no conflict of interest., (Copyright © 2018 the Author(s). Published by PNAS.)