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Several studies have reported a relation between race-related stressors and the poor health of Black Americans. Such findings raise questions regarding the mediating biological mechanisms that might account for this link. The present study investigated elevated systemic inflammation, a factor shown to be a strong predictor of chronic illness and mortality in all ethnic populations, as a possible factor. Using 7 waves of data from the Family and Community Health Study, collected over a 20-year period from over 400 Black Americans, we investigated the extent to which exposure to discrimination and segregation at various points in the life course predicted adult inflammation at age 28. Our analyses examined whether cumulative stress, stress generation, or predictive adaptive response (PAR) models best accounted for any associations that existed between these race-related stressors and adult inflammation. At every wave of data collection, assessments of discrimination and segregation were related to adult inflammation. However, multivariate analyses using structure equation modeling indicated that the PAR model best explained the effect of these race-related stressors on inflammation. Exposure to discrimination and segregation during the juvenile years predicted adult inflammation and amplified the inflammatory effect of adult exposure to these race-related stressors. These effects were considerably more robust than that of traditional health risk factors such as diet, exercise, smoking, and low SES. Implications of these findings are discussed, including the limitations of the widely accepted risk factor approach to increasing the health of Black Americans.

This study investigated how early experience with racial discrimination affected the subsequent risky sexual behaviors of a diverse sample of African American youths (N = 745). The analyses focused on 3 risk-promoting factors thought to mediate the hypothesized discrimination--risky sex relation: negative affect, affiliation with deviant peers, and favorable attitudes toward risky sex. In addition, attentive parenting was examined as a protective factor. Analyses using structural equation modeling revealed that youths who perceived more racial discrimination at age 10 or 11 were engaging in more sexual risk taking at age 18 or 19. This relation was mediated by the hypothesized risk-promoting factors via pathways that were consistent with our conceptual model. Results also indicated a prospective reciprocal relation between parenting and children's deviant affiliations: deviant peer affiliations at age 10 or 11 predicted more attentive parenting behaviors by the parents; this response from the parents, in turn, predicted relatively fewer deviant affiliations when the youths were 15 or 16. Study findings are discussed in terms of their relevance to the disproportionately high rates of sexually transmitted infections among African Americans. (Contains 3 figures, 3 tables, and 6 footnotes.)

The present study tests a developmental model designed to explain the romantic relationship difficulties and reluctance to marry often reported for African Americans. Using longitudinal data from a sample of approximately 400 African American young adults, we examine the manner in which race-related adverse experiences during late childhood and early adolescence give rise to the cynical view of romantic partners and marriage held by many young African Americans. Our results indicate that adverse circumstances disproportionately suffered by African American youth (viz., harsh parenting, family instability, discrimination, criminal victimization, and financial hardship) promote distrustful relational schemas that lead to troubled dating relationships, and that these negative relationship experiences, in turn, encourage a less positive view of marriage. [ABSTRACT FROM PUBLISHER]

Purpose: The timing and social context of pregnancy have significant implications for the well-being of African-American young people. Rarely, however, do studies focus on identifying the developmental processes associated with young people's avoidance of pregnancy until after adolescence., Methods: We tested hypotheses regarding the factors associated with delayed fertility (no experience of a pregnancy by age 19) among a sample of 889 African-American youth recruited at age 11 and assessed longitudinally through age 19. We hypothesized that, during preadolescence (age 11), health-promoting environmental processes would be linked to nurturant-responsive parenting, which in turn would be linked to youths' conventional future orientations and risky sexual behavior in midadolescence (age 16) and to pregnancy experience by late adolescence (age 19). Hypotheses were tested with logistic structural equation modeling., Results: Our conceptual model fit the data well. We identified a cascade process whereby protective environments were associated with nurturant-responsive parenting, which was associated with youths' conventional future orientations; conventional future orientations were associated with avoidance of sexual risk behaviors at age 16 and avoidance of pregnancy by age 19. We identified an additional direct effect between nurturant-responsive parenting and avoidance of risky sexual behavior., Conclusions: The results suggest processes that may be targeted to facilitate delayed fertility among African-American youth., (Copyright © 2013 Society for Adolescent Health and Medicine. Published by Elsevier Inc. All rights reserved.)

Interparental aggression has long been implicated as a cause of child and adolescent antisocial behavior. Four theoretical explanations (viz., an aggressogenic cognition model, general strain theory, an emotional security model, and a spillover model) have been proposed to account for this deleterious effect. To gain a better understanding of the mechanism whereby interparental aggression promotes antisocial behavior, this study tests the competing explanations simultaneously using longitudinal data from a sample of 508 African American families with a child aged 12-14 (53.5% are girls). Using path analysis, the results support both the general strain theory and the emotional security model for girls. The results also show weak support for the spillover model. Whereas, for boys, all of the four explanations were supported, though the support for the spillover model is weak. Thus, the findings suggest that the mechanisms whereby interparental aggression fosters antisocial behavior may differ by gender. Implications and limitations of the current research are discussed.