6 results on '"*NEUROENDOCRINE system"'
Search Results
2. History of Music Therapy and Its Contemporary Applications in Cardiovascular Diseases.
- Author
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Montinari, Maria Rosa, Giardina, Simona, Minelli, Pierluca, and Minelli, Sergio
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MUSIC therapy , *CARDIOVASCULAR diseases , *MENTAL healing , *PALEOLITHIC Period , *NEUROENDOCRINE system , *CARDIOVASCULAR disease treatment , *HISTORY , *POSTOPERATIVE care , *TREATMENT effectiveness - Abstract
Contrary to what is commonly believed, music therapy is an old cure, the use of which is lost in the mists of time. Music always has been perceived to have particular healing powers, and the entire history of civilization contains aspects that link music to physical and mental healing. It seems that the adoption of music for therapeutic purposes harks back to a distant past, probably since the Paleolithic period: it was believed that listening to music could affect the behavior of human beings. In later centuries, the concept of "musical organ-tropism" was born and developed, because according to the type of music, one may affect the cardiovascular, respiratory, and neuroendocrine systems. Studies have shown that music can powerfully evoke and modulate emotions and moods, along with changes in heart activity, blood pressure, and breathing. Indeed, the following findings arise from the literature: heart and respiratory rates are higher in response to exciting music than in the case of tranquilizing music. In addition, music produces activity changes in brain structures (amygdala, hypothalamus, insular and orbitofrontal cortex) known to modulate heart function. This article provides a careful overview of music therapy history from prehistory to the present and a review of the latest applications of music therapy in cardiovascular diseases. [ABSTRACT FROM AUTHOR]
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- 2018
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3. How nurse gender influences patient priority assignments in US emergency departments.
- Author
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Vigil, Jacob Miguel, Coulombe, Patrick, Alcock, Joe, Stith, Sarah See, Kruger, Eric, and Cichowski, Sara
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EMERGENCY medical services , *MEDICAL triage , *ELECTRONIC health records , *NEUROENDOCRINE system , *HEART beat measurement , *PAIN diagnosis , *HEART beat , *HOSPITAL emergency services , *HUMAN reproduction , *NURSING assessment , *PAIN , *RESEARCH funding , *RESPIRATION , *RESEARCH bias , *RETROSPECTIVE studies - Abstract
The goals of this study were to compare whether emergency department (ED) patients' pain intensity (PI) is measured differently by male and female nurses and to determine whether PI, heart rate (HR), and respiratory rate (RR) were used to prioritize patient urgency differently by male and female nurses. The associations between patients' PI|HR|RR and the Emergency Severity Index (ESI) scores they were assigned by attending nurses were analyzed using a national database of electronic medical records of US Veterans Affairs ED patients from 2008 to 2012. A total of 129,991 patients presenting for emergency care (Mage = 59.5, 92% males) and their triage nurses (n = 774, Mage = 47.5, 18% males) were analyzed, resulting in a total of 359,642 patient-provider interactions. Patients' PI did not differ by the nurse's gender; however a cross-classified mixed-effects model showed that nurse gender influenced how PI and RR measurements informed the ESI levels that male patients received. Higher PI levels were associated with more urgent (higher priority) ESI levels by female nurses, yet less urgent ESI levels by male nurses. In contrast, male patients with high RR received more urgent ESI levels by male nurses, whereas the nurse gender did not influence ESI assignments for female patients. These findings show that ED patients receive disparate treatment based on inherent characteristics of their triage nurses, and more standardized (eg, automated) protocols that can account for implicit social factors on health care practice for reliably assessing and prioritizing ED patients may be currently warranted. [ABSTRACT FROM AUTHOR]
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- 2017
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4. A Biopsychosocial Conceptual Framework of Postpartum Depression Risk in Immigrant and U.S.-born Latina Mothers in the United States.
- Author
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Lara-Cinisomo, Sandraluz, Girdler, Susan S., Grewen, Karen, and Meltzer-Brody, Samantha
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POSTPARTUM depression , *BIOMARKERS , *CINAHL database , *CONCEPTUAL structures , *HISPANIC Americans , *IMMIGRANTS , *MEDICAL information storage & retrieval systems , *MEDLINE , *PSYCHOLOGY of mothers , *NEUROBIOLOGY , *ONLINE information services , *PSYCHOLOGICAL stress , *SYSTEMATIC reviews , *NEUROENDOCRINE system - Abstract
Objective In this review, we offer a conceptual framework that identifies risk factors of postpartum depression (PPD) in immigrant and U.S.-born Latinas in the United States by focusing on psychosocial and neuroendocrine factors. Although the evidence of the impact psychosocial stressors have on the development of PPD has been well-documented, less is known about the biological etiology of PPD or how these complex stressors jointly increase the risk of PPD in immigrant and U.S.-born Latinas in the United States. Methods Using PubMed, CINAHL, and Embase, we reviewed the literature from 2000 to 2015 regarding psychosocial and physiological risk factors associated with PPD to develop a conceptual model for Latinas. Results Our search yielded 16 relevant studies. Based on our review of the literature, we developed a biopsychosocial conceptual model of PPD for Latinas in the United States. We make arguments for an integrated model designed to assess psychosocial and physiological risk factors and PPD in a high-risk population. Our framework describes the hypothesized associations between culturally and contextually relevant psychosocial stressors, neurobiological factors (e.g., hypothalamic-pituitary-adrenal [HPA] axis response system and oxytocin signaling), and PPD in Latinas in the United States. Conclusions Future studies should evaluate prospectively the impact psychosocial stressors identified here have on the development of PPD in both immigrant and U.S-born Latinas while examining neuroendocrine function, such as the HPA axis and oxytocin signaling. Our conceptual framework will allow for the reporting of main and indirect effects of psychosocial risk factors and biomarkers (e.g., HPA axis and oxytocin function) on PPD in foreign- and U.S.-born postpartum Latinas. [ABSTRACT FROM AUTHOR]
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- 2016
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5. Neuroendocrine properties of macrophage migration inhibitory factor (MIF).
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Bucala, R, Fingerle-Rowson, Gunter R, and Bucala, Richard
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MACROPHAGES , *NEUROENDOCRINOLOGY - Abstract
Summary The cytokine macrophage migration inhibitory factor (MIF) is produced by neuroendocrine and immune tissues and possesses several features that allow it to be characterized as a neuroendocrine mediator. Its pro-inflammatory action and its pathogenic role in inflammatory diseases, such as septic shock, arthritis and other diseases, have clearly been demonstrated and may be based in part on neuroendocrine mechanisms. Macrophage migration inhibitory factor possesses glucocorticoid-antagonist properties within the immune system and participates in the regulation of several endocrine circuits. This review summarizes the current state of MIF research and focuses on MIF expression and function in nervous and endocrine tissues. [ABSTRACT FROM AUTHOR]
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- 2001
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6. The stress response and the regulation of inflammatory disease.
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Sternberg, Esther M., Chrousos, George P., Wider, Ronald L., Gold, Philip W., Sternberg, E M, Chrousos, G P, Wilder, R L, and Gold, P W
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IMMUNOLOGY , *HOMEOSTASIS , *CORTICOTROPIN releasing hormone , *PHYSIOLOGICAL stress , *HYPOTHALAMUS physiology , *ENDOCRINE gland physiology , *IMMUNITY , *INFLAMMATION , *PSYCHONEUROIMMUNOLOGY , *PSYCHOLOGICAL stress , *THEORY , *NEUROENDOCRINE system - Abstract
The molecular and biochemical bases for interactions between the immune and central nervous systems are described. Immune cytokines not only activate immune function but also recruit central stress-responsive neurotransmitter systems in the modulation of the immune response and in the activation of behaviors that may be adaptive during injury or inflammation. Peripherally generated cytokines, such as interleukin-1, signal hypothalamic corticotropin-releasing hormone (CRH) neurons to activate pituitary-adrenal counter-regulation of inflammation through the potent antiinflammatory effects of glucocorticoids. Corticotropin-releasing hormone not only activates the pituitary-adrenal axis but also sets in motion a coordinated series of behavioral and physiologic responses, suggesting that the central nervous system may coordinate both behavioral and immunologic adaptation during stressful situations. The pathophysiologic perturbation of this feedback loop, through various mechanisms, results in the development of inflammatory syndromes, such as rheumatoid arthritis, and behavioral syndromes, such as depression. Thus, diseases characterized by both inflammatory and emotional disturbances may derive from common alterations in specific central nervous system pathways (for example, the CRH system). In addition, disruptions of this communication by genetic, infectious, toxic, or pharmacologic means can influence the susceptibility to disorders associated with both behavioral and inflammatory components and potentially alter their natural history. These concepts suggest that neuropharmacologic agents that stimulate hypothalamic CRH might potentially be adjunctive therapy for illnesses traditionally viewed as inflammatory or autoimmune. [ABSTRACT FROM AUTHOR]
- Published
- 1992
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