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2. Focal adhesion kinase-YAP signaling axis drives drug-tolerant persister cells and residual disease in lung cancer

7. BMN 673, a Novel and Highly Potent PARP1/2 Inhibitor for the Treatment of Human Cancers with DNA Repair Deficiency

8. Figure S1 from TEAD Inhibition Overcomes YAP1/TAZ-Driven Primary and Acquired Resistance to KRASG12C Inhibitors

9. Table S2 from TEAD Inhibition Overcomes YAP1/TAZ-Driven Primary and Acquired Resistance to KRASG12C Inhibitors

10. TEAD Inhibition Overcomes YAP1/TAZ-Driven Primary and Acquired Resistance to KRASG12C Inhibitors

18. Abstract B009: TEAD inhibition overcomes YAP1/TAZ-driven resistance to RAS inhibitors in KRASG12C-mutant cancers

22. Inhibition of YAP/TAZ-driven TEAD activity prevents growth of NF2-null schwannoma and meningioma

26. Inhibition of YAP/TAZ-driven TEAD activity prevents growth of NF2-null schwannoma and meningioma.

33. A focal adhesion kinase-YAP signaling axis drives drug tolerant persister cells and residual disease in lung cancer

43. Dynamic variations in epithelial-to-mesenchymal transition (EMT), ATM, and SLFN11 govern response to PARP inhibitors and cisplatin in small cell lung cancer

44. Modern Day Robin Hoods?

46. Court Urged To Fulfill Crawford Promise

47. High court may clarify use of out-of-court testimony

48. Beam Me Up: High-tech gadgets add flair to trials

49. High-tech gadgets add flair to trials

50. State Chief Justices Discuss New Threats At Annual Meeting

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