257 results on '"Beres, Stephen B."'
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52. Novel genes required for the fitness ofStreptococcus pyogenesin human saliva
53. Rapidly progressive, fatal, inhalation anthrax-like infection in a human: case report, pathogen genome sequencing, pathology, and coordinated response
54. Population Genomic Analysis of 1,777 Extended-Spectrum Beta-Lactamase-Producing Klebsiella pneumoniae Isolates, Houston, Texas: Unexpected Abundance of Clonal Group 307
55. Interacting networks of resistance, virulence and core machinery genes identified by genome-wide epistasis analysis
56. Rapid Emergence of a New Clone Impacts the Population at Risk and Increases the Incidence of Type emm89 Group A Streptococcus Invasive Disease
57. Genomic Landscape of Intrahost Variation in Group A Streptococcus: Repeated and Abundant Mutational Inactivation of the fabT Gene Encoding a Regulator of Fatty Acid Synthesis
58. Genomic Characteristics Behind the Spread of Bacteremic Group AStreptococcusTypeemm89 in Finland, 2004–2014
59. Transcriptome Remodeling Contributes to Epidemic Disease Caused by the Human Pathogen Streptococcus pyogenes
60. The Majority of 9,729 Group A Streptococcus Strains Causing Disease Secrete SpeB Cysteine Protease: Pathogenesis Implications
61. Novel Genes Required for the Fitness of Streptococcus pyogenes in Human Saliva.
62. Clinical Laboratory Response to a Mock Outbreak of Invasive Bacterial Infections: a Preparedness Study
63. Absence of Patient-to-Patient Intrahospital Transmission of Staphylococcus aureus as Determined by Whole-Genome Sequencing
64. Streptolysin O Rapidly Impairs Neutrophil Oxidative Burst and Antibacterial Responses to Group A Streptococcus.
65. Asymptomatic Carriage of Group A Streptococcus Is Associated with Elimination of Capsule Production
66. Natural Disruption of Two Regulatory Networks in Serotype M3 Group A Streptococcus Isolates Contributes to the Virulence Factor Profile of This Hypervirulent Serotype
67. Streptolysin O Rapidly Impairs Neutrophil Oxidative Burst and Antibacterial Responses to Group A Streptococcus.
68. Human Disease Isolates of Serotype M4 and M22 Group A Streptococcus Lack Genes Required for Hyaluronic Acid Capsule Biosynthesis
69. Polymorphisms in regulator of protease B (RopB) alter disease phenotype and strain virulence of serotype M3 Group A Streptococcus
70. Whole Genome Sequence Analysis of a B. cereus Strain Causing a Fatal Anthrax‐Like Pulmonary Infection
71. Genomic Analysis ofemm59Group AStreptococcusInvasive Strains, United States
72. Evolution of diversity in epidemics revealed by analysis of the human bacterial pathogen group A Streptococcus
73. Virulence of serotype M3 Group AStreptococcusstrains in wax worms (Galleria mellonellalarvae)
74. Genome Sequence of a Lancefield Group C Streptococcus zooepidemicus Strain Causing Epidemic Nephritis: New Information about an Old Disease
75. Contribution of Exogenous Genetic Elements to the Group A Streptococcus Metagenome
76. Genome Sequence of a Serotype M28 Strain of Group AStreptococcus:Potential New Insights into Puerperal Sepsis and Bacterial Disease Specificity
77. Genetic Diversity among Type emm28 Group A Streptococcus Strains Causing Invasive Infections and Pharyngitis
78. Analysis of a Novel Prophage-encoded Group A Streptococcus Extracellular Phospholipase A2
79. Progress toward Characterization of the Group AStreptococcusMetagenome: Complete Genome Sequence of a Macrolide‐Resistant Serotype M6 Strain
80. Genome sequence of a serotype M3 strain of group AStreptococcus: Phage-encoded toxins, the high-virulence phenotype, and clone emergence
81. III: EVOLUTION OF SELECTED PATHOGENIC SPECIES AND MECHANISMS: CHAPTER 14: GROUP A Streptococcus AND Staphylococcus aureus: EVOLUTION, REEMERGENCE, AND STRAIN DIVERSIFICATION.
82. Chapter 16: CONTRIBUTION OF PHAGES TO GROUP A STREPTOCOCCUS GENETIC DIVERSITY AND PATHOGENESIS.
83. Genomic Landscape of Intrahost Variation in Group A Streptococcus: Repeated and Abundant Mutational Inactivation of the fabTGene Encoding a Regulator of Fatty Acid Synthesis
84. Virulence of serotype M3 Group A Streptococcus strains in wax worms (Galleria mellonella larvae).
85. In Vitro Serial Passage of Staphylococcus aureus: Changes in Physiology, Virulence Factor Production,a nd agr Nucleotide Sequence.
86. Asymptomatic Carriage of Group A StreptococcusIs Associated with Elimination of Capsule Production
87. Natural Disruption of Two Regulatory Networks in Serotype M3 Group A StreptococcusIsolates Contributes to the Virulence Factor Profile of This Hypervirulent Serotype
88. Virulence of serotype M3 Group A Streptococcusstrains in wax worms (Galleria mellonella larvae)
89. Genetic Diversity among Type emm28Group A StreptococcusStrains Causing Invasive Infections and Pharyngitis
90. Genetic Basis Underlying the Hyperhemolytic Phenotype of Streptococcus agalactiae Strain CNCTC10/84.
91. Genome-Wide Assessment of Streptococcus agalactiaeGenes Required for Survival in Human Whole Blood and Plasma
92. Population Genomic Molecular Epidemiological Study of Macrolide-Resistant Streptococcus pyogenesin Iceland, 1995 to 2016: Identification of a Large Clonal Population with a pbp2xMutation Conferring Reduced In Vitroß-Lactam Susceptibility
93. Reduced In VitroSusceptibility of Streptococcus pyogenesto ß-Lactam Antibiotics Associated with Mutations in the pbp2xGene Is Geographically Widespread
94. Gene fitness landscape of group A streptococcus during necrotizing myositis.
95. Population Genomic Analysis of 1,777 Extended-Spectrum Beta-Lactamase-Producing Klebsiella pneumoniaeIsolates, Houston, Texas: Unexpected Abundance of Clonal Group 307
96. Absence of Patient-to-Patient Intrahospital Transmission of Staphylococcus aureusas Determined by Whole-Genome Sequencing
97. Genome-Wide Transposon Mutagenesis Screens Identify Group A Streptococcus Genes Affecting Susceptibility to ß-Lactam Antibiotics.
98. A molecular trigger for intercontinental epidemics of group A Streptococcus.
99. Analysis of a Novel Prophage-encoded Group A Streptococcus Extracellular Phospholipase A2.
100. Interacting networks of resistance, virulence and core machinery genes identified by genome-wide epistasis analysis
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