355 results on '"Dichek, David A."'
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52. CONTROL OF THROMBOSIS ON SYNTHETIC GRAFT SURFACES-GENE TRANSFER APPROACH
53. Established Immunity Precludes Adenovirus-Mediated Gene Transfer in Rat Carotid Arteries: Potential for Immunosuppression and Vector Engineering to Overcome Barriers of Immunity
54. DIRECT TRANSFER—VIRAL APPROACHES: MODIFICATION OF THE VASCULAR PHENOTYPE BY VIRAL GENE TRANSFER: THERAPY AND TOXICITY
55. Adenovirus-mediated Gene Transfer into Normal Rabbit Arteries Results in Prolonged Vascular Cell Activation, Inflammation, and Neointimal Hyperplasia
56. Jugular Vein Injection of High-Titer Lentiviral Vectors Does Not Transduce the Aorta—Brief Report.
57. Germline Incorporation of a Replication-Defective Adenoviral Vector in Mice Does Not Alter Immune Responses to Adenoviral Vectors
58. ABCA1 Overexpression in Endothelial CellsIn VitroEnhances ApoAI-Mediated Cholesterol Efflux and Decreases Inflammation
59. A Rabbit Model of Durable Transgene Expression in Jugular Vein to Common Carotid Artery Interposition Grafts
60. In Vivo Gene Transfer to the Rabbit Common Carotid Artery Endothelium
61. Abstract 106: Young Tgfbr2 G357W/+ Mice Have Decreased Expression of TGF-β Ligands and Receptors in the Proximal Aorta, Aortic Root Dilation, and Aortic Elongation
62. Abstract 628: Overexpression of ABCA1 in Cultured Endothelial Cells Using Helper-Dependent Adenovirus Enhances ApoAI-Mediated Cholesterol Efflux and has Anti-Inflammatory Effects
63. Variability in Messenger Rna Levels in Human Umbilical Vein Endothelial Cells of Different Lineage and Time in Culture
64. Plasminogen promotes cholesterol efflux by the ABCA1 pathway
65. Abstract 28: Apolipoprotein A-I Vascular Gene Therapy Provides Durable Protection from Atherosclerosis in Hyperlipidemic Rabbits
66. Abstract 9: Smooth Muscle Cell Tgfbr2 Deletion in Mice Causes Aortic Hypercontractility and Impaired Endothelium-Dependent Relaxation
67. StableIn VivoTransgene Expression in Endothelial Cells with Helper-Dependent Adenovirus: Roles of Promoter and Interleukin-10
68. Postnatal Deletion of the Type II TGF-Beta Receptor in Smooth Muscle Cells Causes Severe Aortopathy in Mice
69. Aortopathy in a Mouse Model of Marfan Syndrome Is Not Mediated by Altered Transforming Growth Factor β Signaling
70. Abstract 432: Endothelial Cells Express miR-33a-5p and Release Extracellular Vesicles Containing miR-33a-5p
71. Abstract 435: Vascular Gene Therapy with Apolipoprotein A-I in a Rabbit Model of Atherosclerosis Regression
72. Abstract 255: Plasminogen Promotes Cholesterol Efflux by the ABCA1
73. ABCA1 Overexpression in Endothelial Cells In Vitro Enhances ApoAI-Mediated Cholesterol Efflux and Decreases Inflammation.
74. IgG1 is the predominant subclass ofin vivo- andin vitro-produced anti-tetanus toxoid antibodies and also serves as the membrane IgG molecule for delivering inhibitory signals to anti-tetanus toxoid antibody-producing B cells
75. Single-Cell Transcriptomics Identifies Selective Lineage-Specific Regulation of Genes in Aortic Smooth Muscle Cells in Mice
76. Postnatal Deletion of the Type II Transforming Growth Factor-β Receptor in Smooth Muscle Cells Causes Severe Aortopathy in Mice
77. Abstract 41: Postnatal Deletion of Vascular Smooth Muscle Cell Transforming Growth Factor beta Receptor 2 in Mice Exacerbates Marfan-syndrome-associated Aortic Dilation
78. Abstract 345: A Proteomic Study of Human and Mouse Plaques Supports Involvement of Proteolysis, Extracellular Matrix, and Cytoskeleton in Atherosclerotic Plaque Rupture
79. Abstract 455: Systemic, But Not Smooth Muscle Cell-Specific, TGF-β Blockade Exacerbates Angiotensin II-Induced Abdominal Aortic Pathology
80. 190. Strategies for Achieving High-Level, Stable, Endothelium-Specific Transgene Expression Using Helper-Dependent Adenovirus
81. β-TGF (Transforming Growth Factor-β) Signaling Protects the Thoracic and Abdominal Aorta From Angiotensin II-Induced Pathology by Distinct Mechanisms.
82. Stable In Vivo Transgene Expression in Endothelial Cells with Helper-Dependent Adenovirus: Roles of Promoter and Interleukin-10.
83. Efficient Gene Transfer and Durable Transgene Expression in Grafted Rabbit Veins
84. Cardiovascular Gene Therapy
85. Abstract 84: Deletion of the Type II TGF-ß Receptor in Smooth Muscle Causes Severe Aortopathy in Mice
86. Abstract 522: Shotgun Proteomics Implicates Matrix Proteolysis, Cell Adhesion and Cell Death in Urokinase-Accelerated Vascular Disease
87. Improved Animal Models for Testing Gene Therapy for Atherosclerosis
88. Abstract 207: Comparison of Two Lines of Mice Expressing Smooth Muscle-Targeted Cre Recombinase from Different Promoters
89. Abstract 249: Delayed Gene Transfer Increases Transgene Expression in Vein Grafts
90. Abstract 138: Treatment of Atherosclerosis by Systemic Protease Inhibition
91. Overexpression of Endothelial Nitric Oxide Synthase Improves Endothelium-Dependent Vasodilation in Arteries Infused with Helper-Dependent Adenovirus
92. Transforming Growth Factor-β Signaling in Myogenic Cells Regulates Vascular Morphogenesis, Differentiation, and Matrix Synthesis
93. Helper-Dependent Adenoviral Vector Achieves Prolonged, Stable Expression of Interleukin-10 in Rabbit Carotid Arteries but Does Not Limit Early Atherogenesis
94. Helper-Dependent Adenovirus Is Superior to First-Generation Adenovirus for Expressing Transgenes in Atherosclerosis-Prone Arteries
95. Overexpression of Urokinase by Plaque Macrophages Causes Histological Features of Plaque Rupture and Increases Vascular Matrix Metalloproteinase Activity in Aged Apolipoprotein E–Null Mice
96. Level of Macrophage uPA Expression Is an Important Determinant of Atherosclerotic Lesion Growth in Apoe −/− Mice
97. TGF-β1 Limits Plaque Growth, Stabilizes Plaque Structure, and Prevents Aortic Dilation in Apolipoprotein E-Null Mice
98. Smooth Muscle Cells Give Rise to Osteochondrogenic Precursors and Chondrocytes in Calcifying Arteries
99. Constriction of carotid arteries by urokinase-type plasminogen activator requires catalytic activity and is independent of NH2-terminal domains
100. Explicit Evidence of Smooth Muscle Cell Transdifferentiation in Calcifying Blood Vessels: an In Vivo Lineage Tracing Study using Matrix Gla Protein Knockout Mice
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