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302 results on '"Diomedi, M."'

51. Shunt-associated migraines respond favorably to changes in work conditions

Author

Magrini, A, Coppeta, L, Pietroiusti, A, Somma, G, Romeo, E, Bergamaschi, A, Diomedi, M, and Coppetta, L

Subjects
Advanced and Specialized Nursing, Adult, Pediatrics, medicine.medical_specialty, Inpatients, business.industry, Heart Septal Defects, Atrial, Pressure, Migraine Disorders, Abdomen, Female, Nursing Care, Humans, Atrial, Heart Septal Defects, Mean age, Shunt (medical), Natural history, medicine, Settore MED/44 - Medicina del Lavoro, Neurology (clinical), Risk factor, Cardiology and Cardiovascular Medicine, business
Abstract

To the Editor: In their case-control study on shunt-associated migraines, Anzola and colleagues convincingly show that atrial septal repair induces a favorable modification of the natural history of the disease.1 Nearly half of their consecutive patients had a history of cerebrovascular disease, and it is plausible that the vast majority of them were active workers, because the mean age of these subjects was

Published
2006

68. Sonno ed epilessia

Author

Gigli, Gian Luigi, Diomedi, M, Baldinetti, F, and Placidi, F.

Published
1993

77. Association of trisomy 9p and band heterotopia

Author

Federico, A., primary, Tomasetti, P., additional, Zollino, M., additional, Diomedi, M., additional, Dotti, M. T., additional, De Stefano, N., additional, Gualdi, G. F., additional, Neri, G., additional, and Gigli, G. L., additional

Published
1999
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79. Sleep microstructure and EEG epileptiform activity in patients with juvenile myoclonic epilepsy

Author

Gigli, Gl, Calia, E, Marciani, Mg, Mazza, Salvatore, Mennuni, Gm, Diomedi, M, Terzano, Mg, Janz, D., Gigli, Gl, Calia, E, Marciani, Mg, Mazza, Salvatore, Mennuni, Gm, Diomedi, M, Terzano, Mg, and Janz, D.

Abstract

Clinical and EEG manifestations of juvenile myoclonic epilepsy (JME) occur in a strict relationship to the sleepwake cycle, particularly to transition phases (awakening, falling asleep, afternoon relaxation after work). JME manifestations are deactivated during sleep. Because arousal fluctuations during NREM sleep may be controlled by the same neurophysiologic mechanisms regulating awakening, we studied the relationship between the cyclic alternating pattern (CAP) and JME manifestations. All-night polysomnographic recordings of 10 JME patients were analyzed for variations of epileptiform EEG abnormalities in relation to sleep stages and to different microstructural variables (NCAP, CAP, phases A and B). CAP rates (ratio between total CAP duration and total NREM sleep duration) were also calculated. Average CAP rate was 46.70%, significantly higher than that (23%) of an age-matched control group. Macrostructural analysis showed only a trend toward a slight predominance of EEG epileptiform activity during slow wave sleep but no significant correlation between spiking rates and sleep stages. Microstructural analysis confirmed the CAP modulation of EEG epileptiform activity, with maximum appearance of epileptiform abnormalities during phase A CAP (normalized spiking rate = 4.00 ? 0.98) and strong inhibition during phase B (0.06 +- 00.6). Intermediate values were noted during NCAP (0.54 ? 0.27). No correlation was noted between spiking rates during NREM sleep and CAP rates, possibly indicating that in JME patients the increased CAP rate may be partially independent of epileptiform EEG activity. Our data suggest that in JME patients CAP may be a neurophysiologic oscillator organizing expression of the epileptiform discharges independent of the tendency of the individual patient to produce epileptiform EEG discharges

Published
1992

94. CagA-positive Helicobacter pyloristrains may influence the natural history of atherosclerotic stroke

Author

Diomedi, M, Pietroiusti, A, Silvestrini, M, Rizzato, B, Cupini, L M., Ferrante, F, Magrini, A, Bergamaschi, A, Galante, A, and Bernardi, G

Abstract

To test the hypothesis that infection with virulent cytotoxin-associated gene-A (CagA)-bearing Helicobacter pyloristrains influences the atherosclerotic process and the clinical course in atherosclerotic stroke patients.

Published
2004
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99. Effect of anticonvulsants on nocturnal sleep in epilepsy

Author

Placidi, F., Diomedi, M., Scalise, A., Marciani, M. G., Andrea Romigi, and Gigli, G. L.

Subjects
Adult, Male, Epilepsy, Cyclohexanecarboxylic Acids, Triazines, Acetates, Middle Aged, Lamotrigine, Circadian Rhythm, Carbamazepine, Humans, Anticonvulsants, Female, Amines, Gabapentin, Sleep, gamma-Aminobutyric Acid
Abstract

Our objective was to determine, in three separate studies, the effects of controlled-release carbamazepine (CBZ-CR), lamotrigine (LTG), and gabapentin (GBP) on nocturnal sleep in epilepsy. Antiepileptic drugs (AEDs) control seizures and also modify hypnic structure. Despite widespread clinical use, their effects on sleep are not well known. PSG was performed in all three studies as follows: CBZ-CR: at baseline, after initial administration of CBZ-CR 400 mg, and after 1 month of CBZ-CR treatment (400 mg BID) in a sample of seven temporal lobe epileptic (TLE) patients. Results were compared with those of nine healthy volunteers; LTG: at baseline, after 3 months of stable treatment with LTG (300 mg/day); GBP: at baseline, after 3 months of stable treatment with GBP (1800 mg/day). Significant findings are as follows for each study. The acute administration of CBZ-CR increased number of stage shifts, reduced REM sleep, and increased REM sleep fragmentation. In the TLE group, these effects were almost completely reversed after chronic treatment. LTG increased REM sleep, reduced number of entries into REM sleep, decreased number of phase shifts, and decreased percentage of slow-wave sleep. GBP increased REM sleep percentage, increased mean duration of REM periods, reduced number of awakenings, and reduced stage 1 sleep percentage. We conclude that CBZ-CR disrupts REM sleep, but only during acute administration. LTG and GBP improve sleep stability while reducing seizures.

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