934 results on '"Finetti, Pascal"'
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52. Supplementary Figure 3 from The Histone Deacetylase Inhibitor Abexinostat Induces Cancer Stem Cells Differentiation in Breast Cancer with Low Xist Expression
53. Supplementary Figure 6 from The Histone Deacetylase Inhibitor Abexinostat Induces Cancer Stem Cells Differentiation in Breast Cancer with Low Xist Expression
54. Supplementary Figure Legend from The Histone Deacetylase Inhibitor Abexinostat Induces Cancer Stem Cells Differentiation in Breast Cancer with Low Xist Expression
55. Supplementary Figures from Uncovering the Molecular Secrets of Inflammatory Breast Cancer Biology: An Integrated Analysis of Three Distinct Affymetrix Gene Expression Datasets
56. Supplementary Figure 2 from The Histone Deacetylase Inhibitor Abexinostat Induces Cancer Stem Cells Differentiation in Breast Cancer with Low Xist Expression
57. Supplementary Figure S1 Legend from Prognosis and Gene Expression Profiling of 20q13-Amplified Breast Cancers
58. Supplementary Table 1 from The Histone Deacetylase Inhibitor Abexinostat Induces Cancer Stem Cells Differentiation in Breast Cancer with Low Xist Expression
59. Supplementary Figure 8 from The Histone Deacetylase Inhibitor Abexinostat Induces Cancer Stem Cells Differentiation in Breast Cancer with Low Xist Expression
60. Supplementary Figure 4 from The Histone Deacetylase Inhibitor Abexinostat Induces Cancer Stem Cells Differentiation in Breast Cancer with Low Xist Expression
61. Supplementary Table 5 from ALDH1-Positive Cancer Stem Cells Predict Engraftment of Primary Breast Tumors and Are Governed by a Common Stem Cell Program
62. Data from Integrated Profiling of Basal and Luminal Breast Cancers
63. Supplementary Figure 2 from Integrated Profiling of Basal and Luminal Breast Cancers
64. Supplemental Figure S5 from Autocrine Secretion of Progastrin Promotes the Survival and Self-Renewal of Colon Cancer Stem–like Cells
65. Supplementary Table 3 from ALDH1-Positive Cancer Stem Cells Predict Engraftment of Primary Breast Tumors and Are Governed by a Common Stem Cell Program
66. Supplementary Figure 4 from Integrated Profiling of Basal and Luminal Breast Cancers
67. Supplementary Figure 8 from Integrated Profiling of Basal and Luminal Breast Cancers
68. Supplementary Figure 9 from Integrated Profiling of Basal and Luminal Breast Cancers
69. Data from EFA6B Antagonizes Breast Cancer
70. Supplementary Table 4 from ALDH1-Positive Cancer Stem Cells Predict Engraftment of Primary Breast Tumors and Are Governed by a Common Stem Cell Program
71. Supplementary Figure 3 from Integrated Profiling of Basal and Luminal Breast Cancers
72. Supplementary Table 7 from ALDH1-Positive Cancer Stem Cells Predict Engraftment of Primary Breast Tumors and Are Governed by a Common Stem Cell Program
73. Supplementary Methods from ALDH1-Positive Cancer Stem Cells Predict Engraftment of Primary Breast Tumors and Are Governed by a Common Stem Cell Program
74. Supplementary Figure 6 from Integrated Profiling of Basal and Luminal Breast Cancers
75. Supplementary Figure 1 from Gene Expression Profiling Shows Medullary Breast Cancer Is a Subgroup of Basal Breast Cancers
76. Supplementary Table 2 from Sixteen–Kinase Gene Expression Identifies Luminal Breast Cancers with Poor Prognosis
77. Supplementary Table 6 from ALDH1-Positive Cancer Stem Cells Predict Engraftment of Primary Breast Tumors and Are Governed by a Common Stem Cell Program
78. Data from Gene Expression Profiling Shows Medullary Breast Cancer Is a Subgroup of Basal Breast Cancers
79. Supplementary Figure 1 from Sixteen–Kinase Gene Expression Identifies Luminal Breast Cancers with Poor Prognosis
80. Supplementary Figure 1C from Integrated Profiling of Basal and Luminal Breast Cancers
81. Supplementary Figures 1 - 14 from ALDH1-Positive Cancer Stem Cells Predict Engraftment of Primary Breast Tumors and Are Governed by a Common Stem Cell Program
82. Supplementary Figure Legends from ALDH1-Positive Cancer Stem Cells Predict Engraftment of Primary Breast Tumors and Are Governed by a Common Stem Cell Program
83. Supplementary Figures 6-10 from Breast Cancer Cell Lines Contain Functional Cancer Stem Cells with Metastatic Capacity and a Distinct Molecular Signature
84. Data Supplement from EFA6B Antagonizes Breast Cancer
85. Supplementary Figure 2 from Gene Expression Profiling Shows Medullary Breast Cancer Is a Subgroup of Basal Breast Cancers
86. Data from Autocrine Secretion of Progastrin Promotes the Survival and Self-Renewal of Colon Cancer Stem–like Cells
87. Supplementary Table 3 from Gene Expression Profiling for Molecular Characterization of Inflammatory Breast Cancer and Prediction of Response to Chemotherapy
88. Supplementary Data 2 from Human Breast Tumor Cells Induce Self-Tolerance Mechanisms to Avoid NKG2D-Mediated and DNAM-Mediated NK Cell Recognition
89. Supplementary Materials from Integrated Profiling of Basal and Luminal Breast Cancers
90. Supplementary Data 4 from Human Breast Tumor Cells Induce Self-Tolerance Mechanisms to Avoid NKG2D-Mediated and DNAM-Mediated NK Cell Recognition
91. Supplementary Tables 1-9 from Integrated Profiling of Basal and Luminal Breast Cancers
92. Supplementary Data 5 from Human Breast Tumor Cells Induce Self-Tolerance Mechanisms to Avoid NKG2D-Mediated and DNAM-Mediated NK Cell Recognition
93. Supplementary Figures 1-5 from Breast Cancer Cell Lines Contain Functional Cancer Stem Cells with Metastatic Capacity and a Distinct Molecular Signature
94. Supplementary Figure 5 from Integrated Profiling of Basal and Luminal Breast Cancers
95. Supplementary Figure 7 from Integrated Profiling of Basal and Luminal Breast Cancers
96. Supplemental Figure Legends from Autocrine Secretion of Progastrin Promotes the Survival and Self-Renewal of Colon Cancer Stem–like Cells
97. Supplementary Data 8 from Human Breast Tumor Cells Induce Self-Tolerance Mechanisms to Avoid NKG2D-Mediated and DNAM-Mediated NK Cell Recognition
98. Supplementary Methods from Human Breast Tumor Cells Induce Self-Tolerance Mechanisms to Avoid NKG2D-Mediated and DNAM-Mediated NK Cell Recognition
99. Data from Human Breast Tumor Cells Induce Self-Tolerance Mechanisms to Avoid NKG2D-Mediated and DNAM-Mediated NK Cell Recognition
100. Supplementary Table 1 from Gene Expression Profiling for Molecular Characterization of Inflammatory Breast Cancer and Prediction of Response to Chemotherapy
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