516 results on '"Murphy, Maureen E."'
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52. Suppl. Figure 8 from HSP70 Inhibition Limits FAK-Dependent Invasion and Enhances the Response to Melanoma Treatment with BRAF Inhibitors
53. Data from A Rare TP53 Mutation Predominant in Ashkenazi Jews Confers Risk of Multiple Cancers
54. Suppl. Figure 1 from HSP70 Inhibition Limits FAK-Dependent Invasion and Enhances the Response to Melanoma Treatment with BRAF Inhibitors
55. Suppl. Figure 5 from HSP70 Inhibition Limits FAK-Dependent Invasion and Enhances the Response to Melanoma Treatment with BRAF Inhibitors
56. Data from ATG5 Mediates a Positive Feedback Loop between Wnt Signaling and Autophagy in Melanoma
57. Supp Materials and Methods from A Novel Inhibitor of HSP70 Induces Mitochondrial Toxicity and Immune Cell Recruitment in Tumors
58. Supplementary Data from A Rare TP53 Mutation Predominant in Ashkenazi Jews Confers Risk of Multiple Cancers
59. Suppl Figure 2 from HSP70 Inhibition Limits FAK-Dependent Invasion and Enhances the Response to Melanoma Treatment with BRAF Inhibitors
60. Suppl. Figure 7 from HSP70 Inhibition Limits FAK-Dependent Invasion and Enhances the Response to Melanoma Treatment with BRAF Inhibitors
61. Figure S1-4, TABLES1, S2 from ATG5 Mediates a Positive Feedback Loop between Wnt Signaling and Autophagy in Melanoma
62. Suppl. Table 1 from HSP70 Inhibition Limits FAK-Dependent Invasion and Enhances the Response to Melanoma Treatment with BRAF Inhibitors
63. Supplemental Figures from A Novel Inhibitor of HSP70 Induces Mitochondrial Toxicity and Immune Cell Recruitment in Tumors
64. Suppl. Figure 6 from HSP70 Inhibition Limits FAK-Dependent Invasion and Enhances the Response to Melanoma Treatment with BRAF Inhibitors
65. Suppl. Figure 3 from HSP70 Inhibition Limits FAK-Dependent Invasion and Enhances the Response to Melanoma Treatment with BRAF Inhibitors
66. Suppl. Figure 4 from HSP70 Inhibition Limits FAK-Dependent Invasion and Enhances the Response to Melanoma Treatment with BRAF Inhibitors
67. Data from The ARF Tumor Suppressor Can Promote the Progression of Some Tumors
68. Supplementary Figure 2 from The ARF Tumor Suppressor Can Promote the Progression of Some Tumors
69. Supplementary Figure Legends 1-2 from The ARF Tumor Suppressor Can Promote the Progression of Some Tumors
70. Supplementary Figure 1 from The ARF Tumor Suppressor Can Promote the Progression of Some Tumors
71. Editorial: Double-edged swords: Important factors connecting metabolic disorders and cancer development – from basic research to translational applications
72. Common activities and predictive gene signature identified for genetic hypomorphs of TP53
73. Small-Molecule Reactivation of Mutant p53 to Wild-Type-like p53 through the p53-Hsp40 Regulatory Axis
74. PLTP is a p53 target gene with roles in cancer growth suppression and ferroptosis
75. Oncogenes and Tumor Suppressor Genes in Autophagy
76. p53, ARF, and the Control of Autophagy
77. Transcriptional Repression by the p53 Tumor Suppressor Protein
78. Targeting ErbB3 and Cellular NADPH/NADP+ Abundance Sensitizes Cutaneous Melanomas to Ferroptosis Inducers
79. A functionally significant SNP in TP53 and breast cancer risk in African-American women
80. Single-Nucleotide Polymorphisms in the P53 Pathway Regulate Fertility in Humans
81. HSP70 Inhibition Blocks Adaptive Resistance and Synergizes with MEK Inhibition for the Treatment of NRAS-Mutant Melanoma
82. The Hsp70 Family of Heat Shock Proteins in Tumorigenesis: From Molecular Mechanisms to Therapeutic Opportunities
83. Fatty acid transporter 2 reprograms neutrophils in cancer
84. Wnt5A promotes an adaptive, senescent-like stress response, while continuing to drive invasion in melanoma cells
85. ARF Induces Autophagy by Virtue of Interaction with Bcl-xl
86. Editor's evaluation: Heat shock factor 1 (HSF1) cooperates with estrogen receptor α (ERα) in the regulation of estrogen action in breast cancer cells
87. Selective abrogation of S6K2 maps lipid homeostasis as a survival vulnerability in MAPKi-resistant NRASMUT melanoma
88. Targeting ErbB3 and Cellular NADPH/NADP+ Abundance Sensitizes Cutaneous Melanomas to Ferroptosis Inducers.
89. Mutant p53 regulates Survivin to foster lung metastasis
90. P53 regulates cellular redox state, ferroptosis and metabolism
91. Editorial: Double-edged swords: Important factors connecting metabolic disorders and cancer development – from basic research to translational applications.
92. RETRACTED ARTICLE: IspH inhibitors kill Gram-negative bacteria and mobilize immune clearance
93. A Novel Inhibitor of HSP70 Induces Mitochondrial Toxicity and Immune Cell Recruitment in Tumors
94. Abstract PR08: African-centric p53 hypomorphs play significant roles in cancer risk and efficacy of therapy
95. Identification of Mubritinib (TAK 165) as an inhibitor of KSHV driven primary effusion lymphoma via disruption of mitochondrial OXPHOS metabolism
96. Increased mTOR activity and metabolic efficiency in mouse and human cells containing the African-centric tumor-predisposing p53 variant Pro47Ser
97. The HSP70 family and cancer
98. A Rare TP53 Mutation Predominant in Ashkenazi Jews Confers Risk of Multiple Cancers
99. Author response: Increased mTOR activity and metabolic efficiency in mouse and human cells containing the African-centric tumor-predisposing p53 variant Pro47Ser
100. Paradoxical Role for Wild-Type p53 in Driving Therapy Resistance in Melanoma
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