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53. C3‐dependent effector functions of complement.

56. Hyperfunctional C3 convertase leads to complement deposition on endothelial cells and contributes to atypical hemolytic uremic syndrome

60. Intracellular Factor H Drives Tumor Progression Independently of the Complement Cascade

61. Complement C1s and C4d as Prognostic Biomarkers in Renal Cancer: Emergence of Noncanonical Functions of C1s

64. Plasma C3d levels as a diagnostic marker for complete complement factor I deficiency

68. C1q and its growing family

69. Anti-Factor H Autoantibodies Assay

71. Interaction of Clq with IgGl, C-reactive protein and pentraxin 3: Mutational studies using recombinant globular head molecules of human Clq A, B, and C chains

73. Role of Ca(super 2+) in the electrostatic stability and the functional activity of the globular domain of human Clq

74. The receptor for advanced glycation end products is a sensor for cell‐free heme

75. B cells and cancer: To B or not to B?

80. Circulating FH Protects Kidneys From Tubular Injury During Systemic Hemolysis

82. The murine Microenvironment Cell Population counter method to estimate abundance of tissue-infiltrating immune and stromal cell populations in murine samples using gene expression

83. Complement activation in sickle cell disease: Dependence on cell density, hemolysis and modulation by hydroxyurea therapy

85. The receptor for advanced glycation end products is a sensor for cell‐free heme.

88. Tumor Cells Hijack Macrophage-Produced Complement C1q to Promote Tumor Growth

90. Heme Drives Susceptibility of Glomerular Endothelium to Complement Overactivation Due to Inefficient Upregulation of Heme Oxygenase-1

92. A Single-Domain Antibody Targeting Complement Component C5 Acts as a Selective Inhibitor of the Terminal Pathway of the Complement System and Thus Functionally Mimicks the C-Terminal Domain of the Staphylococcus aureus SSL7 Protein

93. Both Monoclonal and Polyclonal Immunoglobulin Contingents Mediate Complement Activation in Monoclonal Gammopathy Associated-C3 Glomerulopathy

94. ATYPICAL HEMOLYTIC UREMIC SYNDROME AND C3 GLOMERULOPATHY: CONCLUSIONS FROM A «KIDNEY DISEASE: IMPROVING GLOBAL OUTCOMES» (KDIGO) CONTROVERSIES CONFERENCE

95. Intravascular hemolysis activates complement via cell-free heme and heme-loaded microvesicles

96. Characterization of Renal Injury and Inflammation in an Experimental Model of Intravascular Hemolysis

98. Complement C3 is a novel modulator of the anti-factor VIII immune response

99. C5 nephritic factors drive the biological phenotype of C3 glomerulopathies

100. Existence of different but overlapping IgG- and IgM-binding sites on the globular domain of human C1q

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