68 results on '"Traccis, S"'
Search Results
52. Successful treatment of acquired pendular elliptical nystagmus in multiple sclerosis with isoniazid and baseout prisms
- Author
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Traccis, S., Rosati, G., Monaco, M. F., Aiello, I., and Agnetti, V.
- Abstract
We treated 3 multiple sclerosis patients who had pendular nystagmus with isoniazid (800 to 1,000 mg/d). Isoniazid abolished the nystagmus and relieved oscillopsia in 2 patients but was ineffective in the 3rd in whom the nystagmus was damped with convergence and vision improved with converging (base-out) prisms.
- Published
- 1990
53. ATXN2 is a modifier of phenotype in ALS patients of Sardinian ancestry
- Author
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Borghero, Giuseppe, Pugliatti, Maura, Marrosu, Francesco, Marrosu, Maria Giovanna, Murru, Maria Rita, Floris, Gianluca, Cannas, Antonino, Parish, Leslie D., Cau, Tea B., Loi, Daniela, Ticca, Anna, Traccis, Sebastiano, Manera, Umberto, Canosa, Antonio, Moglia, Cristina, Calvo, Andrea, Barberis, Marco, Brunetti, Maura, Renton, Alan E., Nalls, Mike A., Traynor, Bryan J., Restagno, Gabriella, Chiò, Adriano, Logullo, Francesco O., Simone, Isabella, Logroscino, Giancarlo, Salvi, Fabrizio, Bartolomei, Ilaria, Capasso, Margherita, Caponnetto, Claudia, Mancardi, Gianluigi, Mandich, Paola, Origone, Paola, Conforti, Francesca L., Mora, Gabriele, Marinou, Kalliopi, Sideri, Riccardo, Lunetta, Christian, Penco, Silvana, Mosca, Lorena, Nilo, Riva, Pinter, Giuseppe Lauria, Corbo, Massimo, Volanti, Paolo, Mandrioli, Jessica, Fini, Nicola, Georgoulopoulou, Eleni, Tremolizzo, Lucio, Maria Rosaria Monsurrò, Null, Tedeschi, Gioacchino, Cristillo, Viviana, la Bella, Vincenzo, Spataro, Rossella, Colletti, Tiziana, Sabatelli, Mario, Zollino, Marcella, Conte, Amelia, Luigetti, Marco, Lattante, Serena, Marangi, Giuseppe, Santarelli, Marialuisa, Petrucci, Antonio, Giannini, Fabio, Battistini, Stefania, Ricci, Claudia, Casale, Federico, Marrali, Giuseppe, Fuda, Giuseppe, Ossola, Irene, Cammarosano, Stefania, Ilardi, Antonio, Bertuzzo, Davide, Tanel, Raffaella, Pisano, Fabrizio, Costantino, Emanuela, Pani, Carla, Puddu, Roberta, Caredda, Carla, Piras, Valeria, Tranquilli, Stefania, Cuccu, Stefania, Corongiu, Daniela, Melis, Maurizio, Milia, Antonio, Pirisi, Angelo, Occhineri, Patrizia, Ortu, Enzo, Borghero, G, Pugliatti, M, Marrosu, F, Marrosu, M, Murru, M, Floris, G, Cannas, A, Parish, L, Cau, T, Loi, D, Ticca, A, Traccis, S, Manera, U, Canosa, A, Moglia, C, Calvo, A, Barberis, M, Brunetti, M, Renton, A, Nalls, M, Traynor, B, Restagno, G, Chiò, A, Logullo, F, Simone, I, Logroscino, G, Salvi, F, Bartolomei, I, Capasso, M, Caponnetto, C, Mancardi, G, Mandich, P, Origone, P, Conforti, F, Mora, G, Marinou, K, Sideri, R, Lunetta, C, Penco, S, Mosca, L, Nilo, R, Pinter, G, Corbo, M, Volanti, P, Mandrioli, J, Fini, N, Georgoulopoulou, E, Tremolizzo, L, Monsurròad, M, Tedeschi, G, Cristillo, V, la Bella, V, Spataro, R, Colletti, T, Sabatelli, M, Zollino, M, Conte, A, Luigetti, M, Lattante, S, Marangi, G, Santarelli, M, Petrucci, A, Giannini, F, Battistini, S, Ricci, C, Casale, F, Marrali, G, Fuda, G, Ossola, I, Cammarosano, S, Ilardi, A, Bertuzzo, D, Tanel, R, Pisano, F, Costantino, E, Pani, C, Puddu, R, Caredda, C, Piras, V, Tranquilli, S, Cuccu, S, Corongiu, D, Melis, M, Milia, A, Pirisi, A, Occhineri, P, and Ortu, E
- Subjects
Male ,Aging ,Amyotrophic lateral sclerosis ,Ataxin 2 gene ,Genetic modifier ,Neurology (clinical) ,Neuroscience (all) ,Developmental Biology ,Geriatrics and Gerontology ,Genetic Association Studie ,Biology ,Settore MED/03 - GENETICA MEDICA ,Article ,NO ,03 medical and health sciences ,0302 clinical medicine ,Risk Factors ,medicine ,Humans ,Risk factor ,10. No inequality ,Gene ,Amyotrophic Lateral Sclerosis ,Ataxin-2 ,Female ,Italy ,Middle Aged ,Survival Rate ,Trinucleotide Repeat Expansion ,Genetic Association Studies ,Phenotype ,Amyotrophic lateral sclerosi ,030304 developmental biology ,Genetics ,0303 health sciences ,Risk Factor ,Medicine (all) ,General Neuroscience ,Healthy subjects ,medicine.disease ,Trinucleotide repeat expansion ,030217 neurology & neurosurgery ,Human - Abstract
Intermediate-length CAG expansions (encoding 27-33 glutamines, polyQ) of the Ataxin2 (ATXN2) gene represent a risk factor for amyotrophic lateral sclerosis (ALS). Recently, it has been proposed that >=31 CAG expansions may influence ALS phenotype. We assessed whether ATXN2 intermediate-length polyQ expansions influence ALS phenotype in a series of 375 patients of Sardinian ancestry. Controls were 247 neurologically healthy subjects, resident in the study area, age- and gender-matched to cases. The frequency of >=31 polyQ ATNX2 repeats was significantly more common in ALS cases (4 patients vs. no control, p = 0.0001). All patients with >=31 polyQ repeats had a spinal onset versus 73.3% of patients with =31 polyQ repeats of the ATXN2 gene influenced patients' phenotype, being associated to a spinal onset and a significantly shorter survival.
- Published
- 2015
54. Impact of treatment with dimethyl fumarate on sleep quality in patients with relapsing-remitting multiple sclerosis: A multicentre Italian wearable tracker study.
- Author
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Comi G, Leocani L, Ferini-Strambi L, Radaelli M, Costa GD, Lanzillo R, Lus G, Bianchi V, Traccis S, Capone F, Grimaldi LM, Salemi G, Cardillo A, and Zipoli V
- Abstract
Background: Sleep disorders are common in patients with multiple sclerosis and have a bidirectional interplay with fatigue and depression., Objective: To evaluate the effect of treatment with oral dimethyl fumarate on the quality of sleep in relapsing-remitting multiple sclerosis., Methods: This was a multicentre observational study with 223 relapsing-remitting multiple sclerosis subjects starting treatment with dimethyl fumarate ( n =177) or beta interferon ( n =46). All patients underwent subjective (Pittsburgh Sleep Quality Index) and objective (wearable tracker) measurements of quality of sleep. Fatigue, depression, and quality of life were also investigated and physical activity was monitored., Results: Patients treated with dimethyl fumarate had significant improvement in the quality of sleep as measured with the Pittsburgh Sleep Quality Index ( p <0.001). At all-time points, no significant changes in Pittsburgh Sleep Quality Index score were observed in the interferon group. Total and deep sleep measured by wearable tracker decreased at week 12 with both treatments, then remained stable for the total study duration. Depression significantly improved in patients treated with dimethyl fumarate. No significant changes were observed in mobility, fatigue and quality of life., Conclusion: In patients with relapsing-remitting multiple sclerosis, the treatment with dimethyl fumarate was associated with improvements in patient-reported quality of sleep. Further randomised clinical trials are needed to confirm the benefits of long-term treatment with dimethyl fumarate., (© The Author(s), 2023.)
- Published
- 2023
- Full Text
- View/download PDF
55. TBK1 is associated with ALS and ALS-FTD in Sardinian patients.
- Author
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Borghero G, Pugliatti M, Marrosu F, Marrosu MG, Murru MR, Floris G, Cannas A, Occhineri P, Cau TB, Loi D, Ticca A, Traccis S, Manera U, Canosa A, Moglia C, Calvo A, Barberis M, Brunetti M, Gibbs JR, Renton AE, Errichiello E, Zoledziewska M, Mulas A, Qian Y, Din J, Pliner HA, Traynor BJ, and Chiò A
- Subjects
- Aged, Cohort Studies, Female, Humans, Italy, Male, Amyotrophic Lateral Sclerosis genetics, Frontotemporal Dementia genetics, Genetic Association Studies, Mutation, Protein Serine-Threonine Kinases
- Abstract
Recently, mutations in the TANK-binding kinase 1 (TBK1) gene were identified as a cause for amyotrophic lateral sclerosis (ALS) with or without comorbid frontotemporal dementia. We have assessed the frequency and clinical characteristics of TBK1 mutations in a cohort of ALS patients of Sardinian ancestry. Whole-exome sequencing was performed on Hiseq2000 platform (Illumina). Genome analysis Toolkit was used to align and to code variants according to Human Genome (UCSC hg19). Mutation was confirmed with Sanger sequence. In our screening of 186 Sardinian ALS cases, we found 3 (1.6%) patients carrying 3 distinct novel genetic variants: a nonsynonymous SNV c.1150C>T leading to a p.Arg384Thr change in exon 9; a nonsynonymous SNV c.1331G>A causes a p.Arg444Gln change in exon 11; and a frameshift deletion c.2070delG (p.Met690fs) at the exon 20 of the gene leading to a stop at 693 codon. The latter patients also carried missense mutation c.98C>T of the SQSTM1 gene causing a substitution of an arginine with a valine at the position 33 (p.Arg33Val). All variants were found to be deleterious according to in silico predictions. All cases were apparently sporadic and one of them showed frontotemporal dementia associated to ALS. These mutations were not found in 2 cohorts of 6780 ethnic-matched controls. We have found that TBK1 mutations account for 1.6% of Sardinian ALS cases. Our data support the notion that TBK1 is a novel ALS gene, providing important evidence complementary to the first descriptions., (Copyright © 2016 Elsevier Inc. All rights reserved.)
- Published
- 2016
- Full Text
- View/download PDF
56. ATXN2 is a modifier of phenotype in ALS patients of Sardinian ancestry.
- Author
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Borghero G, Pugliatti M, Marrosu F, Marrosu MG, Murru MR, Floris G, Cannas A, Parish LD, Cau TB, Loi D, Ticca A, Traccis S, Manera U, Canosa A, Moglia C, Calvo A, Barberis M, Brunetti M, Renton AE, Nalls MA, Traynor BJ, Restagno G, and Chiò A
- Subjects
- Amyotrophic Lateral Sclerosis mortality, Female, Humans, Italy, Male, Middle Aged, Risk Factors, Survival Rate, Trinucleotide Repeat Expansion genetics, Amyotrophic Lateral Sclerosis genetics, Ataxin-2 genetics, Genetic Association Studies, Phenotype
- Abstract
Intermediate-length CAG expansions (encoding 27-33 glutamines, polyQ) of the Ataxin2 (ATXN2) gene represent a risk factor for amyotrophic lateral sclerosis (ALS). Recently, it has been proposed that ≥31 CAG expansions may influence ALS phenotype. We assessed whether ATXN2 intermediate-length polyQ expansions influence ALS phenotype in a series of 375 patients of Sardinian ancestry. Controls were 247 neurologically healthy subjects, resident in the study area, age- and gender-matched to cases. The frequency of ≥31 polyQ ATNX2 repeats was significantly more common in ALS cases (4 patients vs. no control, p = 0.0001). All patients with ≥31 polyQ repeats had a spinal onset versus 73.3% of patients with <31 polyQ repeats. Patients with an increased number of polyQ repeats have a shorter survival than those with <31 repeats (1.2 vs. 4.2 years, p = 0.035). In this large series of ALS patients of Sardinian ancestry, we have found that ≥31 polyQ repeats of the ATXN2 gene influenced patients' phenotype, being associated to a spinal onset and a significantly shorter survival., Competing Interests: statement All other authors report no conflicts of interest., (Copyright © 2015 Elsevier Inc. All rights reserved.)
- Published
- 2015
- Full Text
- View/download PDF
57. Genetic architecture of ALS in Sardinia.
- Author
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Borghero G, Pugliatti M, Marrosu F, Marrosu MG, Murru MR, Floris G, Cannas A, Parish LD, Occhineri P, Cau TB, Loi D, Ticca A, Traccis S, Manera U, Canosa A, Moglia C, Calvo A, Barberis M, Brunetti M, Pliner HA, Renton AE, Nalls MA, Traynor BJ, Restagno G, and Chiò A
- Subjects
- Age of Onset, Amyotrophic Lateral Sclerosis epidemiology, C9orf72 Protein, DNA Repeat Expansion, DNA-Binding Proteins genetics, Genotype, Humans, Italy epidemiology, Penetrance, Phenotype, Proteins genetics, RNA-Binding Protein FUS genetics, Superoxide Dismutase genetics, Superoxide Dismutase-1, Amyotrophic Lateral Sclerosis genetics, Genetic Association Studies, Genetic Predisposition to Disease genetics, Mutation
- Abstract
Conserved populations, such as Sardinians, displaying elevated rates of familial or sporadic amyotrophic lateral sclerosis (ALS) provide unique information on the genetics of the disease. Our aim was to describe the genetic profile of a consecutive series of ALS patients of Sardinian ancestry. All ALS patients of Sardinian ancestry, identified between 2008 and 2013 through the Italian ALS Genetic Consortium, were eligible to be included in the study. Patients and controls underwent the analysis of TARDBP, C9ORF72, SOD1, and FUS genes. Genetic mutations were identified in 155 out of 375 Sardinian ALS cases (41.3%), more commonly the p.A382T and p.G295S mutations of TARDBP and the GGGGCC hexanucleotide repeat expansion of C9ORF72. One patient had both p.G295S and p.A382T mutations of TARDBP and 8 carried both the heterozygous p.A382T mutation of TARDBP and a repeat expansion of C9ORF72. Patients carrying the p.A382T and the p.G295S mutations of TARDBP and the C9ORF72 repeat expansion shared distinct haplotypes across these loci. Patients with cooccurrence of C9ORF72 and TARDBP p.A382T missense mutation had a significantly lower age at onset and shorter survival. More than 40% of all cases on the island of Sardinia carry a mutation of an ALS-related gene, representing the highest percentage of ALS cases genetically explained outside of Scandinavia. Clinical phenotypes associated with different genetic mutations show some distinctive characteristics, but the heterogeneity between and among families carrying the same mutations implies that ALS manifestation is influenced by other genetic and nongenetic factors., (Copyright © 2014 Elsevier Inc. All rights reserved.)
- Published
- 2014
- Full Text
- View/download PDF
58. Primary motor cortex hyperexcitability in Fabry's disease.
- Author
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Ortu E, Fancellu L, Sau G, Falchi P, Traccis S, Pes GM, Ganau A, and Sechi G
- Subjects
- Adult, Analysis of Variance, Biophysics, Electromyography, Female, Humans, Magnetic Resonance Imaging, Male, Middle Aged, Neural Conduction physiology, Neural Inhibition physiology, Rest, Transcranial Magnetic Stimulation, Young Adult, Evoked Potentials, Motor physiology, Fabry Disease pathology, Motor Cortex physiopathology
- Abstract
Objective: Involvement of pyramidal cells and/or changes in excitability of brain areas remote from an ischemic stroke has been demonstrated. Since in Fabry disease (FD), specific cerebrovascular lesions are present, we thought to investigate motor cortex excitability, using transcranial magnetic stimulation., Methods: Resting (RMT) and active (AMT) motor threshold, input-output curve (IN-OUT), central motor conduction time (CMCT), cortical silent period (cSP), short and long interval intracortical inhibition (SICI and LICI), intracortical facilitation (ICF), short interval intracortical facilitation (SICF) and short afferent inhibition (SAI) were measured in the cortical representation of the right first dorsal interosseous muscle in 11 patients with FD and 11 sex- and age matched healthy subjects., Results: FD patients showed a significant increase of steepness in IN-OUT, ICF and SICF curves. RMT, AMT, CMCT, SICI, LICI and SAI were normal., Conclusions: Our data documented an increased activity of motor cortex glutamatergic excitatory circuits in FD, evident also in patients without brain MRI lesions. Following enzyme replacement treatment, this abnormality was partly reversed., Significance: We suggest that our findings are expression of subtle "biochemical brain lesions", due to an early involvement of neurons and/or astrocytes by the cascade of pathologic events leading to brain damage in FD., (Copyright © 2013 International Federation of Clinical Neurophysiology. Published by Elsevier Ireland Ltd. All rights reserved.)
- Published
- 2013
- Full Text
- View/download PDF
59. Amyotrophic lateral sclerosis in Sardinia, insular Italy, 1995-2009.
- Author
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Pugliatti M, Parish LD, Cossu P, Leoni S, Ticca A, Saddi MV, Ortu E, Traccis S, Borghero G, Puddu R, Chiò A, and Pirina P
- Subjects
- Adult, Age Factors, Aged, Aged, 80 and over, Female, Humans, Incidence, Italy epidemiology, Male, Middle Aged, Prevalence, Retrospective Studies, Amyotrophic Lateral Sclerosis epidemiology
- Abstract
Recent genetic studies suggest a Sardinian type of amyotrophic lateral sclerosis (ALS). Thus, ALS incidence, prevalence and survival were investigated in a large population of Sardinians aimed to disclose population-specific patterns and their temporal changes. This is a population-based incidence and prevalence study in northern and central Sardinia, insular Italy (over 700,000 population). Incidence rates were computed for the time interval 1995-2009 and by quinquennia. Prevalence was computed for prevalence days 31 December 2004 and 2009. Onset-based survival for 1995-2009 is also reported. All ALS patients (El Escorial Criteria) in the study area were retrospectively included. The ALS crude incidence from 2005-2009 was 2.5 (95 % CIs: 0.1, 4.9), 3.4 in men and 1.6 in women. Onset occurred most often between the age of 65-74 years in men and 55-64 years in women. The ALS incidence tended to increase over the period 1995-2009. The mean age at onset was 61.7 years with no difference based on gender, varying significantly from 59.9 years in 1995-1999 to 63.9 years in 2005-2009. On December 31, 2009, the ALS crude prevalence was 10.8 per 100,000 (95 % CIs: 8.6, 13.1), 13.8 in men and 8.0 in women, whereas it was 6.3 per 100,000 (95 % CIs: 4.1, 8.6) on December 31, 2004 (M:F ratio of 0.95). Mean survival from onset was 37.0 months, with no difference based on gender, and a tendency to decrease during the period 1995-2009, in relation to type and age of onset. The population-based incidence and prevalence data of ALS in Sardinians indicate an increase of the disease occurrence over the past 40 years, providing support for a population-specific variant of ALS in Sardinia.
- Published
- 2013
- Full Text
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60. Variants within the immunoregulatory CBLB gene are associated with multiple sclerosis.
- Author
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Sanna S, Pitzalis M, Zoledziewska M, Zara I, Sidore C, Murru R, Whalen MB, Busonero F, Maschio A, Costa G, Melis MC, Deidda F, Poddie F, Morelli L, Farina G, Li Y, Dei M, Lai S, Mulas A, Cuccuru G, Porcu E, Liang L, Zavattari P, Moi L, Deriu E, Urru MF, Bajorek M, Satta MA, Cocco E, Ferrigno P, Sotgiu S, Pugliatti M, Traccis S, Angius A, Melis M, Rosati G, Abecasis GR, Uda M, Marrosu MG, Schlessinger D, and Cucca F
- Subjects
- Genetic Predisposition to Disease, Genome-Wide Association Study, Humans, Polymorphism, Single Nucleotide, Adaptor Proteins, Signal Transducing genetics, Major Histocompatibility Complex, Multiple Sclerosis genetics, Proto-Oncogene Proteins c-cbl genetics
- Abstract
A genome-wide association scan of approximately 6.6 million genotyped or imputed variants in 882 Sardinian individuals with multiple sclerosis (cases) and 872 controls suggested association of CBLB gene variants with disease, which was confirmed in 1,775 cases and 2,005 controls (rs9657904, overall P = 1.60 x 10(-10), OR = 1.40). CBLB encodes a negative regulator of adaptive immune responses, and mice lacking the ortholog are prone to experimental autoimmune encephalomyelitis, the animal model of multiple sclerosis.
- Published
- 2010
- Full Text
- View/download PDF
61. POEMS syndrome: a case report.
- Author
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Bisail M, Cossu A, Massarelli G, Tanda F, Agnetti V, Traccis S, Cassisa L, Puliga MV, and Bartoli E
- Subjects
- Castleman Disease complications, Hepatomegaly etiology, Humans, Male, Middle Aged, Osteolysis, Splenomegaly etiology, Bone Neoplasms complications, Demyelinating Diseases etiology, Paraneoplastic Syndromes etiology, Plasmacytoma complications
- Abstract
A case of POEMS syndrome in a 43-year-old male with polyneuropathy, osteolytic lesion of the basin due to solitary IgA-lambda plasmacytoma, cutaneous scleroderma-like changes, diffuse lymphadenopathy and hepatosplenomegaly is described. Liver biopsy showed a regenerative process of the parenchyma without laboratory and histologic evidence of necrosis. A peculiar finding was the onset of a right hemiparesis in the absence of signs of vascular disease or other predisposing factors. The possible links between the peculiar elements of the POEMS syndrome are briefly analyzed and discussed.
- Published
- 1990
62. Diagnostic value of extensor digitorum brevis F-wave in L5 root compression.
- Author
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Aiello I, Patraskakis S, Sau GF, Zirattu G, Bissakou M, Patta G, and Traccis S
- Subjects
- Adult, Electromyography methods, Female, Humans, Male, Middle Aged, Nerve Compression Syndromes diagnosis, Nerve Compression Syndromes physiopathology, Spinal Nerve Roots physiopathology
- Abstract
The sensibility of F-wave in detecting lumbosacral radicular compression has been found to range from 65% to 18%. The present study was performed on 24 patients suffering from unilateral L5 compressive radiculopathy. The aim was to verify the reliability of extensor digitorum brevis (EDB) F-wave in the diagnosis of L5 root impairment, by using different parameters such as minimal, mean, maximal latency and the difference of these parameters between the affected and unaffected sides. In all patients conventional needle EMG was also performed bilaterally. While the needle EMG showed abnormalities in L5 innervated muscles of all patients, at least one of the different EDB F-wave parameters was found to be abnormal in only 7 patients (29.2%). Moreover no significant relation was observed between the severity of EMG and F-wave abnormalities. We conclude that conventional needle EMG appears to be the most useful electrophysiological technique in the diagnosis of L5 compressive radiculopathy, while EDB F-wave does not provide additional information.
- Published
- 1990
63. Audio-ocular response: saccadic programming.
- Author
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Traccis S, Abel LA, and Dell'Osso LF
- Subjects
- Adult, Aerospace Medicine, Aircraft instrumentation, Fixation, Ocular, Humans, Time Factors, Acoustic Stimulation, Eye Movements, Ocular Physiological Phenomena, Saccades
- Abstract
The eye movements elicited by auditory stimuli--the audio-ocular response (AOR)--differ from those made in response to a visual target. The movements consist of both monosaccadic and multiple saccadic refixations (MSR). In visual refixation, monosaccadic refixations are always accurate; in AOR, they rarely are. In MSR, many strategies were used in the attempt to find the target but they were not always successful. However, final amplitudes of the total refixation were quite accurate in both MSR and monosaccadic refixations. Velocity profiles of the AOR showed such anomalies as discrete decelerations and multiple, closely-spaced saccades. These data suggest that, without visual feedback, the location of acoustic targets is difficult. In the absence of visual afference, when vigilance may be decreased by the lack of arousal, the velocity profiles also became abnormal, even at small amplitudes. Thus, for cockpit warning devices, a combination of auditory and visual indicators should be used.
- Published
- 1984
64. Unusual manifestation of hereditary neuropathy with liability to pressure palsy.
- Author
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Traccis S, Pirisi A, and Mutani R
- Subjects
- Adolescent, Adult, Bone Neoplasms complications, Female, Humans, Male, Middle Aged, Osteosarcoma complications, Nerve Compression Syndromes genetics
- Published
- 1982
65. Standardization of F wave parameters from extensor digitorum brevis muscle.
- Author
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Aiello I, Sau GF, Traccis S, Patraskakis S, Bissakou M, and Rosati G
- Subjects
- Adolescent, Adult, Female, Forearm, Humans, Male, Middle Aged, Reference Values, Electromyography standards, Muscles physiology
- Published
- 1987
66. Peripheral nerve function in rats chronically treated with carbamazepine.
- Author
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Moglia A, Arrigo A, Zandrini C, Scelsi R, Mutani R, Monaco F, Fassio F, and Traccis S
- Subjects
- Animals, Female, Rats, Rats, Inbred Strains, Carbamazepine toxicity, Peripheral Nervous System Diseases chemically induced
- Published
- 1983
67. Standardization of changes in M-wave area to repetitive nerve stimulation.
- Author
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Aiello I, Sau GF, Bissakou M, Patraskakis S, and Traccis S
- Subjects
- Adolescent, Adult, Aged, Electric Stimulation, Humans, Middle Aged, Ulnar Nerve physiology, Electromyography standards, Muscles innervation, Peripheral Nerves physiology
- Published
- 1986
68. Dissociated patellar reflex in a case of multiple sclerosis.
- Author
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Mutani R, Azzena GB, and Traccis S
- Subjects
- Adolescent, Electrophysiology, Humans, Male, Multiple Sclerosis physiopathology, Patella physiopathology, Reflex physiology
- Published
- 1982
- Full Text
- View/download PDF
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