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104. [P2-252]: ASSOCIATION OF HDL SUBSPECIES WITH OR WITHOUT APOLIPOPROTEIN E WITH ALZHEIMER's DISEASE NEUROPATHOLOGY: THE GINKGO EVALUATION OF MEMORY STUDY

Author

Koch, Manja, primary, Fitzpatrick, Annette L., additional, Furtado, Jeremy D., additional, DeKosky, Steven, additional, Kuller, Lewis H., additional, Lopez, Oscar L., additional, Hughes, Timothy M., additional, Mackey, Rachel H., additional, Mukamal, Kenneth J., additional, and Jensen, Majken K., additional

Published
2017
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105. Human Leukocyte Antigen Shared Epitope and Inflammation, Cardiovascular Disease, Cancer, and Mortality Among Postmenopausal Women in the Women's Health Initiative Rheumatoid Arthritis Study

Author

Birru Talabi, Mehret, primary, Mackey, Rachel H., additional, Kuller, Lewis H., additional, Dorman, Janice S., additional, Deane, Kevin D., additional, Robinson, William H., additional, Walitt, Brian T., additional, Chang, Yuefang, additional, Holers, V. Michael, additional, Liu, Simin, additional, and Moreland, Larry W., additional

Published
2017
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112. Heart disease and stroke statistics--2014 update: a report from the American Heart Association

Author

Go, Alan S, Mozaffarian, Dariush, Roger, Véronique L, Benjamin, Emelia J, Berry, Jarett D, Blaha, Michael J, Dai, Shifan, Ford, Earl S, Fox, Caroline S, Franco, Sheila, Fullerton, Heather J, Gillespie, Cathleen, Hailpern, Susan M, Heit, John A, Howard, Virginia J, Huffman, Mark D, Judd, Suzanne E, Kissela, Brett M, Kittner, Steven J, Lackland, Daniel T, Lichtman, Judith H, Lisabeth, Lynda D, Mackey, Rachel H, Magid, David J, Marcus, Gregory M, Marelli, Ariane, Matchar, David B, McGuire, Darren K, Mohler, Emile R, Moy, Claudia S, Mussolino, Michael E, Neumar, Robert W, Nichol, Graham, Pandey, Dilip K, Paynter, Nina P, Reeves, Matthew J, Sorlie, Paul D, Stein, Joel, Towfighi, Amytis, Turan, Tanya N, Virani, Salim S, Wong, Nathan D, Woo, Daniel, Turner, Melanie B, and American Heart Association Statistics Committee and Stroke Statistics Subcommittee

Subjects
Heart Diseases, Clinical Sciences, Cardiology, American Heart Association, Cardiorespiratory Medicine and Haematology, stroke, United States, cardiovascular diseases, AHA Scientific Statements, Cardiovascular System & Hematology, American Heart Association Statistics Committee and Stroke Statistics Subcommittee, statistics, Public Health and Health Services, Humans, risk factors, epidemiology
Abstract

Each year, the American Heart Association (AHA), in conjunction with the Centers for Disease Control and Prevention, the National Institutes of Health, and other government agencies, brings together the most up-to-date statistics on heart disease, stroke, other vascular diseases, and their risk factors and presents them in its Heart Disease and Stroke Statistical Update. The Statistical Update is a critical resource for researchers, clinicians, healthcare policy makers, media professionals, the lay public, and many others who seek the best available national data on heart disease, stroke, and other cardiovascular disease-related morbidity and mortality and the risks, quality of care, use of medical procedures and operations, and costs associated with the management of these diseases in a single document. Indeed, since 1999, the Statistical Update has been cited >10 500 times in the literature, based on citations of all annual versions. In 2012 alone, the various Statistical Updates were cited ≈3500 times (data from Google Scholar). In recent years, the Statistical Update has undergone some major changes with the addition of new chapters and major updates across multiple areas, as well as increasing the number of ways to access and use the information assembled. For this year's edition, the Statistics Committee, which produces the document for the AHA, updated all of the current chapters with the most recent nationally representative data and inclusion of relevant articles from the literature over the past year. This year's edition includes a new chapter on peripheral artery disease, as well as new data on the monitoring and benefits of cardiovascular health in the population, with additional new focus on evidence-based approaches to changing behaviors, implementation strategies, and implications of the AHA's 2020 Impact Goals. Below are a few highlights from this year's Update. © 2013 American Heart Association, Inc.

Published
2014

115. Lipids, Menopause and Early Atherosclerosis in SWAN Heart Women: Menopausal Transition and Lipids

Author

Woodard, Genevieve A., Brooks, Maria M., Barinas-Mitchell, Emma, Mackey, Rachel H., Matthews, Karen A., and Sutton-Tyrrell, Kim

Subjects
Adult, Cholesterol, HDL, Calcinosis, Middle Aged, Atherosclerosis, Article, Risk Factors, Humans, Women's Health, Carotid Stenosis, Female, Community Health Services, Longitudinal Studies, Menopause, Tunica Intima, Tunica Media
Abstract

The risk of cardiovascular disease increases after menopause. Recent evidence suggests that it is possible for high-density lipoprotein (HDL) to become proatherogenic or dysfunctional in certain situations. Our objective was to evaluate whether the relationship of HDL cholesterol (HDL-C) to subclinical cardiovascular disease differed across the menopausal transition, which would provide insight for this increased risk.Aortic calcification (AC), coronary artery calcification (CAC), carotid plaque, and intima media thickness (IMT) were measured in an ancillary study of the Study of Women's Health Across the Nation. Women not using hormone therapy were stratified into premenopausal or early perimenopausal (Pre/EP, n=316) and late perimenopausal or postmenopausal (LP/Post, n=224).The inverse relationship of HDL-C to subclinical atherosclerosis measures among Pre/EP women was weaker or reversed among LP/Post women, adjusted for age, site, race, systolic blood pressure, glucose, body mass index, smoking, menopause status, and low-density lipoprotein cholesterol. Specifically, multivariable modeling demonstrated an inverse association between HDL-C level and AC and IMT among Pre/EP women; however, the protective effect of HDL-C for AC, left main CAC, carotid plaque, and IMT was not seen in LP/Post women. In a small subset (n=53), LP/Post women had more total and small HDL particles, higher triglyceride levels, and more total low-density lipoprotein particles compared with Pre/EP women (P0.05).These results suggest that the protective effect of HDL may be diminished as women transition in menopause. Future studies should examine whether this may be due to changes in HDL size, functionality, or related changes in other lipids or lipoproteins.

Published
2011

116. Executive Summary: Heart Disease and Stroke Statistics—2016 Update

Author

Mozaffarian, Dariush, primary, Benjamin, Emelia J., additional, Go, Alan S., additional, Arnett, Donna K., additional, Blaha, Michael J., additional, Cushman, Mary, additional, Das, Sandeep R., additional, de Ferranti, Sarah, additional, Després, Jean-Pierre, additional, Fullerton, Heather J., additional, Howard, Virginia J., additional, Huffman, Mark D., additional, Isasi, Carmen R., additional, Jiménez, Monik C., additional, Judd, Suzanne E., additional, Kissela, Brett M., additional, Lichtman, Judith H., additional, Lisabeth, Lynda D., additional, Liu, Simin, additional, Mackey, Rachel H., additional, Magid, David J., additional, McGuire, Darren K., additional, Mohler, Emile R., additional, Moy, Claudia S., additional, Muntner, Paul, additional, Mussolino, Michael E., additional, Nasir, Khurram, additional, Neumar, Robert W., additional, Nichol, Graham, additional, Palaniappan, Latha, additional, Pandey, Dilip K., additional, Reeves, Mathew J., additional, Rodriguez, Carlos J., additional, Rosamond, Wayne, additional, Sorlie, Paul D., additional, Stein, Joel, additional, Towfighi, Amytis, additional, Turan, Tanya N., additional, Virani, Salim S., additional, Woo, Daniel, additional, Yeh, Robert W., additional, and Turner, Melanie B., additional

Published
2016
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118. High‐Density Lipoprotein Subclasses and Noncardiovascular, Noncancer Chronic Inflammatory‐Related Events Versus Cardiovascular Events: The Multi‐Ethnic Study of Atherosclerosis

Author

Duprez, Daniel A., primary, Otvos, James, additional, Tracy, Russell P., additional, Feingold, Kenneth R., additional, Greenland, Philip, additional, Gross, Myron D., additional, Lima, Joao A. C., additional, Mackey, Rachel H., additional, Neaton, James D., additional, Sanchez, Otto A., additional, and Jacobs, David R., additional

Published
2015
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124. Human Leukocyte Antigen Shared Epitope and Inflammation, Cardiovascular Disease, Cancer, and Mortality Among Postmenopausal Women in the Women's Health Initiative Rheumatoid Arthritis Study.

Author

Talabi, Mehret Birru, Mackey, Rachel H., Kuller, Lewis H., Dorman, Janice S., Deane, Kevin D., Robinson, William H., Walitt, Brian T., Yuefang Chang, Holers, V. Michael, Simin Liu, and Moreland, Larry W.

Subjects
*CARDIOVASCULAR diseases, *GENES, *INFLAMMATION, *MORTALITY, *RHEUMATOID arthritis, *TUMORS, *HLA-B27 antigen, *POSTMENOPAUSE
Abstract

Specific alleles of the human leukocyte antigen (HLA)-DRB1 gene (HLA-DRB1) encode a "shared epitope" (SE) associated with rheumatoid arthritis (RA), especially more severe cyclic-citrullinated peptide antibody-positive (anti-CCP+) RA. We evaluated associations of number of SE alleles (0, 1, or 2) with total and cardiovascular disease (CVD) mortality and incident coronary heart disease (CHD), CVD, and cancer over a mean 8.9 (standard deviation, 3.5) years of follow-up, stratifying by baseline anti-CCP status (positive (+) vs. negative (-)). A longitudinal study, the Women's Health Initiative RA Study (1993-2010), sampled postmenopausal women who reported RA at baseline (1993-1998) or follow-up in the Women's Health Initiative, classified as anti-CCP+ RA (n = 556) or anti-CCP- non-RA (n = 1,070). Among anti-CCP+ RA women, SE alleles were not related to age-adjusted risks of CHD, CVD, or cancer or to total or CVD mortality. Among anti-CCP- non-RA women, age-adjusted hazard ratios for 1 and 2 SE alleles versus 0 SE alleles were 0.41 (95% confidence interval (CI): 0.34, 0.50) and 0.44 (95% CI: 0.27, 0.72), respectively, for CVD; 0.43 (95% CI: 0.37, 0.53) and 0.30 (95% CI: 0.16, 0.64), respectively, for CHD; and 0.62 (95% CI: 0.53, 0.73) and 0.52 (95% CI: 0.33, 0.83), respectively, for cancer. Associations persisted after adjustment for CVD risk factors, joint pain, rheumatoid factor positivity, and inflammatory markers (white blood cell count or cytokine level). In future studies, investigators should evaluate SE associations among anti-CCP- adults without RA and potential mechanisms. [ABSTRACT FROM AUTHOR]

Published
2017
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125. Severe obesity, heart disease, and death among white, African American, and Hispanic postmenopausal women.

Author

McTigue, Kathleen M, McTigue, Kathleen M, Chang, Yue-Fang, Eaton, Charles, Garcia, Lorena, Johnson, Karen C, Lewis, Cora E, Liu, Simin, Mackey, Rachel H, Robinson, Jennifer, Rosal, Milagros C, Snetselaar, Linda, Valoski, Alice, Kuller, Lewis H, McTigue, Kathleen M, McTigue, Kathleen M, Chang, Yue-Fang, Eaton, Charles, Garcia, Lorena, Johnson, Karen C, Lewis, Cora E, Liu, Simin, Mackey, Rachel H, Robinson, Jennifer, Rosal, Milagros C, Snetselaar, Linda, Valoski, Alice, and Kuller, Lewis H

Abstract

ObjectiveTo compare mortality, nonfatal coronary heart disease (CHD), and congestive heart failure (CHF) risk across BMI categories in white, African American, and Hispanic women, with a focus on severe obesity (BMI ≥ 40), and examine heterogeneity in weight-related CHD risk.MethodsAmong 156,775 Women's Health Initiative observational study and clinical trial participants (September 1993-12 September 2005), multivariable Cox models estimated relative risk for mortality, CHD, and CHF. CHD incidence was calculated by anthropometry, race, and cardiovascular risk factors (CVRF).ResultsMortality, nonfatal CHD, and CHF incidence generally rose with BMI category. For severe obesity versus normal BMI, hazard ratios (HRs, 95% confidence interval) for mortality were 1.97 (1.77-2.20) in white, 1.55 (1.20-2.00) in African American, and 2.59 (1.55-4.31) in Hispanic women; for CHD, HRs were 2.05 (1.80-2.35), 2.24 (1.57-3.19), and 2.95 (1.60-5.41) respectively; for CHF, HRs were 5.01 (4.33-5.80), 3.60 (2.30-5.62), and 6.05 (2.49-14.69). CVRF variation resulted in substantial variation in CHD rates across BMI categories, even in severe obesity. CHD incidence was similar by race/ethnicity when differences in BMI or CVRF were accounted for.ConclusionsSevere obesity increases mortality, nonfatal CHD, and CHF risk in women of diverse race/ethnicity. CVRF heterogeneity contributes to variation in CHD incidence even in severe obesity.

Published
2014

126. The adipokine profile of metabolically benign obese and at-risk normal weight postmenopausal women: the Women's Health Initiative Observational Study.

Author

Khan, Unab I, Khan, Unab I, Ogorodnikova, Alexandra D, Xu, Linzhi, Wang, Dan, Wassertheil-Smoller, Sylvia, Ho, Gloria YF, Sowers, Mary Fran R, Rajpathak, Swapnil N, Allison, Matthew A, Mackey, Rachel H, Vitolins, Mara Z, Manson, Joann E, Wildman, Rachel P, Khan, Unab I, Khan, Unab I, Ogorodnikova, Alexandra D, Xu, Linzhi, Wang, Dan, Wassertheil-Smoller, Sylvia, Ho, Gloria YF, Sowers, Mary Fran R, Rajpathak, Swapnil N, Allison, Matthew A, Mackey, Rachel H, Vitolins, Mara Z, Manson, Joann E, and Wildman, Rachel P

Abstract

ObjectiveNearly, a third of obese individuals, termed metabolically benign obese, have a low burden of adiposity-related cardiometabolic abnormalities, whereas a substantial proportion of normal-weight individuals possess risk factors.MethodsIn cross-sectional analyses of 699 normal weight and 1,294 overweight/obese postmenopausal women enrolled in a nested case-control stroke study ancillary to the Women's Health Initiative Observational Study, we compared levels of adiponectin, leptin, and resistin among metabolically benign normal weight, at-risk normal weight, metabolically benign obese, and at-risk obese women using components of the ATP III definition of the metabolic syndrome (metabolically benign: ≤1 of the four components; at-risk phenotype: ≥2 components or diabetes).ResultsOverall, 382/699 normal-weight women (54.6%) and 328/1,194 overweight/obese women (27.5%) were metabolically benign. Among normal-weight women, at-risk women had higher leptin and lower adiponectin levels compared to metabolically benign women; multivariate-adjusted odds ratios were significant for having leptin (OR: 2.51; 95% CI: 1.28-5.01) and resistin (1.46; 1.03-2.07) in the top tertile and adiponectin in the bottom tertile (2.64; 1.81-3.84). Compared to metabolically benign overweight/obese women, at-risk obese women had higher odds of having leptin in the top tertile (1.62; 1.24-2.12) and adiponectin in the bottom tertile (2.78; 2.04-3.77).ConclusionsOverall, metabolically benign overweight/obese women had an intermediate adipokine profile (between at-risk obese and metabolically benign normal-weight women), whereas at-risk normal-weight women had a less favorable profile compared to metabolically benign normal-weight women. As adiponectin was the only adipokine independent of BMI, it may be most likely to have a role in the etiological pathway of these phenotypes.

Published
2014

128. Incorrect Values in Table 4 of the Article by Kuller et al (Arthritis Rheumatol, March 2014)

Author

Kuller, Lewis H., primary, Mackey, Rachel H., additional, Walitt, Brian T., additional, Deane, Kevin D., additional, Holers, V. Michael, additional, Robinson, William H., additional, Sokolove, Jeremy, additional, Chang, Yuefang, additional, Liu, Simin, additional, Parks, Christine G., additional, Wright, Nicole C., additional, and Moreland, Larry W., additional

Published
2014
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129. Abstract MP60: Fine Mapping of Body Fat Distribution Loci in African American Populations: The Population Architecture Using Genomics and Epidemiology (PAGE) Study

Author

Yoneyama, Sachiko, primary, Graff, Misa, additional, Lim, Unhee, additional, Gong, Jian, additional, Fornage, Myriam, additional, Isasi, Carmen, additional, Buzkova, Petra, additional, Daviglus, Martha, additional, Wilkens, Lynne, additional, Kooperberg, Charles, additional, Buyske, Steve, additional, Li, Rongling, additional, Hindorff, Lucia, additional, Kuller, Lewis H, additional, Manson, JoAnn E, additional, Sethupathy, Praveen, additional, Mackey, Rachel H, additional, Houston, Denise K, additional, Gu, C. Charles, additional, Cooper, Richard, additional, Lewis, Cora E, additional, Leppert, Mark, additional, Irvin, Marguerite R, additional, Peters, Ulrike, additional, and North, Kari E, additional

Published
2014
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131. Update on Cardiovascular Disease Risk in Patients with Rheumatic Diseases

Author

Mackey, Rachel H., Kuller, Lewis H., and Moreland, Larry W.

Abstract

Cardiovascular disease (CVD) risk is 1.5-fold higher in rheumatoid arthritis (RA), partly due to subclinical atherosclerosis that develops before the diagnosis of RA. Dyslipidemia in RA is better quantified by lipoproteins and apolipoproteins than by cholesterol levels. Current risk factors likely underestimate CVD risk by underestimating prior risk factor levels. Some of the 2-fold higher risk of heart failure and total mortality in RA may be due to myocardial disease caused by inflammation. Per recent recommendations, to reduce CVD risk in RA, control disease activity, reduce inflammation, and aggressively treat CVD risk factors.

Published
2018
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132. Severe obesity, heart disease, and death among white, african american, and hispanic postmenopausal women

Author

McTigue, Kathleen M., primary, Chang, Yue-Fang, additional, Eaton, Charles, additional, Garcia, Lorena, additional, Johnson, Karen C., additional, Lewis, Cora E., additional, Liu, Simin, additional, Mackey, Rachel H., additional, Robinson, Jennifer, additional, Rosal, Milagros C., additional, Snetselaar, Linda, additional, Valoski, Alice, additional, and Kuller, Lewis H., additional

Published
2014
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133. Regional pulse wave velocities and their cardiovascular risk factors among healthy middle-aged men: a cross-sectional population-based study

Author

Choo, Jina, primary, Shin, Chol, additional, Barinas-Mitchell, Emma, additional, Masaki, Kamal, additional, Willcox, Bradley J, additional, Seto, Todd B, additional, Ueshima, Hirotsugu, additional, Lee, Sunghee, additional, Miura, Katsuyuki, additional, Venkitachalam, Lakshmi, additional, Mackey, Rachel H, additional, Evans, Rhobert W, additional, Kuller, Lewis H, additional, Sutton-Tyrrell, Kim, additional, and Sekikawa, Akira, additional

Published
2014
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134. The adipokine profile of metabolically benign obese and at‐risk normal weight postmenopausal women: The Women's Health Initiative Observational Study

Author

Khan, Unab I., primary, Ogorodnikova, Alexandra D., additional, Xu, Linzhi, additional, Wang, Dan, additional, Wassertheil‐Smoller, Sylvia, additional, Ho, Gloria Y.F., additional, Sowers, Mary Fran R., additional, Rajpathak, Swapnil N., additional, Allison, Matthew A., additional, Mackey, Rachel H., additional, Vitolins, Mara Z., additional, Manson, JoAnn E., additional, and Wildman, Rachel P., additional

Published
2013
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135. Abstract P291: Adipocytokines, Lipoproteins & Obesity Among Black & White Postmenopausal Women from the Women’s Health Initiative

Author

Mackey, Rachel H, primary, McTigue, Kathleen M, additional, Barinas-Mitchell, Emma, additional, Evans, Rhobert W, additional, Tinker, Lesley F, additional, Manson, JoAnn E, additional, Liu, Simin, additional, Howard, Barbara V, additional, Robinson, Jennifer G, additional, Stefanick, Marcia L, additional, Phillips, Lawrence S, additional, Allison, Matthew A, additional, Rosal, Milagros C, additional, Beresford, Shirley, additional, and Kuller, Lewis H, additional

Published
2013
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138. The Relationship of Cardiovascular Disease to Physical Functioning in Women Surviving to Age 80 and Above in the Women's Health Initiative.

Author

Stefanick, Marcia L., Johnson, Karen C., LaCroix, Andrea, Robinson, Jennifer G., Seguin, Rebecca A., Tindle, Hilary A., Wassertheil-Smoller, Sylvia, Brunner, Robert L., Leng, X., Hogan, Patricia E., Limacher, Marian C., Bird, Chloe E., Garcia, David O., LaMonte, Michael J., and Mackey, Rachel H.

Subjects
FUNCTIONAL assessment of older women, CARDIOVASCULAR diseases, DISEASES in women, PHYSIOLOGICAL aspects of aging, OLDER women, HEALTH, PHYSIOLOGY, GERIATRIC assessment, AGING, COMPARATIVE studies, FUNCTIONAL assessment, HEALTH status indicators, RESEARCH methodology, MEDICAL cooperation, QUALITY of life, QUESTIONNAIRES, RESEARCH, RESEARCH funding, SURVEYS, WOMEN'S health, EVALUATION research, DISEASE prevalence
Abstract

Background: Cardiovascular disease (CVD) is highly prevalent at ages 80 and above. The association of physical functioning (PF), a key to an optimal aging trajectory, with CVD and specific CVD diagnosis in women who survive to age 80 and above has not been described previously and has important public health significance given our aging population.Methods: Women's Health Initiative participants aged 80 years or older at the time of self-reporting PF (RAND SF-36) were studied in relationship to CVD diagnosis, whether present at study baseline (1993-1998) or diagnosed during follow-up through 2012. Cross-sectional analyses utilized demographic, medical, lifestyle, and psycho-social questionnaire data from baseline or updated at the time of self-reported PF.Results: Among 27,145 older Women's Health Initiative participants, 22.0% (N = 5,959) had been diagnosed with CVD, specifically: 11.3% (N = 3,071) with coronary heart disease; 4.7% (N = 1,279), stroke; 5.2% (N = 1,397), venous thromboembolism; 2.7% (N = 737), peripheral arterial disease; and 2.7% (N = 725), congestive heart failure. PF scores (mean ± SE) were significantly (p < .0001) higher without CVD (60.0 ± 26.9), compared with any CVD (47.9 ± 27.3), and for each specific CVD diagnosis: coronary heart disease (48.8 ± 27.1); stroke (44.8 ± 27.9); venous thromboembolism (48.9 ± 27.4); peripheral arterial disease (41.9 ± 2.2); and congestive heart failure (38.8 ± 26.1). Regardless of CVD diagnosis, higher PF was associated with: younger age at the time of PF assessment; lower body mass index; higher recreational physical activity; better self-reported general health; fewer hip fractures after age 55; no history of arthritis; and no recent use of non-steroidal anti-inflammatory drugs.Conclusions: Older women with any CVD, and particularly women with congestive heart failure or peripheral arterial disease, reported significantly lower PF compared to women with no CVD. Regardless of CVD diagnosis, higher PF was strongly associated with a more active lifestyle and lower body mass index, suggesting potential intervention targets for more optimal aging. [ABSTRACT FROM AUTHOR]

Published
2016
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140. Hormone Therapy, Estrogen Metabolism, and Risk of Breast Cancer in the Women's Health Initiative Hormone Therapy Trial

Author

Mackey, Rachel H., primary, Fanelli, Theresa J., additional, Modugno, Francesmary, additional, Cauley, Jane A., additional, McTigue, Kathleen M., additional, Brooks, Maria Mori, additional, Chlebowski, Rowan T., additional, Manson, JoAnn E., additional, Klug, Thomas L., additional, Kip, Kevin E., additional, Curb, J. David, additional, and Kuller, Lewis H., additional

Published
2012
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142. Abstract P303: Differential Association of Carotid-femoral, Brachial-ankle, and Femoral Ankle Pulse Wave Velocity (PWV) With Lipoprotein Distributions From The Era Jump Study

Author

Vishnu, Abhishek, primary, Shin, Chol, additional, Curb, J. David, additional, Mackey, Rachel H., additional, Barinas-Mitchell, Emma, additional, Masaki, Kamal, additional, El-Saed, Aiman, additional, Seto, Todd B, additional, Choo, Jina, additional, Fujiyoshi, Akira, additional, Miura, Katsuyuki, additional, Edmundowicz, Daniel, additional, Ueshima, Hirotsugu, additional, Kuller, Lewis H, additional, Sutton-Tyrrell, Kim, additional, and Sekikawa, Akira, additional

Published
2012
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