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107. Replication study and meta-analysis in European samples supports association of the 3p21.1 locus with bipolar disorder

Author

Vassos, E., Steinberg, S., Cichon, S., Breen, G., Sigurdsson, E., Andreassen, O.A., Djurovic, S., Morken, G., Grigoroiu-Serbanescu, M., Diaconu, C.C., Czerski, P.M., Hauser, J., Babadjanova, G., Abramova, L.I., Muhleisen, T.W., Nothen, Markus, Rietschel, M., McGuffin, P., St Clair, D., Gustafsson, O., Melle, I., Pietilainen, O.P., Ruggeri, M., Tosato, S., Werge, T., Ophoff, R.A., Consortium, G., Rujescu, D., Borglum, A.D., Mors, O., Mortensen, P.B., Demontis, D., Hollegaard, M.V., van Winkel, R., Kenis, G., De Hert, M., Rethelyi, J.M., Bitter, I., Rubino, I.A., Golimbet, V., Kiemeney, L.A., Berg, L.H. van den, Franke, B., Jonsson, E.G., Farmer, A., Stefansson, H., Stefansson, K., Collier, D.A., Vassos, E., Steinberg, S., Cichon, S., Breen, G., Sigurdsson, E., Andreassen, O.A., Djurovic, S., Morken, G., Grigoroiu-Serbanescu, M., Diaconu, C.C., Czerski, P.M., Hauser, J., Babadjanova, G., Abramova, L.I., Muhleisen, T.W., Nothen, Markus, Rietschel, M., McGuffin, P., St Clair, D., Gustafsson, O., Melle, I., Pietilainen, O.P., Ruggeri, M., Tosato, S., Werge, T., Ophoff, R.A., Consortium, G., Rujescu, D., Borglum, A.D., Mors, O., Mortensen, P.B., Demontis, D., Hollegaard, M.V., van Winkel, R., Kenis, G., De Hert, M., Rethelyi, J.M., Bitter, I., Rubino, I.A., Golimbet, V., Kiemeney, L.A., Berg, L.H. van den, Franke, B., Jonsson, E.G., Farmer, A., Stefansson, H., Stefansson, K., and Collier, D.A.

Abstract

Item does not contain fulltext, BACKGROUND: Common genetic polymorphisms at chromosome 3p21.1, including rs2251219 in polybromo 1 (PBRM1), have been implicated in susceptibility to bipolar affective disorder (BP) through genome-wide association studies. Subsequent studies have suggested that this is also a risk locus for other psychiatric phenotypes, including major depression and schizophrenia. METHODS: To replicate the association, we studied 2562 cases with BP and 25,439 control subjects collected from seven cohorts with either genome-wide association or individual genotyping of rs2251219 and tagging single nucleotide polymorphisms across the PBRM1 gene. Results from the different case-control groups were combined with the inverse variance weighting method. RESULTS: In our dataset, rs2251219 was associated with BP (odds ratio [OR] = .89, p = .003), and meta-analysis of previously published data with our nonoverlapping new data confirmed genome-wide significant association (OR = .875, p = 2.68 x 10(-9)). Genotypic data from the SGENE-plus consortium were used to examine the association of the same variant with schizophrenia in an overall sample of 8794 cases and 25,457 control subjects, but this was not statistically significant (OR = .97, p = .21). CONCLUSIONS: There is strong evidence of association of rs2251219 with BP. However, our data do not support association of this marker with schizophrenia. Because the region of association has high linkage disequilibrium, forming a large haplotype block across many genes, it is not clear which gene is causally implicated in the disorder.

Published
2012

108. Randomised controlled trial of colostrum to improve intestinal function in patients with short bowel syndrome

Author

Lund, Pernille, Sangild, Per Torp, Aunsholt, L., Hartmann, Bolette, Holst, Jens Juul, Mortensen, J., Mortensen, P.B., Jeppesen, P.B., Lund, Pernille, Sangild, Per Torp, Aunsholt, L., Hartmann, Bolette, Holst, Jens Juul, Mortensen, J., Mortensen, P.B., and Jeppesen, P.B.

Abstract

Colostrum is rich in immunoregulatory, antimicrobial and trophic components supporting intestinal development and function in newborns. We assessed whether bovine colostrum could enhance intestinal adaptation and function in adult short bowel syndrome (SBS) patients.

Published
2012

114. Dual association of a TRKA polymorphism with schizophrenia

Author

Schijndel, J.E. van, Zweeden, M. van, Loo, K.M.J. van, Djurovic, S., Andreassen, O.A., Hansen, T., Werge, T., Nyegaard, M., Sorensen, K.M., Nordentoft, M., Mortensen, P.B., Mors, O., Borglum, A.D., Del-Favero, J., Norrback, K.F., Adolfsson, R., Hert, M. De, Claes, S., Cichon, S., Rietschel, M., Nothen, Markus, Kallunki, P., Pedersen, J.T., Martens, G.J.M., Schijndel, J.E. van, Zweeden, M. van, Loo, K.M.J. van, Djurovic, S., Andreassen, O.A., Hansen, T., Werge, T., Nyegaard, M., Sorensen, K.M., Nordentoft, M., Mortensen, P.B., Mors, O., Borglum, A.D., Del-Favero, J., Norrback, K.F., Adolfsson, R., Hert, M. De, Claes, S., Cichon, S., Rietschel, M., Nothen, Markus, Kallunki, P., Pedersen, J.T., and Martens, G.J.M.

Abstract

Contains fulltext : 91735.pdf (Publisher’s version ) (Closed access)

Published
2011

115. Common variants at VRK2 and TCF4 conferring risk of schizophrenia

Author

Steinberg, S., Jong, S. de, Andreassen, O.A., Werge, T., Borglum, A.D., Mors, O., Mortensen, P.B., Gustafsson, O., Costas, J., Pietilainen, O.P.H., Demontis, D., Papiol, S., Huttenlocher, J., Mattheisen, M., Breuer, R., Vassos, E., Giegling, I., Fraser, G., Walker, N., Tuulio-Henriksson, A., Suvisaari, J., Lonnqvist, J., Paunio, T., Agartz, I., Melle, I., Djurovic, S., Strengman, E., Jurgens, G., Glenthoj, B., Terenius, L., Hougaard, D.M., Orntoft, T., Wiuf, C., Didriksen, M., Hollegaard, M.V., Nordentoft, M., Winkel, R. van, Kenis, G., Abramova, L.I., Kaleda, V., Arrojo, M., Sanjuan, J., Arango, C., Sperling, S., Rossner, M., Ribolsi, M., Magni, V., Siracusano, A., Christiansen, C., Kiemeney, L.A.L.M., Veldink, J., Berg, L. van den, Ingason, A., Muglia, P., Murray, R., Nothen, Markus, Sigurdsson, E., Petursson, H., Thorsteinsdottir, U., Kong, A., Rubino, I.A., Hert, M. de, Rethelyi, J.M., Bitter, I., Jonsson, E.G., Golimbet, V., Carracedo, A., Ehrenreich, H., Craddock, N., Owen, M.J., O'Donovan, M.C., Ruggeri, M., Tosato, S., Peltonen, L., Ophoff, R.A., Collier, D.A., St Clair, D., Rietschel, M., Cichon, S., Stefansson, H., Rujescu, D., Stefansson, K., Steinberg, S., Jong, S. de, Andreassen, O.A., Werge, T., Borglum, A.D., Mors, O., Mortensen, P.B., Gustafsson, O., Costas, J., Pietilainen, O.P.H., Demontis, D., Papiol, S., Huttenlocher, J., Mattheisen, M., Breuer, R., Vassos, E., Giegling, I., Fraser, G., Walker, N., Tuulio-Henriksson, A., Suvisaari, J., Lonnqvist, J., Paunio, T., Agartz, I., Melle, I., Djurovic, S., Strengman, E., Jurgens, G., Glenthoj, B., Terenius, L., Hougaard, D.M., Orntoft, T., Wiuf, C., Didriksen, M., Hollegaard, M.V., Nordentoft, M., Winkel, R. van, Kenis, G., Abramova, L.I., Kaleda, V., Arrojo, M., Sanjuan, J., Arango, C., Sperling, S., Rossner, M., Ribolsi, M., Magni, V., Siracusano, A., Christiansen, C., Kiemeney, L.A.L.M., Veldink, J., Berg, L. van den, Ingason, A., Muglia, P., Murray, R., Nothen, Markus, Sigurdsson, E., Petursson, H., Thorsteinsdottir, U., Kong, A., Rubino, I.A., Hert, M. de, Rethelyi, J.M., Bitter, I., Jonsson, E.G., Golimbet, V., Carracedo, A., Ehrenreich, H., Craddock, N., Owen, M.J., O'Donovan, M.C., Ruggeri, M., Tosato, S., Peltonen, L., Ophoff, R.A., Collier, D.A., St Clair, D., Rietschel, M., Cichon, S., Stefansson, H., Rujescu, D., and Stefansson, K.

Abstract

Contains fulltext : 97739.pdf (publisher's version ) (Closed access), Common sequence variants have recently joined rare structural polymorphisms as genetic factors with strong evidence for association with schizophrenia. Here we extend our previous genome-wide association study and meta-analysis (totalling 7 946 cases and 19 036 controls) by examining an expanded set of variants using an enlarged follow-up sample (up to 10 260 cases and 23 500 controls). In addition to previously reported alleles in the major histocompatibility complex region, near neurogranin (NRGN) and in an intron of transcription factor 4 (TCF4), we find two novel variants showing genome-wide significant association: rs2312147[C], upstream of vaccinia-related kinase 2 (VRK2) [odds ratio (OR) = 1.09, P = 1.9 x 10(-9)] and rs4309482[A], between coiled-coiled domain containing 68 (CCDC68) and TCF4, about 400 kb from the previously described risk allele, but not accounted for by its association (OR = 1.09, P = 7.8 x 10(-9)).

Published
2011

116. Expanding the range of ZNF804A variants conferring risk of psychosis

Author

Steinberg, S., Mors, O., Borglum, A.D., Gustafsson, O., Werge, T., Mortensen, P.B., Andreassen, O.A., Sigurdsson, E., Thorgeirsson, T.E., Bottcher, Y., Olason, P., Ophoff, R.A., Cichon, S., Gudjonsdottir, I.H., Pietilainen, O.P.H., Nyegaard, M., Tuulio-Henriksson, A., Ingason, A., Hansen, T., Athanasiu, L., Suvisaari, J., Lonnqvist, J., Paunio, T., Hartmann, A., Jurgens, G., Nordentoft, M., Hougaard, D., Norgaard-Pedersen, B., Breuer, R., Moller, H.J., Giegling, I., Glenthoj, B., Rasmussen, H.B., Mattheisen, M., Bitter, I., Rethelyi, J.M., Sigmundsson, T., Fossdal, R., Thorsteinsdottir, U., Ruggeri, M., Tosato, S., Strengman, E., Kiemeney, L.A.L.M., Melle, I., Djurovic, S., Abramova, L.I., Kaleda, V., Walshe, M., Bramon, E., Vassos, E., Li, T., Fraser, G., Walker, N., Toulopoulou, T., Yoon, J., Freimer, N.B., Cantor, R.M., Murray, R., Kong, A., Golimbet, V., Jonsson, E.G., Terenius, L., Agartz, I., Petursson, H., Nothen, Markus, Rietschel, M., Peltonen, L., Rujescu, D., Collier, D.A., Stefansson, H., St Clair, D., Stefansson, K., Steinberg, S., Mors, O., Borglum, A.D., Gustafsson, O., Werge, T., Mortensen, P.B., Andreassen, O.A., Sigurdsson, E., Thorgeirsson, T.E., Bottcher, Y., Olason, P., Ophoff, R.A., Cichon, S., Gudjonsdottir, I.H., Pietilainen, O.P.H., Nyegaard, M., Tuulio-Henriksson, A., Ingason, A., Hansen, T., Athanasiu, L., Suvisaari, J., Lonnqvist, J., Paunio, T., Hartmann, A., Jurgens, G., Nordentoft, M., Hougaard, D., Norgaard-Pedersen, B., Breuer, R., Moller, H.J., Giegling, I., Glenthoj, B., Rasmussen, H.B., Mattheisen, M., Bitter, I., Rethelyi, J.M., Sigmundsson, T., Fossdal, R., Thorsteinsdottir, U., Ruggeri, M., Tosato, S., Strengman, E., Kiemeney, L.A.L.M., Melle, I., Djurovic, S., Abramova, L.I., Kaleda, V., Walshe, M., Bramon, E., Vassos, E., Li, T., Fraser, G., Walker, N., Toulopoulou, T., Yoon, J., Freimer, N.B., Cantor, R.M., Murray, R., Kong, A., Golimbet, V., Jonsson, E.G., Terenius, L., Agartz, I., Petursson, H., Nothen, Markus, Rietschel, M., Peltonen, L., Rujescu, D., Collier, D.A., Stefansson, H., St Clair, D., and Stefansson, K.

Abstract

Contains fulltext : 95884.pdf (publisher's version ) (Closed access), A trio of genome-wide association studies recently reported sequence variants at three loci to be significantly associated with schizophrenia. No sequence polymorphism had been unequivocally (P<5 x 10(-8)) associated with schizophrenia earlier. However, one variant, rs1344706[T], had come very close. This polymorphism, located in an intron of ZNF804A, was reported to associate with schizophrenia with a P-value of 1.6 x 10(-7), and with psychosis (schizophrenia plus bipolar disorder) with a P-value of 1.0 x 10(-8). In this study, using 5164 schizophrenia cases and 20,709 controls, we replicated the association with schizophrenia (odds ratio OR = 1.08, P = 0.0029) and, by adding bipolar disorder patients, we also confirmed the association with psychosis (added N = 609, OR = 1.09, P = 0.00065). Furthermore, as it has been proposed that variants such as rs1344706[T]-common and with low relative risk-may also serve to identify regions harboring less common, higher-risk susceptibility alleles, we searched ZNF804A for large copy number variants (CNVs) in 4235 psychosis patients, 1173 patients with other psychiatric disorders and 39,481 controls. We identified two CNVs including at least part of ZNF804A in psychosis patients and no ZNF804A CNVs in controls (P = 0.013 for association with psychosis). In addition, we found a ZNF804A CNV in an anxiety patient (P = 0.0016 for association with the larger set of psychiatric disorders).

Published
2011

117. Common variants conferring risk of schizophrenia.

Author

Stefansson, H., Ophoff, R.A., Steinberg, S., Andreassen, O.A., Cichon, S., Rujescu, D., Werge, T., Pietilainen, O.P.H., Mors, O., Mortensen, P.B., Sigurdsson, E., Gustafsson, O., Nyegaard, M., Tuulio-Henriksson, A., Ingason, A., Hansen, T., Suvisaari, J., Lonnqvist, J., Paunio, T., Borglum, A.D., Hartmann, A., Fink-Jensen, A., Nordentoft, M., Hougaard, D., Norgaard-Pedersen, B., Bottcher, Y., Olesen, J., Breuer, R., Moller, H.J., Giegling, I., Rasmussen, H.B., Timm, S., Mattheisen, M., Bitter, I., Rethelyi, J.M., Magnusdottir, B.B., Sigmundsson, T., Olason, P., Masson, G., Gulcher, J.R., Haraldsson, M., Fossdal, R., Thorgeirsson, T.E., Thorsteinsdottir, U., Ruggeri, M., Tosato, S., Franke, B., Strengman, E., Kiemeney, L.A.L.M., Melle, I., Djurovic, S., Abramova, L.I., Kaleda, V., Sanjuan, J., Frutos, R. de, Bramon, E., Vassos, E., Fraser, G., Ettinger, U., Picchioni, M., Walker, N., Toulopoulou, T., Need, A.C., Ge, D., Yoon, J.L., Shianna, K.V., Freimer, N.B., Cantor, R.M., Murray, R., Kong, A., Golimbet, V., Carracedo, A., Arango, C., Costas, J., Jonsson, E.G., Terenius, L., Agartz, I., Petursson, H., Nothen, Markus, Rietschel, M., Matthews, P.M., Muglia, P., Peltonen, L., St Clair, D., Goldstein, D.B, Stefansson, K., Collier, D.A., Stefansson, H., Ophoff, R.A., Steinberg, S., Andreassen, O.A., Cichon, S., Rujescu, D., Werge, T., Pietilainen, O.P.H., Mors, O., Mortensen, P.B., Sigurdsson, E., Gustafsson, O., Nyegaard, M., Tuulio-Henriksson, A., Ingason, A., Hansen, T., Suvisaari, J., Lonnqvist, J., Paunio, T., Borglum, A.D., Hartmann, A., Fink-Jensen, A., Nordentoft, M., Hougaard, D., Norgaard-Pedersen, B., Bottcher, Y., Olesen, J., Breuer, R., Moller, H.J., Giegling, I., Rasmussen, H.B., Timm, S., Mattheisen, M., Bitter, I., Rethelyi, J.M., Magnusdottir, B.B., Sigmundsson, T., Olason, P., Masson, G., Gulcher, J.R., Haraldsson, M., Fossdal, R., Thorgeirsson, T.E., Thorsteinsdottir, U., Ruggeri, M., Tosato, S., Franke, B., Strengman, E., Kiemeney, L.A.L.M., Melle, I., Djurovic, S., Abramova, L.I., Kaleda, V., Sanjuan, J., Frutos, R. de, Bramon, E., Vassos, E., Fraser, G., Ettinger, U., Picchioni, M., Walker, N., Toulopoulou, T., Need, A.C., Ge, D., Yoon, J.L., Shianna, K.V., Freimer, N.B., Cantor, R.M., Murray, R., Kong, A., Golimbet, V., Carracedo, A., Arango, C., Costas, J., Jonsson, E.G., Terenius, L., Agartz, I., Petursson, H., Nothen, Markus, Rietschel, M., Matthews, P.M., Muglia, P., Peltonen, L., St Clair, D., Goldstein, D.B, Stefansson, K., and Collier, D.A.

Abstract

Contains fulltext : 81575.pdf (publisher's version ) (Closed access), Schizophrenia is a complex disorder, caused by both genetic and environmental factors and their interactions. Research on pathogenesis has traditionally focused on neurotransmitter systems in the brain, particularly those involving dopamine. Schizophrenia has been considered a separate disease for over a century, but in the absence of clear biological markers, diagnosis has historically been based on signs and symptoms. A fundamental message emerging from genome-wide association studies of copy number variations (CNVs) associated with the disease is that its genetic basis does not necessarily conform to classical nosological disease boundaries. Certain CNVs confer not only high relative risk of schizophrenia but also of other psychiatric disorders. The structural variations associated with schizophrenia can involve several genes and the phenotypic syndromes, or the 'genomic disorders', have not yet been characterized. Single nucleotide polymorphism (SNP)-based genome-wide association studies with the potential to implicate individual genes in complex diseases may reveal underlying biological pathways. Here we combined SNP data from several large genome-wide scans and followed up the most significant association signals. We found significant association with several markers spanning the major histocompatibility complex (MHC) region on chromosome 6p21.3-22.1, a marker located upstream of the neurogranin gene (NRGN) on 11q24.2 and a marker in intron four of transcription factor 4 (TCF4) on 18q21.2. Our findings implicating the MHC region are consistent with an immune component to schizophrenia risk, whereas the association with NRGN and TCF4 points to perturbation of pathways involved in brain development, memory and cognition.

Published
2009

118. Megafaunal diversity associated with marine landscapes of northern Norway: a preliminary assessment

Author

Mortensen, P.B., Buhl-Mortensen, L., Dolan, M., Dannheim, J., Kröger, K., Mortensen, P.B., Buhl-Mortensen, L., Dolan, M., Dannheim, J., and Kröger, K.

Abstract

The seabed of northern Norway was explored using video and multibeam surveys to assess the distribution of megafauna and examine associations of benthic organisms to their physical environment. The study area was initially divided into ten marine landscapes based on seabed morphology and general water mass distribution. In total 195 taxa were observed during video recordings. The highest number of taxa was found within fjord/coast and upper slope landscapes. Multivariate statistical methods were used to relate bottom environment and taxonomic composition and in order to find the relation between faunal groups and landscapes. Detrended Correspondence Analysis indicated four groups of video transects. The most important environmental factors influencing the groupings were depth, and the frequency of occurrence of mud along the transects. Video transects from canyons and the deep slope formed a distinct group based on faunal composition. The three other groups consisted of transects from banks, fans in outer trough areas, and upper slope/slope terraces respectively. Based on the groups defined by DCA a new set of five landscape classes could be defined. The results indicate that broad-scale topographic features and general hydrography are of relevance for the distribution and composition of megafauna. However to be able to define finer-scaled units of “nature types” more factors (quantitative substrate composition, current regime, etc) must be taken into account, and detailed analyses of the video records must be performed.

Published
2009

119. Risk for Hospitalization With Depression After a Cancer Diagnosis: A Nationwide, Population-Based Study of Cancer Patients in Denmark From 1973 to 2003

Author

Dalton, S.O., Laursen, T.M., Nylandsted, Lone Ross, Mortensen, P.B., Johansen, C., Dalton, S.O., Laursen, T.M., Nylandsted, Lone Ross, Mortensen, P.B., and Johansen, C.

Abstract

Purpose As more people survive cancer, it is necessary to understand the long-term impact of cancer. We investigated whether cancer survivors are at increased risk for hospitalization for depression. Methods We linked data on all 5,703,754 persons living in Denmark on January 1, 1973, or born thereafter to the Danish Cancer Registry and identified 608,591 adults with a diagnosis of cancer. Follow-up for hospitalization for depression in the Danish Psychiatric Central Register from 1973 through 2003 yielded 121,227,396 person-years and 121,304 hospitalizations for depression. The relative risk (RR) for depression among cancer survivors relative to the cancer-free population was estimated by Poisson regression analysis with adjustment for age and period and stratified by sex, site of cancer, and extent of disease. Results The risk for depression in the first year after a cancer diagnosis was increased, with RRs ranging from 1.16 (95% CI, 0.90 to 1.51) in women with colorectal cancer to 3.08 (95% CI, 1.88 to 5.02) in men with brain cancer. Decreasing but still significant excess risks during subsequent years were observed for most specific cancers. The risk remained increased throughout the study period for both men and women surviving hormone-related cancers, for women surviving smoking-related cancers, and for men surviving virus- and immune-related cancers. Conclusion This study confirms an increased risk for depression in patients facing a disruptive event like cancer. Early recognition and effective treatment are needed to prevent admission of cancer survivors for depression Udgivelsesdato: 2009/3/20

Published
2009

126. Estimation of the fermentability of dietary fibre in vitro : a European interlaboratory study

Author

Barry, J.L., HOEBLER, Christine, MacFarlane, G.T., MacFarlane, S., MATHERS, J.C., Reed, K.A., Mortensen, P.B., Nordgaard, I., Rowland, I.R., Rumney, C.J., Laboratoire de technologie appliquée à la nutrition, Institut National de la Recherche Agronomique (INRA), and ProdInra, Migration

Subjects
[SDV.AEN] Life Sciences [q-bio]/Food and Nutrition, METHODE RAPIDE, [SDV.AEN]Life Sciences [q-bio]/Food and Nutrition, ComputingMilieux_MISCELLANEOUS
Abstract

International audience

Published
1995

128. PP099-MON HOME PARENTERAL NUTRITION (HPN) PROVIDED FROM THE INTESTINAL FAILURE (IF) UNIT AT RIGSHOSPITALET, COPENHAGEN, DENMARK, IN THE PERIOD FROM 1973 TO 2009. EXTRACTS FROM THE WORLD'S LARGEST SINGLE CENTRE HPN DATABASE

Author

Brandt, C., primary, Bangsgaard, L., additional, Lund, P., additional, Staun, M., additional, Tjellesen, L., additional, Haderslev, K.V., additional, Holtug, K., additional, Schneider, B., additional, Schou, B., additional, Christensen, D., additional, Christiansen, J., additional, Mortensen, P.B., additional, and Jeppesen, P.B., additional

Published
2011
Full Text
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130. Short-term administration of Glucagon-like peptide-2. Effects on bone mass density and markers of bone remodelling in short bowel patients with no colon

Author

Haderslev, K.V., Jeppesen, P.B., Hartmann, B., Thulesen, Jesper, Sørensen, H.A., Graff, J., Hansen, B.S., Tofteng, F., Poulsen, Steen Seier, Madsen, J.L., Holst, J.J., Staun, M., Mortensen, P.B., Haderslev, K.V., Jeppesen, P.B., Hartmann, B., Thulesen, Jesper, Sørensen, H.A., Graff, J., Hansen, B.S., Tofteng, F., Poulsen, Steen Seier, Madsen, J.L., Holst, J.J., Staun, M., and Mortensen, P.B.

Abstract

Anatomi

Published
2002

134. Treatment of short bowel patients with glucagon-like peptide-2 (GLP-2), a newly discovered intestinotrophic, anti-secretory, and transit modulating peptide

Author

Jeppesen, P.B., Hartmann, Bolette, Thulesen, J., Graff, J., Lohmann, J., Hansen, B.S., Tofteng, F., Poulsen, S.S., Madsen, J.L., Holst, J.J., Mortensen, P.B., Jeppesen, P.B., Hartmann, Bolette, Thulesen, J., Graff, J., Lohmann, J., Hansen, B.S., Tofteng, F., Poulsen, S.S., Madsen, J.L., Holst, J.J., and Mortensen, P.B.

Published
2001

144. Dementia in affective disorder: a case register study.

Author

Kessing, Lars Vedel, Andersen, Elisabeth Anne Wreford, Mortensen, P.B., Andersen, Per Kragh, Kessing, Lars Vedel, Andersen, Elisabeth Anne Wreford, Mortensen, P.B., and Andersen, Per Kragh

Published
1999

146. Breast cancer risk among women with psychiatric admission with affective or neurotic disorders: a nationwide cohort study in Denmark

Author

Hjerl, Karen Margrete, Andersen, Elisabeth Anne Wreford, Keiding, Niels, Sawitz, Allen Ronald, Olsen, J.H., Mortensen, P.B., Jørgensen, Torben, Hjerl, Karen Margrete, Andersen, Elisabeth Anne Wreford, Keiding, Niels, Sawitz, Allen Ronald, Olsen, J.H., Mortensen, P.B., and Jørgensen, Torben

Abstract

neoplasm breast, aetiology, affective disorder, neurotic disorders, alcohol abuse, non-specified abuse

Published
1999

149. Recurrence in affective disorder

Author

Kessing, Lars Vedel, Andersen, Per Kragh, Mortensen, P.B., Bolwig, Tom G., Kessing, Lars Vedel, Andersen, Per Kragh, Mortensen, P.B., and Bolwig, Tom G.

Published
1998

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