Author: "Passiu, G" - Searchworks@Jio Institute Digital Library Search Results

Author: Passiu G
Subjects: Administration, Oral, Auranofin administration & dosage, Auranofin adverse effects, Auranofin pharmacokinetics, Drug Administration Schedule, Humans, Arthritis, Rheumatoid drug therapy, Auranofin therapeutic use
Published: 1986

Author: Carcassi U, Cacace E, Mela Q, Passiu G, Perpignano G, and Pintus A
Subjects: Adolescent, Adult, Aged, Cardiomyopathies complications, Diabetes Complications, Female, Ferritins blood, Genetic Carrier Screening, Glucosephosphate Dehydrogenase Deficiency complications, Glucosephosphate Dehydrogenase Deficiency epidemiology, Glucosephosphate Dehydrogenase Deficiency genetics, Hemoglobin H genetics, Humans, Italy, Male, Middle Aged, Phenotype, Thalassemia complications, Thalassemia epidemiology, Thalassemia genetics
Published: 1982

Author: Sanna A, Passiu G, Binaghi F, Ibba G, Perpinano G, Putzu ML, and Bande A
Subjects: Adult, Aged, Echocardiography, Electrocardiography, Female, Heart Function Tests, Humans, Male, Middle Aged, Rheumatic Heart Disease etiology, Arthritis, Rheumatoid complications, Rheumatic Heart Disease diagnosis
Published: 1981

Author: Passiu G, Mela Q, Perpignano G, and Carcassi U
Subjects: Adult, Glucosephosphate Dehydrogenase Deficiency drug therapy, Half-Life, Humans, Male, Middle Aged, Suprofen therapeutic use, Erythrocyte Aging drug effects, Glucosephosphate Dehydrogenase Deficiency blood, Phenylpropionates adverse effects, Suprofen adverse effects
Published: 1984

Author: Passiu G, Perpignano G, La Nasa G, and Carcassi U
Subjects: Adolescent, Colchicine therapeutic use, Familial Mediterranean Fever drug therapy, Familial Mediterranean Fever genetics, Familial Mediterranean Fever immunology, Humans, Lymphocytes classification, Male, Pedigree, Familial Mediterranean Fever pathology
Abstract: Familial mediterranean fever (FMF) is an hereditary disorder characterized by attacks of febrile serosal inflammation involving pleura or peritoneum and synovium, followed usually by insidious onset of amyloidosis. In other patients amyloidosis of AA-type is the only finding of the disease. This disorder is common in Jews of Sephardi and Ashkenazi ancestry, Arabs, Armenians and Turks. In this work the clinico-biological features and the therapeutical aspects of a patient, suffering from FMF, of Italian ancestry are presented.
Published: 1984

Author: Passiu G
Subjects: Animals, Cartilage cytology, Cartilage metabolism, Humans, Glucosamine pharmacokinetics, Glucosamine physiology, Glucosamine therapeutic use, Glucosamine toxicity
Published: 1988

Author: Frigerio R, Mela Q, Passiu G, Cacace E, La Nasa G, Perpignano G, and Carcassi UE
Subjects: Adult, Female, Humans, Male, Middle Aged, Acetylglucosaminidase blood, Ferritins analysis, Hexosaminidases blood, Iron metabolism, Lysosomes physiology, Thalassemia blood
Abstract: Increased iron storage represents a characteristic condition in patients with beta zero-thalassaemia intermedia. Iron overload is an important factor in cellular damage. Recent studies have shown an enhanced lysosomal fragility due to increased iron storage. 13 patients with beta-thalassaemia intermedia, aged 17-44 years, were studied. Both serum ferritin and serum N-acetyl-beta-D-glucosaminidase levels were evaluated in all subjects studied. A significant linear correlation (P less than 0.05) between serum ferritin and serum N-acetyl-beta-D-glucosaminidase levels were found.
Published: 1984
Full Text: View/download PDF

Searchworks