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592 results on '"Persico, A. M."'

152. Altered Connectivity Between Cerebellum, Visual, and Sensory-Motor Networks in Autism Spectrum Disorder: Results from the EU-AIMS Longitudinal European Autism Project

Author

Oldehinkel, Marianne, Mennes, Maarten, Marquand, Andre, Charman, Tony, Tillmann, Julian, Ecker, Christine, Dell’Acqua, Flavio, Brandeis, Daniel, Banaschewski, Tobias, Baumeister, Sarah, Moessnang, Carolin, Baron-Cohen, Simon, Holt, Rosemary, Bölte, Sven, Durston, Sarah, Kundu, Prantik, Lombardo, Michael V., Spooren, Will, Loth, Eva, Murphy, Declan G.M., Beckmann, Christian F., Buitelaar, Jan K., Ahmad, Jumana, Ambrosino, Sara, Auyeung, Bonnie, Banaschewski, Tobias, Baron-Cohen, Simon, Baumeister, Sarah, Beckmann, Christian F., Bölte, Sven, Bourgeron, Thomas, Bours, Carsten, Brammer, Michael, Brandeis, Daniel, Brogna, Claudia, de Bruijn, Yvette, Buitelaar, Jan K., Chakrabarti, Bhismadev, Charman, Tony, Cornelissen, Ineke, Crawley, Daisy, Dell’Acqua, Flavio, Dumas, Guillaume, Durston, Sarah, Ecker, Christine, Faulkner, Jessica, Frouin, Vincent, Garcés, Pilar, Goyard, David, Ham, Lindsay, Hayward, Hannah, Hipp, Joerg, Holt, Rosemary, Johnson, Mark H., Jones, Emily J.H., Kundu, Prantik, Lai, Meng-Chuan, Liogier D’ardhuy, Xavier, Lombardo, Michael V., Loth, Eva, Lythgoe, David J., Mandl, René, Marquand, Andre, Mason, Luke, Mennes, Maarten, Meyer-Lindenberg, Andreas, Moessnang, Carolin, Mueller, Nico, Murphy, Declan G.M., Oakley, Bethany, O’Dwyer, Laurence, Oldehinkel, Marianne, Oranje, Bob, Pandina, Gahan, Persico, Antonio M., Ruggeri, Barbara, Ruigrok, Amber, Sabet, Jessica, Sacco, Roberto, Cáceres, Antonia San José, Simonoff, Emily, Spooren, Will, Tillmann, Julian, Toro, Roberto, Tost, Heike, Waldman, Jack, Williams, Steve C.R., Wooldridge, Caroline, and Zwiers, Marcel P.

Abstract

Resting-state functional magnetic resonance imaging–based studies on functional connectivity in autism spectrum disorder (ASD) have generated inconsistent results. Interpretation of findings is further hampered by small samples and a focus on a limited number of networks, with networks underlying sensory processing being largely underexamined. We aimed to comprehensively characterize ASD-related alterations within and between 20 well-characterized resting-state networks using baseline data from the EU-AIMS (European Autism Interventions—A Multicentre Study for Developing New Medications) Longitudinal European Autism Project.

Published
2024
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153. Cluster analysis of autistic patients based on principal pathogenetic components

Author

Sacco, Roberto, Lenti, Carlo, Saccani, Monica, Curatolo, Paolo, Manzi, Barbara, Bravaccio, Carmela, Persico, Antonio M., Sacco, R, Lenti, C, Saccani, M, Curatolo, P, Manzi, B, Bravaccio, Carmela, and Persico, A. M.

Subjects
Pervasive developmental disorders, Adult, Male, Child Development Disorders, Adolescent, Developmental Disabilities, Neurodevelopment, Principal component analysis, Chronobiology Disorders, Cluster analysis, Cluster Analysis, Humans, Immune system, Child, Pervasive, Preschool, Female, Immune System Diseases, Italy, Principal Component Analysis, Sensation Disorders, Stereotyped Behavior, Neuroscience (all), Neurology (clinical), Genetics (clinical), Medicine (all), Child Development Disorders, Pervasive, Child, Preschool, Settore MED/39 - Neuropsichiatria Infantile
Abstract

We have recently described four principal pathogenetic components in autism: (I) circadian and sensory dysfunction, (II) immune abnormalities, (III) neurodevelopmental delay, and (IV) stereotypic behaviors. Using hierarchical and k-means clustering, the same 245 patients assessed in our principal component analysis can be partitioned into four clusters: (a) 43 (17.6%) have prominent immune abnormalities accompanied by some circadian and sensory issues; (b) 44 (18.0%) display major circadian and sensory dysfunction, with little or no immune symptoms; (c) stereotypies predominate in 75 (31.0%); and (d) 83 (33.9%) show a mixture of all four components, with greater disruptive behaviors and mental retardation. The "immune" component provides the largest contributions to phenotypic variance (P = 2.7 x 10(-45)), followed by "stereotypic behaviors." These patient clusters may likely differ in genetic and immune underpinnings, developmental trajectories, and response to treatment.

Published
2012

154. Slow Intestinal Transit Contributes to Elevate Urinary p-Cresol Level in Italian Autistic Children

Author

Gabriele, Stefano, Sacco, Roberto, Altieri, Laura, Neri, Cristina, Urbani, Andrea, Bravaccio, Carmela, Riccio, Maria Pia, Iovene, Rosaria Maria, Bombace, Francesca, De Magistris, Laura, Persico, Antonio M., Urbani, Andrea (ORCID:0000-0001-9168-3174), Gabriele, Stefano, Sacco, Roberto, Altieri, Laura, Neri, Cristina, Urbani, Andrea, Bravaccio, Carmela, Riccio, Maria Pia, Iovene, Rosaria Maria, Bombace, Francesca, De Magistris, Laura, Persico, Antonio M., and Urbani, Andrea (ORCID:0000-0001-9168-3174)

Abstract

The uremic toxin p-cresol (4-methylphenol) is either of environmental origin or can be synthetized from tyrosine by cresol-producing bacteria present in the gut lumen. Elevated p-cresol amounts have been previously found in the urines of Italian and French autism spectrum disorder (ASD) children up until 8 years of age, and may be associated with autism severity or with the intensity of abnormal behaviors. This study aims to investigate the mechanism producing elevated urinary p-cresol in ASD. Urinary p-cresol levels were thus measured by High Performance Liquid Chromatography in a sample of 53 Italian ASD children assessed for (a) presence of Clostridium spp. strains in the gut by means of an in vitro fecal stool test and of Clostridium difficile-derived toxin A/B in the feces, (b) intestinal permeability using the lactulose/mannitol (LA/MA) test, (c) frequent use of antibiotics due to recurrent infections during the first 2 years of postnatal life, and (d) stool habits with the Bristol Stool Form Scale. Chronic constipation was the only variable significantly associated with total urinary p-cresol concentration (P < 0.05). No association was found with presence of Clostridium spp. in the gut flora (P = 0.92), augmented intestinal permeability (P = 0.18), or frequent use of antibiotics in early infancy (P = 0.47). No ASD child was found to carry C. difficile in the gut or to release toxin A/B in the feces. In conclusion, urinary p-cresol levels are elevated in young ASD children with increased intestinal transit time and chronic constipation. (C) 2015 International Society for Autism Research, Wiley Periodicals, Inc.

Published
2016

158. Unmet needs in paediatric psychopharmacology: Present scenario and future perspectives

Author

Ontwikkelingsstoornissen Med., Brain, Persico, Antonio M., Arango, Celso, Buitelaar, Jan K., Correll, Christoph U., Glennon, Jeffrey C., Hoekstra, Pieter J., Moreno, Carmen, Vitiello, Benedetto, Vorstman, Jacob, Zuddas, Alessandro, European Child & Adolescent Clinic, Ontwikkelingsstoornissen Med., Brain, Persico, Antonio M., Arango, Celso, Buitelaar, Jan K., Correll, Christoph U., Glennon, Jeffrey C., Hoekstra, Pieter J., Moreno, Carmen, Vitiello, Benedetto, Vorstman, Jacob, Zuddas, Alessandro, and European Child & Adolescent Clinic

Published
2015

159. Identification and validation of biomarkers for autism spectrum disorders

Author

Loth, Eva, primary, Spooren, Will, additional, Ham, Lindsay M., additional, Isaac, Maria B., additional, Auriche-Benichou, Caroline, additional, Banaschewski, Tobias, additional, Baron-Cohen, Simon, additional, Broich, Karl, additional, Bölte, Sven, additional, Bourgeron, Thomas, additional, Charman, Tony, additional, Collier, David, additional, de Andres-Trelles, Fernando, additional, Durston, Sarah, additional, Ecker, Christine, additional, Elferink, Andre, additional, Haberkamp, Marion, additional, Hemmings, Robert, additional, Johnson, Mark H., additional, Jones, Emily J. H., additional, Khwaja, Omar S., additional, Lenton, Sabine, additional, Mason, Luke, additional, Mantua, Valentina, additional, Meyer-Lindenberg, Andreas, additional, Lombardo, Michael V., additional, O'Dwyer, Laurence, additional, Okamoto, Koichi, additional, Pandina, Gahan J., additional, Pani, Luca, additional, Persico, Antonio M., additional, Simonoff, Emily, additional, Tauscher-Wisniewski, Sitra, additional, Llinares-Garcia, Jordi, additional, Vamvakas, Spiros, additional, Williams, Steve, additional, Buitelaar, Jan K., additional, and Murphy, Declan G. M., additional

Published
2015
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160. Autisms

Author

Persico, A. M.

Subjects
Pervasive developmental disorders, PTEN, Rett syndrome, Autism, Neuroligin, Asperger disorder, MET, Autistic disorder, Copy number variant, MECP2, Reelin
Published
2013

161. Unraveling molecular pathways shared by Kabuki and Kabuki‐like syndromes.

Author

Lintas, C. and Persico, A. M.

Subjects
*KABUKI syndrome, *SKELETAL abnormalities, *DELETION mutation, *CHROMATIN, *GENETIC transcription
Abstract

Kabuki syndrome (KS) is a rare genetic syndrome characterized by a typical facial gestalt, variable degrees of intellectual disability, organ malformations, postnatal growth retardation and skeletal abnormalities. So far, KMT2D or KDM6A mutation has been identified as the main cause of KS, accounting for 56%‐75% and 3%‐8% of cases, respectively. Patients without mutations in 1 of the 2 causative KS genes are often referred to as affected by Kabuki‐like syndrome. Overall, they represent approximately 30% of KS cases, pointing toward substantial genetic heterogeneity for this condition. Here, we review all currently available literature describing KS‐like phenotypes (or phenocopies) associated with genetic variants located in loci different from KMT2D and KDM6A . We also report on a new KS phenocopy harboring a 5 Mb de novo deletion in chr10p11.22‐11.21. An enrichment analysis aimed at identifying functional Gene Ontology classes shared by the 2 known KS causative genes and by new candidate genes currently associated with KS‐like phenotypes primarily converges upon abnormal chromatin remodeling and transcriptional dysregulation as pivotal to the pathophysiology of KS phenotypic hallmarks. The identification of mutations in genes belonging to the same functional pathways of KMT2D and KDM6A can help design molecular screenings targeted to KS‐like phenotypes. [ABSTRACT FROM AUTHOR]

Published
2018
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162. An Interstitial 17q11.2 de novo Deletion Involving the CDK5R1 Gene in a High-Functioning Autistic Patient

Author

Lintas, Carla, Sacco, Roberto, Tabolacci, Claudio, Brogna, Claudia, Canali, Marco, Picinelli, Chiara, Tomaiuolo, Pasquale, Castronovo, Paola, Baccarin, Marco, and Persico, Antonio M.

Abstract

We describe a 32-year-old male patient diagnosed with high-functioning autism spectrum disorder carrying a de novo 196-kb interstitial deletion at chromosome 17q11.2. The deletion was detected by array CGH (180K Agilent) and confirmed by quantitative PCR on genomic DNA. The deleted region spans the entire PSMD11and CDK5R1genes and partially the MYO1Dgene. The CDK5R1gene encodes for a regulatory subunit of the cyclin-dependent kinase 5 responsible for its brain-specific activation. This gene has been previously associated with intellectual disability in humans. A reduction in CDK5R1transcript was detected, consistent with the genomic deletion. Based on the functional role of CDK5R1, this gene appears as the best candidate to explain the clinical phenotype of our patient, whose neuropsychological profile has more resemblance with some of the higher brain function anomalies recently described in the CreER-p35 conditional knockout mouse model than previously described patients with intellectual disability.

Published
2019
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173. Deafferentation-induced apoptosis of neurons in thalamic somatosensory nuclei of the newborn rat: critical period and rescue from cell death by peripherally applied neurotrophins

Author

Baldi, A., Calia, E., Ciampini, A., Riccio, Massimo, Vetuschi, A., Persico, A. M., Keller, F., Persico, Antonio, Baldi, Alfonso, Calia, E, Ciampini, A, Riccio, M, Vetuschi, A, Persico, Am, and Keller, F.

Subjects
Thalamocortical neuron, Stereology, LATERAL GENICULATE NEURONS, STEM AUDITORY NUCLEI, INFRAORBITAL NERVE, BRAIN-STEM, AXOPLASMIC-TRANSPORT, NEONATAL TRANSECTION, AFFERENT INFLUENCES, TRIGEMINAL SYSTEM, ACTION-POTENTIALS, PROTEIN-SYNTHESIS, Apoptosis, Cell Count, Brain-derived neurotrophic factor, Electron microscopy, Fluorescent microspheres, Infraorbital nerve, Nerve growth factor, TUNEL, Vinblastine, Neuroscience (all), Axonal Transport, Nerve Growth Factor, Neural Pathways, In Situ Nick-End Labeling, Maxillary Nerve, Animals, Cell Nucleus, Neurons, Fluorescent microsphere, Brain-Derived Neurotrophic Factor, Critical Period, Psychological, Axotomy, Somatosensory Cortex, Antineoplastic Agents, Phytogenic, Chromatin, Rats, Microscopy, Electron, Animals, Newborn, Rats, Inbred Lew, Thalamic Nuclei
Abstract

This study shows that unilateral transection of the infraorbital nerve (ION) in newborn (PO) rats induces apoptosis in the contralateral ventrobasal thalamic (VB) complex, as evidenced by terminal transferase-mediated deoxyuridine triphosphate-biotin nick end labelling (TUNEL) and electron miscroscopy. Double-labelling experiments using retrograde transport of labelled microspheres injected into the barrel cortex, followed by TUNEL staining, show that TUNEL-positive cells are thalamocortical neurons. The number of TUNEL-positive cells had begun to increase by 24 h postlesion, increased further 48 h after nerve section, and decreased to control levels after 120 h. Lesion-induced apoptosis in the VB complex is less pronounced if ION section is performed at P4, and disappears if the lesion is performed at P7. This time course closely matches the critical period of lesion-induced plasticity in the barrel cortex. Nerve growth factor (NGF) or brain-derived neurotrophic factor (BDNF), applied on the ION stump alone or in combination, are able to partially rescue thalamic neurons from apoptosis. Total cell counts in the VB complex of P7 animals that underwent ION section at PO confirm the rescuing effect of BDNF and NGF. Blockade of axonal transport in the ION mimics the effect of ION section. These data suggest that survival-promoting signals from the periphery, maybe neurotrophins, are required for the survival of higher-order neurons in the somatosensory system during the period of fine-tuning of neuronal connections. We also propose that anterograde transneuronal degeneration in the neonatal rat trigeminal system may represent a new animal model for studying the pathways of programmed cell death in vivo.

Published
2000

174. Lack of association between serotonin transporter gene promoter variants and autistic disorder in two ethnically-distinct samples

Author

Persico, A. M., Militerni, R., Bravaccio, C., Schneider, C., Melmed, R., Damiani, V., Baldi, A., and Keller, F.

Subjects
Male, Serotonin Plasma Membrane Transport Proteins, Membrane Glycoproteins, Adolescent, Base Sequence, Membrane Transport Proteins, Nerve Tissue Proteins, United States, Promoter Regions, Haplotypes, Italy, Genetic, Humans, Female, Autistic Disorder, Carrier Proteins, Child, Preschool, DNA Primers
Published
2000

176. BDNF and NT-3 applied in the whisker pad reverse cortical changes after peripheral deafferentation in neonatal rats

Author

Calia, E, Persico, A M, Baldi, A, Keller, F, Calia, E, Persico, Am, Baldi, Alfonso, and Keller, F.

Subjects
Serotonin, animal structures, Plasticity, Acetylcholinesterase, Animals, Newborn, Biomarkers, Brain-Derived Neurotrophic Factor, Child, Preschool, Denervation, Drug Administration Routes, Drug Implants, Histocytochemistry, Humans, Immunohistochemistry, Nerve Growth Factors, Neurons, Afferent, Neurotrophin 3, Peripheral Nerves, Rats, Inbred Lew, Somatosensory Cortex, Vibrissae, Barrel, Cortical specification, Neurons, Afferent, Animals, Newborn, Child, Preschool, Rats, Inbred Lew, Neural development, Neurotrophin
Abstract

It has been known for a long time that subcortical input drives the specification of cortical areas. Molecular signals mediating this instructive effect from the periphery are poorly understood. In foetal or neonatal rats, ablation of whisker follicles, transection of the infraorbital nerve, inhibition of axonal transport, but not impulse activity blockade, prevent formation of barrels in the primary somatosensory cortex (S1). These findings suggest that a chemical signal, possibly arising from the skin or the follicle, may be responsible for somatotopic pattern formation in S1. Neurotrophins promote survival and differentiation of primary sensory neurons, and are expressed in the whisker pad during development. Neonatal rats received gelfoam impregnated with NGF, BDNF or NT-3 under the whisker pad following surgical denervation of whisker rows D and E on P0. Barrel formation in S1 was assessed on P7 by acetylcholinesterase histochemistry and 5-HT-immunohistochemistry. BDNF and NT-3, but not NGF, promoted development of the cortical barrels corresponding to denervated whiskers. Furthermore, BDNF and NT-3 prevented the lesion-induced expansion of row C barrels, while NGF appeared to promote row C expansion. Our results suggest that BDNF and NT-3 arising from the whisker pad are involved in the formation and/or maintenance of the barrel pattern in S1. These findings are potentially relevant for the prevention of sensory disturbances possibly due to reorganization of central sensory circuits after peripheral nerve lesions in humans.

Published
1998

177. Parkinsonian patients report blunted subjective effects of methylphenidate

Author

Persico, A. M., Reich, S., Henningfield, J. E., Kuhar, M. J., and Uhl, G. R.

Subjects
Adult, Male, Depressive Disorder, Dose-Response Relationship, Drug, Parkinson Disease, Middle Aged, Affect, Dopamine Uptake Inhibitors, Reward, Heart Rate, Methylphenidate, Humans, Female, Aged
Abstract

Mesolimbic-mesocortical dopamine brain circuits important for psychostimulant reward in animals are developed to greater extents in humans. Brains of patients with Parkinson's disease show depletion of ventral tegmental area mesolimbic-mesocortical neurons. The authors assessed psychostimulant responses in parkinsonian patients to test whether intact dopaminergic systems are required for subjective psychostimulant effects. Responses to placebo and 15, 20, 25, and 30 mg of methylphenidate were studied in 12 parkinsonian patients and 12 neurologically intact matched controls. Physiological and subjective mood responses were recorded using the Profile of Mood States, Addiction Research Center Inventory, and Visual Analog Scale. Drug-induced changes in "good" feelings and overall drug responses were attenuated in the parkinsonian patients. These results, in conjunction with animal data, provide support for dopamine hypotheses of psychostimulant reward in humans and suggest possible bases for some of the mood disturbances found in many parkinsonian patients.

Published
1998

179. Plasma cytokine profiling in sibling pairs discordant for autism spectrum disorder

Author

Napolioni, Valerio, primary, Ober-Reynolds, Benjamin, additional, Szelinger, Szabolcs, additional, Corneveaux, Jason J, additional, Pawlowski, Traci, additional, Ober-Reynolds, Sharman, additional, Kirwan, Janet, additional, Persico, Antonio M, additional, Melmed, Raun D, additional, Craig, David W, additional, Smith, Christopher J, additional, and Huentelman, Matthew J, additional

Published
2013
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182. Urinary polyomavirus infections in neurodevelopmental disorders

Author

Gentile, Ivan, primary, Altieri, Laura, additional, Lintas, Carla, additional, Sacco, Roberto, additional, Curatolo, Paolo, additional, Benvenuto, Arianna, additional, Muratori, Filippo, additional, Santocchi, Elisa, additional, Bravaccio, Carmela, additional, Lenti, Carlo, additional, Faggioli, Raffaella, additional, Rigardetto, Roberto, additional, Gandione, Marina, additional, Portella, Giuseppe, additional, Zappulo, Emanuela, additional, Borgia, Guglielmo, additional, and Persico, Antonio M., additional

Published
2013
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183. Genotypic association between dopamine transporter gene polymorphisms and schizophrenia

Author

Persico, A M and Macciardi, F

Subjects
Male, Dopamine Plasma Membrane Transport Proteins, Membrane Glycoproteins, Polymorphism, Genetic, Alleles, Carrier Proteins, Case-Control Studies, DNA Primers, Female, Genetic Linkage, Genotype, Homozygote, Humans, Italy, Minisatellite Repeats, Schizophrenia, Membrane Transport Proteins, Nerve Tissue Proteins, Polymorphism, Genetic
Abstract

Dopamine transporter (DAT) gene variants do not appear to provide widespread contributions to the etiology of schizophrenia spectrum disorders, according to linkage studies [Persico et al., 1995: Am J Psychiatry 152:134-136]. They may, however, produce modifying effects, more readily detectable in specific subpopulations of schizophrenics through associations analyses. We therefore compared polymorphic DAT gene variable number tandem repeat (VNTR) distributions in 84 controls and 147 patients, divided according to DSM-IIIR schizophrenia type criteria. No evidence of allelic association between DAT alleles and schizophrenia or any specific schizophrenia subtype was found. Interestingly, the DAT genotype distribution among schizophrenic patients did display a statistically significant departure from the genotype distribution found in controls. Such discrepancies may represent stigmata of assortative mating or may suggest a "modifying" contribution of homozygote DAT genotypes to pathogenetic processes underlying schizophrenia.

Published
1997

192. 2p15–p16.1 microdeletion syndrome: molecular characterization and association of the OTX1 and XPO1 genes with autism spectrum disorders

Author

Liu, Xudong, primary, Malenfant, Patrick, additional, Reesor, Chelsea, additional, Lee, Alana, additional, Hudson, Melissa L, additional, Harvard, Chansonette, additional, Qiao, Ying, additional, Persico, Antonio M, additional, Cohen, Ira L, additional, Chudley, Albert E, additional, Forster-Gibson, Cynthia, additional, Rajcan-Separovic, Evica, additional, Lewis, ME Suzanne, additional, and Holden, Jeanette JA, additional

Published
2011
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194. Urinary p-cresol is elevated in small children with severe autism spectrum disorder

Author

Altieri, Laura, primary, Neri, Cristina, additional, Sacco, Roberto, additional, Curatolo, Paolo, additional, Benvenuto, Arianna, additional, Muratori, Filippo, additional, Santocchi, Elisa, additional, Bravaccio, Carmela, additional, Lenti, Carlo, additional, Saccani, Monica, additional, Rigardetto, Roberto, additional, Gandione, Marina, additional, Urbani, Andrea, additional, and Persico, Antonio M., additional

Published
2011
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196. Decreased serum arylesterase activity in autism spectrum disorders

Author

Gaita, Laura, primary, Manzi, Barbara, additional, Sacco, Roberto, additional, Lintas, Carla, additional, Altieri, Laura, additional, Lombardi, Federica, additional, Pawlowski, Tracy L., additional, Redman, Margot, additional, Craig, David W., additional, Huentelman, Matthew J., additional, Ober-Reynolds, Sharman, additional, Brautigam, Sarah, additional, Melmed, Raun, additional, Smith, Christopher J., additional, Marsillach, Judith, additional, Camps, Jordi, additional, Curatolo, Paolo, additional, and Persico, Antonio M., additional

Published
2010
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197. Family-based association study of ITGB3 in autism spectrum disorder and its endophenotypes

Author

Napolioni, Valerio, primary, Lombardi, Federica, additional, Sacco, Roberto, additional, Curatolo, Paolo, additional, Manzi, Barbara, additional, Alessandrelli, Riccardo, additional, Militerni, Roberto, additional, Bravaccio, Carmela, additional, Lenti, Carlo, additional, Saccani, Monica, additional, Schneider, Cindy, additional, Melmed, Raun, additional, Pascucci, Tiziana, additional, Puglisi-Allegra, Stefano, additional, Reichelt, Karl-Ludvig, additional, Rousseau, Francis, additional, Lewin, Patricia, additional, and Persico, Antonio M, additional

Published
2010
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198. Principal pathogenetic components and biological endophenotypes in autism spectrum disorders

Author

Sacco, Roberto, primary, Curatolo, Paolo, additional, Manzi, Barbara, additional, Militerni, Roberto, additional, Bravaccio, Carmela, additional, Frolli, Alessandro, additional, Lenti, Carlo, additional, Saccani, Monica, additional, Elia, Maurizio, additional, Reichelt, Karl-Ludvig, additional, Pascucci, Tiziana, additional, Puglisi-Allegra, Stefano, additional, and Persico, Antonio M., additional

Published
2010
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199. Mitochondrial pathways to autism

Author

Palmieri, Luigi, primary, Lombardi, Federica, additional, Porcelli, Vito, additional, Sacco, Roberto, additional, Scarcia, Pasquale, additional, and Persico, Antonio M., additional

Published
2010
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