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203. [Practice guidelines 2007 for the treatment of arterial hypertension]

Author

Mancia, G, De Backer, G, Dominiczak, A, Cifkova, R, Fagard, R, Germanò, G, Grassi, G, Heagerty, A, Kjeldsen, S, Laurent, S, Narkiewicz, K, Ruilope, L, Rynkiewicz, A, Schmieder, R, Struijker Boudier, H, Zanchetti, A, Vahanian, A, Camm, J, De Caterina, R, Dean, V, Dickstein, K, Filippatos, G, Funck-Brentano, C, Hellemans, I, Dalby Kristensen, S, Mcgregor, K, Sechtem, U, Silber, S, Tendera, M, Widimsky, P, Zamorano, J, Erdine, S, Kiowski, W, Agabiti-Rosei, E, Ambrosioni, E, Lindholm, L, Manolis, A, Nilsson, P, Redon, J, Viigimaa, M, Mancia, Giuseppe, De Backer, Guy, Dominiczak, Anna, Cifkova, Renata, Fagard, Robert, Germanò, Giuseppe, Grassi, Guido, Heagerty, Anthony M, Kjeldsen, Sverre E, Laurent, Stephane, Narkiewicz, Krzysztof, Ruilope, Luis, Rynkiewicz, Andrzej, Schmieder, Roland E, Struijker Boudier, Harry A J, Zanchetti, Alberto, Vahanian, Alec, Camm, John, De Caterina, Raffaele, Dean, Veronica, Dickstein, Kenneth, Filippatos, Gerasimos, Funck-Brentano, Christian, Hellemans, Irene, Dalby Kristensen, Steen, McGregor, Keith, Sechtem, Udo, Silber, Sigmund, Tendera, Michal, Widimsky, Petr, Zamorano, José Luis, Erdine, Serap, Kiowski, Wolfgang, Agabiti-Rosei, Enrico, Ambrosioni, Ettore, Lindholm, Lars H, Manolis, Athanasios, Nilsson, Peter M, Redon, Josep, Viigimaa, Margus, Mancia, G, De Backer, G, Dominiczak, A, Cifkova, R, Fagard, R, Germanò, G, Grassi, G, Heagerty, A, Kjeldsen, S, Laurent, S, Narkiewicz, K, Ruilope, L, Rynkiewicz, A, Schmieder, R, Struijker Boudier, H, Zanchetti, A, Vahanian, A, Camm, J, De Caterina, R, Dean, V, Dickstein, K, Filippatos, G, Funck-Brentano, C, Hellemans, I, Dalby Kristensen, S, Mcgregor, K, Sechtem, U, Silber, S, Tendera, M, Widimsky, P, Zamorano, J, Erdine, S, Kiowski, W, Agabiti-Rosei, E, Ambrosioni, E, Lindholm, L, Manolis, A, Nilsson, P, Redon, J, Viigimaa, M, Mancia, Giuseppe, De Backer, Guy, Dominiczak, Anna, Cifkova, Renata, Fagard, Robert, Germanò, Giuseppe, Grassi, Guido, Heagerty, Anthony M, Kjeldsen, Sverre E, Laurent, Stephane, Narkiewicz, Krzysztof, Ruilope, Luis, Rynkiewicz, Andrzej, Schmieder, Roland E, Struijker Boudier, Harry A J, Zanchetti, Alberto, Vahanian, Alec, Camm, John, De Caterina, Raffaele, Dean, Veronica, Dickstein, Kenneth, Filippatos, Gerasimos, Funck-Brentano, Christian, Hellemans, Irene, Dalby Kristensen, Steen, McGregor, Keith, Sechtem, Udo, Silber, Sigmund, Tendera, Michal, Widimsky, Petr, Zamorano, José Luis, Erdine, Serap, Kiowski, Wolfgang, Agabiti-Rosei, Enrico, Ambrosioni, Ettore, Lindholm, Lars H, Manolis, Athanasios, Nilsson, Peter M, Redon, Josep, and Viigimaa, Margus

Published
2007

204. 2007 Guidelines for the management of arterial hypertension: The Task Force for the Management of Arterial Hypertension of the European Society of Hypertension (ESH) and of the European Society of Cardiology (ESC)

Author

Mancia, G, De Backer, G, Dominiczak, A, Cifkova, R, Fagard, R, Germano, G, Grassi, G, Heagerty, A, Kjeldsen, S, Laurent, S, Narkiewicz, K, Ruilope, L, Rynkiewicz, A, Schmieder, R, Struijker Boudier, H, Zanchetti, A, Vahanian, A, Camm, J, De Caterina, R, Dean, V, Dickstein, K, Filippatos, G, Funck Brentano, C, Hellemans, I, Kristensen, S, Mcgregor, K, Sechtem, U, Silber, S, Tendera, M, Widimsky, P, Zamorano, J, Erdine, S, Kiowski, W, Agabiti Rosei, E, Ambrosioni, E, Lindholm, L, Manolis, A, Nilsson, P, Redon, J, Viigimaa, M, Adamopoulos, S, Bertomeu, V, Clement, D, Farsang, C, Gaita, D, Lip, G, Mallion, J, O'Brien, E, Ponikowski, P, Ruschitzka, F, Tamargo, J, van Zwieten, P, Waeber, B, Williams, B, The task force for the management of arterial hypertension of the European Society of Hypertension, N, The task force for the management of arterial hypertension of the European Society of Cardiology, N, MANCIA, GIUSEPPE, GRASSI, GUIDO, Heagerty, AM, Kjeldsen, SE, Schmieder, RE, Struijker Boudier, HAJ, Kristensen, SD, McGregor, K, Zamorano, JL, Lindholm, LH, Nilsson, PM, Manolis, AJ, The task force for the management of arterial hypertension of the European Society of Hypertension, n, The task force for the management of arterial hypertension of the European Society of Cardiology, n., Mancia, G, De Backer, G, Dominiczak, A, Cifkova, R, Fagard, R, Germano, G, Grassi, G, Heagerty, A, Kjeldsen, S, Laurent, S, Narkiewicz, K, Ruilope, L, Rynkiewicz, A, Schmieder, R, Struijker Boudier, H, Zanchetti, A, Vahanian, A, Camm, J, De Caterina, R, Dean, V, Dickstein, K, Filippatos, G, Funck Brentano, C, Hellemans, I, Kristensen, S, Mcgregor, K, Sechtem, U, Silber, S, Tendera, M, Widimsky, P, Zamorano, J, Erdine, S, Kiowski, W, Agabiti Rosei, E, Ambrosioni, E, Lindholm, L, Manolis, A, Nilsson, P, Redon, J, Viigimaa, M, Adamopoulos, S, Bertomeu, V, Clement, D, Farsang, C, Gaita, D, Lip, G, Mallion, J, O'Brien, E, Ponikowski, P, Ruschitzka, F, Tamargo, J, van Zwieten, P, Waeber, B, Williams, B, The task force for the management of arterial hypertension of the European Society of Hypertension, N, The task force for the management of arterial hypertension of the European Society of Cardiology, N, MANCIA, GIUSEPPE, GRASSI, GUIDO, Heagerty, AM, Kjeldsen, SE, Schmieder, RE, Struijker Boudier, HAJ, Kristensen, SD, McGregor, K, Zamorano, JL, Lindholm, LH, Nilsson, PM, Manolis, AJ, The task force for the management of arterial hypertension of the European Society of Hypertension, n, and The task force for the management of arterial hypertension of the European Society of Cardiology, n.

Published
2007

205. Central blood pressure measurements and antihypertensive therapy a consensus document

Author

Agabiti Rosei, E, Mancia, G, O'Rourke, M, Roman, M, Safar, M, Smulyan, H, Wang, J, Wilkinson, I, Williams, B, Vlachopoulos, C, O'Rourke, MF, Roman, MJ, Safar, ME, Wang, JG, Wilkinson, IB, Vlachopoulos, C., MANCIA, GIUSEPPE, Agabiti Rosei, E, Mancia, G, O'Rourke, M, Roman, M, Safar, M, Smulyan, H, Wang, J, Wilkinson, I, Williams, B, Vlachopoulos, C, O'Rourke, MF, Roman, MJ, Safar, ME, Wang, JG, Wilkinson, IB, Vlachopoulos, C., and MANCIA, GIUSEPPE

Abstract

The 2003 European Society of Hypertension/European Society of Cardiology guidelines for the management of arterial hypertension1 included 2 important novel recommendations: assessment of the total cardiovascular risk should be taken into account in the management of the hypertensive patient, and quantification of risk should include subclinical target organ damage. These guidelines acknowledged that central (aortic) blood pressure (BP), which is the pressure exerted on the heart and brain, may be different from the pressure that is measured at the arm. They also recognized that central pressure may be predictive of outcome in specific populations2 and differently affected by antihypertensive drugs. However, although these guidelines accepted that central augmentation index and pulse wave velocity may be important as measures of subclinical organ damage, they also stressed the need for prospective trials to establish their predictive values given that such studies were lacking at that time (2003). After publication of these guidelines, additional data have strengthened the pathophysiological importance of central BP. Clinical studies have indicated that central BP may have predictive value independent of the corresponding peripheral (brachial) BP. More importantly, recent large-scale trials have shown that central hemodynamics may provide a worthwhile treatment target. In addition, central hemodynamics can now be reliably assessed noninvasively with a number of devices. Accordingly, because arterial stiffening and central hemodynamics are markers and manifestations of organ damage, the pertinent key question is whether the balance of evidence on their importance and issues related to clinical practice allows for implementation in patient management.

Published
2007

208. Cardiac structural and functional changes during long-term antihypertensive treatment with lacidipine and atenolol in the European Lacidipine Study on Atherosclerosis (ELSA)

Author

Agabiti rosei, E, Trimarco, B, Muiesan, M, Reid, J, Salvetti, A, Tang, R, Hennig, M, Baurecht, H, Parati, G, Mancia, G, Zanchetti, A, Zanchetti, A., PARATI, GIANFRANCO, MANCIA, GIUSEPPE, Agabiti rosei, E, Trimarco, B, Muiesan, M, Reid, J, Salvetti, A, Tang, R, Hennig, M, Baurecht, H, Parati, G, Mancia, G, Zanchetti, A, Zanchetti, A., PARATI, GIANFRANCO, and MANCIA, GIUSEPPE

Abstract

Objectives: To evaluate and correlate the effects of long-term antihypertensive treatment on left ventricular (LV) mass and carotid structural changes in a large group of essential hypertensive patients, participating in the European Lacidipine Study on Atherosclerosis (ELSA). Design: In four (Brescia, Glasgow, Naples and Pisa) of 23 centres participating in the ELSA study, an echocardiographic examination was performed at baseline and repeated, until the end of the 4-year study, in essential hypertensive patients, followed-up for carotid quantitative ultrasound examination of intima-media thickness (IMT), after random allocation to treatment with either lacidipine or atenolol (and added hydrochlorothiazide, as required for control of blood pressure). Methods: M-mode, two-dimensional guided echocardiography was used to measure left ventricular (LV) wall thickness and dimensions, from which LV mass was calculated, using an anatomically validated formula (Penn Convention) and indexed to body surface area (left ventricular mass index, LVMI). The echocardiographic tracings were blindly evaluated in a single reading centre (Brescia). Bilateral IMT was measured at the site of common carotid and bifurcation far walls (CBMmax). Results: At baseline, cardiac and carotid ultrasound scans were available in 278 patients (mean age 54 ± 7 years, 57% males, 22% obese). A significant correlation was observed between baseline LVMI and CBMmax (r = 0.22, P < 0.001), independent of age. In multivariate analysis, CBMmax and mean 24-h pulse pressure were most strongly associated with baseline LVMI. A significant reduction in LVMI was observed both during lacidipine (n = 96) (-12.5% reduction) and atenolol (n = 78) (-13.9% reduction) treatments (up to 4 years) (P < 0.001 for both, without significant differences between treatments). Changes in LVMI were not related to changes in carotid wall thickness. In multivariate analysis, baseline LV mass and mean 24-h systolic blood pressure change

Published
2005

210. 3.5 EFFECT OF SHORT-TERM TREATMENT WITH LERCANIDIPINE ON CIRCULATING ENDOTHELIAL PROGENITOR CELLS AND STRUCTURAL ALTERATIONS IN RETINAL ARTERIOLES

Author

De Ciuceis, C., primary, Rossini, C., primary, La Boria, E., primary, Duse, S., primary, Semeraro, F., primary, Agabiti Rosei, C., primary, Airò, P., primary, Scarsi, M., primary, Tincani, A., primary, Muiesan, M.L., primary, Paini, A., primary, Salvetti, M., primary, Porteri, E., primary, Agabiti Rosei, E., primary, and Rizzoni, D., primary

Published
2012
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212. CIRCULATING INDICES OF INFLAMMATION/OXIDATIVE STRESS IN NORMOTENSIVE AND HYPERTENSIVE OBESE PATIENTS

Author

De Ciuceis, C., primary, Pilu, A., additional, Porteri, E., additional, La Boria, E., additional, Corbellini, C., additional, Boari, G. E.M., additional, Mittempergher, F., additional, Di Betta, E., additional, Casella, C., additional, Agabiti Rosei, C., additional, Ruggeri, G., additional, Rizzoni, D., additional, and Agabiti Rosei, E., additional

Published
2011
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214. RESISTANT HYPERTENSION IN A GENERAL POPULATION IN NORTHERN ITALY: PREVALENCE, ASSOCIATED CARDIOVASCULAR RISK FACTORS AND TARGET ORGAN DAMAGE

Author

Salvetti, M., primary, Muiesan, M. L., additional, Paini, A., additional, Agabiti Rosei, C., additional, Aggiusti, C., additional, Bertacchini, F., additional, Stassaldi, D., additional, Beschi, F., additional, Cobelli, S., additional, Rubagotti, G., additional, Monteduro, C., additional, Castellano, M., additional, and Agabiti Rosei, E., additional

Published
2011
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215. PREVALENCE AND CLINICAL CHARACTERISTICS OF PATIENTS ADMITTED TO AN EMERGENCY DEPARTMENT FOR HYPERTENSIVE EMERGENCIES AND URGENCIES

Author

Paini, A., primary, Salvetti, M., additional, Cobelli, S., additional, Torazzi, L., additional, Agabiti Rosei, C., additional, Aggiusti, C., additional, Zotti, D., additional, Bazza, A., additional, Bertacchini, F., additional, Beschi, F., additional, Stassaldi, D., additional, Concoreggi, C., additional, Agabiti Rosei, E., additional, and Muiesan, M. L., additional

Published
2011
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217. CLOSE RELATIONSHIP BETWEEN MEDIA TO LUMEN RATIO OF SUBCUTANEOUS SMALL ARTERIES AND WALL TO LUMEN RATIO OF RETINAL ARTERIOLES EVALUATED NON INVASIVELY BY SCANNING LASER DOPPLER FLOWMETRY

Author

Rizzoni, D., primary, De Ciuceis, C., additional, Porteri, E., additional, La Boria, E., additional, Duse, S., additional, Semeraro, F., additional, Costagliola, C., additional, Sebastiani, A., additional, Danzi, P., additional, Tiberio, G. A. M., additional, Giulini, S. M., additional, Agabiti Rosei, C., additional, Sarkar, A., additional, Avanzi, D., additional, and Agabiti Rosei, E., additional

Published
2011
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219. HYPERTENSIVE EMERGENCIES AND URGENCIES IN AN EMERGENCY DEPARTMENT. ASSOCIATION WITH SUBSEQUENT CARDIOVASCULAR EVENTS DURING 2 YEARS FOLLOW-UP

Author

Salvetti, M., primary, Paini, A., additional, Torazzi, L., additional, Cobelli, S., additional, Agabiti Rosei, C., additional, Aggiusti, C., additional, Bazza, A., additional, Zotti, D., additional, Bertacchini, F., additional, Stassaldi, D., additional, Concoreggi, C., additional, Agabiti Rosei, E., additional, and Muiesan, M. L., additional

Published
2011
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220. EFFECT OF SHORT-TERM TREATMENT WITH TELMISARTAN OR NIFEDIPINE ON INSULIN SIGNALLING IN LYMPHOMONOCYTES OF ESSENTIAL HYPERTENSIVE PATIENTS

Author

Porteri, E., primary, Flati, V., additional, Pasini, E., additional, De ciuceis, C., additional, Platto, C., additional, Giacomelli, I., additional, La boria, E., additional, Assanelli, D., additional, Speca, S., additional, Donini, C., additional, Rizzoni, D., additional, and Agabiti rosei, E., additional

Published
2011
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232. ASSESSMENT OF VASCULAR STRUCTURAL AND FUNCTIONAL ALTERATIONS IN APOLIPOPROTEIN A-I AMYLOIDOSIS (LEU75PRO): PP.7.301

Author

Muiesan, ML, primary, Salvetti, M, additional, Paini, A, additional, Agabiti Rosei, C, additional, Aggiusti, C, additional, Beschi, F, additional, Monteduro, C, additional, Nardi, M, additional, Obici, L, additional, Negrinelli, A, additional, Gregorini, G, additional, Cancarini, G, additional, and Agabiti Rosei, E, additional

Published
2010
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237. Comparative effects of candesartan and enalapril on left ventricular hypertrophy in patients with essential hypertension: The candesartan assessment in the treatment of cardiac hypertrophy (CATCH) study

Author

Cuspidi, C, Muiesan, M, Valagussa, L, Salvetti, M, Di Biagio, C, Agabiti Rosei, E, Magnani, B, Zanchetti, A, CUSPIDI, CESARE, Zanchetti, A., Cuspidi, C, Muiesan, M, Valagussa, L, Salvetti, M, Di Biagio, C, Agabiti Rosei, E, Magnani, B, Zanchetti, A, CUSPIDI, CESARE, and Zanchetti, A.

Abstract

Background: A limited number of studies have evaluated the effect of angiotensin II receptor antagonists (AIIAs) on left ventricular hypertrophy (LVH) in comparison with other antihypertensive drugs, and no large study has compared AIIAs with angiotensin-converting enzyme inhibitors (ACEIs). Methods and results: The CATCH (Candesartan Assessment in the Treatment of Cardiac Hypertrophy) study was a multicenter prospective randomized double-blind trial comparing the effects of candesartan cilexetil (8-16 mg once daily) and enalapril (10-20 mg once daily) with possible addition of hydrochlorothiazide (12.5-25 mg once daily) on echocardiographic left ventricular mass index (LVMI), in 239 hypertensives with LVH (LVMI>120 g/m2 in men and >100 g/m2 in women). Two-dimensionally guided M-mode echocardiograms were carried out at screening (recruiting scan), randomization (baseline scan) and after 24 and 48 weeks of treatment. Baseline and treatment echocardiograms were read at two central labs without knowledge of the scan time sequence. In intention-to-treat (ITT) analyses (196 patients), systolic and diastolic blood pressures (SBP, DBP) were significantly and equally reduced by the two treatments. Candesartan and enalapril reduced LVMI to the same extent, i.e. by 15.0 and 13.1 g/m2 (-10.9 and -8.4%; P<0.001 for both). The proportion of patients achieving normalization of LVMI was non-significantly higher with candesartan (36.3 versus 28.6%). Similar results were obtained in per-protocol (PP) analyses. Cough incidence was lower with candesartan (P<0.03). Conclusions: CATCH is the first large study comparing the effects of an AIIA and an ACEI on LVMI. Candesartan cilexetil was found to be equally effective as enalapril in reducing SBP, DBP and LVMI in hypertensives with LVH, according to both ITT and PP analyses. © 2002 Lippincott Williams & Wilkins.

Published
2002

238. Midwall mechanics are improved after regression of hypertensive left ventricular hypertrophy and normalization of chamber geometry

Author

Perlini, S, Muiesan, M, Cuspidi, C, Sampieri, L, Trimarco, B, Aurigemma, G, Agabiti Rosei, E, Mancia, G, CUSPIDI, CESARE, MANCIA, GIUSEPPE, Perlini, S, Muiesan, M, Cuspidi, C, Sampieri, L, Trimarco, B, Aurigemma, G, Agabiti Rosei, E, Mancia, G, CUSPIDI, CESARE, and MANCIA, GIUSEPPE

Abstract

Background - It is still unclear whether substantial regression of hypertensive left ventricular hypertrophy (LVH) and normalization of chamber geometry are associated with improved left ventricular (LV) myocardial function. Methods and Results - Midwall mechanics were evaluated in 152 patients undergoing 1 year of effective antihypertensive treatment. Two-dimensionally directed M-mode echocardiography was performed as follows: (1) after a 4-week placebo "run-in" period, (2) after 1 year of treatment with 20 mg/d lisinopril (alone or associated with 12.5 to 25 mg/d hydrochlorothiazide), and (3) after a final 1-month placebo period to allow blood pressure (24-hour average ambulatory monitoring) to return to pretreatment levels. Treatment-induced reductions in blood pressure (from 149±16/95±11 to 131±12/83±10 mm Hg, P<0.05) and circumferential end-systolic wall stress (from 84±22 to 72±19 g/cm2, P±0.05) were associated with a marked reduction in LV mass index (from 159±30 to 133±26 g/m2, P<0.05). LVH regression was accompanied by an increase in midwall fractional shortening (from 19.7±2.7% to 20.9±2.7%, P<0.05) and by a decrease in relative wall thickness (from 48.2±7.7% to 44.1±6.7%, P<0.05). The improvement in midwall function associated with afterload reduction and substantial LVH regression persisted after antihypertensive therapy withdrawal and restoration of the hypertensive state. Despite a significant increase in end-systolic wall stress, further LV chamber remodeling did not occur. The preservation of relative wall thickness was associated with a persistent improvement in midwall systolic function. Conclusions - Regression of concentric LVH is associated with an improvement of midwall systolic function, which is more dependent on the normalization of LV geometry than on the reduction in LV systolic stress.

Published
2001

242. Antihypertensive efficacy of manidipine and enalapril in hypertensive diabetic patients

Author

Mancia, G, Omboni, S, Agabiti Rosei, E, Casati, R, Fogari, R, Leonetti, G, Montemurro, G, Nami, R, Pessina, A, Pirrelli, A, Zanchetti, A, MANCIA, GIUSEPPE, Pessina, AC, Zanchetti, A., Mancia, G, Omboni, S, Agabiti Rosei, E, Casati, R, Fogari, R, Leonetti, G, Montemurro, G, Nami, R, Pessina, A, Pirrelli, A, Zanchetti, A, MANCIA, GIUSEPPE, Pessina, AC, and Zanchetti, A.

Abstract

Recent studies showed that in diabetic hypertensive patients, administration of angiotensin-converting enzyme (ACE)-inhibitors or calcium antagonists can effectively lower blood pressure (BP) and prevent diabetes-related cardiovascular complications with no adverse metabolic effects. We sought to assess the antihypertensive and metabolic effects of the new dihydropyridine calcium antagonist manidipine (M) in patients with diabetes mellitus and essential hypertension as compared with the ACE inhibitor enalapril (E). After 3 weeks of placebo, 101 (62 men; age range, 34-72 years) hypertensives with type IlIdiabetes mellitus were randomized to M 10-20 mg or E 10-20 mg, od. for 74 weeks. At the end of the placebo period and the active-treatment phase, BP was measured with a mercury sphygmomanometer (office, O) and over the 24 h by ambulatory (A) monitoring. ABP recordings were analyzed to obtain 24-h, day (6 a.m. to midnight), and night (midnight to 6 a.m.) average systolic (S) and diastolic (D) BP and heart rate (HR) values. Homogeneity of the antihypertensive effect over the 24 h was assessed by the smoothness index [SI; i.e., the ratio between the average of the 14 hourly BP changes after treatment and the corresponding standard deviation (the higher the SI, the more uniform is the BP control by treatment over the 24 h]. The O SEP and DBP were significantly (p < 0.01) and similarly reduced by M (16 +/- 10 and 13 +/- 6 mm Hg, n = 49) and E (15 +/- 10 and 13 +/- 6 mm Hg, n = 45). The percentage of patients whose O DBP was reduced less than or equal to 85 mm Hg (i.e., the value indicated to be the optimal DBP goal in diabetic hypertensives) was similar for hi (37%) and E (40%). The reduction of 24-h BP also was similar between M (n = 38) and E (n = 38) for bath drugs (systolic, 6 +/- 11 and 8 +/- 10 mm Hg; diastolic, 5 +/- 8 and 5 +/- 7; NS, M vs. E). The antihypertensive effect was distributed in a similar homogeneous fashion throughout the dosing interval, as shown by

Published
2000

245. Impaired Insulin Signalling in the Heart and Skeletal Muscle of Spontaneously Hypertensive Rats, and Effects of Treatment with an Angiotensin Receptor Blocker or with an ACE Inhibitor

Author

Porteri, E, primary, Pasini, E, additional, Flati, V, additional, Paiardi, S, additional, Assanelli, D, additional, Corsetti, G, additional, Speca, S, additional, Bianchi, R, additional, Rizzardi, N, additional, Rizzoni, D, additional, and Agabiti Rosei, E, additional

Published
2007
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246. Patients with Essential Hypertension Show the Presence of an Altered Structure of Small Cerebral Arteries

Author

Rizzoni, D, primary, De Ciuceis, C, additional, Porteri, E, additional, Boari, G EM, additional, Mortini, P, additional, Cornali, C, additional, Rodella, L, additional, Miclini, M, additional, Zani, F, additional, Rizzardi, N, additional, Platto, C, additional, Paiardi, S, additional, Rezzani, R, additional, Bianchi, R, additional, and Agabiti Rosei, E, additional

Published
2007
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