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5,420 results on '"Boffetta, Paolo"'

201. Association of polygenic risk score with the risk of chronic lymphocytic leukemia and monoclonal B-cell lymphocytosis

Author

Kleinstern, Geffen, Camp, Nicola J., Goldin, Lynn R., Vachon, Celine M., Vajdic, Claire M., de Sanjose, Silvia, Weinberg, J. Brice, Benavente, Yolanda, Casabonne, Delphine, Liebow, Mark, Nieters, Alexandra, Hjalgrim, Henrik, Melbye, Mads, Glimelius, Bengt, Adami, Hans-Olov, Boffetta, Paolo, Brennan, Paul, Maynadie, Marc, McKay, James, Cocco, Pier Luigi, Shanafelt, Tait D., Call, Timothy G., Norman, Aaron D., Hanson, Curtis, Robinson, Dennis, Chaffee, Kari G., Brooks-Wilson, Angela R., Monnereau, Alain, Clavel, Jacqueline, Glenn, Martha, Curtin, Karen, Conde, Lucia, Bracci, Paige M., Morton, Lindsay M., Cozen, Wendy, Severson, Richard K., Chanock, Stephen J., Spinelli, John J., Johnston, James B., Rothman, Nathaniel, Skibola, Christine F., Leis, Jose F., Kay, Neil E., Smedby, Karin E., Berndt, Sonja I., Cerhan, James R., Caporaso, Neil, and Slager, Susan L.

Published
2018
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202. Alcohol intake and gastric cancer: Meta-analyses of published data versus individual participant data pooled analyses (StoP Project)

Author

Ferro, Ana, Morais, Samantha, Rota, Matteo, Pelucchi, Claudio, Bertuccio, Paola, Bonzi, Rossella, Galeone, Carlotta, Zhang, Zuo-Feng, Matsuo, Keitaro, Ito, Hidemi, Hu, Jinfu, Johnson, Kenneth C., Yu, Guo-Pei, Palli, Domenico, Ferraroni, Monica, Muscat, Joshua, Malekzadeh, Reza, Ye, Weimin, Song, Huan, Zaridze, David, Maximovitch, Dmitry, Fernández de Larrea, Nerea, Kogevinas, Manolis, Vioque, Jesus, Navarrete-Muñoz, Eva M., Pakseresht, Mohammadreza, Pourfarzi, Farhad, Wolk, Alicja, Orsini, Nicola, Bellavia, Andrea, Håkansson, Niclas, Mu, Lina, Pastorino, Roberta, Kurtz, Robert C., Derakhshan, Mohammad H., Lagiou, Areti, Lagiou, Pagona, Boffetta, Paolo, Boccia, Stefania, Negri, Eva, La Vecchia, Carlo, Peleteiro, Bárbara, and Lunet, Nuno

Published
2018
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205. Gallbladder disease, cholecystectomy, and pancreatic cancer risk in the International Pancreatic Cancer Case-Control Consortium (PanC4)

Author

Rosato, Valentina, Negri, Eva, Bosetti, Cristina, Malats, Núria, Gomez-Rubio, Paulina, Consortium, PanGenEU, Maisonneuve, Patrick, Miller, Anthony B., Bueno-de-Mesquita, H. Bas, Baghurst, Peter A., Zatonski, Witold, Petersen, Gloria M., Scelo, Ghislaine, Holcatova, Ivana, Fabianova, Eleonora, Serraino, Diego, Olson, Sara H., Vioque, Jesús, Lagiou, Pagona, Duell, Eric J., Boffetta, Paolo, and La Vecchia, Carlo

Published
2020
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206. Genome-wide association study identifies multiple risk loci for chronic lymphocytic leukemia

Author

Berndt, Sonja I, Skibola, Christine F, Joseph, Vijai, Camp, Nicola J, Nieters, Alexandra, Wang, Zhaoming, Cozen, Wendy, Monnereau, Alain, Wang, Sophia S, Kelly, Rachel S, Lan, Qing, Teras, Lauren R, Chatterjee, Nilanjan, Chung, Charles C, Yeager, Meredith, Brooks-Wilson, Angela R, Hartge, Patricia, Purdue, Mark P, Birmann, Brenda M, Armstrong, Bruce K, Cocco, Pierluigi, Zhang, Yawei, Severi, Gianluca, Zeleniuch-Jacquotte, Anne, Lawrence, Charles, Burdette, Laurie, Yuenger, Jeffrey, Hutchinson, Amy, Jacobs, Kevin B, Call, Timothy G, Shanafelt, Tait D, Novak, Anne J, Kay, Neil E, Liebow, Mark, Wang, Alice H, Smedby, Karin E, Adami, Hans-Olov, Melbye, Mads, Glimelius, Bengt, Chang, Ellen T, Glenn, Martha, Curtin, Karen, Cannon-Albright, Lisa A, Jones, Brandt, Diver, W Ryan, Link, Brian K, Weiner, George J, Conde, Lucia, Bracci, Paige M, Riby, Jacques, Holly, Elizabeth A, Smith, Martyn T, Jackson, Rebecca D, Tinker, Lesley F, Benavente, Yolanda, Becker, Nikolaus, Boffetta, Paolo, Brennan, Paul, Foretova, Lenka, Maynadie, Marc, McKay, James, Staines, Anthony, Rabe, Kari G, Achenbach, Sara J, Vachon, Celine M, Goldin, Lynn R, Strom, Sara S, Lanasa, Mark C, Spector, Logan G, Leis, Jose F, Cunningham, Julie M, Weinberg, J Brice, Morrison, Vicki A, Caporaso, Neil E, Norman, Aaron D, Linet, Martha S, De Roos, Anneclaire J, Morton, Lindsay M, Severson, Richard K, Riboli, Elio, Vineis, Paolo, Kaaks, Rudolph, Trichopoulos, Dimitrios, Masala, Giovanna, Weiderpass, Elisabete, Chirlaque, María-Dolores, Vermeulen, Roel CH, Travis, Ruth C, Giles, Graham G, Albanes, Demetrius, Virtamo, Jarmo, Weinstein, Stephanie, Clavel, Jacqueline, Zheng, Tongzhang, Holford, Theodore R, Offit, Kenneth, Zelenetz, Andrew, Klein, Robert J, Spinelli, John J, and Bertrand, Kimberly A

Subjects
Cancer, Case-Control Studies, Chromosomes, Human, Pair 2, Genetic Loci, Genetic Predisposition to Disease, Genome-Wide Association Study, Humans, Leukemia, Lymphocytic, Chronic, B-Cell, Linkage Disequilibrium, Polymorphism, Single Nucleotide, Recombination, Genetic, Risk, Biological Sciences, Medical and Health Sciences, Developmental Biology
Abstract

Genome-wide association studies (GWAS) have previously identified 13 loci associated with risk of chronic lymphocytic leukemia or small lymphocytic lymphoma (CLL). To identify additional CLL susceptibility loci, we conducted the largest meta-analysis for CLL thus far, including four GWAS with a total of 3,100 individuals with CLL (cases) and 7,667 controls. In the meta-analysis, we identified ten independent associated SNPs in nine new loci at 10q23.31 (ACTA2 or FAS (ACTA2/FAS), P=1.22×10(-14)), 18q21.33 (BCL2, P=7.76×10(-11)), 11p15.5 (C11orf21, P=2.15×10(-10)), 4q25 (LEF1, P=4.24×10(-10)), 2q33.1 (CASP10 or CASP8 (CASP10/CASP8), P=2.50×10(-9)), 9p21.3 (CDKN2B-AS1, P=1.27×10(-8)), 18q21.32 (PMAIP1, P=2.51×10(-8)), 15q15.1 (BMF, P=2.71×10(-10)) and 2p22.2 (QPCT, P=1.68×10(-8)), as well as an independent signal at an established locus (2q13, ACOXL, P=2.08×10(-18)). We also found evidence for two additional promising loci below genome-wide significance at 8q22.3 (ODF1, P=5.40×10(-8)) and 5p15.33 (TERT, P=1.92×10(-7)). Although further studies are required, the proximity of several of these loci to genes involved in apoptosis suggests a plausible underlying biological mechanism.

Published
2013

207. Hierarchical modeling identifies novel lung cancer susceptibility variants in inflammation pathways among 10,140 cases and 11,012 controls

Author

Brenner, Darren R, Brennan, Paul, Boffetta, Paolo, Amos, Christopher I, Spitz, Margaret R, Chen, Chu, Goodman, Gary, Heinrich, Joachim, Bickeböller, Heike, Rosenberger, Albert, Risch, Angela, Muley, Thomas, McLaughlin, John R, Benhamou, Simone, Bouchardy, Christine, Lewinger, Juan Pablo, Witte, John S, Chen, Gary, Bull, Shelley, and Hung, Rayjean J

Subjects
Biological Sciences, Biomedical and Clinical Sciences, Oncology and Carcinogenesis, Genetics, Prevention, Lung Cancer, Human Genome, Lung, Cancer, Aetiology, 2.1 Biological and endogenous factors, Alleles, Canada, Case-Control Studies, Europe, Female, Gene Frequency, Genetic Predisposition to Disease, Genetic Variation, Genome-Wide Association Study, Genotype, Humans, Inflammation, Likelihood Functions, Lung Neoplasms, Male, Middle Aged, Models, Statistical, Polymorphism, Single Nucleotide, Risk, United States, White People, Complementary and Alternative Medicine, Paediatrics and Reproductive Medicine, Genetics & Heredity, Reproductive medicine
Abstract

Recent evidence suggests that inflammation plays a pivotal role in the development of lung cancer. In this study, we used a two-stage approach to investigate associations between genetic variants in inflammation pathways and lung cancer risk based on genome-wide association study (GWAS) data. A total of 7,650 sequence variants from 720 genes relevant to inflammation pathways were identified using keyword and pathway searches from Gene Cards and Gene Ontology databases. In Stage 1, six GWAS datasets from the International Lung Cancer Consortium were pooled (4,441 cases and 5,094 controls of European ancestry), and a hierarchical modeling (HM) approach was used to incorporate prior information for each of the variants into the analysis. The prior matrix was constructed using (1) role of genes in the inflammation and immune pathways; (2) physical properties of the variants including the location of the variants, their conservation scores and amino acid coding; (3) LD with other functional variants and (4) measures of heterogeneity across the studies. HM affected the priority ranking of variants particularly among those having low prior weights, imprecise estimates and/or heterogeneity across studies. In Stage 2, we used an independent NCI lung cancer GWAS study (5,699 cases and 5,818 controls) for in silico replication. We identified one novel variant at the level corrected for multiple comparisons (rs2741354 in EPHX2 at 8p21.1 with p value = 7.4 × 10(-6)), and confirmed the associations between TERT (rs2736100) and the HLA region and lung cancer risk. HM allows for prior knowledge such as from bioinformatic sources to be incorporated into the analysis systematically, and it represents a complementary analytical approach to the conventional GWAS analysis.

Published
2013

208. Previous Lung Diseases and Lung Cancer Risk: A Pooled Analysis From the International Lung Cancer Consortium

Author

Brenner, Darren R, Boffetta, Paolo, Duell, Eric J, Bickeböller, Heike, Rosenberger, Albert, McCormack, Valerie, Muscat, Joshua E, Yang, Ping, Wichmann, H-Erich, Brueske-Hohlfeld, Irene, Schwartz, Ann G, Cote, Michele L, Tjønneland, Anne, Friis, Søren, Le Marchand, Loic, Zhang, Zuo-Feng, Morgenstern, Hal, Szeszenia-Dabrowska, Neonila, Lissowska, Jolanta, Zaridze, David, Rudnai, Peter, Fabianova, Eleonora, Foretova, Lenka, Janout, Vladimir, Bencko, Vladimir, Schejbalova, Miriam, Brennan, Paul, Mates, Ioan N, Lazarus, Philip, Field, John K, Raji, Olaide, McLaughlin, John R, Liu, Geoffrey, Wiencke, John, Neri, Monica, Ugolini, Donatella, Andrew, Angeline S, Lan, Qing, Hu, Wei, Orlow, Irene, Park, Bernard J, and Hung, Rayjean J

Subjects
Epidemiology, Health Services and Systems, Health Sciences, Chronic Obstructive Pulmonary Disease, Pneumonia, Prevention, Lung, Tobacco Smoke and Health, Cancer, Tobacco, Rare Diseases, Pneumonia & Influenza, Lung Cancer, Infectious Diseases, Respiratory, Good Health and Well Being, Bronchitis, Chronic, Humans, Logistic Models, Lung Neoplasms, Proportional Hazards Models, Pulmonary Emphysema, Risk, Risk Factors, Self Report, Tuberculosis, Pulmonary, bronchitis, chronic, emphysema, lung diseases, lung neoplasms, meta-analysis, pneumonia, pulmonary disease, chronic obstructive, tuberculosis, Mathematical Sciences, Medical and Health Sciences
Abstract

To clarify the role of previous lung diseases (chronic bronchitis, emphysema, pneumonia, and tuberculosis) in the development of lung cancer, the authors conducted a pooled analysis of studies in the International Lung Cancer Consortium. Seventeen studies including 24,607 cases and 81,829 controls (noncases), mainly conducted in Europe and North America, were included (1984-2011). Using self-reported data on previous diagnoses of lung diseases, the authors derived study-specific effect estimates by means of logistic regression models or Cox proportional hazards models adjusted for age, sex, and cumulative tobacco smoking. Estimates were pooled using random-effects models. Analyses stratified by smoking status and histology were also conducted. A history of emphysema conferred a 2.44-fold increased risk of lung cancer (95% confidence interval (CI): 1.64, 3.62 (16 studies)). A history of chronic bronchitis conferred a relative risk of 1.47 (95% CI: 1.29, 1.68 (13 studies)). Tuberculosis (relative risk = 1.48, 95% CI: 1.17, 1.87 (16 studies)) and pneumonia (relative risk = 1.57, 95% CI: 1.22, 2.01 (12 studies)) were also associated with lung cancer risk. Among never smokers, elevated risks were observed for emphysema, pneumonia, and tuberculosis. These results suggest that previous lung diseases influence lung cancer risk independently of tobacco use and that these diseases are important for assessing individual risk.

Published
2012

210. Diet and the risk of head and neck cancer: a pooled analysis in the INHANCE consortium

Author

Chuang, Shu-Chun, Jenab, Mazda, Heck, Julia E, Bosetti, Cristina, Talamini, Renato, Matsuo, Keitaro, Castellsague, Xavier, Franceschi, Silvia, Herrero, Rolando, Winn, Deborah M, Vecchia, Carlo La, Morgenstern, Hal, Zhang, Zuo-Feng, Levi, Fabio, Maso, Luigino Dal, Kelsey, Karl, McClean, Michael D, Vaughan, Thomas, Lazarus, Philip, Muscat, Joshua, Ramroth, Heribert, Chen, Chu, Schwartz, Stephen M, Eluf-Neto, Jose, Hayes, Richard B, Purdue, Mark, Boccia, Stefania, Cadoni, Gabriella, Zaridze, David, Koifman, Sergio, Curado, Maria Paula, Ahrens, Wolfgang, Benhamou, Simone, Matos, Elena, Lagiou, Pagona, Szeszenia-Dabrowska, Neonilla, Olshan, Andrew F, Fernandez, Leticia, Menezes, Ana, Agudo, Antonio, Daudt, Alexander W, Merletti, Franco, Macfarlane, Gary J, Kjaerheim, Kristina, Mates, Dana, Holcatova, Ivana, Schantz, Stimson, Yu, Guo-Pei, Simonato, Lorenzo, Brenner, Hermann, Mueller, Heiko, Conway, David I, Thomson, Peter, Fabianova, Eleonora, Znaor, Ariana, Rudnai, Peter, Healy, Claire M, Ferro, Gilles, Brennan, Paul, Boffetta, Paolo, and Hashibe, Mia

Subjects
Nutrition, Cancer, Prevention, Clinical Research, Dental/Oral and Craniofacial Disease, Adult, Aged, Case-Control Studies, Diet, Female, Head and Neck Neoplasms, Humans, Male, Middle Aged, Risk Factors, Head and neck cancer, Fruit and vegetable, Red meat, Processed meat, Oncology and Carcinogenesis, Public Health and Health Services, Epidemiology
Abstract

We investigated the association between diet and head and neck cancer (HNC) risk using data from the International Head and Neck Cancer Epidemiology (INHANCE) consortium. The INHANCE pooled data included 22 case-control studies with 14,520 cases and 22,737 controls. Center-specific quartiles among the controls were used for food groups, and frequencies per week were used for single food items. A dietary pattern score combining high fruit and vegetable intake and low red meat intake was created. Odds ratios (OR) and 95% confidence intervals (CI) for the dietary items on the risk of HNC were estimated with a two-stage random-effects logistic regression model. An inverse association was observed for higher-frequency intake of fruit (4th vs. 1st quartile OR = 0.52, 95% CI = 0.43-0.62, p (trend) < 0.01) and vegetables (OR = 0.66, 95% CI = 0.49-0.90, p (trend) = 0.01). Intake of red meat (OR = 1.40, 95% CI = 1.13-1.74, p (trend) = 0.13) and processed meat (OR = 1.37, 95% CI = 1.14-1.65, p (trend) < 0.01) was positively associated with HNC risk. Higher dietary pattern scores, reflecting high fruit/vegetable and low red meat intake, were associated with reduced HNC risk (per score increment OR = 0.90, 95% CI = 0.84-0.97).

Published
2012

211. Xenobiotic Metabolizing Gene Variants and Renal Cell Cancer: A Multicenter Study

Author

Heck, Julia E, Moore, Lee E, Lee, Yuan-Chin A, McKay, James D, Hung, Rayjean J, Karami, Sara, Gaborieau, Valérie, Szeszenia-Dabrowska, Neonila, Zaridze, David G, Mukeriya, Anush, Mates, Dana, Foretova, Lenka, Janout, Vladimir, Kollárová, Helena, Bencko, Vladimir, Rothman, Nathaniel, Brennan, Paul, Chow, Wong-Ho, and Boffetta, Paolo

Subjects
Biomedical and Clinical Sciences, Clinical Sciences, Oncology and Carcinogenesis, Prevention, Genetics, Cancer, Human Genome, Kidney Disease, renal cell cancer, epidemiology, NAT1, NAT2, CYR NQO1, mEH, COMT, CYP, NQO1, Clinical sciences, Oncology and carcinogenesis
Abstract

BackgroundThe countries of Central and Eastern Europe have among the highest worldwide rates of renal cell cancer (RCC). Few studies have examined whether genetic variation in xenobiotic metabolic pathway genes may modify risk for this cancer.MethodsThe Central and Eastern Europe Renal Cell Cancer study was a hospital-based case-control study conducted between 1998 and 2003 across seven centers in Central and Eastern Europe. Detailed data were collected from 874 cases and 2053 controls on demographics, work history, and occupational exposure to chemical agents. Genes [cytochrome P-450 family, N-acetyltransferases,Nad(p)hquinone oxidoreductase I (NQO1), microsomal epoxide hydrolase (mEH), catechol-O-methyltransferase (COMT), uridine diphosphate-glucuronosyltransferase (UGT)] were selected for the present analysis based on their putative role in xenobiotic metabolism. Haplotypes were calculated using fastPhase. Odds ratios and 95% confidence intervals were estimated by unconditional logistic regression adjusted for country of residence, age, sex, smoking, alcohol intake, obesity, and hypertension.ResultsWe observed an increased risk of RCC with one SNP. After adjustment for multiple comparisons it did not remain significant. Neither NAT1 nor NAT2 slow acetylation was associated with disease.ConclusionWe observed no association between this pathway and renal cell cancer.

Published
2012

214. Tobacco smoking as a risk factor of bronchioloalveolar carcinoma of the lung: pooled analysis of seven case–control studies in the International Lung Cancer Consortium (ILCCO)

Author

Boffetta, Paolo, Jayaprakash, Vijayvel, Yang, Ping, Asomaning, Kofi, Muscat, Joshua E, Schwartz, Ann G, Zhang, Zuo-Feng, Le Marchand, Loic, Cote, Michele L, Stoddard, Shawn M, Morgenstern, Hal, Hung, Rayjean J, and Christiani, David C

Subjects
Epidemiology, Health Sciences, Cancer, Lung Cancer, Tobacco, Tobacco Smoke and Health, Lung, Prevention, Respiratory, Good Health and Well Being, Adenocarcinoma, Bronchiolo-Alveolar, Aged, Case-Control Studies, Female, Humans, Lung Neoplasms, Male, Middle Aged, Odds Ratio, Risk Factors, Smoking, Lung cancer, Bronchioloalveolar carcinoma, Tobacco smoking, Oncology and Carcinogenesis, Public Health and Health Services, Oncology and carcinogenesis
Abstract

BackgroundThe International Lung Cancer Consortium (ILCCO) was established in 2004, based on the collaboration of research groups leading large molecular epidemiology studies of lung cancer that are ongoing or have been recently completed. This framework offered the opportunity to investigate the role of tobacco smoking in the development of bronchioloalveolar carcinoma (BAC), a rare form of lung cancer.MethodsOur pooled data comprised seven case-control studies from the United States, with detailed information on tobacco smoking and histology, which contributed 799 cases of BAC and 15,859 controls. We estimated the odds ratio of BAC for tobacco smoking, using never smokers as a referent category, after adjustment for age, sex, race, and study center.ResultsThe odds ratio of BAC for ever smoking was 2.47 (95% confidence interval [CI] 2.08, 2.93); the risk increased linearly with duration, amount, and cumulative cigarette smoking and persisted long after smoking cessation. The proportion of BAC cases attributable to smoking was 0.47 (95% CI 0.39, 0.54).ConclusionsThis analysis provides a precise estimate of the risk of BAC for tobacco smoking.

Published
2011

216. In-Home Coal and Wood Use and Lung Cancer Risk: A Pooled Analysis of the International Lung Cancer Consortium

Author

Hosgood, H Dean, Boffetta, Paolo, Greenland, Sander, Lee, Yuan-Chin Amy, McLaughlin, John, Seow, Adeline, Duell, Eric J, Andrew, Angeline S, Zaridze, David, Szeszenia-Dabrowska, Neonila, Rudnai, Peter, Lissowska, Jolanta, Fabiánová, Eleonóra, Mates, Dana, Bencko, Vladimir, Foretova, Lenka, Janout, Vladimir, Morgenstern, Hal, Rothman, Nathaniel, Hung, Rayjean J, Brennan, Paul, and Lan, Qing

Subjects
Lung, Clinical Research, Prevention, Cancer, Lung Cancer, 2.2 Factors relating to the physical environment, Aetiology, Respiratory, Aged, Air Pollution, Indoor, Coal, Cooking, Female, Heating, Humans, International Agencies, Lung Neoplasms, Male, Middle Aged, Risk Factors, Wood, coal, lung cancer, pooled, risk factor, wood, Environmental Sciences, Medical and Health Sciences, Toxicology
Abstract

BackgroundDomestic fuel combustion from cooking and heating is an important public health issue because roughly 3 billion people are exposed worldwide. Recently, the International Agency for Research on Cancer classified indoor emissions from household coal combustion as a human carcinogen (group 1) and from biomass fuel (primarily wood) as a probable human carcinogen (group 2A).ObjectivesWe pooled seven studies from the International Lung Cancer Consortium (5,105 cases and 6,535 controls) to provide further epidemiological evaluation of the association between in-home solid-fuel use, particularly wood, and lung cancer risk.MethodsUsing questionnaire data, we classified subjects as predominant solid-fuel users (e.g., coal, wood) or nonsolid-fuel users (e.g., oil, gas, electricity). Unconditional logistic regression was used to estimate the odds ratios (ORs) and to compute 95% confidence intervals (CIs), adjusting for age, sex, education, smoking status, race/ethnicity, and study center.ResultsCompared with nonsolid-fuel users, predominant coal users (OR = 1.64; 95% CI, 1.49-1.81), particularly coal users in Asia (OR = 4.93; 95% CI, 3.73-6.52), and predominant wood users in North American and European countries (OR = 1.21; 95% CI, 1.06-1.38) experienced higher risk of lung cancer. The results were similar in never-smoking women and other subgroups.ConclusionsOur results are consistent with previous observations pertaining to in-home coal use and lung cancer risk, support the hypothesis of a carcinogenic potential of in-home wood use, and point to the need for more detailed study of factors affecting these associations.

Published
2010

217. Alcohol intake and pancreatic cancer: a pooled analysis from the pancreatic cancer cohort consortium (PanScan)

Author

Michaud, Dominique S, Vrieling, Alina, Jiao, Li, Mendelsohn, Julie B, Steplowski, Emily, Lynch, Shannon M, Wactawski-Wende, Jean, Arslan, Alan A, Bas Bueno-de-Mesquita, H, Fuchs, Charles S, Gross, Myron, Helzlsouer, Kathy, Jacobs, Eric J, LaCroix, Andrea, Petersen, Gloria, Zheng, Wei, Allen, Naomi, Ammundadottir, Laufey, Bergmann, Manuela M, Boffetta, Paolo, Buring, Julie E, Canzian, Federico, Chanock, Stephen J, Clavel-Chapelon, Françoise, Clipp, Sandra, Freiberg, Matthew S, Michael Gaziano, J, Giovannucci, Edward L, Hankinson, Susan, Hartge, Patricia, Hoover, Robert N, Allan Hubbell, F, Hunter, David J, Hutchinson, Amy, Jacobs, Kevin, Kooperberg, Charles, Kraft, Peter, Manjer, Jonas, Navarro, Carmen, Peeters, Petra HM, Shu, Xiao-Ou, Stevens, Victoria, Thomas, Gilles, Tjønneland, Anne, Tobias, Geoffrey S, Trichopoulos, Dimitrios, Tumino, Rosario, Vineis, Paolo, Virtamo, Jarmo, Wallace, Robert, Wolpin, Brian M, Yu, Kai, Zeleniuch-Jacquotte, Anne, and Stolzenberg-Solomon, Rachael Z

Subjects
Health Services and Systems, Biomedical and Clinical Sciences, Health Sciences, Alcoholism, Alcohol Use and Health, Cancer, Digestive Diseases, Pancreatic Cancer, Rare Diseases, Clinical Research, Substance Misuse, Oral and gastrointestinal, Good Health and Well Being, Aged, Alcohol Drinking, Case-Control Studies, Cohort Studies, Female, Humans, Male, Pancreatic Neoplasms, Prospective Studies, Alcohol, Pancreatic cancer, Pooled analysis, Oncology and Carcinogenesis, Public Health and Health Services, Epidemiology, Oncology and carcinogenesis
Abstract

The literature has consistently reported no association between low to moderate alcohol consumption and pancreatic cancer; however, a few studies have shown that high levels of intake may increase risk. Most single studies have limited power to detect associations even in the highest alcohol intake categories or to examine associations by alcohol type. We analyzed these associations using 1,530 pancreatic cancer cases and 1,530 controls from the Pancreatic Cancer Cohort Consortium (PanScan) nested case-control study. Odds ratios (OR) and 95% confidence intervals (95% CI) were calculated using unconditional logistic regression, adjusting for potential confounders. We observed no significant overall association between total alcohol (ethanol) intake and pancreatic cancer risk (OR = 1.38, 95% CI = 0.86-2.23, for 60 or more g/day vs. >0 to

Published
2010

218. Genome-wide association study of follicular lymphoma identifies a risk locus at 6p21.32

Author

Conde, Lucia, Halperin, Eran, Akers, Nicholas K, Brown, Kevin M, Smedby, Karin E, Rothman, Nathaniel, Nieters, Alexandra, Slager, Susan L, Brooks-Wilson, Angela, Agana, Luz, Riby, Jacques, Liu, Jianjun, Adami, Hans-Olov, Darabi, Hatef, Hjalgrim, Henrik, Low, Hui-Qi, Humphreys, Keith, Melbye, Mads, Chang, Ellen T, Glimelius, Bengt, Cozen, Wendy, Davis, Scott, Hartge, Patricia, Morton, Lindsay M, Schenk, Maryjean, Wang, Sophia S, Armstrong, Bruce, Kricker, Anne, Milliken, Sam, Purdue, Mark P, Vajdic, Claire M, Boyle, Peter, Lan, Qing, Zahm, Shelia H, Zhang, Yawei, Zheng, Tongzhang, Becker, Nikolaus, Benavente, Yolanda, Boffetta, Paolo, Brennan, Paul, Butterbach, Katja, Cocco, Pierluigi, Foretova, Lenka, Maynadié, Marc, de Sanjosé, Silvia, Staines, Anthony, Spinelli, John J, Achenbach, Sara J, Call, Timothy G, Camp, Nicola J, Glenn, Martha, Caporaso, Neil E, Cerhan, James R, Cunningham, Julie M, Goldin, Lynn R, Hanson, Curtis A, Kay, Neil E, Lanasa, Mark C, Leis, Jose F, Marti, Gerald E, Rabe, Kari G, Rassenti, Laura Z, Spector, Logan G, Strom, Sara S, Vachon, Celine M, Weinberg, J Brice, Holly, Elizabeth A, Chanock, Stephen, Smith, Martyn T, Bracci, Paige M, and Skibola, Christine F

Subjects
Lymphoma, Prevention, Cancer, Human Genome, Genetics, Rare Diseases, Hematology, Aetiology, 2.1 Biological and endogenous factors, Disease Susceptibility, Genetic Variation, Genome-Wide Association Study, Humans, Leukemia, Lymphocytic, Chronic, B-Cell, Lymphoma, Follicular, Lymphoma, Non-Hodgkin, Major Histocompatibility Complex, Risk Factors, Biological Sciences, Medical and Health Sciences, Developmental Biology
Abstract

To identify susceptibility loci for non-Hodgkin lymphoma subtypes, we conducted a three-stage genome-wide association study. We identified two variants associated with follicular lymphoma at 6p21.32 (rs10484561, combined P = 1.12 x 10(-29) and rs7755224, combined P = 2.00 x 10(-19); r(2) = 1.0), supporting the idea that major histocompatibility complex genetic variation influences follicular lymphoma susceptibility. We also found confirmatory evidence of a previously reported association between chronic lymphocytic leukemia/small lymphocytic lymphoma and rs735665 (combined P = 4.24 x 10(-9)).

Published
2010

219. Sexual behaviours and the risk of head and neck cancers: a pooled analysis in the International Head and Neck Cancer Epidemiology (INHANCE) consortium

Author

Heck, Julia E, Berthiller, Julien, Vaccarella, Salvatore, Winn, Deborah M, Smith, Elaine M, Shan'gina, Oxana, Schwartz, Stephen M, Purdue, Mark P, Pilarska, Agnieszka, Eluf-Neto, Jose, Menezes, Ana, McClean, Michael D, Matos, Elena, Koifman, Sergio, Kelsey, Karl T, Herrero, Rolando, Hayes, Richard B, Franceschi, Silvia, Wünsch-Filho, Victor, Fernández, Leticia, Daudt, Alexander W, Curado, Maria Paula, Chen, Chu, Castellsagué, Xavier, Ferro, Gilles, Brennan, Paul, Boffetta, Paolo, and Hashibe, Mia

Subjects
Health Services and Systems, Health Sciences, Cancer, Dental/Oral and Craniofacial Disease, Sexually Transmitted Infections, Infectious Diseases, Adult, Age Factors, Aged, Case-Control Studies, Confounding Factors, Epidemiologic, Female, Head and Neck Neoplasms, Health Behavior, Humans, Male, Middle Aged, Odds Ratio, Papillomavirus Infections, Sexual Behavior, Socioeconomic Factors, Sexual practices, head and neck cancer, oropharyngeal neoplasms, homosexual, gay men, risk factors, pooled analyses, Statistics, Public Health and Health Services, Epidemiology, Public health
Abstract

BackgroundSexual contact may be the means by which head and neck cancer patients are exposed to human papillomavirus (HPV).MethodsWe undertook a pooled analysis of four population-based and four hospital-based case-control studies from the International Head and Neck Cancer Epidemiology (INHANCE) consortium, with participants from Argentina, Australia, Brazil, Canada, Cuba, India, Italy, Spain, Poland, Puerto Rico, Russia and the USA. The study included 5642 head and neck cancer cases and 6069 controls. We calculated odds ratios (ORs) of associations between cancer and specific sexual behaviours, including practice of oral sex, number of lifetime sexual partners and oral sex partners, age at sexual debut, a history of same-sex contact and a history of oral-anal contact. Findings were stratified by sex and disease subsite.ResultsCancer of the oropharynx was associated with having a history of six or more lifetime sexual partners [OR = 1.25, 95% confidence interval (CI) 1.01, 1.54] and four or more lifetime oral sex partners (OR = 2.25, 95% CI 1.42, 3.58). Cancer of the tonsil was associated with four or more lifetime oral sex partners (OR = 3.36, 95 % CI 1.32, 8.53), and, among men, with ever having oral sex (OR = 1.59, 95% CI 1.09, 2.33) and with an earlier age at sexual debut (OR = 2.36, 95% CI 1.37, 5.05). Cancer of the base of the tongue was associated with ever having oral sex among women (OR = 4.32, 95% CI 1.06, 17.6), having two sexual partners in comparison with only one (OR = 2.02, 95% CI 1.19, 3.46) and, among men, with a history of same-sex sexual contact (OR = 8.89, 95% CI 2.14, 36.8).ConclusionsSexual behaviours are associated with cancer risk at the head and neck cancer subsites that have previously been associated with HPV infection.

Published
2010

220. Listing Occupational Carcinogens

Author

Siemiatycki, Jack, Richardson, Lesley, Straif, Kurt, Latreille, Benoit, Lakhani, Ramzan, Campbell, Sally, Rousseau, Marie-Claude, and Boffetta, Paolo

Published
2004

224. Applying New Biotechnologies to the Study of Occupational Cancer: A Workshop Summary

Author

Toraason, Mark, Albertini, Richard, Bayard, Steven, Bigbee, William, Blair, Aaron, Boffetta, Paolo, Bonassi, Stefano, Chanock, Steven, Christiani, David, Eastmond, David, Hanash, Samuel, Henry, Carol, Kadlubar, Fred, Mirer, Frank, Nebert, Daniel, Rapport, Stephen, Rest, Kathleen, Rothman, Nathaniel, Ruder, Avima, Savage, Russell, Schulte, Paul, Siemiatycki, Jack, Shields, Peter, Smith, Martyn, Tolbert, Paige, Vermeulen, Roel, Vineis, Paolo, Wacholder, Sholom, Ward, Elizabeth, Waters, Michael, and Weston, Ainsley

Published
2004

229. Exposure–Response Analyses of Asbestos and Lung Cancer Subtypes in a Pooled Analysis of Case–Control Studies

Author

Olsson, Ann C., Vermeulen, Roel, Schüz, Joachim, Kromhout, Hans, Pesch, Beate, Peters, Susan, Behrens, Thomas, Portengen, Lützen, Mirabelli, Dario, Gustavsson, Per, Kendzia, Benjamin, Almansa, Josue, Luzon, Veronique, Vlaanderen, Jelle, Stücker, Isabelle, Guida, Florence, Consonni, Dario, Caporaso, Neil, Landi, Maria Teresa, Field, John, Brüske, Irene, Wichmann, Heinz-Erich, Siemiatycki, Jack, Parent, Marie-Elise, Richiardi, Lorenzo, Merletti, Franco, Jöckel, Karl-Heinz, Ahrens, Wolfgang, Pohlabeln, Hermann, Plato, Nils, Tardón, Adonina, Zaridze, David, McLaughlin, John, Demers, Paul, Szeszenia-Dabrowska, Neonila, Lissowska, Jolanta, Rudnai, Peter, Fabianova, Eleonora, Dumitru, Rodica Stanescu, Bencko, Vladimir, Foretova, Lenka, Janout, Vladimir, Boffetta, Paolo, Bueno-de-Mesquita, Bas, Forastiere, Francesco, Brüning, Thomas, and Straif, Kurt

Published
2017

230. Cigarette Smoking and Pancreatic Cancer: A Pooled Analysis From the Pancreatic Cancer Cohort Consortium

Author

Lynch, Shannon M, Vrieling, Alina, Lubin, Jay H, Kraft, Peter, Mendelsohn, Julie B, Hartge, Patricia, Canzian, Federico, Steplowski, Emily, Arslan, Alan A, Gross, Myron, Helzlsouer, Kathy, Jacobs, Eric J, LaCroix, Andrea, Petersen, Gloria, Zheng, Wei, Albanes, Demetrius, Amundadottir, Laufey, Bingham, Sheila A, Boffetta, Paolo, Boutron-Ruault, Marie-Christine, Chanock, Stephen J, Clipp, Sandra, Hoover, Robert N, Jacobs, Kevin, Johnson, Karen C, Kooperberg, Charles, Luo, Juhua, Messina, Catherine, Palli, Domenico, Patel, Alpa V, Riboli, Elio, Shu, Xiao-Ou, Rodriguez Suarez, Laudina, Thomas, Gilles, Tjønneland, Anne, Tobias, Geoffrey S, Tong, Elissa, Trichopoulos, Dimitrios, Virtamo, Jarmo, Ye, Weimin, Yu, Kai, Zeleniuch-Jacquette, Anne, Bueno-de-Mesquita, H Bas, and Stolzenberg-Solomon, Rachael Z

Subjects
Tobacco, Digestive Diseases, Cancer, Pancreatic Cancer, Rare Diseases, Prevention, Tobacco Smoke and Health, Prevention of disease and conditions, and promotion of well-being, 3.1 Primary prevention interventions to modify behaviours or promote wellbeing, Adenocarcinoma, Adult, Aged, Aged, 80 and over, Case-Control Studies, Cohort Studies, Female, Humans, Male, Middle Aged, Pancreatic Neoplasms, Prospective Studies, Risk, Smoking, Smoking Cessation, United States, pancreas, pancreatic neoplasms, smoking, tobacco use cessation, Mathematical Sciences, Medical and Health Sciences, Epidemiology
Abstract

Smoking is an established risk factor for pancreatic cancer; however, detailed examination of the association of smoking intensity, smoking duration, and cumulative smoking dose with pancreatic cancer is limited. The authors analyzed pooled data from the international Pancreatic Cancer Cohort Consortium nested case-control study (1,481 cases, 1,539 controls). Odds ratios and 95% confidence intervals were calculated by using unconditional logistic regression. Smoking intensity effects were examined with an excess odds ratio model that was linear in pack-years and exponential in cigarettes smoked per day and its square. When compared with never smokers, current smokers had a significantly elevated risk (odds ratio (OR) = 1.77, 95% confidence interval (CI): 1.38, 2.26). Risk increased significantly with greater intensity (> or =30 cigarettes/day: OR = 1.75, 95% CI: 1.27, 2.42), duration (> or =50 years: OR = 2.13, 95% CI: 1.25, 3.62), and cumulative smoking dose (> or =40 pack-years: OR = 1.78, 95% CI: 1.35, 2.34). Risk more than 15 years after smoking cessation was similar to that for never smokers. Estimates of excess odds ratio per pack-year declined with increasing intensity, suggesting greater risk for total exposure delivered at lower intensity for longer duration than for higher intensity for shorter duration. This finding and the decline in risk after smoking cessation suggest that smoking has a late-stage effect on pancreatic carcinogenesis.

Published
2009

231. Associations between Ileal Juice Bile Acids and Colorectal Advanced Adenoma

Author

Luu, Hung N., primary, Tran, Chi Thi-Du, additional, Wang, Renwei, additional, Nguyen, Mai Vu-Tuyet, additional, Tran, Mo Thi, additional, Tuong, Thuy Thi-Van, additional, Tran, Quang Hong, additional, Le, Linh Cu, additional, Pham, Huong Thi-Thu, additional, Vu, Hien Huy, additional, Bui, Nam Chi, additional, Ha, Hien Thi-Thu, additional, Trinh, Dung Tuan, additional, Thomas, Claire E., additional, Adams-Haduch, Jennifer, additional, Velikokhatnaya, Liudmilla, additional, Schoen, Robert E., additional, Xie, Guoxiang, additional, Jia, Wei, additional, Boffetta, Paolo, additional, Clemente, Jose C., additional, and Yuan, Jian-Min, additional

Published
2023
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232. Association between Sleep Duration and Colorectal Adenomas: Findings from a Case–Control Study in Vietnam

Author

Tran, Chi Thi-Du, primary, Paragomi, Pedram, additional, Tran, Mo Thi, additional, Nguyen, Mai Vu-Tuyet, additional, Tuong, Thuy Thi-Van, additional, Tran, Quang Hong, additional, Le, Linh Cu, additional, Pham, Huong Thi-Thu, additional, Ha, Hien Thi-Thu, additional, Bui, Nam Chi, additional, Vu, Hien Huy, additional, Ta, Phuong Que, additional, Shrubsole, Martha J., additional, Cai, Qiuyin, additional, Ye, Fei, additional, Le, Song Huu, additional, Vu, Khien Van, additional, Tran, Huong Thi-Thanh, additional, Tran, Thuan Van, additional, Boffetta, Paolo, additional, Shu, Xiao-Ou, additional, and Luu, Hung N., additional

Published
2023
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233. Supplementary Table from Plasma miR-151-3p as a Candidate Diagnostic Biomarker for Head and Neck Cancer: A Cross-sectional Study within the INHANCE Consortium

Author

Pastorino, Roberta, primary, Sassano, Michele, primary, Danilo Tiziano, Francesco, primary, Giraldi, Luca, primary, Amore, Rosarita, primary, Arzani, Dario, primary, Abiusi, Emanuela, primary, Ahrens, Wolfgang, primary, Vilches, Laia Alemany, primary, Canova, Cristina, primary, Healy, Claire Mary, primary, Holcatova, Ivana, primary, Lagiou, Pagona, primary, Polesel, Jerry, primary, Popovic, Maja, primary, Nygård, Ståle, primary, Cadoni, Gabriella, primary, Znaor, Ariana, primary, Boffetta, Paolo, primary, Matsuo, Keitaro, primary, Oze, Isao, primary, Brennan, Paul, primary, and Boccia, Stefania, primary

Published
2023
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234. Data from Plasma miR-151-3p as a Candidate Diagnostic Biomarker for Head and Neck Cancer: A Cross-sectional Study within the INHANCE Consortium

Author

Pastorino, Roberta, primary, Sassano, Michele, primary, Danilo Tiziano, Francesco, primary, Giraldi, Luca, primary, Amore, Rosarita, primary, Arzani, Dario, primary, Abiusi, Emanuela, primary, Ahrens, Wolfgang, primary, Vilches, Laia Alemany, primary, Canova, Cristina, primary, Healy, Claire Mary, primary, Holcatova, Ivana, primary, Lagiou, Pagona, primary, Polesel, Jerry, primary, Popovic, Maja, primary, Nygård, Ståle, primary, Cadoni, Gabriella, primary, Znaor, Ariana, primary, Boffetta, Paolo, primary, Matsuo, Keitaro, primary, Oze, Isao, primary, Brennan, Paul, primary, and Boccia, Stefania, primary

Published
2023
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235. Dietary Choline and Betaine Intake and Risk of Colorectal Cancer in an Iranian Population

Author

Seyyedsalehi, Monireh Sadat, primary, Rossi, Marta, additional, Hadji, Maryam, additional, Rashidian, Hamideh, additional, Marzban, Maryam, additional, Parpinel, Maria, additional, Fiori, Federica, additional, Naghibzadeh-Tahami, Ahmad, additional, Hannun, Yusuf A., additional, Luberto, Chiara, additional, Zendehdel, Kazem, additional, and Boffetta, Paolo, additional

Published
2023
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236. The effect of body mass index at cancer diagnosis on survival of patients with squamous cell head and neck carcinoma

Author

Pastorino, Roberta, primary, Marafon, Denise Pires, additional, Hoxhaj, Ilda, additional, Grossi, Adriano, additional, Giraldi, Luca, additional, Rondinò, Antonella, additional, Cadoni, Gabriella, additional, Polesel, Jerry, additional, Serraino, Diego, additional, Vecchia, Carlo La, additional, Garavello, Werner, additional, Canova, Cristina, additional, Richiardi, Lorenzo, additional, Lissowska, Jolanta, additional, Pandics, Tamas, additional, Dudding, Tom, additional, Ness, Andy, additional, Thomas, Steve, additional, Pring, Miranda, additional, Kelsey, Karl, additional, McClean, Michael, additional, Bradshaw, Patrick T., additional, Zhang, Zuo-Feng, additional, Morgenstern, Hal, additional, Rozek, Laura, additional, Wolf, Gregory T., additional, Olshan, Andrew F., additional, Liu, Geoffrey, additional, Hung, Rayjean J., additional, Vilensky, Marta, additional, de Carvalho, Marcos Brasilino, additional, Lopez, Rossana Veronica Mendonza, additional, Wunsch-Filho, Victor, additional, Boffetta, Paolo, additional, Hashibe, Mia, additional, Lee, Yuan-Chin Amy, additional, and Boccia, Stefania, additional

Published
2023
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237. Corrigendum: Temporal trends of COVID-19 antibodies in vaccinated healthcare workers undergoing repeated serological sampling: an individual-level analysis within 13 months in the ORCHESTRA cohort

Author

Collatuzzo, Giulia, primary, De Palma, Giuseppe, additional, Violante, Francesco S., additional, Porru, Stefano, additional, Filon, Francesca Larese, additional, Fabianova, Eleonora, additional, Violán, Concepción, additional, Vimercati, Luigi, additional, Leustean, Mihaela, additional, Rodriguez-Suarez, Marta Maria, additional, Sansone, Emanuele, additional, Sala, Emma, additional, Zunarelli, Carlotta, additional, Lodi, Vittorio, additional, Monaco, Maria Grazia Lourdes, additional, Spiteri, Gianluca, additional, Negro, Corrado, additional, Beresova, Jana, additional, Carrasco-Ribelles, LucÌa A., additional, Tafuri, Silvio, additional, Asafo, Shuffield S., additional, Ditano, Giorgia, additional, Abedini, Mahsa, additional, and Boffetta, Paolo, additional

Published
2023
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241. Abstract 4200: Association between reproductive factors with lung cancer incidence and mortality: A pooled analysis of over 308,000 females in the Asia Cohort Consortium

Author

Yin, Xin, primary, Kishida, Rie, additional, Abe, Sarah Krull, additional, Islam, Md. Rashedul, additional, Rahman, Md. Shafiur, additional, Saito, Eiko, additional, Lan, Qing, additional, Bletcher, Batel, additional, Merritt, Melissa, additional, Choi, Ji-Yeob, additional, Shin, Aesun, additional, Katagiri, Ryoko, additional, Shu, Xiao-Ou, additional, Sawada, Norie, additional, Tamakoshi, Akiko, additional, Koh, Woon-Puay, additional, Tsuji, Ichiro, additional, Nagata, Chisato, additional, Park, Sue K., additional, Kweon, Sun-Seog, additional, Gao, Yu-Tang, additional, Tsugane, Shoichiro, additional, Kimura, Takashi, additional, Yuan, Jian-Min, additional, Lu, Yukai, additional, Kanemura, Seiki, additional, Sugawara, Yumi, additional, Wada, Keiko, additional, Shin, Min-Ho, additional, Ahsan, Habibul, additional, Boffetta, Paolo, additional, Chia, Kee Seng, additional, Matsuo, Keitaro, additional, Qiao, You-Lin, additional, Rothman, Nathaniel, additional, Zheng, Wei, additional, Inoue, Manami, additional, Kang, Daehee, additional, and Seow, Wei Jie, additional

Published
2023
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242. Supplementary Data from Oral Health and Risk of Upper Gastrointestinal Cancers in a Large Prospective Study from a High-risk Region: Golestan Cohort Study

Author

Yano, Yukiko, primary, Abnet, Christian C., primary, Poustchi, Hossein, primary, Roshandel, Gholamreza, primary, Pourshams, Akram, primary, Islami, Farhad, primary, Khoshnia, Masoud, primary, Amiriani, Taghi, primary, Norouzi, Alireza, primary, Kamangar, Farin, primary, Boffetta, Paolo, primary, Brennan, Paul, primary, Dawsey, Sanford M., primary, Vogtmann, Emily, primary, Malekzadeh, Reza, primary, and Etemadi, Arash, primary

Published
2023
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243. Data from Oral Health and Risk of Upper Gastrointestinal Cancers in a Large Prospective Study from a High-risk Region: Golestan Cohort Study

Author

Yano, Yukiko, primary, Abnet, Christian C., primary, Poustchi, Hossein, primary, Roshandel, Gholamreza, primary, Pourshams, Akram, primary, Islami, Farhad, primary, Khoshnia, Masoud, primary, Amiriani, Taghi, primary, Norouzi, Alireza, primary, Kamangar, Farin, primary, Boffetta, Paolo, primary, Brennan, Paul, primary, Dawsey, Sanford M., primary, Vogtmann, Emily, primary, Malekzadeh, Reza, primary, and Etemadi, Arash, primary

Published
2023
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245. Occupation and Bladder Cancer among Men in Western Europe

Author

Kogevinas, Manolis, Cordier, Sylvaine, Ranft, Ulrich, González, Carlos A., Vineis, Paolo, Chang-Claude, Jenny, Lynge, Elsebeth, Wahrendorf, Jürgen, Tzonou, Anastasia, Jöckel, Karl-Heinz, Serra, Consol, Porru, Stefano, Hours, Martine, Greiser, Eberhard, and Boffetta, Paolo

Published
2003

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