2,374 results on '"McCormick, Frank"'
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202. Data from Vemurafenib Cooperates with HPV to Promote Initiation of Cutaneous Tumors
203. Supplementary Table 1 from Vemurafenib Cooperates with HPV to Promote Initiation of Cutaneous Tumors
204. Supplementary Figure 1 from PIK3CA Cooperates with Other Phosphatidylinositol 3′-Kinase Pathway Mutations to Effect Oncogenic Transformation
205. Supplementary Figure 3 from PIK3CA Cooperates with Other Phosphatidylinositol 3′-Kinase Pathway Mutations to Effect Oncogenic Transformation
206. supplementary figure 1 from Enhanced MET Translation and Signaling Sustains K-Ras–Driven Proliferation under Anchorage-Independent Growth Conditions
207. Supplementary Figure 2 from Basal Subtype and MAPK/ERK Kinase (MEK)-Phosphoinositide 3-Kinase Feedback Signaling Determine Susceptibility of Breast Cancer Cells to MEK Inhibition
208. Supplemtal figure legends 1-5 from Enhanced MET Translation and Signaling Sustains K-Ras–Driven Proliferation under Anchorage-Independent Growth Conditions
209. Supplementary Tables 1-6 from Basal Subtype and MAPK/ERK Kinase (MEK)-Phosphoinositide 3-Kinase Feedback Signaling Determine Susceptibility of Breast Cancer Cells to MEK Inhibition
210. Supplementary Figure 4 from PIK3CA Cooperates with Other Phosphatidylinositol 3′-Kinase Pathway Mutations to Effect Oncogenic Transformation
211. supplementary figure 6 from Enhanced MET Translation and Signaling Sustains K-Ras–Driven Proliferation under Anchorage-Independent Growth Conditions
212. supplementary figure 2 from Enhanced MET Translation and Signaling Sustains K-Ras–Driven Proliferation under Anchorage-Independent Growth Conditions
213. Supplementary Methods from PIK3CA Cooperates with Other Phosphatidylinositol 3′-Kinase Pathway Mutations to Effect Oncogenic Transformation
214. Supplementary Tables 1-5 from PIK3CA Cooperates with Other Phosphatidylinositol 3′-Kinase Pathway Mutations to Effect Oncogenic Transformation
215. Supplementary Figure Legends 1-2 from Basal Subtype and MAPK/ERK Kinase (MEK)-Phosphoinositide 3-Kinase Feedback Signaling Determine Susceptibility of Breast Cancer Cells to MEK Inhibition
216. Data from Basal Subtype and MAPK/ERK Kinase (MEK)-Phosphoinositide 3-Kinase Feedback Signaling Determine Susceptibility of Breast Cancer Cells to MEK Inhibition
217. AKT inactivation causes persistent drug tolerance to EGFR inhibitors
218. K-Ras Promotes Tumorigenicity through Suppression of Non-canonical Wnt Signaling
219. Trends in Meniscal Allograft Transplantation in the United States, 2007 to 2011
220. Postoperative Rotator Cuff Repair Rehabilitation and Complication Management
221. Biceps tenodesis for long head of the biceps after auto-rupture or failed surgical tenotomy: results in an active population
222. Drug-tolerant persister cancer cells are vulnerable to GPX4 inhibition
223. A Call for Discovery and Therapeutic Development for Cutaneous Neurofibromas
224. ETS-targeted therapy: can it substitute for MEK inhibitors?
225. Biological Augmentation of Meniscus Repair and Restoration
226. A Comprehensive Analysis of Medicare Trends in Utilization and Hospital Economics for Total Knee and Hip Arthroplasty From 2005 to 2011
227. Contributors
228. Author Correction: RAS-targeted therapies: is the undruggable drugged?
229. Destabilizing NF1 variants act in a dominant negative manner through neurofibromin dimerization
230. Projecting the Economic Impact of Compensating Living Kidney Donors in the United States: Cost-Benefit Analysis Demonstrates Substantial Patient and Societal Gains
231. Ras-dependent RAF-MAPK hyperactivation by pathogenic RIT1 is a therapeutic target in Noonan syndrome-associated cardiac hypertrophy
232. DDDR-06. NEUROFIBROMATOSIS TUMOR SUPPRESSORS COOPERATIVELY DRIVE TUMOR DE-DIFFERENTIATION AND MEK INHIBITOR RESISTANCE IN PERIPHERAL NERVOUS SYSTEM TUMORS
233. Unicompartmental Knee Arthroplasty Versus High Tibial Osteotomy: United States Practice Patterns for the Surgical Treatment of Unicompartmental Arthritis
234. Dragging Ras Back in the Ring
235. A synoptic survey of ecosystem services from headwater catchments in the United States
236. Minced Cartilage Techniques
237. Current Status of Evidence-Based Sports Medicine
238. Trends in the Surgical Treatment of Articular Cartilage Lesions in the United States: An Analysis of a Large Private-Payer Database Over a Period of 8 Years
239. The Epidemiology of Interstitial Cystitis/Bladder Pain Syndrome (IC/BPS): How Good Was a Study of 30-Years Ago
240. Reverse total shoulder arthroplasty in patients of varying body mass index
241. The Management of Type II Superior Labral Anterior to Posterior Injuries
242. Rating a Sports Medicine Surgeon’s “Quality” in the Modern Era: an Analysis of Popular Physician Online Rating Websites
243. Quantification and correlation of hip capsular volume to demographic and radiographic predictors
244. Novel Approaches to p53-Based Therapy: ONYX-015
245. Structure of the SHOC2–MRAS–PP1C complex provides insights into RAF activation and Noonan syndrome
246. Impaired Proteolysis of Noncanonical RAS Proteins Drives Clonal Hematopoietic Transformation
247. Novel Regulators of Macropinocytosis-Dependent Growth Revealed by Informer Set Library Screening in Pancreatic Cancer Cells
248. Functional interactions between neurofibromatosis tumor suppressors drive Schwann cell tumor de-differentiation and treatment resistance
249. Development of an Inhibitor of raf Kinase
250. Endoscopic Repair of Full-Thickness Abductor Tendon Tears: Surgical Technique and Outcome at Minimum of 1-Year Follow-up
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