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243 results on '"Sedivy, John M."'

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201. Loss of epigenetic information as a cause of mammalian aging.

202. Regulation of the somatotropic axis by MYC-mediated miRNA repression.

203. LINE-1 retrotransposon expression in cancerous, epithelial and neuronal cells revealed by 5' single-cell RNA-Seq.

204. Loss of epigenetic information as a cause of mammalian aging.

205. Single-Cell Transcriptomics Reveals Global Markers of Transcriptional Diversity across Different Forms of Cellular Senescence.

206. Analysis of Somatic Mutations in Senescent Cells Using Single-Cell Whole-Genome Sequencing.

207. Translational Significance of the LINE-1 Jumping Gene in Skeletal Muscle.

208. Sirt6 regulates lifespan in Drosophila melanogaster .

209. The role of retrotransposable elements in ageing and age-associated diseases.

210. Inflammation, epigenetics, and metabolism converge to cell senescence and ageing: the regulation and intervention.

211. Phase separation of the LINE-1 ORF1 protein is mediated by the N-terminus and coiled-coil domain.

212. CD38 ecto-enzyme in immune cells is induced during aging and regulates NAD + and NMN levels.

213. L1 drives HSC aging and affects prognosis of chronic myelomonocytic leukemia.

214. Enhancing Autophagy Diminishes Aberrant Ca 2+ Homeostasis and Arrhythmogenesis in Aging Rabbit Hearts.

215. LINE1 Derepression in Aged Wild-Type and SIRT6-Deficient Mice Drives Inflammation.

216. L1 drives IFN in senescent cells and promotes age-associated inflammation.

217. Regulation of Cellular Senescence by Polycomb Chromatin Modifiers through Distinct DNA Damage- and Histone Methylation-Dependent Pathways.

218. Mitochondria: Masters of Epigenetics.

219. Interventions to Slow Aging in Humans: Are We Ready?

220. Human Genomics. Sleeping dogs of the genome.

221. A comparison of oncogene-induced senescence and replicative senescence: implications for tumor suppression and aging.

222. The effects of aging on the expression of Wnt pathway genes in mouse tissues.

223. Death by transposition - the enemy within?

224. A link between the accumulation of DNA damage and loss of multi-potency of human mesenchymal stromal cells.

225. Widespread expression of the Supv3L1 mitochondrial RNA helicase in the mouse.

227. Cellular senescence and organismal aging.

228. Aging by epigenetics--a consequence of chromatin damage?

229. Signaling crossroads: the function of Raf kinase inhibitory protein in cancer, the central nervous system and reproduction.

230. Mice lacking Raf kinase inhibitor protein-1 (RKIP-1) have altered sperm capacitation and reduced reproduction rates with a normal response to testicular injury.

231. Telomeres limit cancer growth by inducing senescence: long-sought in vivo evidence obtained.

232. Correlation analysis reveals the emergence of coherence in the gene expression dynamics following system perturbation.

233. Analysis of cell cycle phases and progression in cultured mammalian cells.

234. Accumulation of senescent cells in mitotic tissue of aging primates.

235. Loss of Raf kinase inhibitor protein promotes cell proliferation and migration of human hepatoma cells.

236. Reduced c-Myc signaling triggers telomere-independent senescence by regulating Bmi-1 and p16(INK4a).

237. Regulation of growth arrest in senescence: telomere damage is not the end of the story.

238. Telomere shortening triggers senescence of human cells through a pathway involving ATM, p53, and p21(CIP1), but not p16(INK4a).

239. Engineering the serine/threonine protein kinase Raf-1 to utilise an orthogonal analogue of ATP substituted at the N6 position.

240. Loss of retinoblastoma but not p16 function allows bypass of replicative senescence in human fibroblasts.

241. Random mutagenesis of PDZ(Omi) domain and selection of mutants that specifically bind the Myc proto-oncogene and induce apoptosis.

242. Molecular characterization of human telomerase reverse transcriptase-immortalized human fibroblasts by gene expression profiling: activation of the epiregulin gene.

243. Targeted inactivation of p53 in human cells does not result in aneuploidy.

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