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456 results on '"Takeda, Taizo"'

206. Short-term intensive treatment with ceftizoxime in patients with otitis media, especially otitis media with irritation of the labyrinth.

Author

Takeda, Taizo, primary, Matsubara, Hideharu, additional, Kitajima, Kazutomo, additional, Yazawa, Yoshiro, additional, Uchida, Kaoru, additional, Ikeda, Sei, additional, Minami, Fumika, additional, Tsuchida, Hiroo, additional, Fujishima, Hajime, additional, Sawada, Izumi, additional, Kodama, Akira, additional, Fujikawa, Norio, additional, and Kitahara, Masaaki, additional

Published
1985
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211. Meningogenic meningitis

Author

Saito, Haruo, primary, Kitahara, Masaaki, additional, Kitajima, Kazutomo, additional, Takeda, Taizo, additional, Yazawa, Yoshiro, additional, Matsubara, Hideharu, additional, Kitano, Hitoshi, additional, Kitano, Mayumi, additional, and Kitano, Hiroya, additional

Published
1981
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214. A Multi-channel Telemetric System for the ENT-Ward

Author

Saito, Haruo, primary, Kitahara, Masaaki, additional, Kitajima, Kazutomo, additional, Takeda, Taizo, additional, Yazawa, Yoshiro, additional, Matsubara, Hideharu, additional, Kitano, Hitoshi, additional, Kitano, Mayumi, additional, Kitano, Hiroya, additional, Kodama, Akira, additional, and Mizukami, Chikashi, additional

Published
1981
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227. Onset Type of Meniere's Disease

Author

Kitano, Hitoshi, primary, Saito, Haruo, additional, Kitajima, Kazutomo, additional, Takeda, Taizo, additional, Yazawa, Yoshiro, additional, Matsubara, Hideharu, additional, Kitano, Mayumi, additional, Kitano, Hiroya, additional, Kodama, Akira, additional, and Mizukami, Chikashi, additional

Published
1981
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233. Relapsing Facial Palsy Caused by Tumor Invasion

Author

Saito, Haruo, primary, Kitahara, Masaaki, additional, Kitajima, Kazutomo, additional, Takeda, Taizo, additional, Yazawa, Yoshiro, additional, Matsubara, Hideharu, additional, Kitano, Hitoshi, additional, Kitano, Mayumi, additional, and Kitano, Hiroya, additional

Published
1981
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244. Roentgenlogic Study of Meniere's Disease

Author

Kitano, Hitoshi, primary, Kitahara, Masaaki, additional, Saito, Haruo, additional, Kitajima, Kazutomo, additional, Takeda, Taizo, additional, Yazawa, Yoshiro, additional, and Matsubara, Hideharu, additional

Published
1979
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248. Regulation of inner ear fluid in the rat by vasopressin

Author

Kitano, Hiroya, Suzuki, Mikio, Kitanishi, Tsuyoshi, Yazawa, Yoshiro, Kitajima, Kazutomo, Isono, Takahiro, Takeda, Taizo, Kimura, Hiroshi, and Tooyama, Ikuo

Abstract

THE anti-diuretic hormone vasopressin has been shown to be important in regulating inner ear fluid. The diuretic hormone, CNP, and its receptor, ANP-B receptor, may also function in the regulation of inner ear fluid. To determine whether vasopressin directly affects the fluid level, we infused this hormone to rat and assay of V2–AVP receptor mRNA by semiquantitative RT-PCR demonstrated a significantly lower level of this transcript in vasopressin-infused animals than in saline-infused animals. The levels of CNP and ANP-B receptors mRNA, however, were the same in both groups of rats. Results suggest that high plasma levels of vasopressin may be a principal causal factor of endolymphatic hydrops in Meniere's disease, perhaps by down-regulating the number of vasopressin receptors.

Published
1999

249. Vasopressin and oxytocin receptor mRNAs are expressed in the rat inner ear

Author

Kitano, Hiroya, Takeda, Taizo, Suzuki, Mikio, Kitanishi, Tsuyoshi, Yazawa, Yoshiro, Kitajima, Kazutomo, Kimura, Hiroshi, and Tooyama, Ikuo

Abstract

THE cause of endolymphatic hydrops, a characteristic finding in Menière's disease, is not known. To study the possible involvement of the neurohormones vasopressin and oxytocin in this condition, we investigated whether transcripts of the genes encoding the arginine vasopressin (AVP) and oxytocin receptors are expressed in the rat inner ear. Utilizing the reverse transcription-polymerase chain reaction (RT-PCR) method, primers specific for each receptor showed a single message band of the expected size in the rat inner ear. When the PCR products were cloned, the sequences were identical to those of the real-type (V2) AVP receptor and oxytocin receptor transcripts. The finding of vasopressin and oxytocin receptor mRNAs in the inner ear suggests that these neurohypophyseal hormones may have roles in the regulation of inner ear fluid. In particular, the presence of vasopressin receptor mRNA in the inner ear supports the hypothesis of a relationship between high plasma vasopressin levels and endolymphatic hydrops.

Published
1997

250. Endocytosis of CF in marginal cells of stria vascularis regulated by ROCK and MLCK signaling cascade, but not G-proteins.

Author

Kakigi, Akinobu, Okada, Teruhiko, Takeda, Taizo, Uehara, Natsumi, and Nibu, Ken-ichi

Subjects
*MYOSIN light chain kinase, *BOTULINUM A toxins, *ENDOCYTOSIS, *MYOSIN, *RHO-associated kinases, *PERTUSSIS toxin, *AMIDES, *ANIMALS, *AZEPINES, *BACTERIAL toxins, *BOTULINUM toxin, *CARRIER proteins, *CELL receptors, *CELLULAR signal transduction, *COCHLEA, *CYTOPLASM, *ELECTRON microscopy, *ENZYME inhibitors, *FERRITIN, *GUINEA pigs, *HYDROCARBONS, *HYDROLASES, *PHOSPHOTRANSFERASES, *PYRIDINE, *TRANSFERASES, *CHEMICAL inhibitors, *PHARMACODYNAMICS
Abstract

Objective The endocytosis of cationized feritin (CF) via a clathrin-mediated pathway is regulated by a signaling network. Marginal cells showed the active endocytosis of CF via a clathrin-mediated pathway. The internalization of receptors through this clathrin-mediated pathway is an important regulatory event in signal transduction. Numerous kinases are involved in endocytosis, and each endocytic route is subjected to high-order regulation by cellular signaling mechanisms. In this study, we investigated whether ROCK and MLCK signaling cascades and G-proteins regulate the endocytosis of CF in marginal cells of the stria vascularis. Methods CF was infused into the cochlear duct with pertussis toxin (PTX),Clostridium botulinum C3 toxin (BTX), guanosine(g-thio)-triphosphate (GTP-γS), ML-7, Y-27632. Endocytic activity was measured at 30 min after the start of infusion under an electron microscope. Results In marginal cells, CF was internalized via a clathrin-mediated pathway that depends on F-actin and microtubules (MT). Its processes were controlled by myosin light chain kinase (MLCK) and Rho-associated kinase (ROCK), but not affected by G-protein-coupled receptor (GPCR) or the RhoA signaling cascade. Conclusion Our previous study showed that the main endocytotic pathway of microperoxidase (MPO) did not depend on the Rho/ROCK molecular switch or actin/myosin motor system, but was mainly regulated by the RhoA signaling cascade. The present study results indicate that these signaling cascades regulating CF internalization completely differ from the cascades for MPO internalization. [ABSTRACT FROM AUTHOR]

Published
2019
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