547 results on '"Aguirre-Ghiso, Julio A."'
Search Results
2. The State of Melanoma: Emergent Challenges and Opportunities
3. ZFP281 drives a mesenchymal-like dormancy program in early disseminated breast cancer cells that prevents metastatic outgrowth in the lung
4. 5-Azacytidine- and retinoic-acid-induced reprogramming of DCCs into dormancy suppresses metastasis via restored TGF-β-SMAD4 signaling
5. Stromal changes in the aged lung induce an emergence from melanoma dormancy
6. A tumor-derived type III collagen-rich ECM niche regulates tumor cell dormancy
7. Primary tumor associated macrophages activate programs of invasion and dormancy in disseminating tumor cells
8. Lineage commitment pathways epigenetically oppose oncogenic Gαq/11-YAP signaling in dormant disseminated uveal melanoma
9. Abstract B021: Influenza-induced inflammatory response reactivates and promotes dormant breast cancer cell outgrowth in lungs
10. Immune evasion by dormant disseminated cancer cells: A Fermi paradox?
11. Tissue-resident macrophages provide a pro-tumorigenic niche to early NSCLC cells
12. Bone marrow NG2+/Nestin+ mesenchymal stem cells drive DTC dormancy via TGF-β2
13. Slow proliferation of BAP1-deficient uveal melanoma cells is associated with reduced S6 signaling and resistance to nutrient stress.
14. Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
15. Supplementary Figure S2 from A PERK-Specific Inhibitor Blocks Metastatic Progression by Limiting Integrated Stress Response–Dependent Survival of Quiescent Cancer Cells
16. Supplementary Table S3 from A PERK-Specific Inhibitor Blocks Metastatic Progression by Limiting Integrated Stress Response–Dependent Survival of Quiescent Cancer Cells
17. Targeting cancer cell dormancy
18. The current paradigm and challenges ahead for the dormancy of disseminated tumor cells
19. Identification of markers that functionally define a quiescent multiple myeloma cell sub-population surviving bortezomib treatment.
20. Targeting the dependence on PIK3C3-mTORC1 signaling in dormancy-prone breast cancer cells blunts metastasis initiation
21. How dormant cancer persists and reawakens
22. Epigenetic Regulation of Cancer Dormancy as a Plasticity Mechanism for Metastasis Initiation
23. An IRAK1–PIN1 signalling axis drives intrinsic tumour resistance to radiation therapy
24. Guidelines for the use and interpretation of assays for monitoring autophagy.
25. Dormancy in Breast Cancer
26. Abstract 5131: Pulmonary influenza infection promotes the awakening of dormant metastatic breast cancer cells
27. Supplementary Figure S1 from Effects of Oncogenic Gαq and Gα11 Inhibition by FR900359 in Uveal Melanoma
28. Supplementary Table 1 from Metabolic Adaptations to MEK and CDK4/6 Cotargeting in Uveal Melanoma
29. Data from IGF1R Inhibition Enhances the Therapeutic Effects of Gq/11 Inhibition in Metastatic Uveal Melanoma Progression
30. Figure S2 from IGF1R Inhibition Enhances the Therapeutic Effects of Gq/11 Inhibition in Metastatic Uveal Melanoma Progression
31. Data from Metabolic Adaptations to MEK and CDK4/6 Cotargeting in Uveal Melanoma
32. Data from Effects of Oncogenic Gαq and Gα11 Inhibition by FR900359 in Uveal Melanoma
33. Supplementary Figures 1-5 from Metabolic Adaptations to MEK and CDK4/6 Cotargeting in Uveal Melanoma
34. Supplementary Figure S1 from Bortezomib Enhances the Efficacy of Fulvestrant by Amplifying the Aggregation of the Estrogen Receptor, Which Leads to a Proapoptotic Unfolded Protein Response
35. Supplementary Figure 2 from Inhibition of eIF2α Dephosphorylation Maximizes Bortezomib Efficiency and Eliminates Quiescent Multiple Myeloma Cells Surviving Proteasome Inhibitor Therapy
36. Supplementary Figure 2 from Inducible Nitric Oxide Synthase Drives mTOR Pathway Activation and Proliferation of Human Melanoma by Reversible Nitrosylation of TSC2
37. Supplementary Figure 3 from Dual Function of Pancreatic Endoplasmic Reticulum Kinase in Tumor Cell Growth Arrest and Survival
38. Supplementary Figure 4 from Inducible Nitric Oxide Synthase Drives mTOR Pathway Activation and Proliferation of Human Melanoma by Reversible Nitrosylation of TSC2
39. Data from Inducible Nitric Oxide Synthase Drives mTOR Pathway Activation and Proliferation of Human Melanoma by Reversible Nitrosylation of TSC2
40. Supplementary Figure 5 from Computational Identification of a p38SAPK-Regulated Transcription Factor Network Required for Tumor Cell Quiescence
41. Data from Inhibition of eIF2α Dephosphorylation Maximizes Bortezomib Efficiency and Eliminates Quiescent Multiple Myeloma Cells Surviving Proteasome Inhibitor Therapy
42. Supplementary Figure 2 from Dual Function of Pancreatic Endoplasmic Reticulum Kinase in Tumor Cell Growth Arrest and Survival
43. Data from Computational Identification of a p38SAPK-Regulated Transcription Factor Network Required for Tumor Cell Quiescence
44. Supplementary Figure 6 from Inducible Nitric Oxide Synthase Drives mTOR Pathway Activation and Proliferation of Human Melanoma by Reversible Nitrosylation of TSC2
45. Supplementary Results and Figure Legends 1-3 from Dual Function of Pancreatic Endoplasmic Reticulum Kinase in Tumor Cell Growth Arrest and Survival
46. Supplementary Figure 3 from Computational Identification of a p38SAPK-Regulated Transcription Factor Network Required for Tumor Cell Quiescence
47. Supplementary Figure 5 from Inducible Nitric Oxide Synthase Drives mTOR Pathway Activation and Proliferation of Human Melanoma by Reversible Nitrosylation of TSC2
48. Supplementary Figure 1 from Dual Function of Pancreatic Endoplasmic Reticulum Kinase in Tumor Cell Growth Arrest and Survival
49. Supplementary Figure 1 from Inducible Nitric Oxide Synthase Drives mTOR Pathway Activation and Proliferation of Human Melanoma by Reversible Nitrosylation of TSC2
50. Supplementary Figure Legends 1-2 from Inhibition of eIF2α Dephosphorylation Maximizes Bortezomib Efficiency and Eliminates Quiescent Multiple Myeloma Cells Surviving Proteasome Inhibitor Therapy
Catalog
Books, media, physical & digital resources
Discovery Service for Jio Institute Digital Library
For full access to our library's resources, please sign in.