48 results on '"Ale-Agha, Niloofar"'
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2. Mitochondrial Telomerase Reverse Transcriptase Protects From Myocardial Ischemia/Reperfusion Injury by Improving Complex I Composition and Function
3. Caffeine Inhibits Oxidative Stress- and Low Dose Endotoxemia-Induced Senescence—Role of Thioredoxin-1
4. Unhealthy diet and ultrafine carbon black particles induce senescence and disease associated phenotypic changes
5. Abstract 1236: The vasoprotective transcription factor Grainyhead-like 3 exerts extra-nuclear functions and is regulated by plant-derived agents in young and senescent endothelial cells
6. Abstract 1214: APEX1 (1–20) and Selenoprotein T protect endothelial cells against LPS-induced activation and apoptosis
7. Aryl Hydrocarbon Receptor-Dependent and -Independent Pathways Mediate Curcumin Anti-Aging Effects
8. Protective Effects of Curcumin in Cardiovascular Diseases—Impact on Oxidative Stress and Mitochondria
9. Selenoprotein T Protects Endothelial Cells against Lipopolysaccharide-Induced Activation and Apoptosis
10. Endothelial hyaluronan synthase 3 aggravates acute colitis in an experimental model of inflammatory bowel disease
11. Cellular functions of the dual-targeted catalytic subunit of telomerase, telomerase reverse transcriptase — Potential role in senescence and aging
12. The imbalanced redox status in senescent endothelial cells is due to dysregulated Thioredoxin-1 and NADPH oxidase 4
13. Extra-Nuclear Functions of the Transcription Factor Grainyhead-Like 3 in the Endothelium—Interaction with Endothelial Nitric Oxide Synthase
14. The Aryl Hydrocarbon Receptor (AhR) in the Aging Process: Another Puzzling Role for This Highly Conserved Transcription Factor
15. Loss of gap junctional intercellular communication in rat lung epithelial cells exposed to carbon or silica-based nanoparticles
16. HuR regulates gap junctional intercellular communication by controlling β-catenin levels and adherens junction integrity#
17. Ultraviolet A induced modulation of gap junctional intercellular communication by P38 MAPK activation in human keratinocytes
18. Menadione Diminishes Gap Junctional Intercellular Communication: Role of Adherens Junctions: 103
19. INFLUENCE OF UVB IRRADIATION ON GAP JUNCTIONAL INTERCELLULAR COMMUNICATION IN NORMAL HUMAN KERATINOCYTES: 430
20. Modulation of homologous gap junctional intercellular communication of human dermal fibroblasts via a paracrine factor(s) generated by squamous tumor cells
21. High Concentration of Low-Density Lipoprotein Results in Disturbances in Mitochondrial Transcription and Functionality in Endothelial Cells
22. Induction of a senescent like phenotype and loss of gap junctional intercellular communication by carbon nanoparticle exposure of lung epithelial cells
23. Molekularer Wirkmechanismus von Koffein entschlüsselt
24. CDKN1B/p27 is localized in mitochondria and improves respiration-dependent processes in the cardiovascular system—New mode of action for caffeine
25. Non-Canonical Activation of the Epidermal Growth Factor Receptor by Carbon Nanoparticles
26. The Anti-Apoptotic Properties of APEX1 in the Endothelium Require the First 20 Amino Acids and Converge on Thioredoxin-1
27. Role of Telomerase in the Cardiovascular System
28. The aryl hydrocarbon receptor promotes aging phenotypes across species
29. Loss of gap junctional intercellular communication in rat lung epithelial cells exposed to carbon or silica-based nanoparticles
30. Signalling-Dependent Adverse Health Effects of Carbon Nanoparticles Are Prevented by the Compatible Solute Mannosylglycerate (Firoin) In Vitro and In Vivo
31. Abstract 335: Carbon Nanoparticles Cause Loss of Gap Junctional Intercellular Communication and Nuclear Translocation of ß-Catenin in Endothelial Cells
32. Untersuchungen zum Einfluss von Retinoiden, Flavonoiden und Menadion auf interzelluläre Kommunikation über Gap Junctions
33. Abstract 234: Defining Domains In The Transcription Factor Grainyhead-like 3 Required For Protective Functions In The Endothelium
34. Hydrogen Peroxide Sensing and Signaling by Protein Kinases in the Cardiovascular System
35. Abstract 352: Unhealthy Diet and Air Pollution Induce Cardiovascular Dysfunction
36. Abstract P324: Unhealthy Diet and Air Pollution Compromise Human Cardiovascular Cell Functions: A First Mechanistic Analysis
37. Role of HuR and p38MAPK in Ultraviolet B-induced Post-transcriptional Regulation of COX-2 Expression in the Human Keratinocyte Cell Line HaCaT
38. Loss of gap junctional intercellular communication in rat lung epithelial cells exposed to quartz particles
39. Ultrafine but not fine carbon particles cause loss of gap junctional intercellular communication in rat lung epithelial cells
40. Epidermal growth factor- and stress-induced loss of gap junctional communication is mediated by ERK-1/ERK-2 but not ERK-5 in rat liver epithelial cells
41. CD133+ hepatic stellate cells are progenitor cells
42. (−)-Epicatechin effects in rat liver epithelial cells: stimulation of gap junctional communication and counteraction of its loss due to the tumor promoter 12-O-tetradecanoylphorbol-13-acetate
43. Role of HuR and p38MAPK in Ultraviolet B-induced Post-transcriptional Regulation of COX-2 Expression in the Human Keratinocyte Cell Line HaCaT.
44. CD133+ hepatic stellate cells are progenitor cells
45. HuR regulates gap junctional intercellular communication by controlling beta-catenin levels and adherens junction integrity.
46. 2-Methyl-1,4-naphthoquinone, vitamin K(3), decreases gap-junctional intercellular communication via activation of the epidermal growth factor receptor/extracellular signal-regulated kinase cascade.
47. (-)-Epicatechin effects in rat liver epithelial cells: stimulation of gap junctional communication and counteraction of its loss due to the tumor promoter 12-O-tetradecanoylphorbol-13-acetate.
48. Non-antioxidant properties of carotenoids.
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