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1. Gladiolin produced by pathogenic Burkholderia synergizes with amphotericin B through membrane lipid rearrangements

2. The C-terminal protein interaction domain of the chromatin reader Yaf9 is critical for pathogenesis of Candida albicans

3. Quantitative live-cell imaging of Candida albicans escape from immune phagocytes

4. The short-chain fatty acid crotonate reduces invasive growth and immune escape of Candida albicans by regulating hyphal gene expression

5. Candida auris uses metabolic strategies to escape and kill macrophages while avoiding robust activation of the NLRP3 inflammasome response

6. The proteasome regulator Rpn4 controls antifungal drug tolerance by coupling protein homeostasis with metabolic responses to drug stress.

7. Disruption of Iron Homeostasis and Mitochondrial Metabolism Are Promising Targets to Inhibit Candida auris

8. The RSC (Remodels the Structure of Chromatin) complex of Candida albicans shows compositional divergence with distinct roles in regulating pathogenic traits.

9. Natural Variation in Clinical Isolates of Candida albicans Modulates Neutrophil Responses

10. Metabolic competition between host and pathogen dictates inflammasome responses to fungal infection.

11. The YEATS Domain Histone Crotonylation Readers Control Virulence-Related Biology of a Major Human Pathogen

12. A Metabolic Checkpoint for the Yeast-to-Hyphae Developmental Switch Regulated by Endogenous Nitric Oxide Signaling

13. Candida aurisevades innate immunity by using metabolic strategies to escape and kill macrophages while avoiding antimicrobial inflammation

14. The Mitochondrial GTPase Gem1 Contributes to the Cell Wall Stress Response and Invasive Growth of Candida albicans

15. The Antifungal Plant Defensin HsAFP1 Is a Phosphatidic Acid-Interacting Peptide Inducing Membrane Permeabilization

17. The Endoplasmic Reticulum-Mitochondrion Tether ERMES Orchestrates Fungal Immune Evasion, Illuminating Inflammasome Responses to Hyphal Signals

19. Integration of Posttranscriptional Gene Networks into Metabolic Adaptation and Biofilm Maturation in Candida albicans.

20. The escape of Candida albicans from macrophages is enabled by the fungal toxin candidalysin and two host cell death pathways

21. Characterization of Key Bio–Nano Interactions between Organosilica Nanoparticles and Candida albicans

23. The Pathogen Candida albicans Hijacks Pyroptosis for Escape from Macrophages

24. The short chain fatty acid butyrate prevents intracellular replication of Legionella by regulating cysteine levels in macrophages

25. Preparation of Mitochondria from Candida albicans

26. Candida albicans Mitochondrial Protein Import Assay

27. The RSC (Remodels the Structure of Chromatin) complex of Candida albicans shows compositional divergence with distinct roles in regulating pathogenic traits

28. Metabolic competition between host and pathogen dictates inflammasome responses to fungal infection

29. Immunometabolism in fungal infections: the need to eat to compete

30. Mdivi-1 and mitochondrial fission: recent insights from fungal pathogens

31. Transcriptional profiling of a yeast colony provides new insight into the heterogeneity of multicellular fungal communities.

32. The functions of Mediator in Candida albicans support a role in shaping species-specific gene expression.

33. The yeast PUF protein Puf5 has Pop2-independent roles in response to DNA replication stress.

34. Mitochondrial Control of Fungal Cell Walls: Models and Relevance in Fungal Pathogens

35. The YEATS Domain Histone Crotonylation Readers Control Virulence-Related Biology of a Major Human Pathogen

36. Post‐transcriptional gene regulation in the biology and virulence of Candida albicans

37. Central metabolic interactions of immune cells and microbes: prospects for defeating infections

38. Mitochondrial Control of Fungal Cell Walls: Models and Relevance in Fungal Pathogens

39. PAT-seq: a method to study the integration of 3′-UTR dynamics with gene expression in the eukaryotic transcriptome

40. Surface coatings with covalently attached caspofungin are effective in eliminating fungal pathogens

41. Glucose Homeostasis Is Important for Immune Cell Viability during Candida Challenge and Host Survival of Systemic Fungal Infection

42. Bovine pancreatic trypsin inhibitor is a new antifungal peptide that inhibits cellular magnesium uptake

43. Phospholipase C of Cryptococcus neoformans Regulates Homeostasis and Virulence by Providing Inositol Trisphosphate as a Substrate for Arg1 Kinase

44. ePAT: A simple method to tag adenylated RNA to measure poly(A)-tail length and other 3′ RACE applications

45. Mitochondrial Sorting and Assembly Machinery Subunit Sam37 in Candida albicans: Insight into the Roles of Mitochondria in Fitness, Cell Wall Integrity, and Virulence

46. Mitochondrial Biogenesis: Cell-Cycle-Dependent Investment in Making Mitochondria

47. Mitochondria and Fungal Pathogenesis: Drug Tolerance, Virulence, and Potential for Antifungal Therapy

48. PUF proteins: repression, activation and mRNA localization

49. Cell wall integrity is linked to mitochondria and phospholipid homeostasis in Candida albicans through the activity of the post-transcriptional regulator Ccr4-Pop2

50. Anti-infective surface coatings: design and therapeutic promise against device-associated infections

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