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2. Neurobiology of eating behavior, nutrition and health

Author

Stover, P., Field, M., Andermann, M., Bailey, R., Batterham, R., Cauffman, E., Frühbeck, G., Iversen, P., Starke-Reed, P., Sternson, S., Vinoy, S., Witte, A., https://orcid.org/0000-0001-9054-6688, Zuker, C., and Angelin, B.

Abstract

Eating behavior and food-related decision making are among the most complex of the motivated behaviors, and understanding the neurobiology of eating behavior, and its developmental dynamics, is critical to advancing the nutritional sciences and public health. Recent advances from both human and animal studies are revealing that individual capacity to make health-promoting food decisions varies based on biological and physiological variation in the signaling pathways that regulate the homeostatic, hedonic, and executive functions; past developmental exposures and current life-stage; the food environment; and complications of chronic disease that reinforce the obese state. Eating rate drives increased calorie intake, and represents an important opportunity to lower rates of food consumption and energy intake through product reformulation. Understanding human eating behaviors and nutrition in the context of neuroscience can strengthen the evidence base from which dietary guidelines are derived and cam inform policies, practices, and educational programs in a way that increases the likelihood they are adopted and effective for reducing rates of obesity and other diet-related chronic disease.

Published
2023

37. Renewal of the Journal

Author

HOLM, G., ANGELIN, B., FAGRELL, B., FAIRE, U. DE, and LJUNGHALL, S.

Published
2001

40. Molecular aspects of human lipid metabolism

Author

Angelin, B. and Rudling, M.

Subjects
Cholesterol -- Physiological aspects, Lipid metabolism -- Research, Atherosclerosis -- Risk factors
Abstract

The discovery of metabolic disorders associated with atherosclerosis has led to the further study of human lipid metabolism at the molecular and genetic level. Genetic disorders such as hypobetalipoproteinaemia, familial defective apo B-100, type III hyperlipoproteinemia and familial hypercholesterolaemia were discussed from the molecular perspective and the possibilities for molecular regulation of cholestrol metabolism were explored. The physiological correlation with molecular principles is essential to understanding lipid metabolism.

Published
1992

43. Effect of ursodeoxycholic acid treatment on ileal absorption of bile acids in man as determined by the SeHCAT test

Author

Eusufzai, S., Ericsson, S., Cederlund, T., Einarsson, K., and Angelin, B.

Subjects
Bile acids -- Physiological aspects, Cholesterol -- Physiological aspects, Ursodiol -- Health aspects, Health
Abstract

Bile acids are produced by the liver and released into the small intestine in response to a meal; they aid in the efficient absorption of dietary fat and cholesterol. The bile acids are reabsorbed at a more distal point in the intestine, after they have accomplished their function, and return to the liver via the bloodstream where they await reentry into the cycle. The release and reabsorption of bile acids are part of a tightly controlled feedback cycle in which the rate of bile acid biosynthesis is regulated by both the demand for these compounds by the digestive tract and the rate at which they return to the liver. Ursodeoxycholic acid, a derivative of the major bile acids (and itself a minor constituent of bile) has been used in the treatment of a variety of disorders, including gall stones, primary biliary cirrhosis, and sclerosing cholangitis. The extent to which beneficial effects of ursodeoxycholic acid treatment may be related to its influence on intestinal absorption of bile acids is not known. To further investigate this issue, a newly available tracer technique involving monitoring the absorption of radioactively labelled selenium homocholic acid taurine (an easily measurable analog of naturally occurring bile acids) was used to quantify the effects of ursodeoxycholic acid on bile acid absorption. The radiolabelled acid was given orally, and bile acid absorption inferred from the rate of disappearance of radioactivity over a seven-day period. This was done before and after three weeks of treatment with ursodeoxycholic acid. Following treatment, there was a significant decrease in intestinal absorption of bile acids and blood cholesterol levels; blood levels of ursodeoxycholic acid increased. These results suggest that the beneficial effects of ursodeoxycholic acid treatment result, at least in part, from its suppressive actions on the absorption of bile acids. (Consumer Summary produced by Reliance Medical Information, Inc.)

Published
1991

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