259 results on '"Ankarcrona, Maria"'
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2. Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism
3. Guidelines on experimental methods to assess mitochondrial dysfunction in cellular models of neurodegenerative diseases
4. The Sigma-1 Receptor Mediates Pridopidine Rescue of Mitochondrial Function in Huntington Disease Models
5. Simultaneous detection of membrane contact dynamics and associated Ca2+signals by reversible chemogenetic reporters
6. Center for Alzheimer Research at Karolinska Institutet, Sweden
7. Neuronal cell-based high-throughput screen for enhancers of mitochondrial function reveals luteolin as a modulator of mitochondria-endoplasmic reticulum coupling
8. MIEF1/2 orchestrate mitochondrial dynamics through direct engagement with both the fission and fusion machineries
9. Early mitochondrial dysfunction proceeds neuroinflammation, synaptic alteration, and autophagy impairment in hippocampus ofAppknock-in Alzheimer mouse models
10. Additional file 5 of Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism
11. Additional file 3 of Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism
12. Additional file 8 of Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism
13. Additional file 1 of Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism
14. Additional file 2 of Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism
15. Additional file 7 of Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism
16. Alterations in mitochondria-endoplasmic reticulum connectivity in human brain biopsies from idiopathic normal pressure hydrocephalus patients
17. Mitochondrial Alterations in Neurons Derived from the Murine AppNL-F Knock-In Model of Alzheimer’s Disease
18. Aβ in Mitochondria—One Piece in the Alzheimer’s Disease Puzzle
19. The Potential of Small Molecules to Modulate the Mitochondria–Endoplasmic Reticulum Interplay in Alzheimer’s Disease
20. Isolation of Mitochondria-Associated Membranes (MAM) from Mouse Brain Tissue
21. The Role of Presenilins in Aβ-Induced Cell Death in Alzheimer’s Disease
22. Modulation of the endoplasmic reticulum—mitochondria interface in Alzheimer's disease and related models
23. Mitochondria-Endoplasmic Reticulum Interplay Regulates Exo-Cytosis in Human Neuroblastoma Cells
24. The Amyloid β-Peptide Is Imported into Mitochondria via the TOM Import Machinery and Localized to Mitochondrial Cristae
25. The Molecular Assembly State of Drp1 Controls its Association With the Mitochondrial Recruitment Receptors Mff and MIEF1/2
26. Association of Omi/HtrA2 with γ-secretase in mitochondria
27. I13 Pridopidine restores mitochondrial function and decreases er stress which is mediated through the S1R
28. Mitochondrial hypermetabolism precedes impaired autophagy and synaptic disorganization in Appknock-in Alzheimer mouse models
29. Apoptosis and Necrosis: Two Distinct Events Induced, Respectively, by Mild and Intense Insults with N-Methyl-D-Aspartate or Nitric Oxide/Superoxide in Cortical Cell Cultures
30. Additional file 1 of Neuronal cell-based high-throughput screen for enhancers of mitochondrial function reveals luteolin as a modulator of mitochondria-endoplasmic reticulum coupling
31. Additional file 2 of MIEF1/2 orchestrate mitochondrial dynamics through direct engagement with both the fission and fusion machineries
32. Additional file 1 of MIEF1/2 orchestrate mitochondrial dynamics through direct engagement with both the fission and fusion machineries
33. Amyloid Β-Peptide Increases Mitochondria-Endoplasmic Reticulum Contact Altering Mitochondrial Function and Autophagosome Formation in Alzheimer’s Disease-Related Models
34. Mitochondria and Alzheimer’s disease: amyloid-β peptide uptake and degradation by the presequence protease, hPreP
35. The amyloid [beta]-peptide is imported into mitochondria via the TOM import machinery and localized to mitochondrial cristae
36. Aβ in Mitochondria—One Piece in the Alzheimer’s Disease Puzzle
37. γ-secretase complexes containing caspase-cleaved presenilin-1 increase intracellular Aβ42/Aβ40 ratio
38. Mitochondrial accumulation of APP and Aβ: significance for Alzheimer disease pathogenesis
39. Caspase cleaved presenilin-1 is part of active γ-secretase complexes
40. Chapter 17 Glutamate induced cell death: Apoptosis or necrosis?
41. Additional file 1: of Alterations in mitochondria-endoplasmic reticulum connectivity in human brain biopsies from idiopathic normal pressure hydrocephalus patients
42. Systems biology identifies preserved integrity but impaired metabolism of mitochondria due to a glycolytic defect in Alzheimer's disease neurons
43. Reduction of PINK1 or DJ-1 impair mitochondrial motility in neurites and alter ER-mitochondria contacts
44. The Bri2 and Bri3 BRICHOS Domains Interact Differently with Aβ42 and Alzheimer Amyloid Plaques
45. Mechanism of Peptide Binding and Cleavage by the Human Mitochondrial Peptidase Neurolysin
46. TOM70 Sustains Cell Bioenergetics by Promoting IP3R3-Mediated ER to Mitochondria Ca2+ Transfer
47. Beyond the critical point: An overview of excitotoxicity, calcium overload and the downstream consequences
48. Guidelines on experimental methods to assess mitochondrial dysfunction in cellular models of neurodegenerative diseases
49. THE EFFECT OF AMYLOID β-PEPTIDE ON ER-MITOCHONDRIA CONTACT IN ALZHEIMER’S DISEASE
50. Mitofusin-2 knockdown increases ER-mitochondria contact and decreases amyloid β-peptide production
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