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1. Erratum

2. Highly cytotoxic and neurotoxic acetogenins of the Annonaceae: New putative biological targets of squamocin detected by activity-based protein profiling

3. AIFsh, a Novel Apoptosis-inducing Factor (AIF) Pro-apoptotic Isoform with Potential Pathological Relevance in Human Cancer

4. Cysteine protease inhibition prevents mitochondrial apoptosis-inducing factor (AIF) release

5. Expression of dengue ApoptoM sequence results in disruption of mitochondrial potential and caspase activation

6. AIF-mediated programmed necrosis: a highly regulated way to die

7. Drp1 mediates caspase-independent type III cell death in normal and leukemic cells

8. Expression of cortical and hippocampal apoptosis-inducing factor (AIF) in aging and Alzheimer's disease

9. Identification and characterization of AIFsh2, a mitochondrial apoptosis-inducing factor (AIF) isoform with NADH oxidase activity

10. Use of penetrating peptides interacting with PP1/PP2A proteins as a general approach for a drug phosphatase technology

11. La mitochondrie au coeur du suicide cellulaire

12. Semisynthesis and screening of a small library of pro-apoptotic squamocin analogues: selection and study of a benzoquinone hybrid with an improved biological profile

13. The contribution of apoptosis-inducing factor, caspase-activated DNase, and inhibitor of caspase-activated DNase to the nuclear phenotype and DNA degradation during apoptosis

14. Annonaceous acetogenins: the hydroxyl groups and THF rings are crucial structural elements for targeting the mitochondria, demonstration with the synthesis of fluorescent squamocin analogues

15. Immunogenicity of HIV Type 1 gp120 CD4 Binding Site Phage Mimotopes

16. Mitochondrial effectors in caspase-independent cell death

17. Apoptosis inversely correlates with rabies virus neurotropism

18. Mitochondrial dysfunction in CD47-mediated caspase-independent cell death: ROS production in the absence of cytochrome c and AIF release

19. A dual role of IFN-alpha in the balance between proliferation and death of human CD4(+) T lymphocytes during primary response

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