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1. Author Correction: Mutational signature-based identification of DNA repair deficient gastroesophageal adenocarcinomas for therapeutic targeting

Author

Prosz, Aurel, Sahgal, Pranshu, Huffman, Brandon M., Sztupinszki, Zsofia, Morris, Clare X., Chen, David, Börcsök, Judit, Diossy, Miklos, Tisza, Viktoria, Spisak, Sandor, Likasitwatanakul, Pornlada, Rusz, Orsolya, Csabai, Istvan, Cecchini, Michael, Baca, Yasmine, Elliott, Andrew, Enzinger, Peter, Singh, Harshabad, Ubellaker, Jessalyn, Lazaro, Jean-Bernard, Cleary, James M., Szallasi, Zoltan, and Sethi, Nilay S.

Published
2024
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2. Mutational signature-based identification of DNA repair deficient gastroesophageal adenocarcinomas for therapeutic targeting

Author

Prosz, Aurel, Sahgal, Pranshu, Huffman, Brandon M., Sztupinszki, Zsofia, Morris, Clare X., Chen, David, Börcsök, Judit, Diossy, Miklos, Tisza, Viktoria, Spisak, Sandor, Likasitwatanakul, Pornlada, Rusz, Orsolya, Csabai, Istvan, Cecchini, Michael, Baca, Yasmine, Elliott, Andrew, Enzinger, Peter, Singh, Harshabad, Ubellaker, Jessalyn, Lazaro, Jean-Bernard, Cleary, James M., Szallasi, Zoltan, and Sethi, Nilay S.

Published
2024
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4. Pan-tumor survey of ROS1 fusions detected by next-generation RNA and whole transcriptome sequencing.

Author

Zhang, Shannon, Baca, Yasmine, Xiu, Joanne, Nieva, Jorge, Vanderwalde, Ari, Swensen, Jeffrey, Spetzler, David, Korn, Wolfgang, Raez, Luis, Liu, Stephen, Ou, Sai-Hong, and Nagasaka, Misako

Subjects
Breast cancer, Non-small cell lung cancer, Pan-tumor analysis, Receptor tyrosine kinase fusions, c-ROS1, Humans, Female, Carcinoma, Non-Small-Cell Lung, Lung Neoplasms, Protein-Tyrosine Kinases, Retrospective Studies, Exome Sequencing, Proto-Oncogene Proteins, Breast Neoplasms
Abstract

BACKGROUND: Two ROS1 tyrosine kinase inhibitors have been approved for ROS1 fusion positive (ROS1+) non-small cell lung cancer (NSCLC) tumors. We performed a pan-tumor analysis of the incidence of ROS1 fusions to assess if more ROS1+ patients who could benefit from ROS1 TKIs could be identified. METHODS: A retrospective analysis of ROS1 positive solid malignancies identified by targeted RNA sequencing and whole transcriptome sequencing of clinical tumor samples performed at Caris Life Science (Phoenix, AZ). RESULTS: A total of 259 ROS1+ solid malignancies were identified from approximately 175,350 tumors that underwent next-generation sequencing (12% from targeted RNA sequencing [Archer]; 88% from whole transcriptome sequencing). ROS1+ NSCLC constituted 78.8% of the ROS1+ solid malignancies, follow by glioblastoma (GBM) (6.9%), and breast cancer (2.7%). The frequency of ROS1 fusion was approximately 0.47% among NSCLC, 0.29% for GBM, 0.04% of breast cancer. The mean tumor mutation burden for all ROS1+ tumors was 4.8 mutations/megabase. The distribution of PD-L1 (22C3) expression among all ROS1+ malignancies were 0% (18.6%), 1%-49% (29.4%), and ≥ 50% (60.3%) [for NSCLC: 0% (17.8%); 1-49% (27.7%); ≥ 50% (53.9%). The most common genetic co-alterations of ROS1+ NSCLC were TP53 (29.1%), SETD2 (7.3%), ARIAD1A (6.3%), and U2AF1 (5.6%). CONCLUSIONS: ROS1+ NSCLC tumors constituted the majority of ROS1+ solid malignancies with four major fusion partners. Given that > 20% of ROS1+ solid tumors may benefit from ROS1 TKIs treatment, comprehensive genomic profiling should be performed on all solid tumors.

Published
2023

5. Pan-tumor survey of RET fusions as detected by next-generation RNA sequencing identified RET fusion positive colorectal carcinoma as a unique molecular subset.

Author

Brazel, Danielle, Baca, Yasmine, Xiu, Joanne, Al-Hallak, Mohammed, Kim, Chul, Nieva, Jorge, Swensen, Jeffrey, Spetzler, David, Korn, Wolfgang, Socinski, Mark, Raez, Luis, Halmos, Balazs, Ou, Sai-Hong, and Nagasaka, Misako

Subjects
Next-generation sequencing, Pan-tumor survey, Pralsetinib, RET fusion, RNA sequencing, Selective RET inhibitors, Selpercatinib
Abstract

BACKGROUND: RET fusions are driver alterations in cancer and are most commonly found in non-small cell lung cancer and well-differentiated thyroid cancer. However, RET fusion have been reported in other solid tumors. MATERIAL AND METHODS: A retrospective analysis of RET+ solid malignancies identified by targeted RNA sequencing and whole transcriptome sequencing of clinical tumor samples performed at Caris Life Science (Phoenix, AZ). RESULTS: As of March 22, 2022, a total of 378 RET+ solid malignancies were identified in 15 different tumor types and carcinoma of unknown primary (CUP) that underwent next-generation RNA sequencing. RET+ NSCLC and RET+ thyroid cancer constituted 66.9% and 11.1% of the RET+ solid malignancies, respectively. RET+ colorectal adenocarcinoma and RET+ breast adenocarcinoma constituted 10.1% and 2.6%, respectively. The estimated frequency of RET fusions within specific tumor types were NSCLC 0.7%, thyroid cancer 3.1%, colorectal cancer 0.2% and breast cancer 0.1%. KIF5B (46.8%) was the most common fusion partner followed by CCDC6 (28.3%) and NCOA4 (13.8%) in RET+ solid tumors. KIF5B-RET was the dominant fusion variant in RET+ NSCLC, NCOA4-RET was the dominant variant in RET+ colorectal carcinoma, and CCDC6-RET was the dominant variant in thyroid cancer. The most common single gene alterations in RET+ tumors were TP53 (34.8%), RASA1 (14.3%) and ARIAD1A (11.6%). RET+ CRC had a high median TMB of 20.0 and were commonly MSI-H. CONCLUSIONS: RET fusions were identified in multiple tumor types. With a higher median TMB and commonly MSI-H, RET fusion positive CRC may be a unique molecular subset of CRC.

Published
2023

8. Molecular characterization of Kita-Kyushu lung cancer antigen (KK-LC-1) expressing carcinomas.

Author

Hsu, Robert, Baca, Yasmine, Xiu, Joanne, Wang, Rongfu, Bodor, J, Kim, Chul, Khan, Hina, Mamdani, Hirva, Nagasaka, Misako, Puri, Sonam, Liu, Stephen, Korn, W, and Nieva, Jorge

Subjects
biomarkers for immunotherapy, cancer testis antigen, diagnostic biomarkers, lung cancer, tumor microenvironment
Abstract

Cancer/testis antigens (CTAs) are strongly expressed in some solid tumors but minimally expressed in normal tissue, making them appealing therapeutic targets. KK-LC-1 (CXorf61) has cytoplasmic expression in gastric, breast, and lung cancer. We characterized the molecular subtypes of non-small cell lung cancer (NSCLC) expressing KK-LC-1 to inform rational clinical trials of T-cell receptor therapy (TCR-T) targeting KK-LC-1. 9790 NSCLC tumors that underwent whole transcriptome sequencing (Illumina NovaSeq) and NextGen DNA sequencing (NextSeq, 592 Genes and NovaSEQ, WES) at Caris Life Sciences (Phoenix, AZ) were analyzed. Tumors were split into quartiles based on KK-LC-1 expression and pathological and molecular differences were investigated. Adenocarcinoma had significantly higher KK-LC-1 expression than squamous cell carcinoma (median, 3.25 vs. 1.17 transcripts per million (TPM), p < 0.0001). Tumors with the highest quartile of KK-LC-1 expression had a greater proportion of tumors with high tumor mutation burden (TMB) (≥10 mutations per megabase; 44% vs. 28% in Q1, p < 0.001). Increased KK-LC-1 expression was associated with increased M1 macrophage abundance. Higher levels of KK-LC-1 expression were seen in pan-wild type and KRAS mutated tumors and associated with high TMB. TCR-T therapy directed against KK-LC-1 should be considered in patients whose clinical features reflect these characteristics.

Published
2021

9. Characterization of KRAS Mutation Subtypes in Non-small Cell Lung Cancer.

Author

Judd, Julia, Abdel Karim, Nagla, Khan, Hina, Naqash, Abdul, Baca, Yasmine, Xiu, Joanne, VanderWalde, Ari, Mamdani, Hirva, Raez, Luis, Nagasaka, Misako, Pai, Sachin, Socinski, Mark, Nieva, Jorge, Kim, Chul, Wozniak, Antoinette, Ikpeazu, Chukwuemeka, de Lima Lopes, Gilberto, Spira, Alexander, Korn, W, Kim, Edward, Liu, Stephen, and Borghaei, Hossein

Subjects
Carcinoma, Non-Small-Cell Lung, High-Throughput Nucleotide Sequencing, Humans, Lung Neoplasms, Microsatellite Instability, Mutation, Prognosis, Proto-Oncogene Proteins p21(ras)
Abstract

KRAS is the most commonly mutated oncogene in NSCLC and development of direct KRAS inhibitors has renewed interest in this molecular variant. Different KRAS mutations may represent a unique biologic context with different prognostic and therapeutic impact. We sought to characterize genomic landscapes of advanced, KRAS-mutated non-small cell lung cancer (NSCLC) in a large national cohort to help guide future therapeutic development.Molecular profiles of 17,095 NSCLC specimens were obtained using DNA next-generation sequencing of 592 genes (Caris Life Sciences) and classified on the basis of presence and subtype of KRAS mutations. Co-occurring genomic alterations, tumor mutational burden (TMB), and PD-L1 expression [22C3, tumor proportion score (TPS) score] were analyzed by KRAS mutation type.Across the cohort, 4,706 (27.5%) samples harbored a KRAS mutation. The most common subtype was G12C (40%), followed by G12V (19%) and G12D (15%). The prevalence of KRAS mutations was 37.2% among adenocarcinomas and 4.4% in squamous cell carcinomas. Rates of high TMB (≥10 mutations/Mb) and PD-L1 expression varied across KRAS mutation subtypes. KRAS G12C was the most likely to be PD-L1 positive (65.5% TPS ≥ 1%) and PD-L1 high (41.3% TPS ≥ 50%). STK11 was mutated in 8.6% of KRAS wild-type NSCLC but more frequent in KRAS-mutant NSCLC, with the highest rate in G13 (36.2%). TP53 mutations were more frequent in KRAS wild-type NSCLC (73.6%).KRAS mutation subtypes have different co-occurring mutations and a distinct genomic landscape. The clinical relevance of these differences in the context of specific therapeutic interventions warrants investigation.

Published
2021

10. Predictive value of CDC37 gene expression for targeted therapy in metastatic colorectal cancer

Author

Arai, Hiroyuki, Yang, Yan, Baca, Yasmine, Millstein, Joshua, Denda, Tadamichi, Ou, Fang-Shu, Innocenti, Federico, Takeda, Hiroyuki, Kubota, Yohei, Doi, Ayako, Horie, Yoshiki, Umemoto, Kumiko, Izawa, Naoki, Wang, Jingyuan, Battaglin, Francesca, Jayachandran, Priya, Algaze, Sandra, Soni, Shivani, Zhang, Wu, Goldberg, Richard M., Hall, Michael J., Scott, Aaron James, Hwang, Jimmy J., Lou, Emil, Weinberg, Benjamin A., Marshall, John, Goel, Sanjay, Xiu, Joanne, Michael Korn, W., Venook, Alan P., Sunakawa, Yu, and Lenz, Heinz-Josef

Published
2024
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13. PBRM1 mutations might render a subtype of biliary tract cancers sensitive to drugs targeting the DNA damage repair system

Author

Zimmer, Kai, Kocher, Florian, Untergasser, Gerold, Kircher, Brigitte, Amann, Arno, Baca, Yasmine, Xiu, Joanne, Korn, W. Micheal, Berger, Martin D., Lenz, Heinz-Josef, Puccini, Alberto, Fontana, Elisa, Shields, Anthony F., Marshall, John L., Hall, Michael, El-Deiry, Wafik S., Hsiehchen, David, Macarulla, Teresa, Tabernero, Josep, Pichler, Renate, Khushman, Moh’d, Manne, Upender, Lou, Emil, Wolf, Dominik, Sokolova, Viktorija, Schnaiter, Simon, Zeimet, Alain G., Gulhati, Pat, Widmann, Gerlig, and Seeber, Andreas

Published
2023
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14. Impact of XPO1 mutations on survival outcomes in metastatic non-small cell lung cancer (NSCLC).

Author

Nagasaka, Misako, Asad, Mohammad, Al Hallak, Mohammed, Uddin, Md, Sukari, Ammar, Baca, Yasmine, Xiu, Joanne, Magee, Dan, Mamdani, Hirva, Uprety, Dipesh, Kim, Chul, Xia, Bing, Liu, Stephen, Nieva, Jorge, Lopes, Gilberto, Bepler, Gerold, Borghaei, Hossein, Demeure, Michael, Raez, Luis, Ma, Patrick, Puri, Sonam, Korn, W, and Azmi, Asfar

Subjects
NGS, NSCLC, Nuclear Protein Transport, Tumor Mutational Burden, XPO1, Carcinoma, Non-Small-Cell Lung, Humans, Karyopherins, Lung Neoplasms, Mutation, Receptors, Cytoplasmic and Nuclear
Abstract

BACKGROUND: Nuclear protein transport is essential in guiding the traffic of important proteins and RNAs between the nucleus and cytoplasm. Export of proteins from the nucleus is mostly regulated by Exportin 1 (XPO1). In cancer, XPO1 is almost universally hyperactive and can promote the export of important tumor suppressors to the cytoplasm. Currently, there are no studies evaluating XPO1 amplifications and mutations in NSCLC and the impact on outcomes. METHODS: Tumor samples were analyzed using next-generation sequencing (NGS) (NextSeq, 592 Genes), immunohistochemistry (IHC), and whole transcriptome sequencing (WTS, NovaSeq) (Caris Life Sciences, Phoenix, AZ). Survival was extracted from insurance claims data and calculated from time of tissue collection to last contact using Kaplan-Meier estimate. RESULTS: Among 18,218 NSCLC tumors sequenced, 26 harbored XPO1 mutations and 24 had amplifications. XPO1 mutant tumors were more likely to have high TMB (79% vs. 52%, p = 0.007) and less likely to have high PD-L1 (32% vs. 68%, p = 0.03). KRAS co-mutations were seen in 19% (n = 5) and EGFR co-mutations were rare (n = 2). Among the 17,449 NSCLC tumors with clinical data, there were 24 XPO1 mutant. Comparison of survival between XPO1 mutant and WT showed a negative association with a hazard ratio (HR) of 1.932 (95% CI: 1.144-3.264 p = 0.012). XPO1 amplification was not associated with survival. CONCLUSIONS: XPO1 pathogenic mutations were associated with a poor survival in NSCLC. Although XPO1 mutations are rare in NSCLC, further studies to assess its associations with treatment responses are warranted.

Published
2021

15. Influence of TP53 Comutation on the Tumor Immune Microenvironment and Clinical Outcomes With Immune Checkpoint Inhibitors in STK11-Mutant Non–Small-Cell Lung Cancer

Author

Naqash, Abdul Rafeh, Floudas, Charalampos S., Aber, Etan, Maoz, Asaf, Nassar, Amin H., Adib, Elio, Choucair, Khalil, Xiu, Joanne, Baca, Yasmine, Ricciuti, Biagio, Alessi, Joao V., Awad, Mark M., Kim, Chul, Judd, Julia, Raez, Luis E., Lopes, Gilberto, Nieva, Jorge J., Borghaei, Hossein, Takebe, Naoko, Ma, Patrick C., Halmos, Balazs, Kwiatkowski, David J., Liu, Stephen V., and Mamdani, Hirva

Published
2024
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19. The Effects of HER2 Alterations in EGFR Mutant Non-small Cell Lung Cancer

Author

Nagasaka, Misako, Singh, Vijendra, Baca, Yasmine, Sukari, Ammar, Kim, Chul, Mamdani, Hirva, Spira, Alexander I., Uprety, Dipesh, Bepler, Gerold, Kim, Edward S., Raez, Luis E., Pai, Sachin Gopalkrishna, Ikpeazu, Chukwuemeka, Oberley, Matthew, Feldman, Rebecca, Xiu, Joanne, Korn, W. Michael, Wozniak, Antoinette J., Borghaei, Hossein, and Liu, Stephen V.

Published
2022
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20. Uncovering actionable genetic alterations and immune predictive biomarkers for anal squamous cell carcinomas in the era of immunotherapy: PD-L1 and beyond.

Author

Sharma, Gaurav, primary, Baca, Yasmine, additional, Xiu, Joanne, additional, Farrell, Alex Patrick, additional, Sledge, George W., additional, Saeed, Anwaar, additional, Shields, Anthony F., additional, Prakash, Ajay, additional, Goel, Sanjay, additional, Weinberg, Benjamin Adam, additional, and Lou, Emil, additional

Published
2024
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22. Multiomic Characterization Reveals a Distinct Molecular Landscape in Young-Onset Pancreatic Cancer

Author

Ogobuiro, Ifeanyichukwu, Baca, Yasmine, Ribeiro, Jennifer R., Walker, Phillip, Wilson, Gregory C., Gulhati, Prateek, Marshall, John L., Shroff, Rachna T., Spetzler, David, Oberley, Matthew J., Abbott, Daniel E., Kim, Hong Jin, Kooby, David A., Maithel, Shishir K., Ahmad, Syed A., Merchant, Nipun B., Xiu, Joanne, Hosein, Peter J., and Datta, Jashodeep

Published
2023
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23. Large-scale analysis of KMT2 mutations defines a distinctive molecular subset with treatment implication in gastric cancer

Author

Wang, Jingyuan, Xiu, Joanne, Baca, Yasmine, Battaglin, Francesca, Arai, Hiroyuki, Kawanishi, Natsuko, Soni, Shivani, Zhang, Wu, Millstein, Joshua, Salhia, Bodour, Goldberg, Richard M., Philip, Philip A., Seeber, Andreas, Hwang, Jimmy J., Shields, Anthony F., Marshall, John L., Astsaturov, Igor, Craig Lockhart, A., Gatalica, Zoran, Michael Korn, W., and Lenz, Heinz-Josef

Published
2021
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24. Molecular profile of BRCA-mutated biliary tract cancers

Author

Spizzo, Gilbert, Puccini, Alberto, Xiu, Joanne, Goldberg, Richard M., Grothey, Axel, Shields, Anthony F., Arora, Sukeshi Patel, Khushman, Moh’d, Salem, Mohamed E., Battaglin, Francesca, Baca, Yasmine, El-Deiry, Wafik S., Philip, Philip A., Nassem, Madiha, Hall, Michael, Marshall, John L., Kocher, Florian, Amann, Arno, Wolf, Dominik, Korn, W. Michael, Lenz, Heinz-Josef, and Seeber, Andreas

Published
2020
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25. Molecular differences with therapeutic implications in early-onset compared to average-onset cholangiocarcinoma.

Author

Jayakrishnan, Thejus, primary, Baca, Yasmine, additional, Xiu, Joanne, additional, Patel, Mehrie, additional, Weinberg, Benjamin Adam, additional, Lou, Emil, additional, Datta, Jashodeep, additional, Khushman, Moh'd M., additional, Gulhati, Pat, additional, Goel, Sanjay, additional, Biachi de Castria, Tiago, additional, Florou, Vaia, additional, Nair, Kanika G., additional, Kamath, Suneel Deepak, additional, and Khorana, Alok A., additional

Published
2024
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26. Characterization of the cachexia pathway in pancreatic ductal adenocarcinoma.

Author

Ashouri, Karam, primary, Baca, Yasmine, additional, Xiu, Joanne, additional, Soni, Shivani, additional, Algaze, Sandra, additional, Jayachandran, Priya, additional, Wong, Alexandra, additional, Yang, Yan, additional, Torres-Gonzalez, Lesly, additional, Lo, Jae Ho, additional, Zhang, Wu, additional, Khushman, Moh'd M., additional, Weinberg, Benjamin Adam, additional, Goel, Sanjay, additional, Lou, Emil, additional, Battaglin, Francesca, additional, and Lenz, Heinz-Josef, additional

Published
2024
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27. LMTK3 gene expression and the molecular landscape of colorectal cancer (CRC).

Author

Torres-Gonzalez, Lesly, primary, Battaglin, Francesca, additional, Soni, Shivani, additional, Baca, Yasmine, additional, Xiu, Joanne, additional, Walker, Phillip, additional, Lo, Jae Ho, additional, Algaze, Sandra, additional, Jayachandran, Priya, additional, Mittal, Pooja, additional, Zhang, Wu, additional, Weinberg, Benjamin Adam, additional, Lou, Emil, additional, Shields, Anthony F., additional, Goldberg, Richard M., additional, Marshall, John, additional, Goel, Sanjay, additional, Stebbing, Justin, additional, Giamas, Georgios, additional, and Lenz, Heinz-Josef, additional

Published
2024
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29. Abstract 4253: Pan-cancer analysis of XPO1 R749Q mutations across 217,570 patients reveals association with high tumor mutational burden and therapy resistance

Author

Totiger, Tulasigeri M., primary, Baca, Yasmine, additional, Chiraphapphaiboon, Wannasiri, additional, Chaudhry, Sana, additional, Montoya, Skye, additional, Chojnacka, Monika, additional, Gaidosh, Gabriel, additional, Afaghani, Jumana, additional, Affer, Maurizio, additional, Armstrong, Christopher D., additional, Notti, Ryan, additional, Zabroski, Jenna, additional, Jahn, Jacob, additional, Nawaratne, Vindhya, additional, Verdun, Ramiro, additional, Nguyen, Hai Dang, additional, Nabhan, Chadi, additional, Herzog, Thomas J., additional, Walker, Phil, additional, Elliott, Andrew, additional, Lou, Emil, additional, El-Deiry, Wafik S., additional, Mitchell, Edith, additional, Rodriguez, Jose Antonio, additional, and Taylor, Justin, additional

Published
2023
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30. Data from The Landscape of Glycogen Synthase Kinase-3 Beta Genomic Alterations in Cancer

Author

Borden, Brittany A., primary, Baca, Yasmine, primary, Xiu, Joanne, primary, Tavora, Fabio, primary, Winer, Ira, primary, Weinberg, Benjamin A., primary, Vanderwalde, Ari M., primary, Darabi, Sourat, primary, Korn, W. Michael, primary, Mazar, Andrew P., primary, Giles, Francis J., primary, Crawford, Lorin, primary, Safran, Howard, primary, El-Deiry, Wafik S., primary, and Carneiro, Benedito A., primary

Published
2023
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31. Supplemental File 1 from The Landscape of Glycogen Synthase Kinase-3 Beta Genomic Alterations in Cancer

Author

Borden, Brittany A., primary, Baca, Yasmine, primary, Xiu, Joanne, primary, Tavora, Fabio, primary, Winer, Ira, primary, Weinberg, Benjamin A., primary, Vanderwalde, Ari M., primary, Darabi, Sourat, primary, Korn, W. Michael, primary, Mazar, Andrew P., primary, Giles, Francis J., primary, Crawford, Lorin, primary, Safran, Howard, primary, El-Deiry, Wafik S., primary, and Carneiro, Benedito A., primary

Published
2023
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32. Supplemental Table 1 from The Landscape of Glycogen Synthase Kinase-3 Beta Genomic Alterations in Cancer

Author

Borden, Brittany A., primary, Baca, Yasmine, primary, Xiu, Joanne, primary, Tavora, Fabio, primary, Winer, Ira, primary, Weinberg, Benjamin A., primary, Vanderwalde, Ari M., primary, Darabi, Sourat, primary, Korn, W. Michael, primary, Mazar, Andrew P., primary, Giles, Francis J., primary, Crawford, Lorin, primary, Safran, Howard, primary, El-Deiry, Wafik S., primary, and Carneiro, Benedito A., primary

Published
2023
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33. Figure S2 from Molecular Characterization of Appendiceal Goblet Cell Carcinoid

Author

Arai, Hiroyuki, primary, Baca, Yasmine, primary, Battaglin, Francesca, primary, Kawanishi, Natsuko, primary, Wang, Jingyuan, primary, Soni, Shivani, primary, Zhang, Wu, primary, Millstein, Joshua, primary, Johnston, Curtis, primary, Goldberg, Richard M., primary, Philip, Philip A., primary, Seeber, Andreas, primary, Xiu, Joanne, primary, Hwang, Jimmy J., primary, Shields, Anthony F., primary, Marshall, John L., primary, Korn, W. Michael, primary, and Lenz, Heinz-Josef, primary

Published
2023
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34. Supplementary appendix from Molecular Characterization of Appendiceal Goblet Cell Carcinoid

Author

Arai, Hiroyuki, primary, Baca, Yasmine, primary, Battaglin, Francesca, primary, Kawanishi, Natsuko, primary, Wang, Jingyuan, primary, Soni, Shivani, primary, Zhang, Wu, primary, Millstein, Joshua, primary, Johnston, Curtis, primary, Goldberg, Richard M., primary, Philip, Philip A., primary, Seeber, Andreas, primary, Xiu, Joanne, primary, Hwang, Jimmy J., primary, Shields, Anthony F., primary, Marshall, John L., primary, Korn, W. Michael, primary, and Lenz, Heinz-Josef, primary

Published
2023
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35. Supplemental Figure 1 from Association of Homologous Recombination–DNA Damage Response Gene Mutations with Immune Biomarkers in Gastroesophageal Cancers

Author

Cerniglia, Michael, primary, Xiu, Joanne, primary, Grothey, Axel, primary, Pishvaian, Michael J., primary, Baca, Yasmine, primary, Hwang, Jimmy J., primary, Marshall, John L., primary, VanderWalde, Ari M., primary, Shields, Anthony F., primary, Lenz, Heinz-Josef, primary, Korn, W. Michael, primary, Salem, Mohamed, primary, Philip, Philip A., primary, Goldberg, Richard M., primary, Zeng, Jia, primary, and Kim, Sunnie S., primary

Published
2023
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36. Table S2 from Molecular Characterization of Appendiceal Goblet Cell Carcinoid

Author

Arai, Hiroyuki, primary, Baca, Yasmine, primary, Battaglin, Francesca, primary, Kawanishi, Natsuko, primary, Wang, Jingyuan, primary, Soni, Shivani, primary, Zhang, Wu, primary, Millstein, Joshua, primary, Johnston, Curtis, primary, Goldberg, Richard M., primary, Philip, Philip A., primary, Seeber, Andreas, primary, Xiu, Joanne, primary, Hwang, Jimmy J., primary, Shields, Anthony F., primary, Marshall, John L., primary, Korn, W. Michael, primary, and Lenz, Heinz-Josef, primary

Published
2023
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37. Supplementary Figure Legend from Association of Homologous Recombination–DNA Damage Response Gene Mutations with Immune Biomarkers in Gastroesophageal Cancers

Author

Cerniglia, Michael, primary, Xiu, Joanne, primary, Grothey, Axel, primary, Pishvaian, Michael J., primary, Baca, Yasmine, primary, Hwang, Jimmy J., primary, Marshall, John L., primary, VanderWalde, Ari M., primary, Shields, Anthony F., primary, Lenz, Heinz-Josef, primary, Korn, W. Michael, primary, Salem, Mohamed, primary, Philip, Philip A., primary, Goldberg, Richard M., primary, Zeng, Jia, primary, and Kim, Sunnie S., primary

Published
2023
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38. Supplemental Table 2 from The Landscape of Glycogen Synthase Kinase-3 Beta Genomic Alterations in Cancer

Author

Borden, Brittany A., primary, Baca, Yasmine, primary, Xiu, Joanne, primary, Tavora, Fabio, primary, Winer, Ira, primary, Weinberg, Benjamin A., primary, Vanderwalde, Ari M., primary, Darabi, Sourat, primary, Korn, W. Michael, primary, Mazar, Andrew P., primary, Giles, Francis J., primary, Crawford, Lorin, primary, Safran, Howard, primary, El-Deiry, Wafik S., primary, and Carneiro, Benedito A., primary

Published
2023
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39. Data from Association of Homologous Recombination–DNA Damage Response Gene Mutations with Immune Biomarkers in Gastroesophageal Cancers

Author

Cerniglia, Michael, primary, Xiu, Joanne, primary, Grothey, Axel, primary, Pishvaian, Michael J., primary, Baca, Yasmine, primary, Hwang, Jimmy J., primary, Marshall, John L., primary, VanderWalde, Ari M., primary, Shields, Anthony F., primary, Lenz, Heinz-Josef, primary, Korn, W. Michael, primary, Salem, Mohamed, primary, Philip, Philip A., primary, Goldberg, Richard M., primary, Zeng, Jia, primary, and Kim, Sunnie S., primary

Published
2023
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40. Supplementary Data from Multi-omic Characterization of Pancreatic Ductal Adenocarcinoma Relates CXCR4 mRNA Expression Levels to Potential Clinical Targets

Author

Kocher, Florian, primary, Puccini, Alberto, primary, Untergasser, Gerold, primary, Martowicz, Agnieszka, primary, Zimmer, Kai, primary, Pircher, Andreas, primary, Baca, Yasmine, primary, Xiu, Joanne, primary, Haybaeck, Johannes, primary, Tymoszuk, Piotr, primary, Goldberg, Richard M., primary, Petrillo, Angelica, primary, Shields, Anthony F., primary, Salem, Mohamed E., primary, Marshall, John L., primary, Hall, Michael, primary, Korn, W. Michael, primary, Nabhan, Chadi, primary, Battaglin, Francesca, primary, Lenz, Heinz-Josef, primary, Lou, Emil, primary, Choo, Su-Pin, primary, Toh, Chee-Keong, primary, Gasteiger, Silvia, primary, Pichler, Renate, primary, Wolf, Dominik, primary, and Seeber, Andreas, primary

Published
2023
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41. Supplementary Figure from Multi-omic Characterization of Pancreatic Ductal Adenocarcinoma Relates CXCR4 mRNA Expression Levels to Potential Clinical Targets

Author

Kocher, Florian, primary, Puccini, Alberto, primary, Untergasser, Gerold, primary, Martowicz, Agnieszka, primary, Zimmer, Kai, primary, Pircher, Andreas, primary, Baca, Yasmine, primary, Xiu, Joanne, primary, Haybaeck, Johannes, primary, Tymoszuk, Piotr, primary, Goldberg, Richard M., primary, Petrillo, Angelica, primary, Shields, Anthony F., primary, Salem, Mohamed E., primary, Marshall, John L., primary, Hall, Michael, primary, Korn, W. Michael, primary, Nabhan, Chadi, primary, Battaglin, Francesca, primary, Lenz, Heinz-Josef, primary, Lou, Emil, primary, Choo, Su-Pin, primary, Toh, Chee-Keong, primary, Gasteiger, Silvia, primary, Pichler, Renate, primary, Wolf, Dominik, primary, and Seeber, Andreas, primary

Published
2023
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42. Supplementary Table from Multi-omic Characterization of Pancreatic Ductal Adenocarcinoma Relates CXCR4 mRNA Expression Levels to Potential Clinical Targets

Author

Kocher, Florian, primary, Puccini, Alberto, primary, Untergasser, Gerold, primary, Martowicz, Agnieszka, primary, Zimmer, Kai, primary, Pircher, Andreas, primary, Baca, Yasmine, primary, Xiu, Joanne, primary, Haybaeck, Johannes, primary, Tymoszuk, Piotr, primary, Goldberg, Richard M., primary, Petrillo, Angelica, primary, Shields, Anthony F., primary, Salem, Mohamed E., primary, Marshall, John L., primary, Hall, Michael, primary, Korn, W. Michael, primary, Nabhan, Chadi, primary, Battaglin, Francesca, primary, Lenz, Heinz-Josef, primary, Lou, Emil, primary, Choo, Su-Pin, primary, Toh, Chee-Keong, primary, Gasteiger, Silvia, primary, Pichler, Renate, primary, Wolf, Dominik, primary, and Seeber, Andreas, primary

Published
2023
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43. Data from Multi-omic Characterization of Pancreatic Ductal Adenocarcinoma Relates CXCR4 mRNA Expression Levels to Potential Clinical Targets

Author

Kocher, Florian, primary, Puccini, Alberto, primary, Untergasser, Gerold, primary, Martowicz, Agnieszka, primary, Zimmer, Kai, primary, Pircher, Andreas, primary, Baca, Yasmine, primary, Xiu, Joanne, primary, Haybaeck, Johannes, primary, Tymoszuk, Piotr, primary, Goldberg, Richard M., primary, Petrillo, Angelica, primary, Shields, Anthony F., primary, Salem, Mohamed E., primary, Marshall, John L., primary, Hall, Michael, primary, Korn, W. Michael, primary, Nabhan, Chadi, primary, Battaglin, Francesca, primary, Lenz, Heinz-Josef, primary, Lou, Emil, primary, Choo, Su-Pin, primary, Toh, Chee-Keong, primary, Gasteiger, Silvia, primary, Pichler, Renate, primary, Wolf, Dominik, primary, and Seeber, Andreas, primary

Published
2023
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44. Data from Association of TP53 and CDKN2A Mutation Profile with Tumor Mutation Burden in Head and Neck Cancer

Author

Deneka, Alexander Y., primary, Baca, Yasmine, primary, Serebriiskii, Ilya G., primary, Nicolas, Emmanuelle, primary, Parker, Mitchell I., primary, Nguyen, Theodore T., primary, Xiu, Joanne, primary, Korn, W. Michael, primary, Demeure, Michael J., primary, Wise-Draper, Trisha, primary, Sukari, Ammar, primary, Burtness, Barbara, primary, and Golemis, Erica A., primary

Published
2023
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45. Supplementary Figure from Comprehensive Analysis of R-Spondin Fusions and RNF43 Mutations Implicate Novel Therapeutic Options in Colorectal Cancer

Author

Seeber, Andreas, primary, Battaglin, Francesca, primary, Zimmer, Kai, primary, Kocher, Florian, primary, Baca, Yasmine, primary, Xiu, Joanne, primary, Spizzo, Gilbert, primary, Novotny-Diermayr, Veronica, primary, Rieder, Dietmar, primary, Puccini, Alberto, primary, Swensen, Jeff, primary, Ellis, Michelle, primary, Goldberg, Richard M., primary, Grothey, Axel, primary, Shields, Anthony F., primary, Marshall, John L., primary, Weinberg, Benjamin A., primary, Sackstein, Paul E., primary, Lim, Kiat Hon, primary, Tan, Gek San, primary, Nabhan, Chadi, primary, Korn, W. Michael, primary, Amann, Arno, primary, Trajanoski, Zlatko, primary, Berger, Martin D., primary, Lou, Emil, primary, Wolf, Dominik, primary, and Lenz, Heinz-Josef, primary

Published
2023
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46. Supplementary Table from Comprehensive Analysis of R-Spondin Fusions and RNF43 Mutations Implicate Novel Therapeutic Options in Colorectal Cancer

Author

Seeber, Andreas, primary, Battaglin, Francesca, primary, Zimmer, Kai, primary, Kocher, Florian, primary, Baca, Yasmine, primary, Xiu, Joanne, primary, Spizzo, Gilbert, primary, Novotny-Diermayr, Veronica, primary, Rieder, Dietmar, primary, Puccini, Alberto, primary, Swensen, Jeff, primary, Ellis, Michelle, primary, Goldberg, Richard M., primary, Grothey, Axel, primary, Shields, Anthony F., primary, Marshall, John L., primary, Weinberg, Benjamin A., primary, Sackstein, Paul E., primary, Lim, Kiat Hon, primary, Tan, Gek San, primary, Nabhan, Chadi, primary, Korn, W. Michael, primary, Amann, Arno, primary, Trajanoski, Zlatko, primary, Berger, Martin D., primary, Lou, Emil, primary, Wolf, Dominik, primary, and Lenz, Heinz-Josef, primary

Published
2023
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47. Supplementary Data from Association of TP53 and CDKN2A Mutation Profile with Tumor Mutation Burden in Head and Neck Cancer

Author

Deneka, Alexander Y., primary, Baca, Yasmine, primary, Serebriiskii, Ilya G., primary, Nicolas, Emmanuelle, primary, Parker, Mitchell I., primary, Nguyen, Theodore T., primary, Xiu, Joanne, primary, Korn, W. Michael, primary, Demeure, Michael J., primary, Wise-Draper, Trisha, primary, Sukari, Ammar, primary, Burtness, Barbara, primary, and Golemis, Erica A., primary

Published
2023
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48. Multi-omic characterization reveals a distinct molecular landscape in young-onset pancreatic cancer

Author

Ogobuiro, Ifeanyichukwu, primary, Baca, Yasmine, additional, Ribeiro, Jennifer R, additional, Walker, Phillip, additional, Wilson, Gregory C, additional, Gulhati, Prateek, additional, Marshall, John L, additional, Shroff, Rachna T, additional, Spetzler, David, additional, Oberley, Matthew J, additional, Abbott, Daniel E, additional, Kim, Hong Jin, additional, Kooby, David A, additional, Maithel, Shishir K, additional, Ahmad, Syed A, additional, Merchant, Nipun B., additional, Xiu, Joanne, additional, Hosein, Peter J., additional, and Datta, Jashodeep, additional

Published
2023
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49. PD.01.01 Acquired EGFR Resistant Mutations and Co-mutations in Tumors Of Non-small Cell Lung Cancer Patients Treated With Tyrosine Kinase Inhibitors (TKI)

Author

Raez, Luis E., primary, Baca, Yasmine, additional, Carracedo, Carlos, additional, Vanderwalde, Ari, additional, Nabhan, Chadi, additional, Nagasaka, Misako, additional, Nieva, Jorge, additional, Mandani, Hirva, additional, Borghaei, Hossein, additional, Socinski, Mark, additional, Khan, Hina, additional, Wozniak, Antoinette, additional, Lopes, Gilberto, additional, and Liu, Stephen, additional

Published
2023
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50. Abstract P4-08-06: Clock Genes in Breast Cancer

Author

Jayachandran, Priya, primary, Baca, Yasmine, additional, Xiu, Joanne, additional, Pan, Yuanzhong, additional, Walker, Phil, additional, Battaglin, Francesca, additional, Arai, Hiroyuki, additional, Khushman, Moh’d, additional, Lu, Janice, additional, Spicer, Darcy, additional, Mumenthaler, Shannon, additional, Goldberg, Richard, additional, Weinberg, Benjamin, additional, Lou, Emil, additional, Hall, Michael, additional, Heeke, Arielle L., additional, Korn, W. Michael, additional, Kay, Steve A., additional, Lenz, Heinz-Josef, additional, and Torres, Evanthia T. Roussos, additional

Published
2023
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