235 results on '"Baró, Isabelle"'
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2. Editorial: Methods and applications in cardiac electrophysiology–application to inherited arrhythmias
3. Computer modeling of whole-cell voltage-clamp analyses to delineate guidelines for good practice of manual and automated patch-clamp
4. Arrhythmias precede cardiomyopathy and remodeling of Ca2+ handling proteins in a novel model of long QT syndrome
5. SARS-CoV-2 E and 3a Proteins Are Inducers of Pannexin Currents
6. Predicting hERG repolarization power at 37°C from recordings at room temperature
7. Predicting hERG repolarization power at 37°C from recordings at room temperature
8. HIV-Tat induces a decrease in IKr and IKsvia reduction in phosphatidylinositol-(4,5)-bisphosphate availability
9. Basic Physiology of Ion Channel Function
10. SARS-CoV-2 E and 3a proteins are inducers of pannexin currents
11. Basic Physiology of Ion Channel Function
12. Transforming growth factor β receptor inhibition prevents ventricular fibrosis in a mouse model of progressive cardiac conduction disease
13. Dual Effect of Phosphatidyl (4,5)-Bisphosphate PIP2 on Shaker K+ Channels
14. KCNQ1 Channels Voltage Dependence through a Voltage-dependent Binding of the S4-S5 Linker to the Pore Domain
15. Phosphatidylinositol-4,5-Bisphosphate (PIP2) Stabilizes the Open Pore Conformation of the Kv11.1 (hERG) Channel
16. Neural modulation of ion channels in cardiac arrhythmias: Clinical implications and future investigations
17. Delayed rectifier K + currents and cardiac repolarization
18. A standardised hERG phenotyping pipeline to evaluate KCNH2 genetic variant pathogenicity
19. Marine n-3 PUFAs modulate IKs gating, channel expression, and location in membrane microdomains
20. LQT1-associated Mutations Increase KCNQ1 Proteasomal Degradation Independently of Derlin-1
21. Trafficking-deficient long QT syndrome mutation KCNQ1-T587M confers severe clinical phenotype by impairment of KCNH2 membrane localization: Evidence for clinically significant I Kr-I Ks α-subunit interaction
22. Transfer of Rolf S3-S4 Linker to hERG Eliminates Activation Gating but Spares Inactivation
23. Modeling sudden cardiac death risks factors in COVID-19 patients -the hydroxychloroquine and azithromycin case
24. A consistent arrhythmogenic trait in Brugada syndrome cellular phenotype
25. Modelling sudden cardiac death risks factors in patients with coronavirus disease of 2019: the hydroxychloroquine and azithromycin case
26. Torsades de pointes complicating atrioventricular block: Evidence for a genetic predisposition
27. Complex Brugada syndrome inheritance in a family harbouring compound SCN5A and CACNA1C mutations
28. Infanticide vs. inherited cardiac arrhythmias
29. Computer modeling of whole-cell voltage-clamp analyses to delineate guidelines for good practice of manual and automated patch-clamp
30. KCNE1–KCNQ1 osmoregulation by interaction of phosphatidylinositol-4,5-bisphosphate with Mg2+ and polyamines
31. IKs response to protein kinase A-dependent KCNQ1 phosphorylation requires direct interaction with microtubules
32. Kv7.1 (KCNQ1) properties and channelopathies
33. ATP-sensitive K+ channels regulated by intracellular Ca2+ and phosphorylation in normal (T84) and cystic fibrosis (CFPAC-1) epithelial cells
34. Effects of sulphonylureas on cAMP-stimulated Cl− transport via the cystic fibrosis gene product in human epithelial cells
35. Na + channel mutation leading to loss of function and non-progressive cardiac conduction defects
36. RRAD mutation causes electrical and cytoskeletal defects in cardiomyocytes derived from a familial case of Brugada syndrome
37. Inactivating Properties of Recombinant ROMK2 Channels Expressed in Mammalian Cells
38. Infanticide vs. inherited cardiac arrhythmias.
39. β3-Adrenoceptor Control the Cystic Fibrosis Transmembrane Conductance Regulator through a cAMP/Protein Kinase A-independent Pathway
40. Mutations in a Dominant-Negative Isoform Correlate with Phenotype in Inherited Cardiac Arrhythmias
41. HIV-Tat Induces a Decrease in I Kr and I Ks via Reduction in Phosphatidylinositol-(4,5)-Bisphosphate Availability
42. Multifocal Ectopic Purkinje-Related Premature Contractions: A New SCN5A-Related Cardiac Channelopathy
43. HIV-Tat induces a decrease in I Kr and I Ks via reduction in phosphatidylinositol-(4,5)-bisphosphate availability
44. Dysfunction of the Voltage‐Gated K + Channel β2 Subunit in a Familial Case of Brugada Syndrome
45. A Molecular Substrate for Long QT in HIV Patients: Tat Protein Reduces IKR in Human Induced Pluripotent Stem Cells-Derived Cardiomyocytes
46. Physiological and Pathophysiological Insights of Nav1.4 and Nav1.5 Comparison
47. Multifocal ectopic Purkinje-related premature contractions: a new SCN5A-related cardiac channelopathy
48. Toward Personalized Medicine: Using Cardiomyocytes Differentiated From Urine‐Derived Pluripotent Stem Cells to Recapitulate Electrophysiological Characteristics of Type 2 Long QT Syndrome
49. 0417 : SCN5A+/ΔQKP mice present late sodium current associated with long QT syndrome, dilated cardiomyopathy and ranolazine-sensitive spontaneous ventricular arrhythmias
50. Kv7.1 (KCNQ1) properties and channelopathies
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