148 results on '"Battaglia, George"'
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2. MDMA Effects in Brain: Pharmacologic Profile and Evidence of Neurotoxicity from Neurochemical and Autoradiographic Studies
3. Criteria for Assessing Drug-Induced Biochemical Alterations and Degeneration of Serotonergic Neurons
4. Paroxetine is effective in desensitizing 5-HT1A receptor function in adult offspring exposed prenatally to cocaine
5. Alterations in 5-HT2A receptor signaling in male and female transgenic rats over-expressing either Gq or RGS-insensitive Gq protein
6. Prenatal cocaine exposure potentiates paroxetine-induced desensitization of 5-HT 2A receptor function in adult male rat offspring
7. Prenatal cocaine exposure potentiates paroxetine-induced desensitization of 5-HT2A receptor function in adult male rat offspring
8. Limbic pallidal adaptations following long-term cessation of dopaminergic transmission: lack of upregulation of dopamine receptor function
9. 5-HT1A receptor-mediated activation of neuroendocrine responses and multiple protein kinase pathways in the peripubertal rat hypothalamus
10. Corticotropin-Releasing Hormone (CRH) Receptors in Brain
11. Rats with ischemic heart failure (IHF) exhibit exaggerated conditioned fear and altered β‐adrenergic receptor expression in forebrain regions that govern autonomic responses to fear
12. Alterations in β‐adrenergic receptors in frontal cortex and amygdala in rats with heart failure: Effects of chronic antidepressant treatment
13. Chronic fluoxetine decreases elevated left ventricular 5‐HT receptor density in rats with congestive heart failure.
14. Prolonged 5-HT1A/5-HT2A receptor cross-talk is absent prior to adulthood in rats
15. Sustained treatment with a 5-HT2A receptor agonist causes functional desensitization and reductions in agonist-labeled 5-HT2A receptors despite increases in receptor protein levels in rats
16. 5-HT1A receptors mediate (+)8-OH-DPAT-stimulation of extracellular signal-regulated kinase (MAP kinase) in vivo in rat hypothalamus: Time dependence and regional differences
17. Agonist Induced-Phosphorylation of Gα11Protein Reduces Coupling to 5-HT2AReceptors
18. Withdrawal from a single exposure to cocaine increases 5-HT2A receptor and G protein function
19. Single Exposure to a Serotonin 1A Receptor Agonist, (+)8-Hydroxy-2-(di-n-propylamino)-tetralin, Produces a Prolonged Heterologous Desensitization of Serotonin 2A Receptors in Neuroendocrine Neurons in Vivo
20. Short-Term Cocaine Treatment Causes Neuroadaptive Changes in Gαq and Gα11 Proteins in Rats Undergoing Withdrawal
21. Desensitization of 5-HT1AReceptors by 5-HT2AReceptors in Neuroendocrine Neurons in Vivo
22. Estrogen Reduces Serotonin-1A Receptor-Mediated Oxytocin Release and Gαi/o/z Proteins in the Hypothalamus of Ovariectomized Rats
23. A Region-Specific Increase in Gαq And Gα11 Proteins in Brains of Rats during Cocaine Withdrawal
24. Evidence That 5-HT2AReceptors in the Hypothalamic Paraventricular Nucleus Mediate Neuroendocrine Responses to (−)DOI
25. Ovariectomy-Induced Increases in Hypothalamic Serotonin-1A Receptor Function in Rats Are Prevented by Estradiol
26. Characterization of the Functional Heterologous Desensitization of Hypothalamic 5-HT1A Receptors after 5-HT2AReceptor Activation
27. 5-HT1A and 5-HT2A serotonin receptor turnover in adult rat offspring prenatally exposed to cocaine
28. Estrogen desensitizes 5-HT1A receptors and reduces levels of Gz, Gi1 and Gi3 proteins in the hypothalamus
29. Neurochemical changes in brain serotonin neurons in immature and adult offspring prenatally exposed to cocaine
30. Prenatal cocaine exposure potentiates 5-HT2a receptor function in male and female rat offspring
31. Long-term fluoxetine produces behavioral anxiolytic effects without inhibiting neuroendocrine responses to conditioned stress in rats
32. Serotonin transporter production and degradation rates: studies with RTI-76
33. The effects of cocaine and the amphetamines on brain and behavior: a conference report
34. Prenatal Cocaine Exposure Produces Long-Term Impairments in Brain Serotonin Function in Rat Offspringa
35. WAY-100635 inhibits 8-OH-DPAT-stimulated oxytocin, ACTH and corticosterone, but not prolactin secretion
36. Autoradiographic evidence for differential G-protein coupling of 5-HT1A receptors in rat brain: lack of effect of repeated injections of fluoxetine
37. Attenuation of hormone responses to the 5-HT1A agonist ipsapirone by long-term treatment with fluoxetine, but not desipramine, in male rats
38. Cocaine-induced deficits in ACTH and corticosterone responses in female rat progeny
39. Long-term fluoxetine, but not desipramine, inhibits the ACTH and oxytocin responses to the 5-HT1A agonist, 8-OH-DPAT, in male rats
40. Prenatal methamphetamine attenuates serotonin mediated renin secretion in male and female rat progeny: Evidence for selective long-term dysfunction of serotonin pathways in brain
41. In vivo EEDQ dose-dependently inactivates rat brain 5-HT receptors but not 5-HT uptake sites
42. Prenatal cocaine produces deficits in serotonin mediated neuroendocrine responses in adult rat progeny: Evidence for long‐term functional alterations in brain serotonin pathways
43. Neuroanatomic specificity and time course of alterations in rat brain serotonergic pathways induced by MDMA (3,4‐methylenedioxymethamphetamine): Assessment using quantitative autoradiography
44. Identification and exploitation of the .sigma.-opiate pharmacophore
45. Prolonged 5-HT1A/5-HT2A receptor cross-talk is absent prior to adulthood in rats.
46. Withdrawal from a single exposure to cocaine increases 5-HT2A receptor and G protein function.
47. Neurotoxic Effects of MDMA on Brain Serotonin Neurons: Evidence from Neurochemical and Radioligand Binding Studiesa
48. Fluoxetine treatment of prepubescent rats produces a selective functional reduction in the 5-HT2A receptor-mediated stimulation of oxytocin.
49. Paroxetine is effective in desensitizing 5-HT1A receptor function in adult offspring exposed prenatally to cocaine.
50. Estrogen Reduces Serotonin-1A Receptor-Mediated Oxytocin Release and Gαi/o/z Proteins in the Hypothalamus of Ovariectomized Rats.
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