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2. Splenic marginal zone antigen‐presenting cells are critical for the primary allo‐immune response to therapeutic factor VIII in hemophilia A
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NAVARRETE, A., DASGUPTA, S., DELIGNAT, S., CALIGIURI, G., CHRISTOPHE, O.D., BAYRY, J., NICOLETTI, A., KAVERI, S.V., and LACROIX‐DESMAZES, S.
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- 2009
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3. Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
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Klionsky, DJ, Abdel-Aziz, AK, Abdelfatah, S, Abdellatif, M, Abdoli, A, Abel, S, Abeliovich, H, Abildgaard, MH, Abudu, YP, Acevedo-Arozena, A, Adamopoulos, IE, Adeli, K, Adolph, TE, Adornetto, A, Aflaki, E, Agam, G, Agarwal, A, Aggarwal, BB, Agnello, M, Agostinis, P, Agrewala, JN, Agrotis, A, Aguilar, PV, Ahmad, ST, Ahmed, ZM, Ahumada-Castro, U, Aits, S, Aizawa, S, Akkoc, Y, Akoumianaki, T, Akpinar, HA, Al-Abd, AM, Al-Akra, L, Al-Gharaibeh, A, Alaoui-Jamali, MA, Alberti, S, Alcocer-Gómez, E, Alessandri, C, Ali, M, Alim Al-Bari, MA, Aliwaini, S, Alizadeh, J, Almacellas, E, Almasan, A, Alonso, A, Alonso, GD, Altan-Bonnet, N, Altieri, DC, Álvarez, ÉMC, Alves, S, Alves da Costa, C, Alzaharna, MM, Amadio, M, Amantini, C, Amaral, C, Ambrosio, S, Amer, AO, Ammanathan, V, An, Z, Andersen, SU, Andrabi, SA, Andrade-Silva, M, Andres, AM, Angelini, S, Ann, D, Anozie, UC, Ansari, MY, Antas, P, Antebi, A, Antón, Z, Anwar, T, Apetoh, L, Apostolova, N, Araki, T, Araki, Y, Arasaki, K, Araújo, WL, Araya, 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C, Viret, C, Viscomi, MT, Visnjic, D, Vitale, I, Vocadlo, DJ, Voitsekhovskaja, OV, Volonté, C, Volta, M, Vomero, M, Von Haefen, C, Vooijs, MA, Voos, W, Vucicevic, L, Wade-Martins, R, Waguri, S, Waite, KA, Wakatsuki, S, Walker, DW, Walker, MJ, Walker, SA, Walter, J, Wandosell, FG, Wang, B, Wang, C-Y, Wang, C, Wang, D, Wang, F, Wang, G, Wang, H, Wang, H-G, Wang, J, Wang, K, Wang, L, Wang, MH, Wang, M, Wang, N, Wang, P, Wang, QJ, Wang, Q, Wang, QK, Wang, QA, Wang, W-T, Wang, W, Wang, X, Wang, Y, Wang, Y-Y, Wang, Z, Warnes, G, Warnsmann, V, Watada, H, Watanabe, E, Watchon, M, Wawrzyńska, A, Weaver, TE, Wegrzyn, G, Wehman, AM, Wei, H, Wei, L, Wei, T, Wei, Y, Weiergräber, OH, Weihl, CC, Weindl, G, Weiskirchen, R, Wells, A, Wen, RH, Wen, X, Werner, A, Weykopf, B, Wheatley, SP, Whitton, JL, Whitworth, AJ, Wiktorska, K, Wildenberg, ME, Wileman, T, Wilkinson, S, Willbold, D, Williams, B, Williams, RSB, Williams, RL, Williamson, PR, Wilson, RA, Winner, B, Winsor, NJ, Witkin, SS, Wodrich, H, Woehlbier, U, Wollert, T, Wong, E, Wong, JH, Wong, RW, Wong, VKW, Wong, WW-L, Wu, A-G, Wu, C, Wu, J, Wu, KK, Wu, M, Wu, S-Y, Wu, S, Wu, WKK, Wu, X, Wu, Y-W, Wu, Y, Xavier, RJ, Xia, H, Xia, L, Xia, Z, Xiang, G, Xiang, J, Xiang, M, Xiang, W, Xiao, B, Xiao, G, Xiao, H, Xiao, H-T, Xiao, J, Xiao, L, Xiao, S, Xiao, Y, Xie, B, Xie, C-M, Xie, M, Xie, Y, Xie, Z, Xilouri, M, Xu, C, Xu, E, Xu, H, Xu, J, Xu, L, Xu, WW, Xu, X, Xue, Y, Yakhine-Diop, SMS, Yamaguchi, M, Yamaguchi, O, Yamamoto, A, Yamashina, S, Yan, S, Yan, S-J, Yan, Z, Yanagi, Y, Yang, C, Yang, D-S, Yang, H, Yang, H-T, Yang, J-M, Yang, J, Yang, L, Yang, M, Yang, P-M, Yang, Q, Yang, S, Yang, S-F, Yang, W, Yang, WY, Yang, X, Yang, Y, Yao, H, Yao, S, Yao, X, Yao, Y-G, Yao, Y-M, Yasui, T, Yazdankhah, M, Yen, PM, Yi, C, Yin, X-M, Yin, Y, Yin, Z, Ying, M, Ying, Z, Yip, CK, Yiu, SPT, Yoo, YH, Yoshida, K, Yoshii, SR, Yoshimori, T, Yousefi, B, Yu, B, Yu, H, Yu, J, Yu, L, Yu, M-L, Yu, S-W, Yu, VC, Yu, WH, Yu, Z, Yuan, J, Yuan, L-Q, Yuan, S, Yuan, S-SF, Yuan, Y, Yuan, Z, Yue, J, Yue, Z, Yun, J, Yung, RL, Zacks, DN, Zaffagnini, G, Zambelli, VO, Zanella, I, Zang, QS, Zanivan, S, Zappavigna, S, Zaragoza, P, Zarbalis, KS, Zarebkohan, A, Zarrouk, A, Zeitlin, SO, Zeng, J, Zeng, J-D, Žerovnik, E, Zhan, L, Zhang, B, Zhang, DD, Zhang, H, Zhang, H-L, Zhang, J, Zhang, J-P, Zhang, KYB, Zhang, LW, Zhang, L, Zhang, M, Zhang, P, Zhang, S, Zhang, W, Zhang, X, Zhang, X-W, Zhang, XD, Zhang, Y, Zhang, Y-D, Zhang, Y-Y, Zhang, Z, Zhao, H, Zhao, L, Zhao, S, Zhao, T, Zhao, X-F, Zhao, Y, Zheng, G, Zheng, K, Zheng, L, Zheng, S, Zheng, X-L, Zheng, Y, Zheng, Z-G, Zhivotovsky, B, Zhong, Q, Zhou, A, Zhou, B, Zhou, C, Zhou, G, Zhou, H, Zhou, J, Zhou, K, Zhou, R, Zhou, X-J, Zhou, Y, Zhou, Z-Y, Zhou, Z, Zhu, B, Zhu, C, Zhu, G-Q, Zhu, H, Zhu, W-G, Zhu, Y, Zhuang, H, Zhuang, X, Zientara-Rytter, K, Zimmermann, CM, Ziviani, E, Zoladek, T, Zong, W-X, Zorov, DB, Zorzano, A, Zou, W, Zou, Z, Zuryn, S, Zwerschke, W, Brand-Saberi, B, Dong, XC, Kenchappa, CS, Lin, Y, Oshima, S, Rong, Y, Sluimer, JC, Stallings, CL, and Tong, C-K
- Abstract
In 2008, we published the first set of guidelines for standardizing research in autophagy. Since then, this topic has received increasing attention, and many scientists have entered the field. Our knowledge base and relevant new technologies have also been expanding. Thus, it is important to formulate on a regular basis updated guidelines for monitoring autophagy in different organisms. Despite numerous reviews, there continues to be confusion regarding acceptable methods to evaluate autophagy, especially in multicellular eukaryotes. Here, we present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes. These guidelines are not meant to be a dogmatic set of rules, because the appropriateness of any assay largely depends on the question being asked and the system being used. Moreover, no individual assay is perfect for every situation, calling for the use of multiple techniques to properly monitor autophagy in each experimental setting. Finally, several core components of the autophagy machinery have been implicated in distinct autophagic processes (canonical and noncanonical autophagy), implying that genetic approaches to block autophagy should rely on targeting two or more autophagy-related genes that ideally participate in distinct steps of the pathway. Along similar lines, because multiple proteins involved in autophagy also regulate other cellular pathways including apoptosis, not all of them can be used as a specific marker for bona fide autophagic responses. Here, we critically discuss current methods of assessing autophagy and the information they can, or cannot, provide. Our ultimate goal is to encourage intellectual and technical innovation in the field.
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- 2021
4. Intravenous immunoglobulin and immune response
- Author
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Kaveri, S. V., Lecerf, M., Saha, C., Kazatchkine, M. D., Lacroix-Desmazes, S., and Bayry, J.
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- 2014
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5. COVID-19 critique et anticorps anti-Interféron : série de 11 cas
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Gilardin, L., primary, Nielly, H., additional, Roumier, M., additional, Chauvin, C., additional, Bastard, P., additional, Bousquet, A., additional, Vasse, M., additional, Roth, C., additional, Sakuntabhai, A., additional, Bayry, J., additional, Bourgarit, A., additional, Dubost, C., additional, and Tandjaoui-Lambiotte, Y., additional
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- 2021
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6. IVIg increases interleukin-11 levels, which in turn contribute to increased platelets, VWF and FVIII in mice and humans
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Nguyen, A, primary, Repesse, Y, additional, Ebbo, M, additional, Allenbach, Y, additional, Benveniste, O, additional, Vallat, J M, additional, Magy, L, additional, Deshayes, S, additional, Maigné, G, additional, de Boysson, H, additional, Karnam, A, additional, Delignat, S, additional, Lacroix-Desmazes, S, additional, Bayry, J, additional, and Aouba, A, additional
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- 2021
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7. Mechanisms of action of intravenous immunoglobulin in autoimmune and inflammatory diseases
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Bayry, J., Thirion, M., Misra, N., Thorenoor, N., Delignat, S., Lacroix-Desmazes, S., Bellon, B., Kaveri, S., and Kazatchkine, M. D.
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- 2003
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8. Therapeutic factor VIII does not trigger TLR1.2 and TLR2.6 signalling in vitro
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Teyssandier, M., André, S., Gupta, N., Dasgupta, S., Bayry, J., Kaveri, S. V., and Lacroix-Desmazes, S.
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- 2013
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9. Intravenous immunoglobulins in immunodeficiencies: more than mere replacement therapy
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Kaveri, S. V., Maddur, M. S., Hegde, P., Lacroix-Desmazes, S., and Bayry, J.
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- 2011
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10. Mechanisms of action of IVIg in autoimmune and inflammatory diseases: an update: No.24
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Kaveri, S, Maddur, M, and Bayry, J
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- 2010
11. 6th International Immunoglobulin Symposium: Poster presentations
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Fernandez-Cruz, E., Kaveri, S. V., Peter, H. H., Durandy, A., Cantoni, N., Quinti, I., Sorensen, R., Bussel, J. B., Danieli, M. G., Winkelmann, A., Bayry, J., Käsermann, F., Späth, P., Helbert, M., Salama, A., van Schaik, I. N., and Yuki, N.
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- 2009
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12. LDL receptor-related protein, a catabolic receptor for factor VIII, is not critical for factor VIII uptake by professional antigen presenting cells: 14 PO 377
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DASGUPTA, S, KAVERI, S, LACROIX-DESMAZES, S, KAZATCHKINE, M, BAYRY, J, and WOOTLA, B
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- 2006
13. FVIII-hydrolyzing antibodies in acquired hemophilia: 01 FP 01
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WOOTLA, B, KAVERI, S, LACROIX-DESMAZES, S, BAYRY, J, VALAKUNJA, N, and LÉVESQUE, H
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- 2006
14. Natural Autoantibodies as Tools to Predict the Outcome of Immune Response?
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Kohler, H., Bayry, J., Nicoletti, A., and Kaveri, S. V.
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- 2003
15. High responder patients with severe haemophilia A generate anti-FVIII alloantibodies with hydrolytic activity towards FVIII
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LACROIX-DESMAZES, S, ROSE, S, ARTAUD, C, BAYRY, J, MISRA, N, KAZATCHKINE, M D, and SRINI, V KAVERI
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- 2002
16. Pathophysiology of inhibitors to factor VIII in patients with haemophilia A
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LACROIX-DESMAZES, S., MISRA, N., BAYRY, J., ARTAUD, C., DRAYTON, B., KAVERI, S. V., and KAZATCHKINE, M D.
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- 2002
17. The concept of idiotypic vaccination against factor VIII inhibitors in haemophilia A
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LACROIX-DESMAZES, S., BAYRY, J., MISRA, N., KAVERI, S. V., and KAZATCHKINE, M. D.
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- 2002
18. Mechanisms of action of intravenous immunoglobulin in autoimmune and inflammatory diseases
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Bayry, J, Misra, N, Latry, V, Prost, F, Delignat, S, Lacroix-Desmazes, S, Kazatchkine, M.D, and Kaveri, S.V
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- 2003
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19. Early antibody responses of cattle for foot-and-mouth disease quadrivalent double oil emulsion vaccine
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Patil, P.K., Bayry, J., Nair, S.P., Gopalakrishna, S., Sajjanar, C.M., Misra, L.D., and Natarajan, C.
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- 2002
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20. Immune responses of goats against foot-and-mouth disease quadrivalent vaccine: comparison of double oil emulsion and aluminium hydroxide gel vaccines in eliciting immunity
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Patil, P.K., Bayry, J., Ramakrishna, C., Hugar, B., Misra, L.D., and Natarajan, C.
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- 2002
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21. Immuno affinity purification of foot and mouth disease virus type specific antibodies using recombinant protein adsorbed to polystyrene wells
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Bayry, J, Prabhudas, K, Bist, P, Reddy, G.R, and Suryanarayana, V.V.S
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- 1999
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22. Orientation de la réponse immune par les basophiles
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Galeotti, C., Stephen-Victor, E., Sharma, M., Kaveri, S.V., Bayry, J., HAL-UPMC, Gestionnaire, Service de Pédiatrie Générale et Rhumatologie Pédiatrique, Université Paris-Sud - Paris 11 (UP11)-Assistance publique - Hôpitaux de Paris (AP-HP) (AP-HP)-Hôpital Bicêtre, Centre de Recherche des Cordeliers (CRC), Université Pierre et Marie Curie - Paris 6 (UPMC)-École pratique des hautes études (EPHE), Université Paris sciences et lettres (PSL)-Université Paris sciences et lettres (PSL)-Université Paris Diderot - Paris 7 (UPD7)-Université Paris Descartes - Paris 5 (UPD5)-Institut National de la Santé et de la Recherche Médicale (INSERM), Immunologie et immuno-intervention thérapeutique, and Université Paris sciences et lettres (PSL)-Université Paris sciences et lettres (PSL)-Université Paris Diderot - Paris 7 (UPD7)-Université Paris Descartes - Paris 5 (UPD5)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Pierre et Marie Curie - Paris 6 (UPMC)-École pratique des hautes études (EPHE)
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[SDV.IMM] Life Sciences [q-bio]/Immunology ,parasitic diseases ,Antigen-presenting cells ,IL-4 ,[SDV.IMM]Life Sciences [q-bio]/Immunology ,chemical and pharmacologic phenomena ,hemic and immune systems ,Th17 ,[SDV.BC]Life Sciences [q-bio]/Cellular Biology ,antigen presenting cells ,[SDV.BC] Life Sciences [q-bio]/Cellular Biology ,Th2 response ,Basophils - Abstract
Basophils are infrequent granulocytes which are important for Th2 responses, protection against helminthic parasites and the pathogenesis of allergy and other inflammatory conditions. Recent reports suggest that in mice basophils function as antigen-presenting cells for mediation of Th2 responses. However, human basophils lack features of antigen-presenting cells., Les basophiles sont de rares granulocytes et ont un rôle important dans les réponses Th2, dans la protection contre les helminthes et dans la pathogenèse de l’allergie et d’autres maladies inflammatoires. Des études récentes ont suggéré que les basophiles fonctionnent comme des cellules présentatrices d’antigènes pour médier les réponses Th2 chez la souris. Cependant, les basophiles humains n’ont pas les caractéristiques des cellules présentatrices d’antigène.
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- 2016
23. The european hematology association roadmap for european hematology research: A consensus document
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Engert, A. Balduini, C. Brand, A. Coiffier, B. Cordonnier, C. Döhner, H. De Wit, T.D. Eichinger, S. Fibbe, W. Green, T. De Haas, F. Iolascon, A. Jaffredo, T. Rodeghiero, F. Sall Es, G. Schuringa, J.J. André, M. Andre-Schmutz, I. Bacigalupo, A. Bochud, P.-Y. Den Boer, M. Bonini, C. Camaschella, C. Cant, A. Cappellini, M.D. Cazzola, M. Celso, C.L. Dimopoulos, M. Douay, L. Dzierzak, E. Einsele, H. Ferreri, A. De Franceschi, L. Gaulard, P. Gottgens, B. Greinacher, A. Gresele, P. Gribben, J. De Haan, G. Hansen, J.-B. Hochhaus, A. Kadir, R. Kaveri, S. Kouskoff, V. Kühne, T. Kyrle, P. Ljungman, P. Maschmeyer, G. Méndez-Ferrer, S. Milsom, M. Mummery, C. Ossenkoppele, G. Pecci, A. Peyvandi, F. Philipsen, S. Reitsma, P. Ribera, J.M. Risitano, A. Rivella, S. Ruf, W. Schroeder, T. Scully, M. Socie, G. Staal, F. Stanworth, S. Stauder, R. Stilgenbauer, S. Tamary, H. Theilgaard-Mönch, K. Thein, S.L. Tilly, H. Trneny, M. Vainchenker, W. Vannucchi, A.M. Viscoli, C. Vrielink, H. Zaaijer, H. Zanella, A. Zolla, L. Zwaginga, J.J. Martinez, P.A. Van Den Akker, E. Allard, S. Anagnou, N. Andolfo, I. Andrau, J.-C. Angelucci, E. Anstee, D. Aurer, I. Avet-Loiseau, H. Aydinok, Y. Bakchoul, T. Balduini, A. Barcellini, W. Baruch, D. Baruchel, A. Bayry, J. Bento, C. Van Den Berg, A. Bernardi, R. Bianchi, P. Bigas, A. Biondi, A. Bohonek, M. Bonnet, D. Borchmann, P. Borregaard, N. Brækkan, S. Van Den Brink, M. Brodin, E. Bullinger, L. Buske, C. Butzeck, B. Cammenga, J. Campo, E. Carbone, A. Cervantes, F. Cesaro, S. Charbord, P. Claas, F. Cohen, H. Conard, J. Coppo, P. Vives Corron, J.-L. Da Costa, L. Davi, F. Delwel, R. Dianzani, I. Domanović, D. Donnelly, P. Drnovšek, T.D. Dreyling, M. Du, M.-Q. Dufour, C. Durand, C. Efremov, D. Eleftheriou, A. Elion, J. Emonts, M. Engelhardt, M. Ezine, S. Falkenburg, F. Favier, R. Federico, M. Fenaux, P. Fitzgibbon, J. Flygare, J. Foà, R. Forrester, L. Galacteros, F. Garagiola, I. Gardiner, C. Garraud, O. Van Geet, C. Geiger, H. Geissler, J. Germing, U. Ghevaert, C. Girelli, D. Godeau, B. Gökbuget, N. Goldschmidt, H. Goodeve, A. Graf, T. Graziadei, G. Griesshammer, M. Gruel, Y. Guilhot, F. Von Gunten, S. Gyssens, I. Halter, J. Harrison, C. Harteveld, C. Hellström-Lindberg, E. Hermine, O. Higgs, D. Hillmen, P. Hirsch, H. Hoskin, P. Huls, G. Inati, A. Johnson, P. Kattamis, A. Kiefel, V. Kleanthous, M. Klump, H. Krause, D. Hovinga, J.K. Lacaud, G. Lacroix-Desmazes, S. Landman-Parker, J. Legouill, S. Lenz, G. Von Lilienfeld-Toal, M. Von Lindern, M. Lopez-Guillermo, A. Lopriore, E. Lozano, M. Macintyre, E. Makris, M. Mannhalter, C. Martens, J. Mathas, S. Matzdorff, A. Medvinsky, A. Menendez, P. Migliaccio, A.R. Miharada, K. Mikulska, M. Minard, V. Montalbán, C. De Montalembert, M. Montserrat, E. Morange, P.-E. Mountford, J. Muckenthaler, M. Müller-Tidow, C. Mumford, A. Nadel, B. Navarro, J.-T. El Nemer, W. Noizat-Pirenne, F. O’Mahony, B. Oldenburg, J. Olsson, M. Oostendorp, R. Palumbo, A. Passamonti, F. Patient, R. De Latour, R.P. Pflumio, F. Pierelli, L. Piga, A. Pollard, D. Raaijmakers, M. Radford, J. Rambach, R. Koneti Rao, A. Raslova, H. Rebulla, P. Rees, D. Ribrag, V. Rijneveld, A. Rinalducci, S. Robak, T. Roberts, I. Rodrigues, C. Rosendaal, F. Rosenwald, A. Rule, S. Russo, R. Saglio, G. Sanchez, M. Scharf, R.E. Schlenke, P. Semple, J. Sierra, J. So-Osman, C. Soria, J.M. Stamatopoulos, K. Stegmayr, B. Stunnenberg, H. Swinkels, D. Barata, J.P.T. Taghon, T. Taher, A. Terpos, E. Thachil, J. Tissot, J.D. Touw, I. Toye, A. Trappe, R. Traverse-Glehen, A. Unal, S. Vaulont, S. Viprakasit, V. Vitolo, U. Van Wijk, R. Wójtowicz, A. Zeerleder, S. Zieger, B. EHA Roadmap for European Hematology Research
- Abstract
The European Hematology Association (EHA) Roadmap for European Hematology Research highlights major achievements in diagnosis and treatment of blood disorders and identifies the greatest unmet clinical and scientific needs in those areas to enable better funded, more focused European hematology research. Initiated by the EHA, around 300 experts contributed to the consensus document, which will help European policy makers, research funders, research organizations, researchers, and patient groups make better informed decisions on hematology research. It also aims to raise public awareness of the burden of blood disorders on European society, which purely in economic terms is estimated at ∈ European Hematology Association (EHA) Roadmap for European Hematology Research highlights major achievements in diagnosis and treatment of blood disorders and identifies the greatest unmet clinical and scientific needs in those areas to enable better fu treatments, sometimes in revolutionary ways. This progress highlights the potential of focused basic research programs such as this EHA Roadmap. The EHA Roadmap identifies nine ‘sections’ in hematology: normal hematopoiesis, malignant lymphoid and myeloid diseases, anemias and related diseases, platelet disorders, blood coagulation and hemostatic disorders, transfusion medicine, infections in hematology, and hematopoietic stem cell transplantation. These sections span 60 smaller groups of diseases or disorders. The EHA Roadmap identifies priorities and needs across the field of hematology, including those to develop targeted therapies based on genomic profiling and chemical biology, to eradicate minimal residual malignant disease, and to develop cellular immunotherapies, combination treatments, gene therapies, hematopoietic stem cell treatments, and treatments that are better tolerated by elderly patients. © 2016 Ferrata Storti Foundation.
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- 2016
24. Dissecting the anti-inflammatory effects of Viscum album: inhibition of cytokine-induced expression of cyclooxygenase-2 and secretion of prostaglandin E2
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Elluru, S.R., Saha, C., Hedge, P., Friboulet, A., Bayry, J., Kaveri, S.V., Immunopathologie et immunointervention thérapeutique, Université Pierre et Marie Curie - Paris 6 ( UPMC ) -IFR58-Institut National de la Santé et de la Recherche Médicale ( INSERM ), Génie Enzymatique et Cellulaire ( GEC ), Université de Technologie de Compiègne ( UTC ) -Université de Picardie Jules Verne ( UPJV ) -Centre National de la Recherche Scientifique ( CNRS ), Centre de Recherche des Cordeliers ( CRC ), Université Paris Diderot - Paris 7 ( UPD7 ) -École pratique des hautes études ( EPHE ) -Université Pierre et Marie Curie - Paris 6 ( UPMC ) -Université Paris Descartes - Paris 5 ( UPD5 ) -Institut National de la Santé et de la Recherche Médicale ( INSERM ), Université Pierre et Marie Curie - Paris 6 (UPMC)-IFR58-Institut National de la Santé et de la Recherche Médicale (INSERM), Génie Enzymatique et Cellulaire (GEC), Université de Technologie de Compiègne (UTC)-Université de Picardie Jules Verne (UPJV)-Centre National de la Recherche Scientifique (CNRS), Centre de Recherche des Cordeliers (CRC), Université Paris Diderot - Paris 7 (UPD7)-École pratique des hautes études (EPHE), Université Paris sciences et lettres (PSL)-Université Paris sciences et lettres (PSL)-Université Pierre et Marie Curie - Paris 6 (UPMC)-Université Paris Descartes - Paris 5 (UPD5)-Institut National de la Santé et de la Recherche Médicale (INSERM), Université Pierre et Marie Curie - Paris 6 (UPMC)-École Pratique des Hautes Études (EPHE), Université Paris sciences et lettres (PSL)-Université Paris sciences et lettres (PSL)-Université Paris Diderot - Paris 7 (UPD7)-Université Paris Descartes - Paris 5 (UPD5)-Institut National de la Santé et de la Recherche Médicale (INSERM), David, Chantal, and Université Paris Diderot - Paris 7 (UPD7)-École pratique des hautes études (EPHE)-Université Pierre et Marie Curie - Paris 6 (UPMC)-Université Paris Descartes - Paris 5 (UPD5)-Institut National de la Santé et de la Recherche Médicale (INSERM)
- Subjects
[SDV] Life Sciences [q-bio] ,[ SDV ] Life Sciences [q-bio] ,[SDV]Life Sciences [q-bio] - Abstract
International audience; Reports unraveling the beneficial effects of Viscum album (VA) preparations as complementary therapies for cancer have increasingly revealed the underlying molecular and cellular mechanisms, which encompass cytotoxic properties, induction of apoptosis, inhibition of angiogenesis and several other immunomodulatory mechanisms. In addition to their propitious relevance to cancer therapy, VA preparations are also relevant for the treatment of several inflammatory pathologies. In view of the intricate association of inflammation and cancer and the fact that several anti-tumor phytotherapeutics exert potent anti-inflammatory effects, we believe that an anti-inflammatory effect is responsible for the therapeutic benefits of VA preparations. One of the underlying molecular mechanisms of this inflammatory response is the selective down-regulation of the cyclo-oxygenase (COX)-2-mediated cytokine-induced secretion of prostaglandin E2 (PGE2). This inhibitory action has been associated with reduced expression of COX-2, without modulating COX-1 expression. This mechanism is associated with VA-induced destabilization of COX-2 mRNA, thereby depleting the functional COX-2 mRNA available for protein synthesis and for subsequent induction of secretion of PGE2. Together, these results demonstrate a novel anti-inflammatory mechanism of action of VA preparation, wherein VAQUSpez an anti-inflammatory effect by inhibiting cytokine-induced PGE2 via selective inhibition of COX-2 and destabilization of COX-2 mRNA.
- Published
- 2015
25. Orientation de la réponse immune par les basophiles
- Author
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Galeotti, C., primary, Stephen-Victor, E., additional, Sharma, M., additional, Kaveri, S.V., additional, and Bayry, J., additional
- Published
- 2016
- Full Text
- View/download PDF
26. Intravenous Ig inhibits invariant NKT cell-mediated allergic airway inflammation through FcγRIIIA-dependent mechanisms
- Author
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Araujo, L.M., Chauvineau, A., Zhu, R., Diem, S., Bourgeois, E.A., Levescot, A., Huerre, M., Gombert, J.M., Bayry, J., Daëron, M., Ruhns, P., Kaveri, S.V., Herbelin, A., Cytokines, hématopoïèse et réponse immune (CHRI), Université Paris Descartes - Paris 5 (UPD5)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), Modèles de Cellules Souches Malignes et Thérapeutiques, Université Paris-Sud - Paris 11 (UP11)-Institut National de la Santé et de la Recherche Médicale (INSERM), DME (Dept. of Mech. Eng.), Hong Kong University of Science and Technology (HKUST), Institut de biologie et chimie des protéines [Lyon] (IBCP), Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Centre National de la Recherche Scientifique (CNRS), Université Paris Descartes - Paris 5 (UPD5) - Institut National de la Santé et de la Recherche Médicale (INSERM) - Centre National de la Recherche Scientifique (CNRS), Université Paris-Sud - Paris 11 (UP11) - Institut National de la Santé et de la Recherche Médicale (INSERM), Université de Lyon - Université de Lyon - Centre National de la Recherche Scientifique (CNRS), and Slama, Catherine
- Subjects
ComputingMilieux_MISCELLANEOUS - Abstract
International audience
- Published
- 2011
27. FVIII hydrolysis mediated by anti-FVIII autoantibodies in acquired hemophilia
- Author
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Wootla, B., Dasgupta, S., Dimitrov, J.D., Bayry, J., Levesque, H., Borg, J.Y., Borel-Derlon, A., Rao, D.N., Friboulet, A., Kaveri, S.V., Lacroix-Desmazes, S., Immunopathologie et immunointervention thérapeutique, Université Pierre et Marie Curie - Paris 6 (UPMC)-IFR58-Institut National de la Santé et de la Recherche Médicale (INSERM), Génie Enzymatique et Cellulaire (GEC), and Université de Technologie de Compiègne (UTC)-Université de Picardie Jules Verne (UPJV)-Centre National de la Recherche Scientifique (CNRS)
- Subjects
congenital, hereditary, and neonatal diseases and abnormalities ,animal diseases ,hemic and lymphatic diseases - Abstract
International audience; Acquired hemophilia is a rare hemorrhagic disorder caused by the spontaneous appearance of inhibitory autoantibodies directed against endogenous coagulation factor VIII (FVIII). Inhibitory Abs also arise in patients with congenital hemophilia A as alloantibodies directed to therapeutic FVIII. Both autoimmune and alloimmune inhibitors neutralize FVIII by steric hindrance. We have described FVIII-hydrolyzing IgG in 50% of inhibitor-positive patients with severe hemophilia A that inactivate therapeutic FVIII. In this study, we investigated the presence of autoimmune FVIII-hydrolyzing IgG in patients with acquired hemophilia. Pooled IgG from healthy donors demonstrated moderate FVIII-hydrolyzing activity (56 ± 26 µmol/min/mol). Purified IgG from 21 of 45 patients with acquired hemophilia demonstrated FVIII hydrolysis rates (mean 219 ± 94 µmol/min/mol) significantly greater than that of control IgG. Three of four patients followed over the course of the disease had rates of FVIII hydrolysis that co-evolved with inhibitory titers in plasma, suggesting that IgG-mediated FVIII hydrolysis participates, in part, in FVIII inactivation. The present work extends the scope of the diseases associated with FVIII proteolysis and points toward the importance of FVIII as a key target substrate for hydrolytic immunoglobulins. Our data suggest that elevated levels of FVIII-hydrolyzing IgG in acquired hemophilia result from the exacerbation of a physiological catalytic immune response.
- Published
- 2008
28. Induction of maturation and activation of human dendritic cells: A mechanism underlying the beneficial effect of Viscum album as complimentary therapy in cancer
- Author
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Elluru, S.R., Duong van Huyen, J.P., Delignat, S., Kazatchkine, M.D., Friboulet, A., Kaveri, S.V., Bayry, J., Immunopathologie et immunointervention thérapeutique, Université Pierre et Marie Curie - Paris 6 (UPMC)-IFR58-Institut National de la Santé et de la Recherche Médicale (INSERM), Génie Enzymatique et Cellulaire (GEC), and Université de Technologie de Compiègne (UTC)-Université de Picardie Jules Verne (UPJV)-Centre National de la Recherche Scientifique (CNRS)
- Abstract
Viscum album (VA) preparations have been used as a complimentary therapy in cancer. In addition to their cytotoxic properties, they have also been shown to have immunomodulatory properties that facilitate tumor regression. During the tumor growth, the dendritic cells (DCs) are inactivated by the production of tumor factors such as VEGF, IL-10 and PGE2. In the present study, we examine the hypothesis that the effect of VA preparations on DCs may be one of their immune modulatory mechanisms that facilitates effective tumor regression. The CD14 +ve monocytes were isolated from healthy donors by Ficoll followed by positive isolation using magnetic beads. The day 4 old immature DCs were treated with VA Qu Spez and VA M Spez at 5 and 10 microgram/ml for 48hrs. The expression of various surface molecules was analysed by flow cytometry and the cytokine secretion by BD CBA Human Inflammation kit. The functional effect of VA Qu Spez and VA M Spez was analysed by mixed lymphocyte reaction (MLR) with allogenic CD4+ T cells. DCs treated with VA M Spez and VA Qu Spez at 10 microgram/ml displayed an increased expression of antigen presenting HLA-DR molecule and co-stimulatory CD40, CD80 and CD86 molecules accompanied with an increased IL-6, IL-8 and IL-1beta secretion. The ability of the DCs treated with VA to induce the proliferation of allogeneic CD4+ T cells further substantiates the immunostimulatory effect of these preparations. The VA preparations stimulate the expression of various molecules and cytokine secretion by the human DCs. This information should assist in understanding the immunomodulatory properties of VA preparations and improving the therapeutic strategies.
- Published
- 2008
29. Catalytic IgG from patients with hemophila A inactivate therapeutic factor VIII
- Author
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Lacroix-Desmazes, S., Wootla, B., Dasgupta, S., Delignat, S., Bayry, J., Reinbold, J., Hoebeke, J., Saenko, E., Kazatchkine, M.D., Friboulet, A., Christophe, O., Nagaraja, V., Kaveri, S.N., Immunopathologie et immunointervention thérapeutique, Université Pierre et Marie Curie - Paris 6 (UPMC)-IFR58-Institut National de la Santé et de la Recherche Médicale (INSERM), Immunologie et chimie thérapeutiques (ICT), Cancéropôle du Grand Est-Centre National de la Recherche Scientifique (CNRS), School of Medicine, University of Maryland [Baltimore County] (UMBC), University of Maryland System-University of Maryland System, Génie Enzymatique et Cellulaire (GEC), Université de Technologie de Compiègne (UTC)-Université de Picardie Jules Verne (UPJV)-Centre National de la Recherche Scientifique (CNRS), Hémostase et biologie vasculaire, Université Paris-Sud - Paris 11 (UP11)-IFR93-Institut National de la Santé et de la Recherche Médicale (INSERM), Department of Microbiology and Cell Biology, Indian Institute of Science, and David, Chantal
- Subjects
congenital, hereditary, and neonatal diseases and abnormalities ,animal diseases ,hemic and lymphatic diseases - Abstract
Factor VIII (FVIII) inhibitors are anti-FVIII IgG that arise in up to 50% of the patients with hemophilia A, upon therapeutic administration of exogenous FVIII. Factor VIII inhibitors neutralize the activity of the administered FVIII by sterically hindering its interaction with molecules of the coagulation cascade, or by forming immune complexes with FVIII and accelerating its clearance from the circulation. We have shown previously that a subset of anti-factor VIII IgG hydrolyzes FVIII. FVIII-hydrolyzing IgG are detected in over 50% of inhibitor-positive patients with severe hemophilia A, and are not found in inhibitor-negative patients. Although human proficient catalytic Abs have been described in a number of inflammatory and autoimmune disorders, their pathological relevance remains elusive. We demonstrate here that the kinetics of FVIII degradation by FVIII-hydrolyzing IgG are compatible with a pathogenic role for IgG catalysts. We also report that FVIII-hydrolyzing IgG from each patient exhibit multiple cleavage sites on FVIII and that, while the specificity of cleavage varies from one patient to another, catalytic IgG preferentially hydrolyze peptide bonds containing basic amino acids.
- Published
- 2006
30. Catalytic IgG from patients with hemophilia A inactivate therapeutic factor VIII
- Author
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S. Wootla B. Dasgupta S. Delignat S. Bayry J. Reinbolt J. Hoebeke J. Saenko E. Kazatchkine M.D. Friboulet A. Christophe O. Nagaraja V. & Kaveri S.V., Lacroix-Demazes and Wencker, Suzanne
- Published
- 2006
31. Inhibition of Programmed Death 1 Ligand 1 on Dendritic Cells Enhances Mycobacterium-Mediated Interferon (IFN- ) Production Without Modulating the Frequencies of IFN- -Producing CD4+ T Cells
- Author
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Stephen-Victor, E., primary, Saha, C., additional, Sharma, M., additional, Holla, S., additional, Balaji, K. N., additional, Kaveri, S. V., additional, and Bayry, J., additional
- Published
- 2014
- Full Text
- View/download PDF
32. Mediation of T-Helper 17 Responses to Schistosomes by Dendritic Cells but Not Basophils
- Author
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Sharma, M., primary, Lecerf, M., additional, Friboulet, A., additional, Kaveri, S. V., additional, Dissous, C., additional, and Bayry, J., additional
- Published
- 2014
- Full Text
- View/download PDF
33. TLR3 essentially promotes protective class I-restricted memory CD8+ T-cell responses to Aspergillus fumigatus in hematopoietic transplanted patients
- Author
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BAYRY, J., MOUTHON, L., and KAVERI, S. V.
- Published
- 2012
- Full Text
- View/download PDF
34. Indian Science: Steps to Excellence
- Author
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Bayry, J., primary, Kaveri, S. V., additional, and Follette, P., additional
- Published
- 2011
- Full Text
- View/download PDF
35. Enhancement of the Affinity of Glucocorticoid Receptors as a Mechanism Underlying the Steroid-sparing Effect of Intravenous Immunoglobulin
- Author
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PASHOV, A., DELIGNAT, S., BAYRY, J., and KAVERI, S. V.
- Published
- 2011
- Full Text
- View/download PDF
36. Parameters that influence the prediction of epidemiological benefits of more-effective tuberculosis vaccines, drugs, and diagnosis
- Author
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Vani, J., primary, Maddur, M. S., additional, Lacroix-Desmazes, S., additional, Kaveri, S. V., additional, and Bayry, J., additional
- Published
- 2009
- Full Text
- View/download PDF
37. Factor VIII bypasses CD91/LRP for endocytosis by dendritic cells leading to T-cell activation
- Author
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Dasgupta, S., primary, Navarrete, A. M., additional, Andre, S., additional, Wootla, B., additional, Delignat, S., additional, Repesse, Y., additional, Bayry, J., additional, Nicoletti, A., additional, Saenko, E. L., additional, d'Oiron, R., additional, Jacquemin, M., additional, Saint-Remy, J.-M., additional, Kaveri, S. V., additional, and Lacroix-Desmazes, S., additional
- Published
- 2008
- Full Text
- View/download PDF
38. Comparison of the immunogenicity of different therapeutic preparations of human factor VIII in the murine model of hemophilia A
- Author
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Delignat, S., primary, Dasgupta, S., additional, Andre, S., additional, Navarrete, A.-M., additional, Kaveri, S. V., additional, Bayry, J., additional, Andre, M.-H., additional, Chtourou, S., additional, Tellier, Z., additional, and Lacroix-Desmazes, S., additional
- Published
- 2007
- Full Text
- View/download PDF
39. Rescuing CD4+CD25+ regulatory T-cell functions in rheumatoid arthritis by cytokine-targeted monoclonal antibody therapy
- Author
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BAYRY, J, primary, SIBERIL, S, additional, TRIEBEL, F, additional, TOUGH, D, additional, and KAVERI, S, additional
- Published
- 2007
- Full Text
- View/download PDF
40. Sialylated therapeutic IgG: a sweet remedy for inflammatory diseases?
- Author
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Dimitrov, J. D., primary, Bayry, J., additional, Siberil, S., additional, and Kaveri, S. V., additional
- Published
- 2007
- Full Text
- View/download PDF
41. 523 POSTER The anti-angiogenic properties of Mistletoe extracts in associated with endothelial cytotoxicity
- Author
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Elluru, S., primary, Van Huyen, J. Duong, additional, Delignat, S., additional, Prost, F., additional, Bayry, J., additional, and Kaveri, S., additional
- Published
- 2006
- Full Text
- View/download PDF
42. Is RNA interference feasible for the control of foot-and-mouth disease outbreaks?
- Author
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BAYRY, J, primary and TOUGH, D, additional
- Published
- 2005
- Full Text
- View/download PDF
43. Common variable immunodeficiency is associated with defective functions of dendritic cells
- Author
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Bayry, J., primary
- Published
- 2004
- Full Text
- View/download PDF
44. Immunoglobulin-Dependent Regulation of Dendritic Cells in the Context of Autoimmune Responses.
- Author
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Ephrem, A., Bayry, J., Misra, N., Dasgupta, S., Wootla, B., Huyen, J-P. D.V., Hassan, G., Delignat, S., Chamat, S., and Kaveri, S.V.
- Published
- 2005
- Full Text
- View/download PDF
45. Natural Autoantibodies as Tools to Predict the Outcome of Immune Response?
- Author
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KohlerKohler, H., Bayry, J., Nicoletti, A., and Kaveri, S.V.
- Subjects
- *
B cells , *AUTOANTIBODIES , *IMMUNOGLOBULINS - Abstract
Abstract Natural autoantibodies (NAbs), produced by B-1 B-cells, are directed against autoantigens and pathogens. NAbs can capture and present antigen to T helper cells thereby initiating adaptive immunities. It is proposed that screening for NAbs against pathogens will predict the strength of an antigen-induced immune response and could be used as a tool for vaccine development. [ABSTRACT FROM AUTHOR]
- Published
- 2003
- Full Text
- View/download PDF
46. Biological Functions of Catalytic Antibodies
- Author
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Latry, V., Bayry, J., Thorenoor, N., Kazatchkine, M.D., Kaveri, S.V., and Lacroix-Desmazes, S.
- Abstract
The extent of the repertoire of antibodies that may be potentially produced by the organism is determined by the number of V
H , DH , JH , VL and JL genes that encode the variable regions of immunoglobulins and by the junctional diversity that occurs at the time of somatic rearrangement. This potential repertoire includes antibodies the antigen binding site of which may recognize external as well as autologous antigens (the later being referred to as natural antibodies), or may structurally resemble the active site of enzymes and be endowed with enzymatic activity. Under physiological conditions, B-cell clones that produce antibodies naturally endowed with catalytic activity are negatively regulated and subjected to apoptosis. Catalytic antibodies are expressed only following active immunization of the organism using haptens that are analogs of transitions states for chemical reactions, or if the physiological regulatory mechanisms that control the expression of catalytic antibody-producing B-cell clones are perturbed, e.g. in the context of pregnancy or in the course of autoimmune diseases.- Published
- 2003
- Full Text
- View/download PDF
47. Mycobacterium tuberculosis Promotes Regulatory T-Cell Expansion via Induction of Programmed Death-1 Ligand 1 (PD-L1, CD274) on Dendritic Cells.
- Author
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Trinath J, Maddur MS, Kaveri SV, Balaji KN, and Bayry J
- Published
- 2012
48. Desensitization of HLA-incompatible kidney recipients.
- Author
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Bayry J, Kaveri SV, Bayry, Jagadeesh, and Kaveri, Srini V
- Published
- 2011
- Full Text
- View/download PDF
49. Do activated human dendritic cells diminish the suppressive functions of CD4+,CD25+ regulatory T cells? Comment on the article by van Amelsfort et al.
- Author
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Bayry J, Kaveri SV, and Tough DF
- Published
- 2007
- Full Text
- View/download PDF
50. Induction of apoptosis of endothelial cells by Viscum album: A role for anti-tumoral properties of mistletoe lectins
- Author
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Huyen, J. P. D., Bayry, J., Delignat, S., Gaston, A. T., Michel, O., Bruneval, P., Kazatchkine, M. D., Antonino Nicoletti, and Kaveri, S. V.
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