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1. The neurophysiological brain-fingerprint of Parkinson’s disease

2. Data and Tools Integration in the Canadian Open Neuroscience Platform

3. Subjective Cognitive Decline Is Associated With Altered Default Mode Network Connectivity in Individuals With a Family History of Alzheimer’s Disease

6. Primary Care Interventions and Strategies to Mitigate Overcrowding of Emergency Departments: A Scoping review protocol

7. Triage interventions involving primary healthcare providers to improve patient flow in emergency departments: a systematic review

8. Impact of employing primary healthcare professionals in emergency department triage on patient flow outcomes: a systematic review and meta-analysis

10. Interventions and strategies involving primary healthcare professionals to manage emergency department overcrowding: a scoping review

11. Amyloid and Tau Pathology Associations With Personality Traits, Neuropsychiatric Symptoms, and Cognitive Lifestyle in the Preclinical Phases of Sporadic and Autosomal Dominant Alzheimer’s Disease

12. White matter abnormalities and structural parietal disconnections in Alzheimer's disease

14. Regional association maps describe dynamic architecture of amyloidosis and hypometabolism in Alzheimer’s disease:Alzheimer's & Dementia: The Journal of the Alzheimer's Association

18. P4-198: Cognitive Decline is Driven By the Regional Synergism Between Amyloid Load and Hypometabolism in Mild Cognitive Impairment

22. O5‐02‐06: The Association between white Matter Hyperintensities and Memory Impairment Depends on P‐TAU Levels in a High‐Risk Cohort of Aging Cognitively Normal Persons

23. P3-251: Novel Toolbox for Performing Voxel-Wise Generalized Linear Regression with Mulitple Volumetric Covariates in Longitudinal Data

24. O4-01-03: AMYLOID-B AND HYPERPHOSPHORYLATED TAU SYNERGY DRIVES CLINICAL PROGRESSION TO ALZHEIMER’S DISEASE

26. IC-P-157: Novel Toolbox for Performing Voxel-Wise Generalized Linear Regression With Mulitple Volumetric Covariates in Longitudinal Data

29. Association between cortical thickness and CSF biomarkers in mild cognitive impairment and Alzheimer’s disease

32. P1‐058: Genome‐wide association study in PET imaging longitudinal data

33. IC-P-155: Baseline CSF p-tau and fibrillary amyloid load predict mesial temporal hypometabolism in 24-months follow-up in cognitively normal subjects

34. P3-180: Comparison of global and voxel-based diagnostic classification using [18 F]florbetapir ROC estimates

36. O5-01-06: Baseline CSF p-tau and fibrillary amyloid load predict mesial temporal hypometabolism in 24 months' follow-up in cognitively normal subjects

37. P4-097: Should a global or a regional measure of amyloidosis be used in a longitudinal study?

38. IC-P-012: Should a global or a regional measure of amyloidosis be used in a longitudinal study?

39. IC-P-038: Genome-wide association study in PET imaging longitudinal data

40. IC-P-011: Comparison of global and voxel-based diagnostic classification using [18 F]florbetapir ROC estimates

42. P3-186: REGIONAL ASSOCIATION MAPS DESCRIBE DYNAMIC ARCHITECTURE OF AMYLOIDOSIS AND HYPOMETABOLISM IN ALZHEIMER'S DISEASE

43. IC-P-043: NEURODEGENERATION AND CORTICAL ATROPHY IN [18F]FLORBETAPIR ACCUMULATORS AND NON-ACCUMULATORS

44. P4-130: CORRELATIONS BETWEEN VARIOUS MOCA COGNITIVE DOMAIN ASSESSMENTS AND REGIONAL BRAIN FDG-PET HYPOMETABOLISM

46. P4-141: NEURODEGENERATION ASSOCIATED WITH LONGITUDINAL CHANGES OFAβ1-42 AND FIBRILLARY AMYLOID

47. IC-P-194: REGIONAL ASSOCIATION MAPS DESCRIBE DYNAMIC ARCHITECTURE OF AMYLOIDOSIS AND HYPOMETABOLISM IN ALZHEIMER'S DISEASE

48. O2-03-05: HYPOMETABOLISM AND CORTICAL ATROPHY IN [18F]FLORBETAPIR ACCUMULATORS AND NON-ACCUMULATORS

49. P1-049: EPISTASIS ANALYSES INDICATE ASSOCIATION BETWEEN CEREBRAL AMYLOID DEPOSITION AND GENES INVOLVED IN IMMUNORESPONSE

50. P4-132: WHITE MATTER ABNORMALITIES AND STRUCTURAL PARIETAL DISCONNECTIONS IN ALZHEIMER'S DISEASE

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