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1,739 results on '"Bente, K."'

1. Optical properties of Cu(In, Ga)(S, Se)2 films for solar cells

Author

Tivanov, M., Zareţcaia, E., Gremenok, V., Ivanova, V., Mudrîi, A., Ivaniukovici, A., Zalesski, V., Zikotinski, S., and Bente, K.

Subjects
Physics, QC1-999, Electronics, TK7800-8360
Abstract

In this paper, we present structural and optical properties of single-phase Cu(In, Ga)(S, Se)2 alloys, which have been prepared using a novel selenization/sulfurization growth process to react copper-indium-gallium alloy films. The grown scheme differs critically from standard two-step grown processes and was carried out without toxic H2S and H2Se gases. The calculated band gap values for layers with varying sulfur content (i.e. S/(S Se) = 0.16 and 0.19), determined from optical transmission and reflectance measurements, were found to be 1.17 and 1.23 eV respectively. The low temperature PL measurements also confirmed the shift in the band gap of the CIGSS absorber films with sulfur incorporation. In summary, this reaction process produced single-phase CIGSS thin films with controlled sulfur amount suitable for photovoltaic application.

Published
2007

6. Effects of different doses of exercise and diet-induced weight loss on beta-cell function in type 2 diabetes (DOSE-EX): a randomized clinical trial

Author

Legaard, Grit E., Lyngbæk, Mark P. P., Almdal, Thomas P., Karstoft, Kristian, Bennetsen, Sebastian L., Feineis, Camilla S., Nielsen, Nina S., Durrer, Cody G., Liebetrau, Benedikte, Nystrup, Ulrikke, Østergaard, Martin, Thomsen, Katja, Trinh, Beckey, Solomon, Thomas P. J., Van Hall, Gerrit, Brønd, Jan Christian, Holst, Jens J., Hartmann, Bolette, Christensen, Robin, Pedersen, Bente K., and Ried-Larsen, Mathias

Published
2023
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7. Exercise training remodels inguinal white adipose tissue through adaptations in innervation, vascularization, and the extracellular matrix

Author

Nigro, Pasquale, Vamvini, Maria, Yang, Jiekun, Caputo, Tiziana, Ho, Li-Lun, Carbone, Nicholas P., Papadopoulos, Danae, Conlin, Royce, He, Jie, Hirshman, Michael F., White, Joseph D., Robidoux, Jacques, Hickner, Robert C., Nielsen, Søren, Pedersen, Bente K., Kellis, Manolis, Middelbeek, Roeland J.W., and Goodyear, Laurie J.

Published
2023
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9. Motion of magnetotactic bacteria swarms in an external field

Author

Bente, K., Kitenbergs, G., Kriman, D., Erglis, K., Belovs, M., Faivre, D., and Cebers, A.

Subjects
Condensed Matter - Soft Condensed Matter
Abstract

Magnetotactic bacteria moving on circular orbits form hydrodynamically bound states. When close to a surface and with the tilting of the field in a direction close to the perpendicular to this surface these swarms move perpendicularly to the tilting angle. We describe quantitatively this motion by a continuum model with couple stress arising from the torques produced by the rotary motors of the amphitrichous magnetotactic bacteria. The model not only correctly describes the change of direction of swarm motion while inverting the tangential field but also predicts reasonable value of the torque produced by the rotary motors.

Published
2017

10. Exerkines in health, resilience and disease

Author

Chow, Lisa S., Gerszten, Robert E., Taylor, Joan M., Pedersen, Bente K., van Praag, Henriette, Trappe, Scott, Febbraio, Mark A., Galis, Zorina S., Gao, Yunling, Haus, Jacob M., Lanza, Ian R., Lavie, Carl J., Lee, Chih-Hao, Lucia, Alejandro, Moro, Cedric, Pandey, Ambarish, Robbins, Jeremy M., Stanford, Kristin I., Thackray, Alice E., Villeda, Saul, Watt, Matthew J., Xia, Ashley, Zierath, Juleen R., Goodpaster, Bret H., and Snyder, Michael P.

Published
2022
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11. Exercise suppresses tumor growth independent of high fat food intake and associated immune dysfunction

Author

Pernille Hojman, Rikke Stagaard, Emi Adachi-Fernandez, Atul S. Deshmukh, Andreas Mund, Caroline H. Olsen, Lena Keller, Bente K. Pedersen, and Julie Gehl

Subjects
Medicine, Science
Abstract

Abstract Epidemiological data suggest that exercise training protects from cancer independent of BMI. Here, we aimed to elucidate mechanisms involved in voluntary wheel running-dependent control of tumor growth across chow and high-fat diets. Access to running wheels decreased tumor growth in B16F10 tumor-bearing on chow (− 50%) or high-fat diets (− 75%, p

Published
2022
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13. Altered brown fat thermoregulation and enhanced cold-induced thermogenesis in young, healthy, winter-swimming men

Author

Søberg, Susanna, Löfgren, Johan, Philipsen, Frederik E., Jensen, Michal, Hansen, Adam E., Ahrens, Esben, Nystrup, Kristin B., Nielsen, Rune D., Sølling, Christine, Wedell-Neergaard, Anne-Sophie, Berntsen, Marianne, Loft, Annika, Kjær, Andreas, Gerhart-Hines, Zachary, Johannesen, Helle H., Pedersen, Bente K., Karstoft, Kristian, and Scheele, Camilla

Published
2021
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17. Skeletal muscle action of estrogen receptor α is critical for the maintenance of mitochondrial function and metabolic homeostasis in females

Author

Ribas, Vicent, Drew, Brian G, Zhou, Zhenqi, Phun, Jennifer, Kalajian, Nareg Y, Soleymani, Teo, Daraei, Pedram, Widjaja, Kevin, Wanagat, Jonathan, de Aguiar Vallim, Thomas Q, Fluitt, Amy H, Bensinger, Steven, Le, Thuc, Radu, Caius, Whitelegge, Julian P, Beaven, Simon W, Tontonoz, Peter, Lusis, Aldons J, Parks, Brian W, Vergnes, Laurent, Reue, Karen, Singh, Harpreet, Bopassa, Jean C, Toro, Ligia, Stefani, Enrico, Watt, Matthew J, Schenk, Simon, Akerstrom, Thorbjorn, Kelly, Meghan, Pedersen, Bente K, Hewitt, Sylvia C, Korach, Kenneth S, and Hevener, Andrea L

Subjects
Medical Biotechnology, Engineering, Biomedical and Clinical Sciences, Biomedical Engineering, Genetics, Women's Health, Estrogen, Nutrition, Obesity, Diabetes, Aging, 2.1 Biological and endogenous factors, 1.1 Normal biological development and functioning, 5.1 Pharmaceuticals, Animals, Autophagy, Calcium-Binding Proteins, DNA Replication, DNA, Mitochondrial, Dynamins, Estrogen Receptor alpha, Female, Gene Deletion, Glucose, Homeostasis, Humans, Insulin, Intracellular Signaling Peptides and Proteins, Lipid Metabolism, Mice, Mice, Knockout, Mitochondria, Muscle, Mitochondrial Dynamics, Muscle Proteins, Muscle, Skeletal, Organ Specificity, Oxidation-Reduction, Oxidative Stress, Reactive Oxygen Species, Signal Transduction, Biological Sciences, Medical and Health Sciences, Medical biotechnology, Biomedical engineering
Abstract

Impaired estrogen receptor α (ERα) action promotes obesity and metabolic dysfunction in humans and mice; however, the mechanisms underlying these phenotypes remain unknown. Considering that skeletal muscle is a primary tissue responsible for glucose disposal and oxidative metabolism, we established that reduced ERα expression in muscle is associated with glucose intolerance and adiposity in women and female mice. To test this relationship, we generated muscle-specific ERα knockout (MERKO) mice. Impaired glucose homeostasis and increased adiposity were paralleled by diminished muscle oxidative metabolism and bioactive lipid accumulation in MERKO mice. Aberrant mitochondrial morphology, overproduction of reactive oxygen species, and impairment in basal and stress-induced mitochondrial fission dynamics, driven by imbalanced protein kinase A-regulator of calcineurin 1-calcineurin signaling through dynamin-related protein 1, tracked with reduced oxidative metabolism in MERKO muscle. Although muscle mitochondrial DNA (mtDNA) abundance was similar between the genotypes, ERα deficiency diminished mtDNA turnover by a balanced reduction in mtDNA replication and degradation. Our findings indicate the retention of dysfunctional mitochondria in MERKO muscle and implicate ERα in the preservation of mitochondrial health and insulin sensitivity as a defense against metabolic disease in women.

Published
2016

18. Pharmacological but not physiological GDF15 suppresses feeding and the motivation to exercise

Author

Anders B. Klein, Trine S. Nicolaisen, Niels Ørtenblad, Kasper D. Gejl, Rasmus Jensen, Andreas M. Fritzen, Emil L. Larsen, Kristian Karstoft, Henrik E. Poulsen, Thomas Morville, Ronni E. Sahl, Jørn W. Helge, Jens Lund, Sarah Falk, Mark Lyngbæk, Helga Ellingsgaard, Bente K. Pedersen, Wei Lu, Brian Finan, Sebastian B. Jørgensen, Randy J. Seeley, Maximilian Kleinert, Bente Kiens, Erik A. Richter, and Christoffer Clemmensen

Subjects
Science
Abstract

The physiological role of GDF15 remains poorly defined. Here, the authors show that circulating GDF15 increases in response to prolonged exercise, but that this exercise-induced GDF15, unlike pharmacological GDF15, does not affect post-exercise food intake or exercise motivation.

Published
2021
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20. The interaction between metformin and physical activity on postprandial glucose and glucose kinetics: a randomised, clinical trial

Author

Pilmark, Nanna S., Lyngbæk, Mark, Oberholzer, Laura, Elkjær, Ida, Petersen-Bønding, Christina, Kofoed, Katja, Siebenmann, Christoph, Kellenberger, Katja, van Hall, Gerrit, Abildgaard, Julie, Ellingsgaard, Helga, Lauridsen, Carsten, Ried-Larsen, Mathias, Pedersen, Bente K., Hansen, Katrine B., and Karstoft, Kristian

Published
2021
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24. Reply to ‘Lactate as a major myokine and exerkine’

Author

Chow, Lisa S., Gerszten, Robert E., Taylor, Joan M., Pedersen, Bente K., van Praag, Henriette, Trappe, Scott, Febbraio, Mark A., Galis, Zorina S., Gao, Yunling, Haus, Jacob M., Lanza, Ian R., Lavie, Carl J., Lee, Chih-Hao, Lucia, Alejandro, Moro, Cedric, Pandey, Ambarish, Robbins, Jeremy M., Stanford, Kristin I., Thackray, Alice E., Villeda, Saul, Watt, Matthew J., Xia, Ashley, Zierath, Juleen R., Goodpaster, Bret H., and Snyder, Michael

Published
2022
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26. The effects of different doses of exercise on pancreatic β-cell function in patients with newly diagnosed type 2 diabetes: study protocol for and rationale behind the “DOSE-EX” multi-arm parallel-group randomised clinical trial

Author

Lyngbaek, Mark P. P., Legaard, Grit E., Bennetsen, Sebastian L., Feineis, Camilla S., Rasmussen, Villads, Moegelberg, Nana, Brinkløv, Cecilie F., Nielsen, Anette B., Kofoed, Katja S., Lauridsen, Carsten A., Ewertsen, Caroline, Poulsen, Henrik E., Christensen, Robin, Van Hall, Gerrit, Karstoft, Kristian, Solomon, Thomas P. J., Ellingsgaard, Helga, Almdal, Thomas P., Pedersen, Bente K., and Ried-Larsen, Mathias

Published
2021
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27. Metabolic profile in patients with newly diagnosed bipolar disorder and their unaffected first-degree relatives

Author

Klara Coello, Maj Vinberg, Filip K. Knop, Bente K. Pedersen, Roger S. McIntyre, Lars V. Kessing, and Klaus Munkholm

Subjects
Bipolar disorder, Insulin resistance, Metabolic syndrome X, Co-morbidity, Neurosciences. Biological psychiatry. Neuropsychiatry, RC321-571, Neurophysiology and neuropsychology, QP351-495
Abstract

Abstract Objective The prevalence of metabolic syndrome and insulin resistance is twice as high in patients with bipolar disorder compared with the general population, and possibly associated with a disabling illness trajectory of bipolar disorder, an increased risk of cardiovascular disease and premature death. Despite these detrimental effects, the prevalence of metabolic syndrome and insulin resistance in patients newly diagnosed with bipolar disorder and their unaffected first-degree relatives is largely unknown. Methods In a cross-sectional study of 206 patients with newly diagnosed bipolar disorder, 50 of their unaffected first-degree relatives and 109 healthy age- and sex-matched individuals, we compared the prevalence of metabolic syndrome and insulin resistance (HOMA-IR). In patients with bipolar disorder, we further investigated illness and medication variables associated with the metabolic syndrome and insulin resistance. Results Higher rates of metabolic syndrome (odds ratio = 3.529, 95% CI 1.378–9.041, P = 0.009) and levels of insulin resistance (B = 1.203, 95% CI 1.059–1.367, P = 0.005) were found in patients newly diagnosed with bipolar disorder, but not in their unaffected first-degree relatives compared with matched healthy individuals (data adjusted for sex and age). Most patients with bipolar disorder (94.7%) were diagnosed within the preceding 2 years, and the average illness duration, defined as time from first mood episode, was 10 years. Conclusion Our findings of elevated prevalence of metabolic syndrome and insulin resistance in patients with newly diagnosed bipolar disorder highlight the importance of screening for these conditions at an early stage to employ adequate and early care reducing the risk of cardiovascular disease and premature death.

Published
2019
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36. TGF-β2 is an exercise-induced adipokine that regulates glucose and fatty acid metabolism

Author

Takahashi, Hirokazu, Alves, Christiano R. R., Stanford, Kristin I., Middelbeek, Roeland J. W., Nigro, Pasquale, Ryan, Rebecca E., Xue, Ruidan, Sakaguchi, Masaji, Lynes, Matthew D., So, Kawai, Mul, Joram D., Lee, Min-Young, Balan, Estelle, Pan, Hui, Dreyfuss, Jonathan M., Hirshman, Michael F., Azhar, Mohamad, Hannukainen, Jarna C., Nuutila, Pirjo, Kalliokoski, Kari K., Nielsen, Søren, Pedersen, Bente K., Kahn, C. Ronald, Tseng, Yu-Hua, and Goodyear, Laurie J.

Published
2019
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38. Dysregulation of a novel miR-23b/27b-p53 axis impairs muscle stem cell differentiation of humans with type 2 diabetes

Author

Tora I. Henriksen, Peter K. Davidsen, Maria Pedersen, Heidi S. Schultz, Ninna S. Hansen, Therese J. Larsen, Allan Vaag, Bente K. Pedersen, Søren Nielsen, and Camilla Scheele

Subjects
Satellite cell, MicroRNA, Diabetes, Muscle, Human, Myogenesis, muscle stem cells, p53, miR-23b, miR-27b, Internal medicine, RC31-1245
Abstract

Objective: MicroRNAs (miRNAs) are increasingly recognized as fine-tuning regulators of metabolism, and are dysregulated in several disease conditions. With their capacity to rapidly change gene expression, miRNAs are also important regulators of development and cell differentiation. In the current study, we describe an impaired myogenic capacity of muscle stem cells isolated from humans with type 2 diabetes (T2DM) and assess whether this phenotype is regulated by miRNAs. Methods: We measured global miRNA expression during in vitro differentiation of muscle stem cells derived from T2DM patients and healthy controls. Results: The mir-23b/27b cluster was downregulated in the cells of the patients, and a pro-myogenic effect of these miRNAs was mediated through the p53 pathway, which was concordantly dysregulated in the muscle cells derived from humans with T2DM. Conclusions: Our results indicate that we have identified a novel pathway for coordination of myogenesis, the miR-23b/27b-p53 axis that, when dysregulated, potentially contributes to a sustained muscular dysfunction in T2DM.

Published
2017
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44. List of Contributors

Author

Aguirre, Leixuri, primary, Alemán-Mateo, Heliodoro, additional, Alway, Stephen E., additional, Antoun, Sami, additional, Arena, Ross, additional, Atkins, Janice L., additional, Bigard, Xavier, additional, Boirie, Yves, additional, Bond, Samantha, additional, Borghi Silva, Audrey, additional, Breuillard, Charlotte, additional, Capel, Frederic, additional, Cruzat, Vinicius F., additional, Dardevet, Dominique, additional, De Bandt, Jean-Pascal, additional, Domingues-Faria, Carla, additional, Farges, Marie-Chantal, additional, Fernández-Quintela, Alfredo, additional, Girgis, Christian M., additional, Gómez-Zorita, Saioa, additional, Goncalves-Mendes, Nicolas, additional, González, Marcela, additional, González-Arnay, Emilio, additional, González-Reimers, Emilio, additional, Gorissen, Stefan H.M., additional, Goron, Arthur, additional, Guillet, Christelle, additional, Handschin, Christoph, additional, Jeffries, Owen, additional, Le Bacquer, Olivier, additional, Margaritis, Irène, additional, Martín-González, Candelaria, additional, Mazzuco, Adriana, additional, Milton-Laskibar, Iñaki, additional, Moinard, Christophe, additional, Moro, Cedric, additional, Mosoni, Laurent, additional, Papet, Isabelle, additional, Patterson, Stephen D., additional, Pedersen, Bente K., additional, Pérez-Schindler, Joaquín, additional, Peyron, Marie-Agnès, additional, Phillips, Shane A., additional, Phillips, Stuart M., additional, Polakof, Sergio, additional, Portillo, Maria P., additional, Quintero-Platt, Geraldine, additional, Rémond, Didier, additional, Romero-Acevedo, Lucía, additional, Rossi, Andrea P., additional, Rubele, Sofia, additional, Ruiz Valenzuela, Roxana E., additional, Santolaria-Fernández, Francisco, additional, Savary-Auzeloux, Isabelle, additional, Scott, David, additional, Severin, Richard, additional, Smith, Gordon I., additional, Sorrentino, Matthew J., additional, Tachibana, Nobuhiko, additional, Waldron, Mark, additional, Walrand, Stéphane, additional, Wanezaki, Satoshi, additional, Yagasaki, Kazumi, additional, and Zamboni, Mauro, additional

Published
2019
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46. List of Contributors

Author

Bache, Robert J., primary, Baldwin, Kenneth M., additional, Barclay, Chris J., additional, Benson, Alan P., additional, Bowen, T. Scott, additional, Burnley, Mark, additional, Celichowski, Jan, additional, Chabowski, Adrian, additional, Chapman, Brian, additional, Craig, Jesse C., additional, Davis, Jonathan P., additional, Davison, Glen, additional, Degens, Hans, additional, di Prampero, Pietro E., additional, Duchateau, Jacques, additional, Duda, Krzysztof, additional, Duncker, Dirk J., additional, Enoka, Roger M., additional, Fell, Marc J., additional, Fisher, James P., additional, Gao, Lie, additional, Górski, Jan, additional, Grassi, Bruno, additional, Haddad, Fadia, additional, Hammond, Kelly M., additional, Han, June-Chiew, additional, Hearris, Mark A., additional, Heymsfield, Steven B., additional, Janssen, Paul M.L., additional, Jones, Andrew M., additional, Jones, Arwel Wyn, additional, Kemmler, Wolfgang, additional, Kemp, Graham J., additional, Kilarski, Wincenty, additional, Koltai, Erika, additional, Krutki, Piotr, additional, Laughlin, M. Harold, additional, Legård, Grit E., additional, Loiselle, Denis S., additional, Majerczak, Joanna, additional, Merkus, Daphne, additional, Morton, James P., additional, Nieckarz, Zenon, additional, Osgnach, Cristian, additional, Pedersen, Bente K., additional, Poole, David C., additional, Radak, Zsolt, additional, Roseguini, Bruno Tesini, additional, Rossiter, Harry B., additional, Secher, Niels H., additional, Severs, Nicholas J., additional, Szkutnik, Zbigniew, additional, Taberner, Andrew J., additional, Tikunova, Svetlana B., additional, Tran, Kenneth, additional, Vanhatalo, Anni, additional, von Stengel, Simon, additional, Wang, Hanjun, additional, Zoladz, Jerzy A., additional, and Zucker, Irving H., additional

Published
2019
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48. Effects of different doses of exercise and diet-induced weight loss on beta-cell function in type 2 diabetes (DOSE-EX):a randomized clinical trial

Author

Legaard, Grit E., Lyngbæk, Mark P.P., Almdal, Thomas P., Karstoft, Kristian, Bennetsen, Sebastian L., Feineis, Camilla S., Nielsen, Nina S., Durrer, Cody G., Liebetrau, Benedikte, Nystrup, Ulrikke, Østergaard, Martin, Thomsen, Katja, Trinh, Beckey, Solomon, Thomas P.J., Van Hall, Gerrit, Brønd, Jan Christian, Holst, Jens J., Hartmann, Bolette, Christensen, Robin, Pedersen, Bente K., Ried-Larsen, Mathias, Legaard, Grit E., Lyngbæk, Mark P.P., Almdal, Thomas P., Karstoft, Kristian, Bennetsen, Sebastian L., Feineis, Camilla S., Nielsen, Nina S., Durrer, Cody G., Liebetrau, Benedikte, Nystrup, Ulrikke, Østergaard, Martin, Thomsen, Katja, Trinh, Beckey, Solomon, Thomas P.J., Van Hall, Gerrit, Brønd, Jan Christian, Holst, Jens J., Hartmann, Bolette, Christensen, Robin, Pedersen, Bente K., and Ried-Larsen, Mathias

Abstract

Diet-induced weight loss is associated with improved beta-cell function in people with type 2 diabetes (T2D) with remaining secretory capacity. It is unknown if adding exercise to diet-induced weight loss improves beta-cell function and if exercise volume is important for improving beta-cell function in this context. Here, we carried out a four-armed randomized trial with a total of 82 persons (35% females, mean age (s.d.) of 58.2 years (9.8)) with newly diagnosed T2D (<7 years). Participants were randomly allocated to standard care (n = 20), calorie restriction (25% energy reduction; n = 21), calorie restriction and exercise three times per week (n = 20), or calorie restriction and exercise six times per week (n = 21) for 16 weeks. The primary outcome was beta-cell function as indicated by the late-phase disposition index (insulin secretion multiplied by insulin sensitivity) at steady-state hyperglycemia during a hyperglycemic clamp. Secondary outcomes included glucose-stimulated insulin secretion and sensitivity as well as the disposition, insulin sensitivity, and secretion indices derived from a liquid mixed meal tolerance test. We show that the late-phase disposition index during the clamp increases more in all three intervention groups than in standard care (diet control group, 58%; 95% confidence interval (CI), 16 to 116; moderate exercise dose group, 105%; 95% CI, 49 to 182; high exercise dose group, 137%; 95% CI, 73 to 225) and follows a linear dose–response relationship (P > 0.001 for trend). We report three serious adverse events (two in the control group and one in the diet control group), as well as adverse events in two participants in the diet control group, and five participants each in the moderate and high exercise dose groups. Overall, adding an exercise intervention to diet-induced weight loss improves glucose-stimulated beta-cell function in people with newly diagnosed T2D in an exercise dose-dependent manner (NCT03769883).

Published
2023

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