36 results on '"Bertuccio, Claudia A."'
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2. Histone deacetylase inhibitors (HDACi) increase expression of KCa2.3 (SK3) in primary microvascular endothelial cells
3. Intermediate conductance, Ca2+-activated K+ channels: a novel target for chronic renal diseases
4. KCa3.1 in Epithelia
5. Polycystin-1 cleavage and the regulation of transcriptional pathways
6. Mechanisms of Na+-K+-ATPase phosphorylation by PKC in the medullary thick ascending limb of Henle in the rat
7. Endogenous vasopressin regulates Na-K-ATPase and [Na.sup.+]-[K.sup.+]-[Cl.sup.-] cotransporter rbsc-1 in rat outer medulla
8. Plasma membrane insertion of KCa2.3 (SK3) is dependent upon the SNARE proteins, syntaxin-4 and SNAP23
9. Drug Authorization for Sofosbuvir/Ledipasvir (Harvoni) for Chronic HCV Infection in a Real-World Cohort: A New Barrier in the HCV Care Cascade
10. Relevance of VEGF and Nephrin Expression in Glomerular Diseases
11. Plasma Membrane Insertion of KCa2.3 (SK3) is Dependent upon the SNARE Proteins, Syntaxin‐4 and SNAP‐23
12. Intermediate conductance, Ca2+-activated K+ channels: a novel target for chronic renal diseases
13. Anterograde Trafficking of KCa3.1 in Polarized Epithelia Is Rab1- and Rab8-Dependent and Recycling Endosome-Independent
14. Mechanisms of PKC-Dependent Na+K+ATPase Phosphorylation in the Rat Kidney with Chronic Renal Failure
15. Polycystin-1 cleavage and the regulation of transcriptional pathways
16. Rab22 is involved in regulation of the epithelial sodium channel (ENaC)
17. Characterization of the trafficking of a Ca2+‐activated K+ channel (KCa3.1) in polarized epithelial cells
18. Dynamin- and Rab5-Dependent Endocytosis of a Ca2+-Activated K+ Channel, KCa2.3
19. Acute Podocyte Vascular Endothelial Growth Factor (VEGF-A) Knockdown Disrupts alphaVbeta3 Integrin Signaling in the Glomerulus
20. Vascular Endothelial Growth Factor Receptor 2 Direct Interaction with Nephrin Links VEGF-A Signals to Actin in Kidney Podocytes
21. Induction of Podocyte VEGF164 Overexpression at Different Stages of Development Causes Congenital Nephrosis or Steroid-Resistant Nephrotic Syndrome
22. Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease
23. Polycystin-1 C-terminal Cleavage Is Modulated by Polycystin-2 Expression
24. Regulation of Polycystin‐1 C terminal cleavage by Polycystin‐2
25. Mechanisms of Na + -K + -ATPase phosphorylation by PKC in the medullary thick ascending limb of Henle in the rat
26. Endogenous vasopressin regulates Na-K-ATPase and Na+-K+-Cl−cotransporterrbsc-1in rat outer medulla
27. Intermediate conductance, Ca2+-activated K+channels: a novel target for chronic renal diseases
28. POLYCYSTIN-1 C TERMINUS CLEAVAGE AND ITS RELATION WITH POLYCYSTIN-2, TWO PROTEINS INVOLVED IN POLYCYSTIC KIDNEY DISEASE.
29. Dynamin- and Rab5-Dependent Endocytosis of a Ca2+-Activated K+ Channel, KCa2.3.
30. Acute Podocyte Vascular Endothelial Growth Factor (VEGF-A) Knockdown Disrupts alphaVbeta3 Integrin Signaling in the Glomerulus.
31. Mechanisms of PKC-Dependent Na K ATPase Phosphorylation in the Rat Kidney with Chronic Renal Failure.
32. Mechanisms of Na+-K+-ATPase phosphorylation by PKC in the medullary thick ascending limb of Henle in the rat.
33. Endogenous vasopressin regulates Na-K-ATPase and Na[sup +]-K[sup +]-Cl[sup -] cotransporter rbsc1 in rat outer medulla.
34. Induction of Podocyte VEGF164Overexpression at Different Stages of Development Causes Congenital Nephrosis or Steroid-Resistant Nephrotic Syndrome
35. Histone deacetylase inhibitors (HDACi) increase expression of KCa2.3 (SK3) in primary microvascular endothelial cells.
36. Polycystin-1C terminus cleavage and its relation with polycystin-2, two proteins involved in polycystic kidney disease.
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