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6. Glucose-dependent partitioning of arginine to the urea cycle protects β-cells from inflammation

9. Data from MDM2 and MDM4 Are Therapeutic Vulnerabilities in Malignant Rhabdoid Tumors

10. Supplementary Figures S1-S7 from MDM2 and MDM4 Are Therapeutic Vulnerabilities in Malignant Rhabdoid Tumors

11. Supplementary Tables S1-S7 from MDM2 and MDM4 Are Therapeutic Vulnerabilities in Malignant Rhabdoid Tumors

12. Supplementary Methods on Data Analysis from MDM2 and MDM4 Are Therapeutic Vulnerabilities in Malignant Rhabdoid Tumors

18. Generation of Potent and Stable GLP-1 Analogues Via “Serine Ligation”

21. Mucosal delivery of a double-stapled RSV peptide prevents nasopulmonary infection

22. Glucose metabolism and pyruvate carboxylase enhance glutathione synthesis and restrict oxidative stress in pancreatic islets

24. A stapled BIM peptide overcomes apoptotic resistance in hematologic cancers

25. BAX activation is initiated at a novel interaction site

26. Dual role of proapoptotic BAD in insulin secretion and beta cell survival

29. Binding and transport of SFPQ-RNA granules by KIF5A/KLC1 motors promotes axon survival

30. Manipulation of temperature to improve solubility of hydrophobic proteins and cocrystallization with matrix for analysis by MALDI-TOF mass spectrometry

33. Fast Transport of RNA Granules by Direct Interactions with KIF5A/KLC1 Motors Prevents Axon Degeneration

34. Abstract 2867: MDM2 and MDM4 are therapeutic vulnerabilities in malignant rhabdoid tumors

35. MDM2 and MDM4 Are Therapeutic Vulnerabilities in Malignant Rhabdoid Tumors

38. Genome-scale CRISPR-Cas9 screen identifies druggable dependencies in TP53 wild-type Ewing sarcoma

40. Dynamic Regulation of Long-Chain Fatty Acid Oxidation by a Noncanonical Interaction between the MCL-1 BH3 Helix and VLCAD

50. Allosteric inhibition of antiapoptotic MCL-1

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