174 results on '"Björkman, Lena"'
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2. Larixol is not an inhibitor of Gαi containing G proteins and lacks effect on signaling mediated by human neutrophil expressed formyl peptide receptors
3. GRK2 selectively attenuates the neutrophil NADPH-oxidase response triggered by β-arrestin recruiting GPR84 agonists
4. Minor Genetic Overlap Among Rheumatoid Arthritis, Myocardial Infarction, and Myocardial Infarction Risk Determinants.
5. A Novel Fluorescent Immuno-Lectin Assay to Identify Osteoarthritis Associated Glycoforms of Lubricin
6. Function and regulation of GPR84 in human neutrophils.
7. Genome-wide investigation of persistence with methotrexate treatment in early rheumatoid arthritis.
8. Author Correction: Cathepsin g Degrades Both Glycosylated and Unglycosylated Regions of Lubricin, a Synovial Mucin
9. Cathepsin g Degrades Both Glycosylated and Unglycosylated Regions of Lubricin, a Synovial Mucin
10. Genome-wide investigation of persistence with methotrexate treatment in early rheumatoid arthritis
11. AZ2158 is a more potent formyl peptide receptor 1 inhibitor than the commonly used peptide antagonists in abolishing neutrophil chemotaxis
12. Severe chronic non-bacterial osteomyelitis in combination with total MPO deficiency and responsiveness to TNFα inhibition.
13. Function and regulation of GPR84 in human neutrophils
14. Functional characteristics of circulating granulocytes in severe congenital neutropenia caused by ELANE mutations
15. The ketone body acetoacetate activates human neutrophils through FFA2R
16. G protein coupled pattern recognition receptors expressed in neutrophils : Recognition, activation/modulation, signaling and receptor regulated functions
17. Truncated lubricin glycans in osteoarthritis stimulate the synoviocyte secretion of VEGFA, IL-8, and MIP-1α: Interplay between O-linked glycosylation and inflammatory cytokines
18. The ketone body acetoacetate activates human neutrophils through FFAR2.
19. G protein coupled pattern recognition receptors expressed in neutrophils: Recognition, activation/modulation, signaling and receptor regulated functions.
20. Collection of In Vivo Transmigrated Neutrophils from Human Skin
21. Neutrophils produce interleukin-17B in rheumatoid synovial tissue
22. The level of synovial human VEGFA, IL-8 and MIP-1α correlate with truncation of lubricin glycans in osteoarthritis
23. The Two Formyl Peptide Receptors Differently Regulate GPR84-Mediated Neutrophil NADPH Oxidase Activity
24. The two formyl peptide receptors differently regulate GPR84-mediated neutrophil NADPH-oxidase activity
25. Decrease of core 2 O-glycans on synovial lubricin in osteoarthritis reduces galectin-3 mediated crosslinking
26. The neutrophil subset defined by CD177 expression is preferentially recruited to gingival crevicular fluid in periodontitis
27. Interdependent allosteric free fatty acid receptor 2 modulators synergistically induce functional selective activation and desensitization in neutrophils
28. In Vivo Transmigrated Human Neutrophils Are Highly Primed for Intracellular Radical Production Induced by Monosodium Urate Crystals
29. Neutrophil Signaling That Challenges Dogmata of G Protein-Coupled Receptor Regulated Functions
30. Increased CD11b and Decreased CD62L in Blood and Airway Neutrophils from Long-Term Smokers with and without COPD
31. Core-2 O-glycans are required for galectin-3 interaction with the osteoarthritis related protein lubricin
32. The proinflammatory activity of recombinant serum amyloid A is not shared by the endogenous protein in the circulation
33. Interdependent Allosteric FFA2R Modulators Synergistically Induce Functional Selective Activation and Desensitization in Neutrophils
34. Cathepsin g degrades synovial fluid lubricin: relevance for osteoarthritis pathogenesis
35. Data on the NADPH-oxidase activity induced by WKYMVm and galectin-3 in bone marrow derived and exudated neutrophils isolated from four different mouse strains
36. Phagocyte-derived reactive oxygen species as suppressors of inflammatory disease
37. Functional selective ATP receptor signaling controlled by the free fatty acid receptor 2 through a novel allosteric modulation mechanism
38. Neutrophil recruitment to inflamed joints can occur without cellular priming
39. The neutrophil subset defined by CD177 expression is preferentially recruited to gingival crevicular fluid in periodontitis.
40. The two neutrophil members of the formylpeptide receptor family activate the NADPH-oxidase through signals that differ in sensitivity to a gelsolin derived phosphoinositide-binding peptide
41. Lubricin binds cartilage proteins, cartilage oligomeric matrix protein, fibronectin and collagen II at the cartilage surface
42. Neutrophil priming that turns natural FFA2R agonists into potent activators of the superoxide generating NADPH-oxidase
43. Similarities and differences between the responses induced in human phagocytes through activation of the medium chain fatty acid receptor GPR84 and the short chain fatty acid receptor FFA2R
44. Allosteric receptor modulation of FFA2R turns natural agonists into potent activators of the superoxide generating neutrophil NADPH-oxidase
45. Lubricin binds cartilage proteins, cartilage oligomeric matrix protein, fibronectin and collagen II at the cartilage surface
46. The Neutrophil Response Induced by an Agonist for Free Fatty Acid Receptor 2 (GPR43) Is Primed by Tumor Necrosis Factor Alpha and by Receptor Uncoupling from the Cytoskeleton but Attenuated by Tissue Recruitment
47. Neutrophil recruitment to inflamed joints can occur without cellular priming.
48. Neutrophils from patients with SAPHO syndrome show no signs of aberrant NADPH oxidase-dependent production of intracellular reactive oxygen species
49. The Role Of Serum Amyloid A In Inflammatory Disease - Proinflammatory Mediator Or Inert Biomarker?
50. The Human Neutrophil Subsets Defined by the Presence or Absence of OLFM4 Both Transmigrate into Tissue In Vivo and Give Rise to Distinct NETs In Vitro
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