730 results on '"Bo, X."'
Search Results
2. THE ADSORPTION CHARACTERISTICS OF SALT MODIFIED ZEOLITE TO AMMONIA NITROGEN.
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BO, X., GUO, M.-X., LI, X.-C., WU, J.-J., LIU, S.-X., and WANG, C.-Y.
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ZEOLITES ,SOLUTION (Chemistry) ,SODIUM tungstate ,AMMONIA ,NITROGEN in water ,NITROGEN - Abstract
To improve the adsorption capacity of natural zeolite for ammonia nitrogen, zeolite was modified by sodium chloride (NaCl), sodium tungstate (Na2WO4), and sodium citrate (C6H5Na3O7) solutions, and the adsorption characteristics of zeolite before and after modification for ammonia nitrogen in water were studied. The properties and structure of zeolite were characterized and analyzed. The adsorption mechanism of modified zeolite for ammonia nitrogen and the effects of environmental factors on the adsorption were studied. The results showed that the modification of NaCl, Na2WO4, and C6H5Na3O7 increased the adsorption capacity of zeolite by 60.76%, 58.23%, and 54.43%, respectively. The zeolite modified by NaCl solution had the best adsorption effect for ammonia nitrogen. The optimum concentration, temperature, and time of NaCl modifier was 1.5 mol/L, 85, and 12 h. The adsorption of ammonia nitrogen on zeolites before and after modification was more consistent with the pseudo-secondorder kinetic model. Under the conditions of particle size of 60-80 mesh, pH of 7, temperature of 30, and dosage of 10 g/L, the removal rate of ammonia nitrogen by zeolites increased from 54.89% to 90.39% after adsorption of wastewater with initial ammonia nitrogen concentration of 50 mg/L. The adsorption of ammonia nitrogen in wastewater by natural zeolite modified by salt solution is an environmentally friendly and effective method. [ABSTRACT FROM AUTHOR]
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- 2024
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3. ARQ-197 enhances the antitumor effect of sorafenib in hepatocellular carcinoma cells via decelerating its intracellular clearance
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Gao X, Chen H, Huang X, Li H, Liu Z, and Bo X
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advanced hepatocellular carcinoma ,molecular targeted agents ,ARQ-197 and sorafenib ,drug clearance ,epithelial-mesenchymal transition ,multi-drug resistance ,Neoplasms. Tumors. Oncology. Including cancer and carcinogens ,RC254-282 - Abstract
Xudong Gao,1,2 Hebing Chen,1 Xin Huang,1 Hao Li,1 Zhen Liu,1 Xiaochen Bo1 1Beijing Institute of Radiation Medicine, Beijing 100850, People’s Republic of China; 2The 5th Medical Center of PLA General Hospital, Beijing 100039, People’s Republic of China Background: Hepatocellular carcinoma (HCC) is one of the heaviest malignant burdens in China. Molecular targeting agent, sorafenib, is the main therapeutic option for antitumor therapy of advanced HCC, but it is currently too expensive for the public and its therapeutic effect does not satisfy initial expectation. Therefore, it is important to develop more effective molecular targeted therapeutic strategies for advanced HCC. Materials and methods: The antitumor effects of sorafenib or ARQ-197, an antagonist of c-MET (tyrosine-protein kinase Met or hepatocyte growth factor receptor), were examined by MTT or in murine tumor model. The effect of ARQ-197 on epithelial–mesenchymal transition (EMT) or multidrug resistance (MDR) was examined by quantitative real-time PCR for the expression of related genes. The clearance of sorafenib in HCC cells was detected by liquid chromatography-mass spectrometry/mass spectrometry. Results: ARQ-197 treatment enhanced the sensitivity of HCC cells to sorafenib. Mechanistic studies indicated that ARQ-197 inhibited the expression of EMT- and MDR-related genes. Moreover, ARQ-197 treatment decelerated the clearance of sorafenib in cultured HCC cells and subcutaneous HCC tumors in nude mice. Conclusion: In the present work, our data suggested that ARQ-197 decelerated the clearance of sorafenib in HCC cells and enhanced the antitumor effect of sorafenib. Keywords: advanced hepatocellular carcinoma, molecular targeted agents, ARQ-197 and sorafenib, drug clearance, epithelial–mesenchymal transition, multidrug resistance
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- 2019
4. Potential therapeutic value of primary tumor resection in ampullary cancer patients with distant metastases at initial diagnosis: a population-based study
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Wang J, Bo X, Lu P, Suo T, Ni X, Liu H, Pan H, Shen S, Li M, Zhang D, Wang Y, and Liu HB
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ampullary cancer ,PSM ,primary tumor resection ,chemotherapy. ,Neoplasms. Tumors. Oncology. Including cancer and carcinogens ,RC254-282 - Abstract
Jie Wang,1,* Xiaobo Bo,1,* Pinxiang Lu,2,* Tao Suo,1 Xiaoling Ni,1 Han Liu,1 Hongtao Pan,1 Sheng Shen,1 Min Li,1 Dexiang Zhang,2 Yueqi Wang,1 Houbao Liu1 1Department of General Surgery, Zhongshan Hospital, Fudan University, Shanghai, China; 2Department of General Surgery, Xuhui Central Hospital, Shanghai, China *These authors contributed equally to this work Objective: To evaluate the therapeutic value of primary tumor resection (PTR) in metastatic ampullary cancer at the initial presentation. Patients and methods: Patients with metastatic ampullary cancer were identified from Surveillance, Epidemiology and End Results database. Propensity score matching (PSM) was performed to balance the characteristics of our cohort. Kaplan–Meier analyses, log-rank tests and multivariate Cox regression models were employed to evaluate the therapeutic value of PTR. Results: A total of 346 patients with metastatic ampullary cancer were identified from 2004 to 2014 and 90 patients were screened by PSM. PTR was associated with favorable overall survival (OS) and cancer-specific survival (CSS) after PSM (PTR vs no-PTR: 16.0, 95% CI: 9.0–22.0 vs 8.0, 95% CI: 5.0–11.0 for median OS; 22.0, 95% CI: 13.0–33.0 vs 9.0, 95% CI: 5.0–11.0 for median CSS; both log-rank P
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- 2018
5. Automatic detection of microseismic waveform based on machine learning
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Qiao, J., primary, Xue, Y., additional, Li, J., additional, Chen, X., additional, Li, Z., additional, and Bo, X., additional
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- 2023
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6. Behavior Recognition of a Broiler Chicken using Long Short-Term Memory with Convolution Neural Networks
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Xie, Bo X., primary and Chang, Chung L., additional
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- 2022
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7. Evolution mechanism of compression deformation of carbon black reinforced epoxy resin composites
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Wu, Z., primary, Jang, H.F., additional, Bo, X., additional, Yang, C.M., additional, Zhang, Y., additional, and Wan, J.Q., additional
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- 2022
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8. Behavior Recognition of a Broiler Chicken using Long Short-Term Memory with Convolution Neural Networks
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Bo X. Xie and Chung L. Chang
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- 2022
9. A high-power long lifetime beam dump for the Thomson scattering diagnostic system in the XuanLong-50 experiment
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Liu, J. H., primary, Li, S. J., additional, Li, H. Y., additional, Xie, Q. F., additional, Bai, R. H., additional, Bo, X. K., additional, and Deng, B. H., additional
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- 2022
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10. Contributors
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ALOthman, Z.A., primary, An, J., additional, Batmunkh, M., additional, Biggs, M.J., additional, Bo, X., additional, Chen, P., additional, Chen, T., additional, Cheng, L., additional, Fang, S., additional, Grace, T., additional, Guo, L.-H., additional, Hao, B., additional, He, S., additional, Hou, J., additional, Hu, G., additional, Jian, M.Q., additional, Jiang, K., additional, Kang, L.X., additional, Li, D., additional, Li, N., additional, Li, Q., additional, Liu, T., additional, Lv, T., additional, Ma, P.C., additional, Peng, H., additional, Qin, D., additional, Qiu, H., additional, Shapter, J.G., additional, Shearer, C., additional, Shen, H., additional, Shen, L., additional, Sun, X., additional, Tune, D., additional, Wagberg, T., additional, Wan, B., additional, Wang, F., additional, Wang, L., additional, Wang, X., additional, Wang, Z., additional, Wu, H., additional, Xia, K.L., additional, Xie, H.H., additional, Xue, Y., additional, Yang, J., additional, Yao, Y., additional, Yong, Z.Z., additional, Yu, L., additional, Yuan, W., additional, Yuan, Q., additional, Zhan, Z., additional, Zhang, X., additional, Zhang, X.H., additional, Zhang, Y., additional, Zhang, Y.Y., additional, Zheng, L., additional, and Zhou, M., additional
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- 2017
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11. Wearable Carbon Nanotube Devices for Sensing
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Shen, H., primary, Liu, T., additional, Qin, D., additional, Bo, X., additional, Wang, L., additional, Wang, F., additional, Yuan, Q., additional, Wagberg, T., additional, Hu, G., additional, and Zhou, M., additional
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- 2017
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12. Thomson scattering diagnostic system for the XuanLong-50 experiment
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Li, H. Y., primary, Li, S. J., additional, Xie, Q. F., additional, Liu, J. H., additional, Bai, R. H., additional, Tao, R. Y., additional, Lun, X. C., additional, Li, N., additional, Bo, X. K., additional, Liu, C. Q., additional, Han, L., additional, and Deng, B. H., additional
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- 2022
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13. Volatile organic compounds constituents of a typical integrated iron and steel plant and influence on O3 pollution
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Wang, Y., primary, Zhu, R., additional, Bo, X., additional, Dan, M., additional, and Shu, M., additional
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- 2022
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14. Volatile organic compounds constituents of a typical integrated iron and steel plant and influence on O3 pollution.
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Wang, Y., Zhu, R., Bo, X., Dan, M., and Shu, M.
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STEEL mills ,VOLATILE organic compounds ,IRON ,AIR heaters ,COKE (Coal product) ,POLLUTION - Abstract
A typical integrated iron and steel plant in the Beijing–Tianjin–Hebei (BTH) of China was selected to measure volatile organic compound (VOC) constituents from five exhaust stacks belonging to the air heaters, coking, converters, heads, and tails of the sintering machines. The VOC source profiles were established based on the process, and the contribution of VOCs to ozone formation was analyzed in combination with the ozone formation potential (OFP). The concentration of total VOCs (TVOCs) emitted by different processes in the steel plant was 140.5–364.2 μg/m
3 . The sequence of TVOC concentrations was as follows: sinter-tail > coking ≈ sinter-head > converter > air heater. Alkanes and aromatic compounds were the predominant groups constituting 48.3% and 36.6% of the VOC concentration, respectively. However, the calculated results with OFP were different, and the contribution of aromatics reached a maximum of 59.8%, followed by alkanes (30.7%). The sequence of OFP for different processes was sinter-head > sinter-tail > coking > converter > air heater. The sinter-head became the segment that contributed the most to ozone formation in the plant; the reason was that the exhaust from sinter-head contained a large amount of aromatic species, which tend to have a greater maximum incremental reactivity (MIR) due to the difference in chemical reactivity. [ABSTRACT FROM AUTHOR]- Published
- 2023
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15. Cyber-physical systems of smart agriculture driven by data and knowledge
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Wang, X., primary, Bo, X., additional, and Zhang, S., additional
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- 2022
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16. Differential antibody responses in sows and finishing pigs naturally infected with African swine fever virus under field conditions
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Luong, Hung Q., primary, Lai, Huong TL., additional, Do, Luc D., additional, Ha, Bo X., additional, Nguyen, Giap V., additional, and Vu, Hiep LX., additional
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- 2022
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17. Structure-Oriented Laplacian Pyramid Algorithm Based on 3D Structure Adaptive Anisotropic Gaussian Kernel
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Bo, X., primary, Chen, X., additional, Li, J., additional, and Guo, K., additional
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- 2022
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18. Two cytokine signaling molecules co-operate to promote axonal transport and growth
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Miao, T., Wu, D., Wheeler, A., Wang, P., Zhang, Y., Bo, X., Yeh, J.S., Subang, M.C., and Richardson, P.M.
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- 2011
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19. A numerical study of hollow water drop breakup during freezing
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Vu, Truong V., primary, Pham, Binh D., additional, Pham, Phuc H., additional, Vu, Hung V., additional, and Tran, Bo X., additional
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- 2021
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20. Structural variant selection for high-altitude adaptation using single-molecule long-read sequencing
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Jia Q, Song X, Jia Zhilong, Xiliang Wang, Manolis Kellis, Xiao Y, Cui S, Shi J, Zhong Q, Liang F, Bo X, Sun J, Wu S, Zhao C, Chen Q, Wu J, Yu K, Liu Y, Park M, Xian Zhao, Wu Z, Kunlun He, and Wang D
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education.field_of_study ,Population ,Single-nucleotide polymorphism ,Computational biology ,Adaptation ,Biology ,Enhancer ,Indel ,education ,human activities ,Gene ,Selection (genetic algorithm) ,Epigenomics - Abstract
Structural variants (SVs) can be important drivers of human adaptation with strong effects, but previous studies have focused primarily on common variants with weak effects. Here, we used large-scale single-molecule long-read sequencing of 320 Tibetan and Han samples, to show that SVs are key drivers of selection under high-altitude adaptation. We expand the landscape of global SVs, apply robust models of selection and population differentiation combining SVs, SNPs and InDels, and use epigenomic analyses to predict driver enhancers, target genes, upstream regulators, and biological functions, which we validate using enhancer reporter and DNA pull-down assays. We reveal diverse Tibetan-specific SVs affecting the cis- and trans-regulatory circuitry of diverse biological functions, including hypoxia response, energy metabolism, lung function, etc. Our study greatly expands the global SV landscape, reveals the central role of gene-regulatory circuitry rewiring in human adaptation, and illustrates the diverse functional roles that SVs can play in human biology.
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- 2021
21. Differential antibody responses in sows and finishing pigs naturally infected with African swine fever virus under field conditions
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Bo X. Ha, Huong Tl. Lai, Giap Van Nguyen, Luc Duc Do, Hiep Lx. Vu, and Hung Q. Luong
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Cancer Research ,biology ,Swine ,Lectin ,Virulence ,Antibodies, Viral ,biology.organism_classification ,African Swine Fever Virus ,African swine fever virus ,Virology ,Infectious Diseases ,Immune system ,Antigen ,Antibody Formation ,Humoral immunity ,biology.protein ,Animals ,Female ,Lectins, C-Type ,Antibody ,African Swine Fever ,Field conditions - Abstract
Antibody profile of pigs naturally infected with a virulent African swine fever virus (ASFV) strain under field conditions was studied. Twenty-three serum samples were collected from pigs surviving a natural ASFV infection: 17 samples from finishing pigs (∼7 months old) and 6 samples from sows (between 12 and 36 months old). Additionally, 24 serum samples were collected from ASFV-naïve pigs to serve as negative controls. All sera from ASFV-surviving pigs tested positive while all sera from control pigs tested negative by two different commercial ELISA kits. Antibody reactivity of each serum sample was simultaneously measured against six selected ASFV antigens including p12, p32, p54, pp62, C-type lectin and CD2v. All ASFV-surviving pigs had antibody against p32, p54 and pp62 while 91.3% surviving pigs had antibody against p12. Only small portions of ASFV-surviving pigs exhibited antibodies against C-type lectin (34.8%) and CD2v (26.1%). While antibodies against p12, p32, p54 and pp62 were similarly detected in both finishing pigs and sows, antibodies against C-type lectin and CD2v were mainly detected in sows but not in finishing pigs. These results suggest a differential humoral immune response to ASFV infection in sows and finishing pigs. Further studies are needed to better understand the nature of immune responses to ASFV infection in different pig populations.
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- 2022
22. Substantial emission reductions from Chinese power plants after the introduction of ultra-low emissions standards
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Tang, L, Qu, J, Mi, Z, Bo, X, Chang, X, Anadon, LD, Wang, S, Xue, X, Li, S, Wang, X, Zhao, X, Tang, L [0000-0002-2522-9675], Mi, Z [0000-0001-8106-0694], Bo, X [0000-0001-8650-1882], Chang, X [0000-0001-9225-0477], Wang, S [0000-0001-5773-998X], Xue, X [0000-0002-0392-014X], and Apollo - University of Cambridge Repository
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13 Climate Action ,4008 Electrical Engineering ,40 Engineering ,4017 Mechanical Engineering - Abstract
In 2014, China introduced an ultra-low emissions (ULE) standards policy for renovating coal-fired power-generating units to limit SO2, NOX and PM emissions to 35, 50 and 10 mg m-3, respectively. The ULE standard policy had ambitious levels (surpassing those of all other countries) and implementation timeline. We estimate emission reductions associated with the ULE policy by constructing a nationwide, unit-level, hourly-frequency emissions dataset using data from a continuous emission monitoring systems network covering 96-98% of Chinese thermal power capacity during 2014-2017. We find that between 2014 and 2017 China’s annual power emissions of SO2, NOX and PM dropped by 65%, 60% and 72%, respectively. Our estimated emissions using actual monitoring data are 18-92% below other recent estimates. We detail the technologies used to meet the ULE standards and the determinants of compliance, underscoring the importance of ex-post evaluation and providing insights for other countries wishing to reduce their power emissions.
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- 2019
23. Joint PP-PS Time-Lapse Difference Inversion Based on Improved Blocky Constraint
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Tang, W., primary, Li, J., additional, Geng, W., additional, Zhang, J., additional, and Bo, X., additional
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- 2021
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24. Ideological formation, national development, and the “mass line”: the historical-political complexity of cultural policy and Chinese socialism
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Chen, Nick, primary and Bo, X, additional
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- 2021
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25. HIT-scISOseq: High-throughput and High-accuracy Single-cell Full-length Isoform Sequencing for Corneal Epithelium
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Chong Tang, Yingfeng Zheng, Bo X, Kaishun Hu, Y. Chen, Jiawei Zhong, Yang Liu, Shang-Qian Xie, Wenjun Shi, Changxi Wang, Zhichao Chen, Shi Z, Feng Luo, and Chuan-Le Xiao
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Gene isoform ,Transcriptome ,medicine.anatomical_structure ,Biotinylation ,Complementary DNA ,RNA splicing ,Cell ,medicine ,Computational biology ,Biology ,Throughput (business) ,Insert (molecular biology) - Abstract
Single-cell isoform sequencing can reveal transcriptomic dynamics in individual cells invisible to bulk- and single-cell RNA analysis based on short-read sequencing. However, current long-read single-cell sequencing technologies have been limited by low throughput and high error rate. Here we introduce HIT-scISOseq for high-throughput single-cell isoform sequencing. This method was made possible by full-length cDNA capture using biotinylated PCR primers, and by our novel library preparation procedure that combines head-to-tail concatemeric full-length cDNAs into a long SMRTbell insert for high-accuracy PacBio sequencing. HIT-scISOseq yields > 10 million high-accuracy full-length isoforms in a single PacBio Sequel II 8M SMRT Cell, providing > 8 times more data output than the standard single-cell isoform PacBio sequencing protocol. We exemplified HIT-scISOseq by first studying transcriptome profiles of 4,000 normal and 8,000 injured corneal epitheliums from cynomolgus monkeys. We constructed dynamic transcriptome landscapes of known and rare cell types, revealed novel isoforms, and identified injury-related splicing and switching events that are previously not accessible with low throughput isoform sequencing. HIT-scISOseq represents a high-throughput, cost-effective, and technically simple method to accelerate the burgeoning field of long-read single-cell transcriptomics.
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- 2020
26. P-124 SHP-2 deficiency in myeloid cells promotes metastasis of esophageal carcinoma via increasing cathepsins and inflammatory factor secretion
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Li, Q., Min, X., Tang, K., Bai, J., Bo, X., and Guo, P.
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- 2023
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27. Effect of annealing on electrical properties of radio-frequency-sputtered ZnO films
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Alivov, Y. I., Bo, X., Akarca-Biyikli, S., Fan, Q., Xie, J., Biyikli, N., Zhu, K., Johnstone, D., and Morkoç, H.
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- 2006
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28. A numerical study of hollow water drop breakup during freezing
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Binh D. Pham, Hung V. Vu, Bo X. Tran, Phuc Hong Pham, and Truong V. Vu
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Fluid Flow and Transfer Processes ,Physics ,Gravity (chemistry) ,Mechanical Engineering ,Drop (liquid) ,Bubble ,Computational Mechanics ,Shell (structure) ,Mechanics ,Condensed Matter Physics ,Breakup ,Mechanics of Materials ,Stefan number ,Wetting ,Phase diagram - Abstract
We present a numerical investigation of the breakup and freezing of a pendant hollow water drop beneath a cold curved surface. The drop contains a bubble surrounded by a shell of water that forms at an outer wetting angle of θo with the surface. The freezing begins on the cold curved surface and evolves in the direction of gravity. As it freezes, the water accumulates and forms a bulbous end at the bottom of the drop. Breakup can occur, inducing a daughter water drop. As a result, the freezing process of the remaining hollow drop attached to the surface takes less time, and the frozen drop shrinks. However, this breakup has no effect on the bubble. The various parameters under consideration include the Bond number Bo, the Stefan number St, the size of the bubble, and the angle θo. It is found that the breakup of the drop during freezing takes place for increasing Bo (from 0.1 to 3.0), increasing θo (from 60° to 120°), decreasing St (in the range of 0.01–0.64), or decreasing bubble size. On the other hand, the shape of the curved surface has little influence on the breakup of the drop. Phase diagrams of θo vs Bo and of St vs Bo are also presented to provide a more general picture of the breakup and freezing of the water drop.
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- 2021
29. Double-antigen sandwich ELISA for detection of antibodies to SARS-associated coronavirus in human serum
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Chen, S., Lu, D., Zhang, M., Che, J., Yin, Z., Zhang, S., Zhang, W., Bo, X., Ding, Y., and Wang, S.
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- 2005
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30. Deformation of an Elastic Thin Plate using ULE in Lateral Flow of Fluid
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Hao, Y. J., Bai, X. Z., Bo, X. H., Yao, Z. H., and Yuan, M. W.
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- 2009
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31. Deformation of an Elastic Thin Plate using ULE in Lateral Flow of Fluid
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Hao, Y. J., primary, Bai, X. Z., additional, and Bo, X. H., additional
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- 2007
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32. Incidence of Atrial Fibrillation after Lung Transplantation in 50 Cases
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Yang, J, Wu, X Q, Chen, J Y, Zhen, M F, Bo, X P, and Lu, W H
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- 2010
33. Effect of clearance jet on aerodynamic performance of centrifugal fan
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Zhu, G J, primary, Bo, X Y, additional, Feng, J J, additional, and Wu, G K, additional
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- 2019
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34. EpiNet as a way of involving more physicians and patients in epilepsy research: Validation study and accreditation process
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Bergin, P. S., Beghi, E., Sadleir, L. G., Brockington, A., Tripathi, M., Richardson, M. P., Bianchi, E., Srivastava, K., Jayabal, J., Legros, B., Ossemann, M., Mcgrath, N., Verrotti, A., Tan, H. J., Beretta, S., Frith, R., Iniesta, I., Whitham, E., Wanigasinghe, J., Ezeala-Adikaibe, B., Striano, P., Rosemergy, I., Walker, E. B., Alkhidze, M., Rodriguez-Leyva, I., Ramirez Gonzalez, J. A., D'Souza, W. J., Calle, A., Palacios, C., Cairns, A., Carney, P., Craig, D., Gill, D., Gupta, S., Lander, C., Laue-Gizzi, H., Hitchens, N., Kiley, M., Lawn, N., Reyneke, E., Riney, K., Tan, M., Thieban, M., Wong, C., van Rijckevorsel, G., Ferrari Strang, A. G., Gifoni, A., Helio, L., Monnerat, B., Brna, P., Donner, E., Jacques, S., Jette, N., Mclachlan, R., Mohamed, I., Tran, T. P. Y., Bo, X., Fan, S., Guang, Y., Li, M., Wang, K., Zhang, S., Ladino, L., Christensen, J., Kӧlmel, M. S., Nikanorova, M., Uusitalo, A., Vieira, P., Auvin, S., Ediberidze, T., Gogatishvili, N., Jishkariani, T., Dennig, D., Grimmer, A., Michaelis, R., Schubert-Bast, S., Stephani, C., Stodieck, S., Vollbrandt, M., Zellner, A., Zafeiriou, D., Fogarasi, A., Halasz, P., Chaurasia, R. N., Jain, S., Nair, R., Passi, P., Rajadhyaksha, S., Sattaluri, S. J., Shah, H., Udani, V., Costello, D., Aguglia, U., Bartocci, A., Benna, P., Ferlazzo, E., Laino, D., Spalice, A., Zanchi, C., Ali, A., Lim, K. S., Ramirez, A., Anderson, N., Barber, A., Cariga, P., Cleland, J., Child, N., Davis, S., Dayal, V., Dickson, C., Doran, J., Duncan, R., Giri, P., Herd, M., Hutchinson, D., Jones, B., Kao, J., Kilfoyle, D., Mottershead, J., Muir, C., Nolan, M., Pereira, J., Ranta, A., Sadani, S., Simpson, M., Spooner, C., Timmings, P., Walker, E., Wei, D., Willoughby, E., Wong, E., Wu, T., Olusola, T., Mahmud, H., Mogul, Z., Espinoza, J., Vizarreta, J. H., Baeta, E. M., Teotonio, R., Jocic-Jakubi, B., Lukic, S., Korosec, M., Zgur, T., Eguilaz, M. G., Asztely, F., Sithinamsuwan, P., Anderson, J., Auce, P., Desurkar, A., Hamandi, K., Kelso, A., Sanchez, V., Sidra, A., Smith, P., Wehner, T., Winston, G., Andrade, E., Bensalem-Owen, M., Boudreau, M., Caller, T., Chapman, K., Chari, G., Davis, K., Droker, B., El-Hagrassy, M., Eliashiv, D., Eze, C., Heck, C., Kabir, A., Kolesnik, D., Lam, A., Lopez, J., Maamoon, T., Cohen, J. M., Maganti, R., Nwankwo, C., Park, K., Proteasa, S., Sandok, E., Seinfield, S., Toub, J., Wirrell, E., Arbildi, M., Thien, T. T., UCL - SSS/IONS/NEUR - Clinical Neuroscience, and UCL - (MGD) Service de neurologie
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0301 basic medicine ,medicine.medical_specialty ,Validation study ,education ,Alternative medicine ,Multicenter collaboration ,Diagnostic accuracy ,Accreditation ,03 medical and health sciences ,Epilepsy ,Clinical trials ,0302 clinical medicine ,Clinical Trials ,Multicentre Collaboration ,medicine ,health care economics and organizations ,Kappa value ,business.industry ,medicine.disease ,Invited Original Research ,Clinical trial ,030104 developmental biology ,Neurology ,Family medicine ,Etiology ,Neurology (clinical) ,business ,030217 neurology & neurosurgery ,Cohort study ,Biomedical engineering - Abstract
Objective\ud \ud EpiNet was established to encourage epilepsy research. EpiNet is used for multicenter cohort studies and investigator‐led trials. Physicians must be accredited to recruit patients into trials. Here, we describe the accreditation process for the EpiNet‐First trials.\ud Methods\ud \ud Physicians with an interest in epilepsy were invited to assess 30 case scenarios to determine the following: whether patients have epilepsy; the nature of the seizures (generalized, focal); and the etiology. Information was presented in two steps for 23 cases. The EpiNet steering committee determined that 21 cases had epilepsy. The steering committee determined by consensus which responses were acceptable for each case. We chose a subset of 18 cases to accredit investigators for the EpiNet‐First trials. We initially focused on 12 cases; to be accredited, investigators could not diagnose epilepsy in any case that the steering committee determined did not have epilepsy. If investigators were not accredited after assessing 12 cases, 6 further cases were considered. When assessing the 18 cases, investigators could be accredited if they diagnosed one of six nonepilepsy patients as having possible epilepsy but could make no other false‐positive errors and could make only one error regarding seizure classification.\ud Results\ud \ud Between December 2013 and December 2014, 189 physicians assessed the 30 cases. Agreement with the steering committee regarding the diagnosis at step 1 ranged from 47% to 100%, and improved when information regarding tests was provided at step 2. One hundred five of the 189 physicians (55%) were accredited for the EpiNet‐First trials. The kappa value for diagnosis of epilepsy across all 30 cases for accredited physicians was 0.70.\ud Significance\ud \ud We have established criteria for accrediting physicians using EpiNet. New investigators can be accredited by assessing 18 case scenarios. We encourage physicians with an interest in epilepsy to become EpiNet‐accredited and to participate in these investigator‐led clinical trials.
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- 2017
35. The pick-up stick transistor
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Blanchet, Graciela B., Bo, X.-Z., Lee, C.Y., Strano, M.S., and Nuckolls, C.
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- 2005
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36. Antisense oligonucleotides targeted against asialoglycoprotein receptor 1 block human hepatitis B virus replication
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Yang, J., Bo, X. C., Ding, X. R., Dai, J. M., Zhang, M. L., Wang, X. H., and Wang, S. Q.
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- 2006
37. Differentially expressed cellular genes following HBV: potential targets of anti-HBV drugs?
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Yang, J., Bo, X. C., Yao, J., Yang, N. M., and Wang, S. Q.
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- 2005
38. Tumour necrosis factor α impairs function of liver derived T lymphocytes and natural killer cells in patients with primary sclerosing cholangitis
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Bo, X, Broome, U, Remberger, M, and Sumitran-Holgersson, S
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- 2001
39. Locally maximal idempotent-generated subsemigroups of singular orientation-preserving transformation semigroups
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Zhao, P., Bo, X., and Mei, Y.
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- 2008
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40. Design and pharmacological characterization of selective P2-purinoceptor antagonists
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Lambrecht, G., primary, Ardanuy, U., additional, Bäumert, H.G., additional, Bo, X., additional, Hoyle, C.H.V., additional, Nickel, P., additional, Pfaff, O., additional, Ralevic, V., additional, Windscheif, U., additional, Ziganshin, A.U., additional, Ziyal, R., additional, Mutschler, E., additional, and Burnstock, G., additional
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- 1996
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41. Oncolytic Viruses for Tumor Precision Imaging and Radiotherapy
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Ying Xiang, Xian-Wang Wang, Hong-Wu Xin, Zhaowu Ma, Jiafu Ji, Xiao-Chun Peng, Yanling Zhang, Zi J. Wu, Bo X. Ren, Xiao Q. Liu, Jingbo Kang, and Feng R. Tang
- Subjects
0301 basic medicine ,Genetic enhancement ,medicine.medical_treatment ,Genetic Vectors ,Gendicine ,medicine.disease_cause ,Adenoviridae ,03 medical and health sciences ,0302 clinical medicine ,Neoplasms ,Genetics ,Medicine ,Humans ,Simplexvirus ,Virotherapy ,Molecular Biology ,Oncolytic Virotherapy ,business.industry ,Melanoma ,Granulocyte-Macrophage Colony-Stimulating Factor ,medicine.disease ,Recombinant Proteins ,Oncolytic virus ,Radiation therapy ,Oncolytic Viruses ,030104 developmental biology ,Herpes simplex virus ,030220 oncology & carcinogenesis ,Cancer research ,Molecular Medicine ,Tumor Suppressor Protein p53 ,business ,Talimogene laherparepvec - Abstract
In 2003 in China, Peng et al. invented the recombinant adenovirus expressing p53 (Gendicine) for clinical tumor virotherapy. This was the first clinically approved gene therapy and tumor virotherapy drug in the world. An oncolytic herpes simplex virus expressing granulocyte-macrophage colony-stimulating factor (Talimogene laherparepvec) was approved for melanoma treatment in the United States in 2015. Since then, oncolytic viruses have been attracting more and more attention in the field of oncology, and may become novel significant modalities of tumor precision imaging and radiotherapy after further improvement. Oncolytic viruses carrying reporter genes can replicate and express genes of interest selectively in tumor cells, thus improving in vivo noninvasive precision molecular imaging and radiotherapy. Here, the latest developments and molecular mechanisms of tumor imaging and radiotherapy using oncolytic viruses are reviewed, and perspectives are given for further research. Various types of tumors are discussed, and special attention is paid to gastrointestinal tumors.
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- 2017
42. Chapter 7 - Wearable Carbon Nanotube Devices for Sensing
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Shen, H., Liu, T., Qin, D., Bo, X., Wang, L., Wang, F., Yuan, Q., Wagberg, T., Hu, G., and Zhou, M.
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- 2017
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43. The Surgical Treatment of Three Young Chronic Subdural Hematoma Patients with Different Causes
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Kun Hou, Bo X Zhu, Yang Zhang, and Chen G Li
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medicine.medical_specialty ,business.industry ,General Neuroscience ,Case Report ,Disease ,Chronic subdural hematoma ,medicine.disease ,Insidious onset ,Surgery ,Arachnoid cyst ,Young adult ,Cerebrospinal fluid leakage ,medicine ,Neurology (clinical) ,Risk factor ,Surgical treatment ,Intracranial Hypotension ,business ,Intracranial hypotension - Abstract
Chronic subdural hematoma (CSDH), which rarely happens in the young, is thought to be a disease of the elderly. Whereas unspecific symptoms and insidious onset in juveniles and young adults, as a result of its relative low morbidity, CSDH is usually neglected even undertreated in the young. Through the three cases and review of the current literature on this subject, we tried to illustrate the clinical and etiopathological characteristics of this entity and find out the most appropriate treatment strategy. We report three young CSDH patients with different but similar symptoms. The present histories, tests and examinations revealed different predisposing factors accounting for the genesis of CSDH. Their preoperative symptoms were all resolved with burr hole and drainage operation. Juveniles and young adults suffering from CSDH differ from that of their elderly counterparts in their clinical and etiopathological characteristics. Although trauma is the most important risk factor in young and old CSDH patients, some other predisposing factors may exist. Burr hole and drainage surgery could resolve the problem most of the time. But further tests and examinations even specific management should be made in some cases.
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- 2014
44. In-plane optical anisotropy induced by asymmetrically δ-doping in (001) GaAs/AlGaAs quantum wells studied by reflectance difference spectroscopy.
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Yu, J. L., Chen, Y. H., Bo, X., Jiang, C. Y., Ye, X. L., Wu, S. J., and Gao, H. S.
- Subjects
ANISOTROPY ,QUANTUM wells ,REFLECTANCE spectroscopy ,EXTRAPOLATION ,SEMICONDUCTOR doping - Abstract
In-plane optical anisotropy (IPOA) in modulation-doped (001) GaAs/AlGaAs quantum wells (QWs) has been studied by reflectance difference spectroscopy (RDS). By changing the position of the δ-doping layer, we introduce an asymmetric potential into the quantum well system, which results in an additional IPOA. Compared to symmetrically doped and undoped structure, the asymmetrically doped QWs exhibit larger IPOA, which is clearly demonstrated both by the RDS results measured at 80 K and the linear extrapolation of the RDS signal under uniaxial strain measured at room temperature. Numerical calculations within the envelope function framework show that the asymmetric potential induced by asymmetrically doping will introduce additional hole-mixing coefficients. This work demonstrates that the IPOA of QWs can be tailored by changing the delta-doping position. [ABSTRACT FROM AUTHOR]
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- 2013
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45. MiR-126 on mice with coronary artery disease by targeting S1PR2.
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FAN, J.-L., ZHANG, L., and BO, X.-H.
- Abstract
OBJECTIVE: To screen the differentially expressed micro ribonucleic acids (miRNAs) in the serum of coronary atherosclerosis patients, and to investigate their possible mechanisms of action. PATIENTS AND METHODS: The differentially expressed serum miRNAs were screened from 3 coronary artery disease (CAD) patients and 3 healthy controls using miRNA expression profiles, which were verified using low-throughput quantitative Reverse Transcription-Polymerase Chain Reaction (RT-qPCR) assay. 60 apolipoprotein E (ApoE)-/- mice were divided into model group, agomir-126 group, agomir-control (con) group, and antagomir-126 group using a random number table. They were fed with high-fat diets (21% fat and 0.15% cholesterol) ad libitum for 15 weeks to establish the mouse model of CAD. Then, hematoxylin and eosin (HE) staining was applied to detect the impact of miR-126 expression level on the tissue morphology in the thoracic aortic region. The influences of miR-126 expression level on the secretion levels of tumor necrosis factor-alpha (TNF-a), interleukin-1 beta (IL-1ß), and IL-10 were determined via enzyme-linked immunosorbent assay (ELISA). Western blotting assay was performed to examine the effects of miR-126 expression level on the expression levels of nuclear factor-kappa B (NF-κB) and vascular cell adhesion molecule-1 (VACM-1) in the tissues of the thoracic aortic region of the mice. The correlation between miR-126 expression level and sphingosine-1-phosphate receptor 2 (S1PR2) in the serum of CAD patients and animal models was analyzed by the Pearson correlation coefficient method. The targets of miR-126 were predicted using the bioinformatics method, and the direct targets were verified through investigations. Western blotting assay and ELISA were adopted to detect the impacts of miR-126 expression level on the expression and secretion levels of TNF-a, IL-1ß, and IL-10 in S1P + oxidized low-density lipoprotein (ox-LDL)-induced human umbilical vein endothelial cells (HUVECs). Lentivirus-small hairpin RNA (shRNA) was utilized to knock down the expression level of S1RP2 to determine whether miR-126 affected the increase in the inflammation level in S1P + ox-LDL-induced HUVECs by targeting S1RP2. RESULTS: Compared with those in control group, 4 miRNAs (miR-126, miR-206, miR-4297, and miR-3646) in the serum of CAD patients exhibited the most significant expression differences, which increased by 6.72, 7.11, 13.57, and 21.22 times, respectively. The verification results of low-throughput RT-qPCR assay indicated that there were remarkable changes in the expression levels of the 4 selected miRNAs with differential expressions in comparison with those in control group, displaying statistically significant differences (p<0.01). The results of HE staining manifested that the coronary atherosclerotic plaques were reduced markedly in agomir-126 group, while notably more coronary atherosclerotic plaques were formed in the thoracic aortic region in antagomir-126 group. Meanwhile, the elevated expression level of miR-126 evidently lowered the expressions of serum TNF-a and IL-1ß, but significantly increased the expression of IL-10 in the mouse model of CAD. According to the analysis results of the Pearson correlation coefficient method, the miR-126 expression level was negatively correlated with S1PR2 expression level in the serum of both CAD patients and animal models (r=-0.6123, r=-5.37). It was shown in bioinformatics prediction and luciferase reporter gene assay that miR-126 negatively regulated the S1PR2 expression by targeting the 3' untranslated region (UTR) of S1PR2 messenger RNA (mRNA). In the in vitro inflammation model, the increased expression level of miR-126 could relieve the inflammation in cells induced by S1P + ox-LDL. Based on the results of both Western blotting assay and ELISA, the differences in the expression and secretion levels of TNF-a, IL-1ß, and IL-10, as well as the expression levels of signaling molecules of the NF-κB signaling pathway, in the cells were not statistically significant among miR-126 mimic treatment group, sh-S1PR2 group, and miR-126 mimic + sh-S1PR2 group, indicating that miR-126 affects the inflammation level in HUVECs by targeting S1PR2. CONCLUSIONS: MiR-126 represses the progression of coronary atherosclerosis in the mice by binding to S1PR2. The results of this research may propose a new mechanism of miR-126 in exerting its therapeutic effects and possess potential value for the treatment of CAD in the future. [ABSTRACT FROM AUTHOR]
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- 2020
46. Oncolytic Viruses for Tumor Precision Imaging and Radiotherapy
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Wu, Zi J., primary, Tang, Feng R., additional, Ma, Zhao-Wu, additional, Peng, Xiao-Chun, additional, Xiang, Ying, additional, Zhang, Yanling, additional, Kang, Jingbo, additional, Ji, Jiafu, additional, Liu, Xiao Q., additional, Wang, Xian-Wang, additional, Xin, Hong-Wu, additional, and Ren, Bo X., additional
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- 2018
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47. Production of charged pions, kaons and protons at large transverse momenta in pp and Pb–Pb collisions at √sNN = 2.76 TeV
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Piano, Stefano, Lea, Ramona, Camerini, Paolo, Luparello, Grazia, Margagliotti, Giacomo, Rui, Rinaldo, Abelev bt, B., Adam ak, J., Adamová cb, D., Aggarwal cf, M. M., Aglieri Rinella ah, G., Agnello cm, M., Agostinelli z, A., Agrawal ar, N., Ahammed dw, Z., Ahmad r, N., Ahmad Masoodi r, A., Ahmed o, I., Ahn bm, S. U., Ahn bm, S. A., Aimo cm, I., Aiola eb, S., Ajaz o, M., Akindinov bc, A., Aleksandrov cs, D., Alessandro dd, B., Alexandre cu, D., Alici cx, A., L, Alkin c, A., Alme ai, J., Alt am, T., Altini ae, V., Altinpinar q, S., Altsybeev dv, I., Alves Garcia Prado dl, C., Andrei bw, C., Andronic cp, A., Anguelov cl, V., Anielski ay, J., Anticˇic ́ cq, T., Antinori da, F., Antonioli cx, P., Aphecetche df, L., Appelshäuser aw, H., Arbor bp, N., Arcelli z, S., Armesto p, N., Arnaldi dd, R., Aronsson eb, T., Arsene u, I. C., Arslandok aw, M., Augustinus ah, A., Averbeck cp, R., Awes cc, T. C., Azmi r, M. D., Bach am, M., Badalà cz, A., Baek an, Y. W., Bagnasco dd, S., Bailhache aw, R., Bairathi cj, V., Bala ci, R., Baldisseri n, A., Baltasar Dos Santos Pedrosa ah, F., Bán bd, J., Baral bf, R. C., Barbera aa, R., Barile ae, F., Barnaföldi ea, G. G., Barnby cu, L. S., Barret bo, V., Bartke di, J., Basile z, M., Bastid bo, N., Basu dw, S., Bathen ay, B., Batigne df, G., Batyunya bk, B., Batzing u, P. C., Baumann aw, C., Bearden by, I. G., Beck aw, H., Bedda cm, C., Behera ar, N. K., Belikov az, I., Bellini z, F., Bellwied dn, R., Belmont Moreno bi, E., Bencedi ea, G., Beole y, S., Berceanu bw, I., Bercuci bw, A., Berdnikov cd, Y., 1, Berenyi ea, D., Berger ck, M. E., Bertens bb, R. A., Berzano y, D., Betev ah, L., Bhasin ci, A., Bhati cf, A. K., Bhattacharjee ao, B., Bhom ds, J., Bianchi y, L., Bianchi bq, N., Bianchin bb, C., Bielcˇík ak, J., Bielcˇíková cb, J., Bilandzic by, A., Bjelogrlic bb, S., Blanco j, F., Blau cs, D., Blume aw, C., Bock cl, F., Bogdanov bu, A., Bøggild by, H., Bogolyubsky de, M., Böhmer ck, F. V., Boldizsár ea, L., Bombara al, M., Book aw, J., Borel n, H., Borissov dz, A., Bornschein am, J., Bossú bj, F., Botje bz, M., Botta y, E., Böttger av, S., Braun Munzinger cp, P., Bregant dl, M., Breitner av, T., Broker aw, T. A., Browning cn, T. A., Broz aj, M., Bruna dd, E., Bruno ae, G. E., Budnikov cr, D., Buesching aw, H., Bufalino dd, S., Buncic ah, P., Busch cl, O., Buthelezi bj, Z., Caffarri ab, D., Cai g, X., Caines eb, H., Caliva bb, A., Calvo Villar cv, E., Canoa Roman ah, V., Carena ah, F., Carena ah, W., Carminati ah, F., Casanova Díaz bq, A., Castillo Castellanos n, J., Casula w, E. A. R., Catanescu bw, V., Cavicchioli ah, C., Ceballos Sanchez i, C., Cepila ak, J., Cerello dd, P., Chang do, B., Chapeland ah, S., Charvet n, J. L., Chattopadhyay dw, S., Chattopadhyay ct, S., Cherney ce, M., Cheshkov du, C., Cheynis du, B., Chibante Barroso ah, V., Chinellato dn, D. D., Chochula ah, P., Chojnacki by, M., Choudhury dw, S., Christakoglou bz, P., Christensen by, C. H., Christiansen af, P., Chujo ds, T., Chung co, S. U., Cicalo cy, C., Cifarelli l, L., Z, Cindolo cx, F., Cleymans ch, J., Colamaria ae, F., Colella ae, D., Collu w, A., Colocci z, M., Conesa Balbastre bp, G., Conesa del Valle au, Z., Connors eb, M. E., Contin x, G., Contreras k, J. G., Cormier cc, T. M., Corrales Morales y, Y., Cortese ad, P., Cortés Maldonado b, I., Cosentino bs, M. R., Costa ah, F., Crochet bo, P., Cruz Albino k, R., Cuautle bh, E., Cunqueiro bq, L., Dainese da, A., Dang g, R., Danu bg, A., Das ct, D., Das au, I., Das ct, K., Das d, S., Dash dm, A., Dash ar, S., De dw, S., Delagrange df, H., 2, Deloff bv, A., Dénes ea, E., D’Erasmo ae, G., de Barros dl, G. O. V., De Caro l, A., de Cataldo cw, G., de Cuveland am, J., De Falco w, A., De Gruttola ac, D., De Marco dd, N., De Pasquale ac, S., de Rooij bb, R., Diaz Corchero j, M. A., Dietel ay, T., Divià ah, R., Di Bari ae, D., Di Liberto db, S., Di Mauro ah, A., Di Nezza bq, P., Djuvsland q, Ø., Dobrin bb, A., Dobrowolski bv, T., Domenicis Gimenez dl, D., Dönigus aw, B., Dordic u, O., Dørheim ck, S., Dubey dw, A. K., Dubla bb, A., Ducroux du, L., Dupieux bo, P., Dutta Majumdar ct, A. K., Ehlers eb, R. J., Elia cw, D., Engel av, H., Erazmus ah, B., Erdal ai, H. A., Eschweiler am, D., Espagnon au, B., Estienne df, M., Esumi ds, S., Evans cu, D., Evdokimov de, S., Eyyubova u, G., Fabris da, D., Faivre bp, J., Falchieri z, D., Fantoni bq, A., Fasel cl, M., Fehlker q, D., Feldkamp ay, L., Felea bg, D., Feliciello dd, A., Feofilov dv, G., Ferencei cb, J., Fernández Téllez b, A., Ferreiro p, E. G., Ferretti y, A., Festanti ab, A., Figiel di, J., Figueredo dl, M. A. S., Filchagin cr, S., Finogeev ba, D., Fionda ae, F. M., Fiore ae, E. M., Floratos cg, E., Floris ah, M., Foertsch bj, S., Foka cp, P., Fokin cs, S., Fragiacomo dc, E., Francescon ab, A., Frankenfeld cp, U., Fuchs ah, U., Furget bp, C., Fusco Girard ac, M., Gaardhøje by, J. J., Gagliardi y, M., Gago cv, A. M., Gallio y, M., Gangadharan s, D. R., Ganoti cg, P., Garabatos cp, C., Garcia Solis m, E., Gargiulo ah, C., Garishvili bt, I., Gerhard am, J., Germain df, M., Gheata ah, A., Gheata bg, M., Ghidini ae, B., Ghosh dw, P., Ghosh d, S. K., Gianotti bq, P., Giubellino ah, P., Gladysz Dziadus di, E., Glässel cl, P., Gomez k, R., González Zamora j, P., Gorbunov am, S., Görlich di, L., Gotovac dh, S., Graczykowski dy, L. K., Grajcarek cl, R., Grelli bb, A., Grigoras ah, A., Grigoras ah, C., Grigoriev bu, V., Grigoryan a, A., Grigoryan bk, S., Grinyov c, B., Grion dc, N., Grosse Oetringhaus ah, J. F., Grossiord du, J. Y., Grosso ah, R., Guber ba, F., Guernane bp, R., Guerzoni z, B., Guilbaud du, M., Gulbrandsen by, K., Gulkanyan a, H., Gunji dr, T., Gupta ci, A., Gupta ci, R., Khan o, K. H., Haake ay, R., Haaland q, Ø., Hadjidakis au, C., Haiduc bg, M., Hamagaki dr, H., Hamar ea, G., Hanratty cu, L. D., Hansen by, A., Harris eb, J. W., Hartmann am, H., Harton m, A., Hatzifotiadou cx, D., Hayashi dr, S., Heckel aw, S. T., Heide ay, M., Helstrup ai, H., Herghelegiu bw, A., Herrera Corral k, G., Hess ag, B. A., Hetland ai, K. F., Hicks eb, B., Hippolyte az, B., Hladky be, J., Hristov ah, P., Huang q, M., Humanic s, T. J., Hutter am, D., Hwang t, D. S., Ilkaev cr, R., Ilkiv bv, I., Inaba ds, M., Incani w, E., Innocenti y, G. M., Ionita ah, C., Ippolitov cs, M., Irfan r, M., Ivanov cp, M., Ivanov cd, V., Ivanytskyi c, O., Jachołkowski aa, A., Jacobs bs, P. M., Jahnke dl, C., Jang bm, H. J., Janik dy, M. A., Jayarathna dn, P. H. S. Y., Jena ar, S., Jimenez Bustamante bh, R. T., Jones cu, P. G., Jung an, H., Jusko cu, A., Kalcher am, S., Kalinak bd, P., Kalweit ah, A., Kamin aw, J., Kang ec, J. H., Kaplin bu, V., Kar dw, S., Karasu Uysal bn, A., Karavichev ba, O., Karavicheva ba, T., Karpechev ba, E., Kebschull av, U., Keidel ed, R., Ketzer ck, B., Khan r, M. M., 3, Khan ct, P., Khan dw, S. A., Khanzadeev cd, A., Kharlov de, Y., Kileng ai, B., Kim ec, B., Kim bm, D. W., Kim do, D. J., Kim an, J. S., Kim an, M., Kim ec, M., Kim t, S., Kim ec, T., Kirsch am, S., Kisel am, I., Kiselev bc, S., Kisiel dy, A., Kiss ea, G., Klay f, J. L., Klein cl, J., Klein Bösing ay, C., Kluge ah, A., Knichel cp, M. L., Knospe dj, A. G., Kobdaj ah, C., Kofarago ah, M., Köhler cp, M. K., Kollegger am, T., Kolojvari dv, A., Kondratiev dv, V., Kondratyeva bu, N., Konevskikh ba, A., Kovalenko dv, V., Kowalski ah, M., Kox bp, S., Koyithatta Meethaleveedu ar, G., Kral do, J., Králik bd, I., Kramer aw, F., Kravcˇáková al, A., Krelina ak, M., Kretz am, M., Krivda cu, M., Krizek cb, F., Krus ak, M., Kryshen cd, E., Krzewicki cp, M., Kucˇera cb, V., Kucheriaev cs, Y., Kugathasan ah, T., Kuhn az, C., Kuijer bz, P. G., Kulakov aw, I., Kumar ar, J., Kurashvili bv, P., Kurepin ba, A., Kurepin ba, A. B., Kuryakin cr, A., Kushpil cb, S., Kushpil cb, V., Kweon cl, M. J., Kwon ec, Y., Ladron de Guevara bh, P., Lagana Fernandes dl, C., Lakomov au, I., Langoy dx, R., Lara av, C., Lardeux df, A., Lattuca y, A., La Pointe dd, S. L., La Rocca aa, P., Leardini cl, L., Lee cu, G. R., Legrand ah, I., Lehnert aw, J., Lemmon ca, R. C., Lenhardt cp, M., Lenti cw, V., Leogrande bb, E., Leoncino y, M., León Monzón dk, I., Lévai ea, P., Li g, S., Lien dx, J., Lietava cu, R., Lindal u, S., Lindenstruth am, V., Lippmann cp, C., Lisa s, M. A., Ljunggren af, H. M., Lodato bb, D. F., Loenne q, P. I., Loggins dz, V. R., Loginov bu, V., Lohner cl, D., Loizides bs, C., Lopez bo, X., López Torres i, E., Lu cl, X. G., Luettig aw, P., Lunardon ab, M., Luo g, J., Luzzi ah, C., Ma eb, R., Maevskaya ba, A., Mager ah, M., Mahapatra bf, D. P., Maire cl, A., Malaev cd, M., Maldonado Cervantes bh, I., Malinina bk, L., 4, Mal’Kevich bc, D., Malzacher cp, P., Mamonov cr, A., Manceau dd, L., Manko cs, V., Manso bo, F., Manzari ah, V., Marchisone y, M., Mareš be, J., Margotti cx, A., Marín cp, A., Markert ah, C., Marquard aw, M., Martashvili dq, I., Martin cp, N. A., Martinengo ah, P., Martínez b, M. I., Martínez García df, G., Martin Blanco df, J., Martynov c, Y., Mas df, A., Masciocchi cp, S., Masera y, M., Masoni cy, A., Massacrier df, L., Mastroserio ae, A., Matyja di, A., Mayer di, C., Mazer dq, J., Mazumder as, R., Mazzoni db, M. A., Meddi v, F., Menchaca Rocha bi, A., Meninno ac, E., Mercado Pérez cl, J., Meres aj, M., Miake ds, Y., Mikhaylov bc, K., Milano ah, L., Milosevic u, J., 5, Mischke bb, A., Mishra as, A. N., Mis ́kowiec cp, D., Mitu bg, C. M., Mlynarz dz, J., Mohanty dw, B., Molnar az, L., Montaño Zetina k, L., Montes j, E., Morando ab, M., Moreira De Godoy dl, D. A., Moretto ab, S., Morreale do, A., Morsch ah, A., Muccifora bq, V., Mudnic dh, E., Muhuri dw, S., Mukherjee dw, M., Müller ah, H., Munhoz dl, M. G., Murray ch, S., Musa ah, L., Musinsky bd, J., Nandi ar, B. K., Nania cx, R., Nappi cw, E., Nattrass dq, C., Nayak dw, T. K., Nazarenko cr, S., Nedosekin bc, A., Nicassio cp, M., Niculescu bg, M., Nielsen by, B. S., Nikolaev cs, S., Nikulin cs, S., Nikulin cd, V., Nilsen ce, B. S., Noferini l, F., Nomokonov bk, P., Nooren bb, G., Nyanin cs, A., Nystrand q, J., Oeschler cl, H., Oh eb, S., Oh bl, S. K., An, 6, Okatan bn, A., Olah ea, L., Oleniacz dy, J., Oliveira Da Silva dl, A. C., Onderwaater cp, J., Oppedisano dd, C., Ortiz Velasquez af, A., Oskarsson af, A., Otwinowski cp, J., Oyama cl, K., Pachmayer cl, Y., Pachr ak, M., Pagano ac, P., Paic ́ bh, G., Painke am, F., Pajares p, C., Pal dw, S. K., Palmeri cz, A., Pant ar, D., Papikyan a, V., Pappalardo cz, G. S., Park cp, W. J., Passfeld ay, A., Patalakha de, D. I., Paticchio cw, V., Paul ct, B., Pawlak dy, T., Peitzmann bb, T., Pereira Da Costa n, H., Pereira De Oliveira Filho dl, E., Peresunko cs, D., Pérez Lara bz, C. E., Peryt dy, W., Pesci cx, A., Pestov e, Y., Petrácˇek ak, V., Petran ak, M., Petris bw, M., Petrovici bw, M., Petta aa, C., Pikna aj, M., Pillot df, P., Pinazza ah, O., Pinsky dn, L., Piyarathna dn, D. B., Płoskon ́ bs, M., Planinic cq, M., Pluta dy, J., Pochybova ea, S., Podesta Lerma dk, P. L. M., Poghosyan ah, M. G., Pohjoisaho ap, E. H. O., Polichtchouk de, B., Poljak cq, N., Pop bw, A., Porteboeuf Houssais bo, S., Porter bs, J., Pospisil ak, V., Potukuchi ci, B., Prasad d, S. K., Preghenella cx, R., Prino dd, F., Pruneau dz, C. A., Pshenichnov ba, I., Puddu w, G., Punin cr, V., Putschke dz, J., Qvigstad u, H., Rachevski dc, A., Raha d, S., Rak do, J., Rakotozafindrabe n, A., Ramello ad, L., Raniwala cj, R., Raniwala cj, S., Räsänen ap, S. S., Rascanu aw, B. T., Rathee cf, D., Rauf o, A. W., Razazi w, V., Read dq, K. F., Real bp, J. S., Redlich bv, K., 7, Reed eb, R. J., Rehman q, A., Reichelt aw, P., Reicher bb, M., Reidt cl, F., Renfordt aw, R., Reolon bq, A. R., Reshetin ba, A., Rettig am, F., Revol ah, J. P., Reygers cl, K., Riabov cd, V., Ricci br, R. A., Richert af, T., Richter u, M., Riedler ah, P., Riegler ah, W., Riggi aa, F., Rivetti dd, A., Rocco bb, E., Rodríguez Cahuantzi b, M., Rodriguez Manso bz, A., Røed u, K., Rogochaya bk, E., Rohni ci, S., Rohr am, D., Röhrich q, D., Romita dp, R., Ronchetti bq, F., Ronflette df, L., Rosnet bo, P., Rossegger ah, S., Rossi ah, A., Roukoutakis cg, F., Roy as, A., Roy az, C., Roy ct, P., Rubio Montero j, A. J., Russo y, R., Ryabinkin cs, E., Ryabov cd, Y., Rybicki di, A., Sadovsky de, S., Šafarˇík ah, K., Sahlmuller aw, B., Sahoo as, R., Sahu bf, P. K., Saini dw, J., Salgado p, C. A., Salzwedel s, J., Sambyal ci, S., Samsonov cd, V., Sanchez Castro az, X., Sánchez Rodríguez dk, F. J., Šándor bd, L., Sandoval bi, A., Sano ds, M., Santagati aa, G., Sarkar dw, D., Scapparone cx, E., Scarlassara ab, F., Scharenberg cn, R. P., Schiaua bw, C., Schicker cl, R., Schmidt cp, C., Schmidt ag, H. R., Schuchmann aw, S., Schukraft ah, J., Schulc ak, M., Schuster eb, T., Schutz ah, Y., Schwarz cp, K., Schweda cp, K., Scioli z, G., Scomparin dd, E., Scott cu, P. A., Scott dq, R., Segato ab, G., Seger ce, J. E., Selyuzhenkov cp, I., Seo co, J., Serradilla j, E., Sevcenco bg, A., Shabetai df, A., Shabratova bk, G., Shahoyan ah, R., Shangaraev de, A., Sharma dq, N., Sharma ci, S., Shigaki aq, K., Shtejer y, K., Sibiriak cs, Y., Siddhanta cy, S., Siemiarczuk bv, T., Silvermyr cc, D., Silvestre bp, C., Simatovic dt, G., Singaraju dw, R., Singh ci, R., Singha bx, S., Singhal dw, V., Sinha dw, B. C., Sinha ct, T., Sitar aj, B., Sitta ad, M., Skaali u, T. B., Skjerdal q, K., Smakal ak, R., Smirnov eb, N., Snellings bb, R. J. M., Søgaard af, C., Soltz bt, R., Song co, J., Song ec, M., Soramel ab, F., Sorensen dq, S., Spacek ak, M., Sputowska di, I., Spyropoulou Stassinaki cg, M., Srivastava cn, B. K., Stachel cl, J., Stan bg, I., Stefanek bv, G., Steinpreis s, M., Stenlund af, E., Steyn bj, G., Stiller cl, J. H., Stocco df, D., Stolpovskiy de, M., Strmen aj, P., Suaide dl, A. A. P., Subieta Vasquez y, M. A., Sugitate aq, T., Suire au, C., Suleymanov o, M., Sultanov bc, R., Šumbera cb, M., Susa cq, T., Symons bs, T. J. M., Szanto de Toledo dl, A., Szarka aj, I., Szczepankiewicz ah, A., Szymanski dy, M., Takahashi dm, J., Tangaro ae, M. A., Tapia Takaki au, J. D., 8, Tarantola Peloni aw, A., Tarazona Martinez ah, A., Tarzila bw, M. G., Tauro ah, A., Tejeda Muñoz b, G., Telesca ah, A., Terrevoli w, C., Ter Minasyan bu, A., Thäder cp, J., Thomas bb, D., Tieulent du, R., Timmins dn, A. R., Toia da, A., Torii dr, H., Trubnikov c, V., Trzaska do, W. H., Tsuji dr, T., Tumkin cr, A., Turrisi da, R., Tveter u, T. S., Ulery aw, J., Ullaland q, K., Uras du, A., Usai w, G. L., Vajzer cb, M., Vala bk, M., Valencia Palomo au, L., Vallero y, S., Vande Vyvre ah, P., Vannucci br, L., Van Der Maarel bb, J., Van Hoorne ah, J. W., van Leeuwen bb, M., Vargas b, A., Varma ar, R., Vasileiou cg, M., Vasiliev cs, A., Vechernin dv, V., Veldhoen bb, M., Velure q, A., Venaruzzo x, M., Vercellin y, E., Vergara Limón b, S., Vernet h, R., Verweij dz, M., Vickovic dh, L., Viesti ab, G., Viinikainen do, J., Vilakazi bj, Z., Villalobos Baillie cu, O., Vinogradov cs, A., Vinogradov dv, L., Vinogradov cr, Y., Virgili ac, T., Viyogi dw, Y. P., Vodopyanov bk, A., Völkl cl, M. A., Voloshin bc, K., Voloshin dz, S. A., Volpe ah, G., von Haller ah, B., Vorobyev dv, I., Vranic cp, D., Vrláková al, J., Vulpescu bo, B., Vyushin cr, A., Wagner q, B., Wagner cp, J., Wagner ak, V., Wang g, M., Wang cl, Y., Watanabe ds, D., Weber dn, M., Wessels ay, J. P., Westerhoff ay, U., Wiechula ag, J., Wikne u, J., Wilde ay, M., Wilk bv, G., Wilkinson cl, J., Williams cx, M. C. S., Windelband cl, B., Winn cl, M., Xiang g, C., Yaldo dz, C. G., Yamaguchi dr, Y., Yang bb, H., Yang g, P., Yang q, S., Yano aq, S., Yasnopolskiy cs, S., Yi co, J., Yin g, Z., Yoo co, I. K., Yushmanov cs, I., Zaccolo by, V., Zach ak, C., Zaman o, A., Zampolli cx, C., Zaporozhets bk, S., Zarochentsev dv, A., Závada be, P., Zaviyalov cr, N., Zbroszczyk dy, H., Zgura bg, I. S., Zhalov cd, M., Zhang g, F., Zhang g, H., Zhang bo, X., Bs, G, Zhangg, Y., Zhaou, C., Zhoug, D., Zhoug, F., Zhoubb, Y., Zhug, H., Zhudf, J., G, Zhug, J., Zhug, X., Zichichil, A., Zimmermann cl, A., Zimmermann ay, M. B., Zinovjev c, G., Zoccarato du, Y., Zynovyev c, M., Zyzak aw, M., Piano, Stefano, Lea, Ramona, Camerini, Paolo, Luparello, Grazia, Margagliotti, Giacomo, Rui, Rinaldo, B., Abelev bt, J., Adam ak, D., Adamová cb, M. M., Aggarwal cf, G., Aglieri Rinella ah, M., Agnello cm, Dd, A., Agostinelli z, N., Agrawal ar, Z., Ahammed dw, N., Ahmad r, A., Ahmad Masoodi r, I., Ahmed o, S. U., Ahn bm, S. A., Ahn bm, I., Aimo cm, S., Aiola eb, M., Ajaz o, A., Akindinov bc, D., Aleksandrov c, B., Alessandro dd, D., Alexandre cu, A., Alici cx, L, A., Alkin c, J., Alme ai, T., Alt am, V., Altini ae, S., Altinpinar q, I., Altsybeev dv, C., Alves Garcia Prado dl, C., Andrei bw, A., Andronic cp, V., Anguelov cl, J., Anielski ay, T., Anticˇic ́ cq, F., Antinori da, P., Antonioli cx, L., Aphecetche df, H., Appelshäuser aw, N., Arbor bp, S., Arcelli z, N., Armesto p, R., Arnaldi dd, T., Aronsson eb, I. C., Arsene u, Cp, M., Arslandok aw, A., Augustinus ah, R., Averbeck cp, T. C., Awes cc, M. D., Azmi r, Ch, M., Bach am, A., Badalà cz, Y. W., Baek an, Bo, S., Bagnasco dd, R., Bailhache aw, V., Bairathi cj, R., Bala ci, A., Baldisseri n, F., Baltasar Dos Santos Pedrosa ah, J., Bán bd, R. C., Baral bf, R., Barbera aa, F., Barile ae, G. G., Barnaföldi ea, L. S., Barnby cu, V., Barret bo, J., Bartke di, M., Basile z, N., Bastid bo, S., Basu dw, B., Bathen ay, G., Batigne df, B., Batyunya bk, P. C., Batzing u, C., Baumann aw, I. G., Bearden by, H., Beck aw, C., Bedda cm, N. K., Behera ar, I., Belikov az, F., Bellini z, R., Bellwied dn, E., Belmont Moreno bi, G., Bencedi ea, S., Beole y, I., Berceanu bw, A., Bercuci bw, Y., Berdnikov cd, D., Berenyi ea, M. E., Berger ck, R. A., Bertens bb, D., Berzano y, L., Betev ah, A., Bhasin ci, A. K., Bhati cf, B., Bhattacharjee ao, J., Bhom d, L., Bianchi y, N., Bianchi bq, C., Bianchin bb, J., Bielcˇík ak, J., Bielcˇíková cb, A., Bilandzic by, S., Bjelogrlic bb, F., Blanco j, D., Blau c, C., Blume aw, F., Bock cl, Bs, A., Bogdanov bu, H., Bøggild by, M., Bogolyubsky de, F. V., Böhmer ck, L., Boldizsár ea, M., Bombara al, J., Book aw, H., Borel n, A., Borissov dz, Co, J., Bornschein am, F., Bossú bj, M., Botje bz, E., Botta y, S., Böttger av, P., Braun Munzinger cp, M., Bregant dl, T., Breitner av, T. A., Broker aw, T. A., Browning cn, M., Broz aj, Ak, E., Bruna dd, G. E., Bruno ae, D., Budnikov cr, H., Buesching aw, S., Bufalino dd, P., Buncic ah, O., Busch cl, Z., Buthelezi bj, D., Caffarri ab, X., Cai g, H., Caines eb, A., Caliva bb, E., Calvo Villar cv, V., Canoa Roman ah, F., Carena ah, W., Carena ah, F., Carminati ah, A., Casanova Díaz bq, J., Castillo Castellanos n, E. A. R., Casula w, V., Catanescu bw, C., Cavicchioli ah, C., Ceballos Sanchez i, J., Cepila ak, P., Cerello dd, B., Chang do, S., Chapeland ah, J. L., Charvet n, S., Chattopadhyay dw, S., Chattopadhyay ct, M., Cherney ce, C., Cheshkov du, B., Cheynis du, V., Chibante Barroso ah, D. D., Chinellato dn, Dm, P., Chochula ah, M., Chojnacki by, S., Choudhury dw, P., Christakoglou bz, C. H., Christensen by, P., Christiansen af, T., Chujo d, S. U., Chung co, C., Cicalo cy, L., Cifarelli l, Z, F., Cindolo cx, J., Cleymans ch, F., Colamaria ae, D., Colella ae, A., Collu w, M., Colocci z, G., Conesa Balbastre bp, Z., Conesa del Valle au, Ah, M. E., Connors eb, Contin x, G., J. G., Contreras k, T. M., Cormier cc, Dz, Y., Corrales Morales y, P., Cortese ad, I., Cortés Maldonado b, M. R., Cosentino b, Dl, F., Costa ah, P., Crochet bo, R., Cruz Albino k, E., Cuautle bh, L., Cunqueiro bq, A., Dainese da, R., Dang g, A., Danu bg, D., Das ct, I., Das au, K., Das ct, S., Das d, A., Dash dm, S., Dash ar, S., De dw, H., Delagrange df, A., Deloff bv, E., Dénes ea, G., D’Erasmo ae, G. O. V., de Barros dl, A., De Caro l, Ac, G., de Cataldo cw, J., de Cuveland am, A., De Falco w, D., De Gruttola ac, N., De Marco dd, S., De Pasquale ac, R., de Rooij bb, M. A., Diaz Corchero j, T., Dietel ay, R., Divià ah, D., Di Bari ae, S., Di Liberto db, A., Di Mauro ah, P., Di Nezza bq, Ø., Djuvsland q, A., Dobrin bb, T., Dobrowolski bv, D., Domenicis Gimenez dl, B., Dönigus aw, O., Dordic u, S., Dørheim ck, A. K., Dubey dw, A., Dubla bb, L., Ducroux du, P., Dupieux bo, A. K., Dutta Majumdar ct, R. J., Ehlers eb, D., Elia cw, H., Engel av, B., Erazmus ah, Df, H. A., Erdal ai, D., Eschweiler am, B., Espagnon au, M., Estienne df, S., Esumi d, D., Evans cu, S., Evdokimov de, G., Eyyubova u, D., Fabris da, J., Faivre bp, D., Falchieri z, A., Fantoni bq, M., Fasel cl, D., Fehlker q, L., Feldkamp ay, D., Felea bg, A., Feliciello dd, G., Feofilov dv, J., Ferencei cb, A., Fernández Téllez b, E. G., Ferreiro p, A., Ferretti y, A., Festanti ab, J., Figiel di, M. A. S., Figueredo dl, Dp, S., Filchagin cr, D., Finogeev ba, F. M., Fionda ae, E. M., Fiore ae, E., Floratos cg, M., Floris ah, S., Foertsch bj, P., Foka cp, S., Fokin c, E., Fragiacomo dc, A., Francescon ab, U., Frankenfeld cp, U., Fuchs ah, C., Furget bp, M., Fusco Girard ac, J. J., Gaardhøje by, M., Gagliardi y, A. M., Gago cv, M., Gallio y, D. R., Gangadharan, P., Ganoti cg, Cc, C., Garabatos cp, E., Garcia Solis m, C., Gargiulo ah, I., Garishvili bt, J., Gerhard am, M., Germain df, A., Gheata ah, M., Gheata bg, B., Ghidini ae, P., Ghosh dw, S. K., Ghosh d, P., Gianotti bq, P., Giubellino ah, E., Gladysz Dziadus di, P., Glässel cl, R., Gomez k, P., González Zamora j, S., Gorbunov am, L., Görlich di, S., Gotovac dh, L. K., Graczykowski dy, R., Grajcarek cl, A., Grelli bb, A., Grigoras ah, C., Grigoras ah, V., Grigoriev bu, A., Grigoryan a, S., Grigoryan bk, B., Grinyov c, N., Grion dc, J. F., Grosse Oetringhaus ah, J. Y., Grossiord du, R., Grosso ah, F., Guber ba, R., Guernane bp, B., Guerzoni z, M., Guilbaud du, K., Gulbrandsen by, H., Gulkanyan a, T., Gunji dr, A., Gupta ci, R., Gupta ci, K. H., Khan o, R., Haake ay, Ø., Haaland q, C., Hadjidakis au, M., Haiduc bg, H., Hamagaki dr, G., Hamar ea, L. D., Hanratty cu, A., Hansen by, J. W., Harris eb, H., Hartmann am, A., Harton m, D., Hatzifotiadou cx, S., Hayashi dr, S. T., Heckel aw, M., Heide ay, H., Helstrup ai, A., Herghelegiu bw, G., Herrera Corral k, B. A., Hess ag, K. F., Hetland ai, B., Hicks eb, B., Hippolyte az, J., Hladky be, P., Hristov ah, M., Huang q, T. J., Humanic, D., Hutter am, D. S., Hwang t, R., Ilkaev cr, I., Ilkiv bv, M., Inaba d, E., Incani w, G. M., Innocenti y, C., Ionita ah, M., Ippolitov c, M., Irfan r, M., Ivanov cp, V., Ivanov cd, O., Ivanytskyi c, A., Jachołkowski aa, P. M., Jacobs b, C., Jahnke dl, H. J., Jang bm, M. A., Janik dy, P. H. S. Y., Jayarathna dn, S., Jena ar, Dn, R. T., Jimenez Bustamante bh, P. G., Jones cu, H., Jung an, A., Jusko cu, S., Kalcher am, P., Kalinak bd, A., Kalweit ah, J., Kamin aw, J. H., Kang ec, V., Kaplin bu, S., Kar dw, A., Karasu Uysal bn, O., Karavichev ba, T., Karavicheva ba, E., Karpechev ba, U., Kebschull av, R., Keidel ed, B., Ketzer ck, M. M., Khan r, P., Khan ct, S. A., Khan dw, A., Khanzadeev cd, Y., Kharlov de, B., Kileng ai, B., Kim ec, D. W., Kim bm, An, D. J., Kim do, J. S., Kim an, M., Kim an, M., Kim ec, S., Kim t, T., Kim ec, S., Kirsch am, I., Kisel am, S., Kiselev bc, A., Kisiel dy, G., Kiss ea, J. L., Klay f, J., Klein cl, C., Klein Bösing ay, A., Kluge ah, M. L., Knichel cp, A. G., Knospe dj, C., Kobdaj ah, Dg, M., Kofarago ah, M. K., Köhler cp, T., Kollegger am, A., Kolojvari dv, V., Kondratiev dv, N., Kondratyeva bu, A., Konevskikh ba, V., Kovalenko dv, M., Kowalski ah, Di, S., Kox bp, G., Koyithatta Meethaleveedu ar, J., Kral do, I., Králik bd, F., Kramer aw, A., Kravcˇáková al, M., Krelina ak, M., Kretz am, M., Krivda cu, Bd, F., Krizek cb, Ap, M., Krus ak, E., Kryshen cd, M., Krzewicki cp, V., Kucˇera cb, Y., Kucheriaev c, T., Kugathasan ah, C., Kuhn az, P. G., Kuijer bz, I., Kulakov aw, J., Kumar ar, P., Kurashvili bv, A., Kurepin ba, A. B., Kurepin ba, A., Kuryakin cr, S., Kushpil cb, V., Kushpil cb, M. J., Kweon cl, At, Y., Kwon ec, P., Ladron de Guevara bh, C., Lagana Fernandes dl, I., Lakomov au, R., Langoy dx, C., Lara av, A., Lardeux df, A., Lattuca y, S. L., La Pointe dd, Bb, P., La Rocca aa, L., Leardini cl, G. R., Lee cu, I., Legrand ah, J., Lehnert aw, R. C., Lemmon ca, M., Lenhardt cp, V., Lenti cw, E., Leogrande bb, M., Leoncino y, I., León Monzón dk, P., Lévai ea, S., Li g, J., Lien dx, R., Lietava cu, S., Lindal u, V., Lindenstruth am, C., Lippmann cp, M. A., Lisa, H. M., Ljunggren af, D. F., Lodato bb, P. I., Loenne q, V. R., Loggins dz, V., Loginov bu, D., Lohner cl, C., Loizides b, X., Lopez bo, E., López Torres i, X. G., Lu cl, P., Luettig aw, M., Lunardon ab, J., Luo g, C., Luzzi ah, R., Ma eb, A., Maevskaya ba, M., Mager ah, D. P., Mahapatra bf, A., Maire cl, Az, M., Malaev cd, I., Maldonado Cervantes bh, L., Malinina bk, D., Mal’Kevich bc, P., Malzacher cp, A., Mamonov cr, L., Manceau dd, V., Manko c, F., Manso bo, V., Manzari ah, Cw, M., Marchisone y, J., Mareš be, A., Margotti cx, A., Marín cp, C., Markert ah, Dj, M., Marquard aw, I., Martashvili dq, N. A., Martin cp, P., Martinengo ah, M. I., Martínez b, G., Martínez García df, J., Martin Blanco df, Y., Martynov c, A., Mas df, S., Masciocchi cp, M., Masera y, A., Masoni cy, L., Massacrier df, A., Mastroserio ae, A., Matyja di, C., Mayer di, J., Mazer dq, R., Mazumder a, M. A., Mazzoni db, F., Meddi v, A., Menchaca Rocha bi, E., Meninno ac, J., Mercado Pérez cl, M., Meres aj, Y., Miake d, K., Mikhaylov bc, Bk, L., Milano ah, J., Milosevic u, A., Mischke bb, A. N., Mishra a, D., Mis ́kowiec cp, C. M., Mitu bg, J., Mlynarz dz, B., Mohanty dw, Bx, L., Molnar az, L., Montaño Zetina k, E., Montes j, M., Morando ab, D. A., Moreira De Godoy dl, S., Moretto ab, A., Morreale do, A., Morsch ah, V., Muccifora bq, E., Mudnic dh, S., Muhuri dw, M., Mukherjee dw, H., Müller ah, M. G., Munhoz dl, S., Murray ch, L., Musa ah, J., Musinsky bd, B. K., Nandi ar, R., Nania cx, E., Nappi cw, C., Nattrass dq, T. K., Nayak dw, S., Nazarenko cr, A., Nedosekin bc, M., Nicassio cp, M., Niculescu bg, B. S., Nielsen by, S., Nikolaev c, S., Nikulin c, V., Nikulin cd, B. S., Nilsen ce, F., Noferini l, Cx, P., Nomokonov bk, G., Nooren bb, A., Nyanin c, J., Nystrand q, H., Oeschler cl, Ax, S., Oh eb, S. K., Oh bl, An, 6, A., Okatan bn, L., Olah ea, J., Oleniacz dy, A. C., Oliveira Da Silva dl, J., Onderwaater cp, C., Oppedisano dd, A., Ortiz Velasquez af, A., Oskarsson af, J., Otwinowski cp, K., Oyama cl, Y., Pachmayer cl, M., Pachr ak, P., Pagano ac, G., Paic ́ bh, F., Painke am, C., Pajares p, S. K., Pal dw, A., Palmeri cz, D., Pant ar, V., Papikyan a, G. S., Pappalardo cz, W. J., Park cp, A., Passfeld ay, D. I., Patalakha de, V., Paticchio cw, B., Paul ct, T., Pawlak dy, T., Peitzmann bb, H., Pereira Da Costa n, E., Pereira De Oliveira Filho dl, D., Peresunko c, C. E., Pérez Lara bz, W., Peryt dy, A., Pesci cx, Y., Pestov e, V., Petrácˇek ak, M., Petran ak, M., Petris bw, M., Petrovici bw, C., Petta aa, M., Pikna aj, P., Pillot df, O., Pinazza ah, L., Pinsky dn, D. B., Piyarathna dn, M., Płoskon ́ b, M., Planinic cq, Dt, J., Pluta dy, S., Pochybova ea, P. L. M., Podesta Lerma dk, M. G., Poghosyan ah, Ce, E. H. O., Pohjoisaho ap, B., Polichtchouk de, N., Poljak cq, A., Pop bw, S., Porteboeuf Houssais bo, J., Porter b, V., Pospisil ak, B., Potukuchi ci, S. K., Prasad d, R., Preghenella cx, F., Prino dd, C. A., Pruneau dz, I., Pshenichnov ba, G., Puddu w, V., Punin cr, J., Putschke dz, H., Qvigstad u, A., Rachevski dc, S., Raha d, J., Rak do, A., Rakotozafindrabe n, L., Ramello ad, R., Raniwala cj, S., Raniwala cj, S. S., Räsänen ap, B. T., Rascanu aw, D., Rathee cf, A. W., Rauf o, V., Razazi w, K. F., Read dq, J. S., Real bp, K., Redlich bv, R. J., Reed eb, A., Rehman q, P., Reichelt aw, M., Reicher bb, F., Reidt cl, R., Renfordt aw, A. R., Reolon bq, A., Reshetin ba, F., Rettig am, J. P., Revol ah, K., Reygers cl, V., Riabov cd, R. A., Ricci br, T., Richert af, M., Richter u, P., Riedler ah, W., Riegler ah, F., Riggi aa, A., Rivetti dd, E., Rocco bb, M., Rodríguez Cahuantzi b, A., Rodriguez Manso bz, K., Røed u, E., Rogochaya bk, S., Rohni ci, D., Rohr am, D., Röhrich q, R., Romita dp, Ca, F., Ronchetti bq, L., Ronflette df, P., Rosnet bo, S., Rossegger ah, A., Rossi ah, F., Roukoutakis cg, A., Roy a, C., Roy az, P., Roy ct, A. J., Rubio Montero j, R., Russo y, E., Ryabinkin c, Y., Ryabov cd, A., Rybicki di, S., Sadovsky de, K., Šafarˇík ah, B., Sahlmuller aw, R., Sahoo a, P. K., Sahu bf, J., Saini dw, C. A., Salgado p, J., Salzwedel, S., Sambyal ci, V., Samsonov cd, X., Sanchez Castro az, Bh, F. J., Sánchez Rodríguez dk, L., Šándor bd, A., Sandoval bi, M., Sano d, G., Santagati aa, D., Sarkar dw, E., Scapparone cx, F., Scarlassara ab, R. P., Scharenberg cn, C., Schiaua bw, R., Schicker cl, C., Schmidt cp, H. R., Schmidt ag, S., Schuchmann aw, J., Schukraft ah, M., Schulc ak, T., Schuster eb, Y., Schutz ah, K., Schwarz cp, K., Schweda cp, G., Scioli z, E., Scomparin dd, P. A., Scott cu, R., Scott dq, G., Segato ab, J. E., Seger ce, I., Selyuzhenkov cp, J., Seo co, E., Serradilla j, Bi, A., Sevcenco bg, A., Shabetai df, G., Shabratova bk, R., Shahoyan ah, A., Shangaraev de, N., Sharma dq, Bf, S., Sharma ci, K., Shigaki aq, K., Shtejer y, Y., Sibiriak c, S., Siddhanta cy, T., Siemiarczuk bv, D., Silvermyr cc, C., Silvestre bp, G., Simatovic dt, R., Singaraju dw, R., Singh ci, S., Singha bx, Dw, V., Singhal dw, B. C., Sinha dw, T., Sinha ct, B., Sitar aj, M., Sitta ad, T. B., Skaali u, K., Skjerdal q, R., Smakal ak, N., Smirnov eb, R. J. M., Snellings bb, C., Søgaard af, R., Soltz bt, J., Song co, M., Song ec, F., Soramel ab, S., Sorensen dq, M., Spacek ak, I., Sputowska di, M., Spyropoulou Stassinaki cg, B. K., Srivastava cn, J., Stachel cl, I., Stan bg, G., Stefanek bv, M., Steinpreis, E., Stenlund af, G., Steyn bj, J. H., Stiller cl, D., Stocco df, M., Stolpovskiy de, P., Strmen aj, A. A. P., Suaide dl, M. A., Subieta Vasquez y, T., Sugitate aq, C., Suire au, M., Suleymanov o, R., Sultanov bc, M., Šumbera cb, T., Susa cq, T. J. M., Symons b, A., Szanto de Toledo dl, I., Szarka aj, A., Szczepankiewicz ah, M., Szymanski dy, J., Takahashi dm, M. A., Tangaro ae, J. D., Tapia Takaki au, A., Tarantola Peloni aw, A., Tarazona Martinez ah, M. G., Tarzila bw, A., Tauro ah, G., Tejeda Muñoz b, A., Telesca ah, C., Terrevoli w, A., Ter Minasyan bu, J., Thäder cp, D., Thomas bb, R., Tieulent du, A. R., Timmins dn, A., Toia da, Aw, H., Torii dr, V., Trubnikov c, W. H., Trzaska do, T., Tsuji dr, A., Tumkin cr, R., Turrisi da, T. S., Tveter u, J., Ulery aw, K., Ullaland q, A., Uras du, G. L., Usai w, M., Vajzer cb, M., Vala bk, L., Valencia Palomo au, S., Vallero y, Cl, P., Vande Vyvre ah, L., Vannucci br, J., Van Der Maarel bb, J. W., Van Hoorne ah, M., van Leeuwen bb, A., Vargas b, R., Varma ar, M., Vasileiou cg, A., Vasiliev c, V., Vechernin dv, M., Veldhoen bb, A., Velure q, M., Venaruzzo x, E., Vercellin y, S., Vergara Limón b, R., Vernet h, M., Verweij dz, L., Vickovic dh, G., Viesti ab, J., Viinikainen do, Z., Vilakazi bj, O., Villalobos Baillie cu, A., Vinogradov c, L., Vinogradov dv, Y., Vinogradov cr, T., Virgili ac, Y. P., Viyogi dw, A., Vodopyanov bk, M. A., Völkl cl, K., Voloshin bc, S. A., Voloshin dz, G., Volpe ah, B., von Haller ah, I., Vorobyev dv, D., Vranic cp, J., Vrláková al, B., Vulpescu bo, A., Vyushin cr, B., Wagner q, J., Wagner cp, V., Wagner ak, M., Wang g, Y., Wang cl, D., Watanabe d, M., Weber dn, J. P., Wessels ay, U., Westerhoff ay, J., Wiechula ag, J., Wikne u, M., Wilde ay, G., Wilk bv, J., Wilkinson cl, M. C. S., Williams cx, B., Windelband cl, M., Winn cl, C., Xiang g, C. G., Yaldo dz, Y., Yamaguchi dr, H., Yang bb, P., Yang g, S., Yang q, S., Yano aq, S., Yasnopolskiy c, J., Yi co, Z., Yin g, I. K., Yoo co, I., Yushmanov c, Zaccolo by, V., C., Zach ak, A., Zaman o, C., Zampolli cx, S., Zaporozhets bk, A., Zarochentsev dv, P., Závada be, N., Zaviyalov cr, H., Zbroszczyk dy, I. S., Zgura bg, M., Zhalov cd, F., Zhang g, H., Zhang g, X., Zhang bo, G, B, Y., Zhangg, C., Zhaou, D., Zhoug, F., Zhoug, Y., Zhoubb, H., Zhug, J., Zhudf, G, J., Zhug, X., Zhug, A., Zichichil, A., Zimmermann cl, M. B., Zimmermann ay, G., Zinovjev c, Y., Zoccarato du, M., Zynovyev c, and M., Zyzak aw
- Subjects
Particle ratios ,ALICE ,High pT ,Nuclear modification factor ,Identified particle production ,Particle ratio ,LHC ,Baryon anomaly - Abstract
Transverse momentum spectra of π±, K± and p(p ̄) up to pT = 20 GeV/c at mid-rapidity in pp, peripheral (60–80%) and central (0–5%) Pb–Pb collisions at √sNN = 2.76 TeV have been measured using the ALICE detector at the Large Hadron Collider. The proton-to-pion and the kaon-to-pion ratios both show a distinct peak at pT ≈ 3 GeV/c in central Pb–Pb collisions. Below the peak, pT < 3 GeV/c, both ratios are in good agreement with hydrodynamical calculations, suggesting that the peak itself is dominantly the result of radial flow rather than anomalous hadronization processes. For pT > 10 GeV/c particle ratios in pp and Pb–Pb collisions are in agreement and the nuclear modification factors for π±, K± and p(p ̄) indicate that, within the systematic and statistical uncertainties, the suppression is the same. This suggests that the chemical composition of leading particles from jets in the medium is similar to that of vacuum jets.
- Published
- 2014
48. Control of fine particulate emissions from coal-fired utility boilers: Spin filter collection device (rotary cyclone)
- Author
-
He, Bo X., primary
- Published
- 1990
- Full Text
- View/download PDF
49. Culture-independent analysis of microflora in Gayals (Bos frontalis) feces
- Author
-
Run-chi, G, Zun-xi, H, Chang-fei, W, Bo, X, and Xiao-yan, W
- Subjects
Gayal, culture-independent, fecal, phylogenetic analysis, 16S/18S rDNA - Abstract
Comparative DNA sequence analysis of 16S/18S rRNA genes (rDNA) were undertaken to further our understanding of the make-up of micro-organisms communities in the feces of Gayals. Total DNA wereextracted from the feces of 5 Gayals. Two rDNA libraries (16S/18S rDNA) were constructed. In the 16S rDNA library, phylogenetic and sequence similarity analyses of the resultant 71 clone sequences revealed the presence of 67 operational taxonomic units (OTUs) or phylotypes and defined as having more than 97% of sequence similarity. The sequences were affiliated with the following phyla: Firmicutes (34.3%), Bacteroidetes (6.0%), Proteobacteria (4.5%), and uncultured bacteria (55.2%). A setof 58 sequences were analyzed in the 18S rDNA library, which were classified into 27 OTUs. They were mainly affiliated with the following phyla: Protozoa (25.9%), Basidiomycota (3.7%), Ascomycota (11.1%),and uncultured eukaryotes (59.3%). The sequence analysis indicated that more than half of the species, harbored in Gayals fecal belonged to the not-yet-cultured groups at 90% 16S/18S similarity levels with cultured species. In addition, micro-organisms of Chytridiomycetes was one of the most significant cellulose producing species obtained from the Gayal feces as well.
- Published
- 2012
50. Characterization of 6 Bacillus subtilis -mannanases and their genes
- Author
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Bo, X, Lei, D, Xiang-hua, T, Jun-jun, L, Yun-juan, Y, Yue-lin, M, and Zunxi, H
- Subjects
mannanase, enzyme characterization, gene cloning.FLT - Abstract
No Abstract
- Published
- 2011
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