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1. Metabolic Reprogramming and Cancer

19. Low RECK Expression Is Part of the Cervical Carcinogenesis Mechanisms

23. SOM-20-0113-R2_SupMaterial_revised_version_FINAL – Supplemental material for Long-term single-cell passaging of human iPSC fully supports pluripotency and high-efficient trilineage differentiation capacity

24. Higher expression and activity of metalloproteinases in human cervical carcinoma cell lines is associated with HPV presence

25. Human Papillomavirus 16 E7 Promotes EGFR/PI3K/AKT1/NRF2 Signaling Pathway Contributing to PIR/NF-κB Activation in Oral Cancer Cells

26. Long-term single-cell passaging of human iPSC fully supports pluripotency and high-efficient trilineage differentiation capacity

27. Human papillomavirus type 18 E5 oncoprotein cooperates with E6 and E7 in promoting cell viability and invasion and in modulating the cellular redox state

28. Antiviral activity of curcumin-nanoemulsion associated with photodynamic therapy in vulvar cell lines transducing different variants of HPV-16

39. Swainsonine, an alpha-mannosidase inhibitor, may worsen cervical cancer progression through the increase in myeloid derived suppressor cells population

40. Lactate secreted by cervical cancer cells modulates macrophage phenotype

41. HPV-Mediated Resistance to TNF and TRAIL Is Characterized by Global Alterations in Apoptosis Regulatory Factors, Dysregulation of Death Receptors, and Induction of ROS/RNS

42. Tobacco Exposure Enhances Human Papillomavirus 16 Oncogene Expression via EGFR/PI3K/Akt/c-Jun Signaling Pathway in Cervical Cancer Cells

43. Characterization of global transcription profile of normal and HPV-immortalized keratinocytes and their response to TNF treatment

44. Additional file 2: Table S2. of Co-infection of sexually transmitted pathogens and Human Papillomavirus in cervical samples of women of Brazil

45. Additional file 1: Table S1. of Co-infection of sexually transmitted pathogens and Human Papillomavirus in cervical samples of women of Brazil

46. Supplementary Tables and Figures from Upregulation of PIR gene expression induced by human papillomavirus E6 and E7 in epithelial oral and cervical cells

48. Strong SOD2 expression and HPV-16/18 positivity are independent events in cervical cancer

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