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41 results on '"Bohring, Axel"'

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2. Mutations in MAP3K7 that Alter the Activity of the TAK1 Signaling Complex Cause Frontometaphyseal Dysplasia

3. Expanding the clinical spectrum of recessive truncating mutations of KLHL7 to a Bohring-Opitz-like phenotype

7. Mutations in CD96, a member of the immunoglobulin superfamily, cause a form of the C (optiz trigonocephaly) syndrome

8. ANKRD11 variants: KBG syndrome and beyond

9. Novel FOXF1 Mutations in Sporadic and Familial Cases of Alveolar Capillary Dysplasia with Misaligned Pulmonary Veins Imply a Role for its DNA Binding Domain

11. The male phenotype in osteopathia striata congenita with cranial sclerosis

13. The face of Noonan syndrome: Does phenotype predict genotype

18. Genotype-phenotype correlation in eight new patients with a deletion encompassing 2q31.1

19. Expanding the clinical spectrum of recessive truncating mutations ofKLHL7to a Bohring-Opitz-like phenotype

20. Autosomal dominant frontometaphyseal dysplasia: Delineation of the clinical phenotype

21. 47 patients with FLNA associated periventricular nodular heterotopia

22. 47 patients with FLNA associated periventricular nodular heterotopia

23. Novel FOXF1 mutations in sporadic and familial cases of alveolar capillary dysplasia with misaligned pulmonary veins imply a role for its DNA binding domain

24. Increasing the Yield in Targeted Next-Generation Sequencing by Implicating CNV Analysis, Non-Coding Exons and the Overall Variant Load: The Example of Retinal Dystrophies

26. Increasing the Yield in Targeted Next-Generation Sequencing by Implicating CNV Analysis, Non-Coding Exons and the Overall Variant Load: The Example of Retinal Dystrophies

27. NovelFOXF1Mutations in Sporadic and Familial Cases of Alveolar Capillary Dysplasia with Misaligned Pulmonary Veins Imply a Role for its DNA Binding Domain

28. Spectrum of novel mutations found in Waardenburg syndrome types 1 and 2: implications for molecular genetic diagnostics

31. Germline mutations in WTX cause a sclerosing skeletal dysplasia but do not predispose to tumorigenesis

32. Aberrant splicing is a common mutational mechanism inMKS1, a key player in Meckel-Gruber syndrome

34. Expanding the clinical spectrum of recessive truncating mutations of KLHL7to a Bohring-Opitz-like phenotype

35. Polytopic anomalies with agenesis of the lower vertebral column

40. Germline mutations in WTX cause a sclerosing skeletal dysplasia but do not predispose to tumorigenesis.

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