48 results on '"Breiderhoff, Tilman"'
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2. Strikingly conserved gene expression changes of polyamine regulating enzymes among various forms of acute and chronic kidney injury
3. Pan-claudin family interactome analysis reveals shared and specific interactions
4. Urinary Acidification Does Not Explain the Absence of Nephrocalcinosis in a Mouse Model of Familial Hypomagnesaemia with Hypercalciuria and Nephrocalcinosis (FHHNC)
5. Cyclin M2 (CNNM2) knockout mice show mild hypomagnesaemia and developmental defects
6. Modulation of intestinal IL-37 expression and its impact on the epithelial innate immune response and barrier integrity
7. Neuronal Sorting Protein-Related Receptor sorLA/LR11 Regulates Processing of the Amyloid Precursor Protein
8. Mosaic expression of claudins in thick ascending limbs of Henle results in spatial separation of paracellular Na⁺ and Mg 2+ transport
9. Furosemide rescues hypercalciuria in familial hypomagnesaemia with hypercalciuria and nephrocalcinosis model
10. Furosemide rescues hypercalciuria in familial hypomagnesaemia with hypercalciuria and nephrocalcinosis model
11. One gene, two paracellular ion channels—claudin-10 in the kidney
12. Unrecognized role of claudin‐10b in basolateral membrane infoldings of the thick ascending limb
13. Impact of claudin‐10 deficiency on amelogenesis: Lesson from a HELIX tooth
14. Claudin-10a Deficiency Shifts Proximal Tubular Cl- Permeability to Cation Selectivity via Claudin-2 Redistribution
15. Deletion of claudin-10 (Cldn10) in the thick ascending limb impairs paracellular sodium permeability and leads to hypermagnesemia and nephrocalcinosis
16. Sort1, Encoded by the Cardiovascular Risk Locus 1p13.3, Is a Regulator of Hepatic Lipoprotein Export
17. Differential localization patterns of claudin 10, 16, and 19 in human, mouse, and rat renal tubular epithelia
18. Targeted deletion of murine cldn16 identifies extra- and intrarenal compensatory mechanisms of [Ca.sup.2+] and [Mg.sup.2+] wasting
19. Roles for the pro-neurotrophin receptor sortilin in neuronal development, aging and brain injury
20. The Sorting Receptor Sortilin Exhibits a Dual Function in Exocytic Trafficking of Interferon-γ and Granzyme A in T Cells
21. Claudin-10a Deficiency Shifts Proximal Tubular Cl-Permeability to Cation Selectivity viaClaudin-2 Redistribution
22. Differential localization patterns of claudin 10, 16, and 19 in human, mouse, and rat renal tubular epithelia.
23. Altered paracellular cation permeability due to a rare CLDN10B variant causes anhidrosis and kidney damage
24. Deletion of claudin-10 rescues claudin-16–deficient mice from hypomagnesemia and hypercalciuria
25. SORCS 1 and SORCS 3 control energy balance and orexigenic peptide production
26. Claudin-16 deficiency impairs tight junction function in ameloblasts, leading to abnormal enamel formation
27. Claudin Loss-of-Function Disrupts Tight Junctions and Impairs Amelogenesis
28. HDAC as Versatile Regulators of the Intestinal Barrier Integrity and Epithelial Regeneration in Inflammatory Bowel Disease
29. Structural Basis of the Oncogenic Interaction of Phosphatase PRL-1 with the Magnesium Transporter CNNM2
30. Mosaic expression of claudins in thick ascending limbs of Henle results in spatial separation of paracellular Na + and Mg 2+ transport
31. One gene, two paracellular ion channels—claudin-10 in the kidney
32. Role of Sortilin in Models of Autoimmune Neuroinflammation
33. Claudin‐16 Deficiency Impairs Tight Junction Function in Ameloblasts, Leading to Abnormal Enamel Formation
34. SORCS1 and SORCS3 control energy balance and orexigenic peptide production.
35. Sort1, encoded by the cardiovascular risk locus 1p13.3, is a regulator of hepatic lipoprotein export
36. Tu1783 - HDAC as Versatile Regulators of the Intestinal Barrier Integrity and Epithelial Regeneration in Inflammatory Bowel Disease
37. Mosaic expression of claudins in thick ascending limbs of Henle results in spatial separation of paracellular Na+ and Mg2+ transport.
38. Sortilin-Related Receptor SORCS3 Is a Postsynaptic Modulator of Synaptic Depression and Fear Extinction
39. Targeted deletion of murineCldn16identifies extra- and intrarenal compensatory mechanisms of Ca2+and Mg2+wasting
40. Hyporesponsiveness to Glucocorticoids in Mice Genetically Deficient for the Corticosteroid Binding Globulin
41. Disruption of ClC-3, a Chloride Channel Expressed on Synaptic Vesicles, Leads to a Loss of the Hippocampus
42. Mutational Analysis Demonstrates That ClC-4 and ClC-5 Directly Mediate Plasma Membrane Currents
43. Deletion of claudin-10 [Cldn 10) in the thick ascending limb impairs paracellular sodium permeability and leads to hypermagnesemia and nephrocalcinosis.
44. Targeted deletion of murine Cldn16 identifies extra- and intrarenal compensatory mechanisms of Ca2+ and Mg2+ wasting.
45. Targeted deletion of murine Cldn16identifies extra- and intrarenal compensatory mechanisms of Ca2+and Mg2+wasting
46. Hyporesponsiveness to Glucocorticoids in Mice Genetically Deficient for the Corticosteroid Binding Globulin.
47. Mosaic expression of claudins in thick ascending limbs of Henle results in spatial separation of paracellular Na+ and Mg2+ transport.
48. Claudin-16 Deficiency Impairs Tight Junction Function in Ameloblasts, Leading to Abnormal Enamel Formation.
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