Search

Your search keyword '"Buckup, Mark"' showing total 21 results
Start Over Author "Buckup, Mark"
21 results on '"Buckup, Mark"'

1. An IL-4 signalling axis in bone marrow drives pro-tumorigenic myelopoiesis

Author

LaMarche, Nelson M., Hegde, Samarth, Park, Matthew D., Maier, Barbara B., Troncoso, Leanna, Le Berichel, Jessica, Hamon, Pauline, Belabed, Meriem, Mattiuz, Raphaël, Hennequin, Clotilde, Chin, Theodore, Reid, Amanda M., Reyes-Torres, Iván, Nemeth, Erika, Zhang, Ruiyuan, Olson, Oakley C., Doroshow, Deborah B., Rohs, Nicholas C., Gomez, Jorge E., Veluswamy, Rajwanth, Hall, Nicole, Venturini, Nicholas, Ginhoux, Florent, Liu, Zhaoyuan, Buckup, Mark, Figueiredo, Igor, Roudko, Vladimir, Miyake, Kensuke, Karasuyama, Hajime, Gonzalez-Kozlova, Edgar, Gnjatic, Sacha, Passegué, Emmanuelle, Kim-Schulze, Seunghee, Brown, Brian D., Hirsch, Fred R., Kim, Brian S., Marron, Thomas U., and Merad, Miriam

Published
2024
Full Text
View/download PDF

2. SU086, an inhibitor of HSP90, impairs glycolysis and represents a treatment strategy for advanced prostate cancer

Author

Rice, Meghan A, Kumar, Vineet, Tailor, Dhanir, Garcia-Marques, Fernando Jose, Hsu, En-Chi, Liu, Shiqin, Bermudez, Abel, Kanchustambham, Vijayalakshmi, Shankar, Vishnu, Inde, Zintis, Alabi, Busola Ruth, Muruganantham, Arvind, Shen, Michelle, Pandrala, Mallesh, Nolley, Rosalie, Aslan, Merve, Ghoochani, Ali, Agarwal, Arushi, Buckup, Mark, Kumar, Manoj, Going, Catherine C, Peehl, Donna M, Dixon, Scott J, Zare, Richard N, Brooks, James D, Pitteri, Sharon J, Malhotra, Sanjay V, and Stoyanova, Tanya

Subjects
Biomedical and Clinical Sciences, Clinical Sciences, Oncology and Carcinogenesis, Prostate Cancer, Biotechnology, Aging, Clinical Research, Cancer, Urologic Diseases, Development of treatments and therapeutic interventions, 5.1 Pharmaceuticals, Cell Proliferation, Glycolysis, HSP90 Heat-Shock Proteins, Humans, Male, Prostatic Neoplasms, Proteomics, HSP90, combination therapy, glycolysis, metabolism, prostate cancer, therapeutics, therapy, Biomedical and clinical sciences
Abstract

Among men, prostate cancer is the second leading cause of cancer-associated mortality, with advanced disease remaining a major clinical challenge. We describe a small molecule, SU086, as a therapeutic strategy for advanced prostate cancer. We demonstrate that SU086 inhibits the growth of prostate cancer cells in vitro, cell-line and patient-derived xenografts in vivo, and ex vivo prostate cancer patient specimens. Furthermore, SU086 in combination with standard of care second-generation anti-androgen therapies displays increased impairment of prostate cancer cell and tumor growth in vitro and in vivo. Cellular thermal shift assay reveals that SU086 binds to heat shock protein 90 (HSP90) and leads to a decrease in HSP90 levels. Proteomic profiling demonstrates that SU086 binds to and decreases HSP90. Metabolomic profiling reveals that SU086 leads to perturbation of glycolysis. Our study identifies SU086 as a treatment for advanced prostate cancer as a single agent or when combined with second-generation anti-androgens.

Published
2022

3. Intratumoral dendritic cell–CD4+ T helper cell niches enable CD8+ T cell differentiation following PD-1 blockade in hepatocellular carcinoma

Author

Magen, Assaf, Hamon, Pauline, Fiaschi, Nathalie, Soong, Brian Y., Park, Matthew D., Mattiuz, Raphaël, Humblin, Etienne, Troncoso, Leanna, D’souza, Darwin, Dawson, Travis, Kim, Joel, Hamel, Steven, Buckup, Mark, Chang, Christie, Tabachnikova, Alexandra, Schwartz, Hara, Malissen, Nausicaa, Lavin, Yonit, Soares-Schanoski, Alessandra, Giotti, Bruno, Hegde, Samarth, Ioannou, Giorgio, Gonzalez-Kozlova, Edgar, Hennequin, Clotilde, Le Berichel, Jessica, Zhao, Zhen, Ward, Stephen C., Fiel, Isabel, Kou, Baijun, Dobosz, Michael, Li, Lianjie, Adler, Christina, Ni, Min, Wei, Yi, Wang, Wei, Atwal, Gurinder S., Kundu, Kunal, Cygan, Kamil J., Tsankov, Alexander M., Rahman, Adeeb, Price, Colles, Fernandez, Nicolas, He, Jiang, Gupta, Namita T., Kim-Schulze, Seunghee, Gnjatic, Sacha, Kenigsberg, Ephraim, Deering, Raquel P., Schwartz, Myron, Marron, Thomas U., Thurston, Gavin, Kamphorst, Alice O., and Merad, Miriam

Published
2023
Full Text
View/download PDF

4. Spatial CRISPR genomics identifies regulators of the tumor microenvironment

Author

Dhainaut, Maxime, Rose, Samuel A., Akturk, Guray, Wroblewska, Aleksandra, Nielsen, Sebastian R., Park, Eun Sook, Buckup, Mark, Roudko, Vladimir, Pia, Luisanna, Sweeney, Robert, Le Berichel, Jessica, Wilk, C. Matthias, Bektesevic, Anela, Lee, Brian H., Bhardwaj, Nina, Rahman, Adeeb H., Baccarini, Alessia, Gnjatic, Sacha, Pe’er, Dana, Merad, Miriam, and Brown, Brian D.

Published
2022
Full Text
View/download PDF

5. Neoadjuvant cemiplimab for resectable hepatocellular carcinoma: a single-arm, open-label, phase 2 trial

Author

Marron, Thomas U, Fiel, Maria Isabel, Hamon, Pauline, Fiaschi, Nathalie, Kim, Edward, Ward, Stephen C, Zhao, Zhen, Kim, Joel, Kennedy, Paul, Gunasekaran, Ganesh, Tabrizian, Parissa, Doroshow, Deborah, Legg, Meredith, Hammad, Ashley, Magen, Assaf, Kamphorst, Alice O, Shareef, Muhammed, Gupta, Namita T, Deering, Raquel, Wang, Wei, Wang, Fang, Thanigaimani, Pradeep, Mani, Jayakumar, Troncoso, Leanna, Tabachnikova, Alexandra, Chang, Christie, Akturk, Guray, Buckup, Mark, Hamel, Steven, Ioannou, Giorgio, Hennequin, Clotilde, Jamal, Hajra, Brown, Haley, Bonaccorso, Antoinette, Labow, Daniel, Sarpel, Umut, Rosenbloom, Talia, Sung, Max W, Kou, Baijun, Li, Siyu, Jankovic, Vladimir, James, Nicola, Hamon, Sara C, Cheung, Hung Kam, Sims, Jennifer S, Miller, Elizabeth, Bhardwaj, Nina, Thurston, Gavin, Lowy, Israel, Gnjatic, Sacha, Taouli, Bachir, Schwartz, Myron E, and Merad, Miriam

Published
2022
Full Text
View/download PDF

9. An IL-4 signalling axis in bone marrow drives pro-tumorigenic myelopoiesis

Author

LaMarche, Nelson M., primary, Hegde, Samarth, additional, Park, Matthew D., additional, Maier, Barbara B., additional, Troncoso, Leanna, additional, Le Berichel, Jessica, additional, Hamon, Pauline, additional, Belabed, Meriem, additional, Mattiuz, Raphaël, additional, Hennequin, Clotilde, additional, Chin, Theodore, additional, Reid, Amanda M., additional, Reyes-Torres, Iván, additional, Nemeth, Erika, additional, Zhang, Ruiyuan, additional, Olson, Oakley C., additional, Doroshow, Deborah B., additional, Rohs, Nicholas C., additional, Gomez, Jorge E., additional, Veluswamy, Rajwanth, additional, Hall, Nicole, additional, Venturini, Nicholas, additional, Ginhoux, Florent, additional, Liu, Zhaoyuan, additional, Buckup, Mark, additional, Figueiredo, Igor, additional, Roudko, Vladimir, additional, Miyake, Kensuke, additional, Karasuyama, Hajime, additional, Gonzalez-Kozlova, Edgar, additional, Gnjatic, Sacha, additional, Passegué, Emmanuelle, additional, Kim-Schulze, Seunghee, additional, Brown, Brian D., additional, Hirsch, Fred R., additional, Kim, Brian S., additional, Marron, Thomas U., additional, and Merad, Miriam, additional

Published
2023
Full Text
View/download PDF

10. 688 Prediction of HCC response to neoadjuvant immunotherapy using multiparametric magnetic resonance imaging: a preliminary study

Author

Bane, Octavia, primary, Bhuiyan, Enamul, additional, Kennedy, Paul, additional, Shareef, Muhammed, additional, Hamon, Pauline, additional, Buckup, Mark, additional, Gnjatic, Sacha, additional, Hectors, Stefanie, additional, Cheung, Hung Kam, additional, Miller, Elizabeth, additional, Fiel, Maria Isabel, additional, Ward, Stephen, additional, Schwartz, Myron, additional, Marron, Thomas, additional, Merad, Miriam, additional, and Taouli, Bachir, additional

Published
2022
Full Text
View/download PDF

11. The interaction of CD4+ helper T cells with dendritic cells shapes the tumor microenvironment and immune checkpoint blockade response

Author

Hubrecht Institute with UMC, Cohen, Merav, Giladi, Amir, Barboy, Oren, Hamon, Pauline, Li, Baoguo, Zada, Mor, Gurevich-Shapiro, Anna, Beccaria, Cristian Gabriel, David, Eyal, Maier, Barbara B., Buckup, Mark, Kamer, Iris, Deczkowska, Aleksandra, Le Berichel, Jessica, Bar, Jair, Iannacone, Matteo, Tanay, Amos, Merad, Miriam, Amit, Ido, Hubrecht Institute with UMC, Cohen, Merav, Giladi, Amir, Barboy, Oren, Hamon, Pauline, Li, Baoguo, Zada, Mor, Gurevich-Shapiro, Anna, Beccaria, Cristian Gabriel, David, Eyal, Maier, Barbara B., Buckup, Mark, Kamer, Iris, Deczkowska, Aleksandra, Le Berichel, Jessica, Bar, Jair, Iannacone, Matteo, Tanay, Amos, Merad, Miriam, and Amit, Ido

Published
2022

12. Intratumoral mregDC and CXCL13 T helper niches enable local differentiation of CD8 T cells following PD-1 blockade

Author

Magen, Assaf, primary, Hamon, Pauline, additional, Fiaschi, Nathalie, additional, Troncoso, Leanna, additional, Humblin, Etienne, additional, D’souza, Darwin, additional, Dawson, Travis, additional, Park, Matthew D., additional, Kim, Joel, additional, Hamel, Steven, additional, Buckup, Mark, additional, Chang, Christie, additional, Tabachnikova, Alexandra, additional, Schwartz, Hara, additional, Malissen, Nausicaa, additional, Lavin, Yonit, additional, Soares-Schanoski, Alessandra, additional, Giotti, Bruno, additional, Hegde, Samarth, additional, Mattiuz, Raphaël, additional, Hennequin, Clotilde, additional, Berichel, Jessica Le, additional, Zhao, Zhen, additional, Ward, Stephen, additional, Fiel, Isabel, additional, Price, Colles, additional, Fernandez, Nicolas, additional, He, Jiang, additional, Kou, Baijun, additional, Dobosz, Michael, additional, Li, Lianjie, additional, Adler, Christina, additional, Ni, Min, additional, Wei, Yi, additional, Wang, Wei, additional, Gupta, Namita T., additional, Kundu, Kunal, additional, Cygan, Kamil, additional, Deering, Raquel P., additional, Tsankov, Alex, additional, Kim-Schulze, Seunghee, additional, Gnjatic, Sacha, additional, Kenigsberg, Ephraim, additional, Schwartz, Myron, additional, Marron, Thomas U., additional, Thurston, Gavin, additional, Kamphorst, Alice O., additional, and Merad, Miriam, additional

Published
2022
Full Text
View/download PDF

13. Shift of lung macrophage composition is associated with COVID-19 disease severity and recovery

Author

Chen, Steven T., Park, Matthew D., Del Valle, Diane Marie, Buckup, Mark, Tabachnikova, Alexandra, Simons, Nicole W., Mouskas, Konstantinos, Lee, Brian, Geanon, Daniel, D’Souza, Darwin, Dawson, Travis, Marvin, Robert, Nie, Kai, Thompson, Ryan C., Zhao, Zhen, LeBerichel, Jessica, Chang, Christie, Jamal, Hajra, Chaddha, Udit, Mathews, Kusum, Acquah, Samuel, Brown, Stacey-Ann, Reiss, Michelle, Harkin, Timothy, Feldmann, Marc, Powell, Charles A., Hook, Jaime L., Kim-Schulze, Seunghee, Rahman, Adeeb H., Brown, Brian D., Beckmann, Noam D., Gnjatic, Sacha, Kenigsberg, Ephraim, Charney, Alexander W., and Merad, Miriam

Subjects
Macrophages, Macrophages, Alveolar, COVID-19, Humans, Lung, Article, Monocytes
Abstract

Although it has been more than 2 years since the start of the coronavirus disease 2019 (COVID-19) pandemic, COVID-19 continues to be a worldwide health crisis. Despite the development of preventive vaccines, therapies to treat COVID-19 and other inflammatory diseases remain a major unmet need in medicine. Our study sought to identify drivers of disease severity and mortality to develop tailored immunotherapy strategies to halt disease progression. We assembled the Mount Sinai COVID-19 Biobank, which was composed of almost 600 hospitalized patients followed longitudinally through the peak of the pandemic in 2020. Moderate disease and survival were associated with a stronger antigen presentation and effector T cell signature. In contrast, severe disease and death were associated with an altered antigen presentation signature, increased numbers of inflammatory immature myeloid cells, and extrafollicular activated B cells that have been previously associated with autoantibody formation. In severely ill patients with COVID-19, lung tissue-resident alveolar macrophages not only were drastically depleted but also had an altered antigen presentation signature, which coincided with an influx of inflammatory monocytes and monocyte-derived macrophages. In addition, we found that the size of the alveolar macrophage pool correlated with patient outcome and that alveolar macrophage numbers and functionality were restored to homeostasis in patients who recovered from COVID-19. These data suggest that local and systemic myeloid cell dysregulation are drivers of COVID-19 severity and modulation of alveolar macrophage numbers and activity in the lung may be a viable therapeutic strategy for the treatment of critical inflammatory lung diseases.

Published
2022

14. 685 Characterization of molecular and spatial diversity of macrophages in hepatocellular carcinoma

Author

Hamon, Pauline, primary, Magen, Assaf, additional, Kim, Joel, additional, Buckup, Mark, additional, Troncoso, Leanna, additional, Hamel, Steven, additional, Berichel, Jessica Le, additional, Barboy, Oren, additional, David, Eyal, additional, Tabachnikova, Alexandra, additional, Chang, Christie, additional, Zhao, Zhen, additional, Cohen, Merav, additional, Giladi, Amir, additional, Malissen, Nausicaa, additional, Desland, Fiona, additional, Amit, Ido, additional, Kenigsberg, Ephraim, additional, Schwartz, Myron, additional, Marron, Thomas, additional, and Merad, Miriam, additional

Published
2021
Full Text
View/download PDF

15. Perturb-map enables CRISPR genomics with spatial resolution and identifies regulators of tumor immune composition

Author

Dhainaut, Maxime, primary, Rose, Samuel A, additional, Akturk, Guray, additional, Wroblewska, Aleksandra, additional, Park, Eun Sook, additional, Nielsen, Sebastian R, additional, Buckup, Mark, additional, Roudko, Vladimir, additional, Pia, Luisanna, additional, Le Berichel, Jessica, additional, Bektesevic, Anela, additional, Lee, Brian H, additional, Baccarini, Alessia, additional, Bhardwaj, Nina, additional, Rahman, Adeeb H, additional, Gnjatic, Sacha, additional, Pe’er, Dana, additional, Merad, Miriam, additional, and Brown, Brian D, additional

Published
2021
Full Text
View/download PDF

16. Abstract 64: Characterization of molecular and spatial diversity of macrophages in hepatocellular carcinoma

Author

Hamon, Pauline, primary, Magen, Assaf, additional, Cohen, Merav, additional, Tabachnikova, Alexandra, additional, Chang, Christie, additional, Buckup, Mark, additional, Troncoso, Leanna, additional, Zhao, Zhen, additional, Kim, Joel, additional, Giladi, Amir, additional, Malissen, Nausicaa, additional, Desland, Fiona, additional, Berichel, Jessica Le, additional, Amit, Ido, additional, Kenigsberg, Ephraim, additional, Schwartz, Myron, additional, Marron, Thomas, additional, and Merad, Miriam, additional

Published
2021
Full Text
View/download PDF

17. Intratumoral dendritic cell–CD4+T helper cell niches enable CD8+T cell differentiation following PD-1 blockade in hepatocellular carcinoma

Author

Magen, Assaf, Hamon, Pauline, Fiaschi, Nathalie, Soong, Brian Y., Park, Matthew D., Mattiuz, Raphaël, Humblin, Etienne, Troncoso, Leanna, D’souza, Darwin, Dawson, Travis, Kim, Joel, Hamel, Steven, Buckup, Mark, Chang, Christie, Tabachnikova, Alexandra, Schwartz, Hara, Malissen, Nausicaa, Lavin, Yonit, Soares-Schanoski, Alessandra, Giotti, Bruno, Hegde, Samarth, Ioannou, Giorgio, Gonzalez-Kozlova, Edgar, Hennequin, Clotilde, Le Berichel, Jessica, Zhao, Zhen, Ward, Stephen C., Fiel, Isabel, Kou, Baijun, Dobosz, Michael, Li, Lianjie, Adler, Christina, Ni, Min, Wei, Yi, Wang, Wei, Atwal, Gurinder S., Kundu, Kunal, Cygan, Kamil J., Tsankov, Alexander M., Rahman, Adeeb, Price, Colles, Fernandez, Nicolas, He, Jiang, Gupta, Namita T., Kim-Schulze, Seunghee, Gnjatic, Sacha, Kenigsberg, Ephraim, Deering, Raquel P., Schwartz, Myron, Marron, Thomas U., Thurston, Gavin, Kamphorst, Alice O., and Merad, Miriam

Abstract

Despite no apparent defects in T cell priming and recruitment to tumors, a large subset of T cell rich tumors fail to respond to immune checkpoint blockade (ICB). We leveraged a neoadjuvant anti-PD-1 trial in patients with hepatocellular carcinoma (HCC), as well as additional samples collected from patients treated off-label, to explore correlates of response to ICB within T cell-rich tumors. We show that ICB response correlated with the clonal expansion of intratumoral CXCL13+CH25H+IL-21+PD-1+CD4+T helper cells (“CXCL13+TH”) and Granzyme K+PD-1+effector-like CD8+T cells, whereas terminally exhausted CD39hiTOXhiPD-1hiCD8+T cells dominated in nonresponders. CD4+and CD8+T cell clones that expanded post-treatment were found in pretreatment biopsies. Notably, PD-1+TCF-1+(Progenitor-exhausted) CD8+T cells shared clones mainly with effector-like cells in responders or terminally exhausted cells in nonresponders, suggesting that local CD8+T cell differentiation occurs upon ICB. We found that these Progenitor CD8+T cells interact with CXCL13+THwithin cellular triads around dendritic cells enriched in maturation and regulatory molecules, or “mregDC”. These results suggest that discrete intratumoral niches that include mregDC and CXCL13+THcontrol the differentiation of tumor-specific Progenitor exhasuted CD8+T cells following ICB.

Published
2023
Full Text
View/download PDF

18. A shift in lung macrophage composition is associated with COVID-19 severity and recovery.

Author

Chen, Steven T., Park, Matthew D., Del Valle, Diane Marie, Buckup, Mark, Tabachnikova, Alexandra, Thompson, Ryan C., Simons, Nicole W., Mouskas, Konstantinos, Lee, Brian, Geanon, Daniel, D'Souza, Darwin, Dawson, Travis, Marvin, Robert, Nie, Kai, Zhao, Zhen, LeBerichel, Jessica, Chang, Christie, Jamal, Hajra, Akturk, Guray, and Chaddha, Udit

Subjects
COVID-19, ALVEOLAR macrophages, MYELOID cells, ANTIGEN presentation, AUTOANTIBODIES, COVID-19 pandemic, B cells, MACROPHAGES
Abstract

Although it has been more than 2 years since the start of the coronavirus disease 2019 (COVID-19) pandemic, COVID-19 continues to be a worldwide health crisis. Despite the development of preventive vaccines, therapies to treat COVID-19 and other inflammatory diseases remain a major unmet need in medicine. Our study sought to identify drivers of disease severity and mortality to develop tailored immunotherapy strategies to halt disease progression. We assembled the Mount Sinai COVID-19 Biobank, which was composed of almost 600 hospitalized patients followed longitudinally through the peak of the pandemic in 2020. Moderate disease and survival were associated with a stronger antigen presentation and effector T cell signature. In contrast, severe disease and death were associated with an altered antigen presentation signature, increased numbers of inflammatory immature myeloid cells, and extrafollicular activated B cells that have been previously associated with autoantibody formation. In severely ill patients with COVID-19, lung tissue–resident alveolar macrophages not only were drastically depleted but also had an altered antigen presentation signature, which coincided with an influx of inflammatory monocytes and monocyte-derived macrophages. In addition, we found that the size of the alveolar macrophage pool correlated with patient outcome and that alveolar macrophage numbers and functionality were restored to homeostasis in patients who recovered from COVID-19. These data suggest that local and systemic myeloid cell dysregulation are drivers of COVID-19 severity and modulation of alveolar macrophage numbers and activity in the lung may be a viable therapeutic strategy for the treatment of critical inflammatory lung diseases. Modulation of macrophages: Understanding why some individuals develop severe disease after SARS-CoV-2 infection remains a high priority. Here, Chen et al. evaluated factors associated with disease severity and survival in samples from 600 individuals hospitalized with COVID-19 during 2020. The authors found that severe disease and death were associated with altered antigen presentation signatures, as well as a distinct macrophage profile in the peripheral blood. They also studied lung macrophages, finding that those with severe COVID-19 had increased inflammatory monocytes and monocyte-derived macrophage infiltration, with a corresponding decrease in the alveolar macrophage population. Together, these data suggest that restoring macrophage homeostasis may be a strategy for treating COVID-19. [ABSTRACT FROM AUTHOR]

Published
2022
Full Text
View/download PDF

19. SU086, a New Inhibitor of HSP90, Impairs Glycolysis and Represents a New Treatment Strategy for Advanced Prostate Cancer

Author

Rice, Meghan A., primary, Kumar, Vineet, additional, Tailor, Dhanir, additional, Garcia-Marques, Fernando Jose, additional, Bermudez, Abel, additional, Kanchustambham, Vijayalakshmi, additional, Shankar, Vishnu, additional, Inde, Zintis, additional, Pandrala, Mallesh, additional, Nolley, Rosalie, additional, Liu, Shiqin, additional, Aslan, Merve, additional, Ghoochani, Ali, additional, Agarwal, Arushi, additional, Buckup, Mark, additional, Hsu, En-Chi, additional, Going, Catherine C., additional, Peehl, Donna M., additional, Dixon, Scott J., additional, Zare, Richard N., additional, Brooks, James D., additional, Pitteri, Sharon J., additional, Malhotra, Sanjay V., additional, and Stoyanova, Tanya, additional

Published
2021
Full Text
View/download PDF

20. Plectin is a regulator of prostate cancer growth and metastasis

Author

Buckup, Mark, primary, Rice, Meghan A., additional, Hsu, En-Chi, additional, Garcia-Marques, Fernando, additional, Liu, Shiqin, additional, Aslan, Merve, additional, Bermudez, Abel, additional, Huang, Jiaoti, additional, Pitteri, Sharon J., additional, and Stoyanova, Tanya, additional

Published
2020
Full Text
View/download PDF

21. Sampling the host response to SARS-CoV-2 in hospitals under siege

Author

Charney, Alexander W., Simons, Nicole W., Mouskas, Konstantinos, Lepow, Lauren, Cheng, Esther, Le Berichel, Jessica, Chang, Christie, Marvin, Robert, Del Valle, Diane Marie, Calorossi, Sharlene, Lansky, Alona, Walker, Laura, Patel, Manishkumar, Xie, Hui, Yi, Nancy, Yu, Alex, Kang, Gurpawan, Mendoza, Anthony, Liharska, Lora E., Moya, Emily, Hartnett, Matthew, Hatem, Sandra, Wilkins, Lillian, Eaton, Melody, Jamal, Hajra, Tuballes, Kevin, Chen, Steven T., Tabachnikova, Alexandra, Chung, Jonathan, Harris, Jocelyn, Batchelor, Craig, Lacunza, Jose, Yishak, Mahlet, Argueta, Kimberly, Karekar, Neha, Lee, Brian, Kelly, Geoffrey, Geanon, Daniel, Handler, Diana, Leech, John, Stefanos, Hiyab, Dawson, Travis, Scott, Ieisha, Francoeur, Nancy, Johnson, Jessica S., Vaid, Akhil, Glicksberg, Benjamin S., Nadkarni, Girish N., Schadt, Eric E., Gelb, Bruce D., Rahman, Adeeb, Sebra, Robert, Martin, Glenn, Agashe, Charuta, Agrawal, Priyal, Akyatan, Alara, Alesso-Carra, Kasey, Alibo, Eziwoma, Alvarez, Kelvin, Amabile, Angelo, Ascolillo, Steven, Bailey, Rasheed, Begani, Priya, Correra, Paloma Bravo, Brown, Stacey-Ann, Buckup, Mark, Burka, Larissa, Cambron, Lena, Carrara, Gina, Chang, Serena, Chien, Jonathan, Chowdhury, Mashkura, Bozkus, Cansu Cimen, Comella, Phillip, Cosgrove, Dana, Cossarini, Francesca, Cotter, Liam, Dave, Arpit, Dayal, Bheesham, Dhainaut, Maxime, Dornfeld, Rebecca, Dul, Katie, Eber, Nissan, Elaiho, Cordelia, Fabris, Frank, Faith, Jeremiah, Falci, Dominique, Feng, Susie, Fennessy, Brian, Fernandes, Marie, Gangadharan, Sandeep, Grabowska, Joanna, Gyimesi, Gavin, Hamdani, Maha, Herbinet, Manon, Herrera, Elva, Hochman, Arielle, Hoffman, Gabriel E., Hook, Jaime, Horta, Laila, Humblin, Etienne, Karim, Subha, Kim, Jessica, Lebovitch, Dannielle, Lee, Grace, Lee, Gyu Ho, Lee, Jacky, Leventhal, Mike, Lindblad, Katherine, Livanos, Alexandra, Machado, Rosalie, Mahmood, Zafar, Mar, Kelcey, Maskey, Shrisha, Matthews, Paul, Meckel, Katherine, Mehandru, Saurabh, Mercedes, Cynthia, Meyer, Dara, Mollaoglu, Gurkan, Morris, Sarah, Nie, Kai, Nisenholtz, Marjorie, Ofori-Amanfo, George, Onel, Kenan, Ounadjela, Merouane, Patel, Vishwendra, Pruitt, Cassandra, Rathi, Shivani, Redes, Jamie, Reyes-Torres, Ivan, Rodrigues, Alcina, Rodriguez, Alfonso, Roudko, Vladimir, Ruiz, Evelyn, Scalzo, Pearl, Silva, Pedro, Schanoski, Alessandra Soares, Straw, Meghan, Tabachnikova, Sasha, Teague, Collin, Upadhyaya, Bhaskar, Van Der Heide, Verena, Vaninov, Natalie, Wacker, Daniel, Walsh, Hadley, Wilk, C. Matthias, Wilson, Jessica, Wilson, Karen M., Xue, Li, Yeboah, Naa-akomaah, Young, Sabina, Zaks, Nina, Zha, Renyuan, Marron, Thomas, Beckmann, Noam, Kim-Schulze, Seunghee, Gnjatic, Sacha, and Merad, Miriam

Subjects
Siege, 2019-20 coronavirus outbreak, Coronavirus disease 2019 (COVID-19), biology, business.industry, Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), Host response, Sampling (statistics), General Medicine, biology.organism_classification, Virology, General Biochemistry, Genetics and Molecular Biology, Pandemic, Medicine, business, Betacoronavirus
Published
2020
Full Text
View/download PDF

Catalog

Books, media, physical & digital resources
See catalog results