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1. VWF–ADAMTS13 axis dysfunction in children with sickle cell disease treated with hydroxycarbamide vs blood transfusion

3. von Willebrand factor neutralizing and non-neutralizing alloantibodies in 213 subjects with type 3 von Willebrand disease enrolled in 3WINTERS-IPS

6. Von Willebrand factor propeptide and pathophysiological mechanisms in European and Iranian patients with type 3 von Willebrand disease enrolled in the 3WINTERS‐IPS study

8. Clinical and biochemical endpoints and predictors of response to plasma exchange in septic shock: results from a randomized controlled trial

10. Genotypes of European and Iranian patients with type 3 von Willebrand disease enrolled in 3WINTERS-IPS

11. Shear-induced unfolding and enzymatic cleavage of full-length VWF multimers

13. Bleeding symptoms in patients diagnosed as type 3 von Willebrand disease: Results from 3WINTERS‐IPS, an international and collaborative cross‐sectional study

17. Shear-Induced Unfolding Activates von Willebrand Factor A2 Domain for Proteolysis

23. Von Willebrand factor regulates complement on endothelial cells

25. von Willebrand factor is dimerized by protein disulfide isomerase

27. Elevated von Willebrand factor levels in multiple myeloma: Dysregulated mechanisms of both secretion and clearance

28. ADAMTS-13 activity in stroke of known and unknown cause: Relation to vascular risk factor burden

34. Blood Transfusion Ameliorates Free Haem-Induced Loss of Thrombomodulin and Endothelial Cell Activation in Paediatric Sickle Cell Anaemia

38. Treatment Algorithm for Patients With von Willebrand Syndrome Type 2A and Congenital Heart Disease—A Treatment Algorithm May Reduce Perioperative Blood Loss in Children With Congenital Heart Disease

39. Clinical and biochemical endpoints and predictors of response to plasma exchange in septic shock: results from a randomized controlled trial

41. MO149: Effect of Plasma Exchange on COVID-19 Associated Excess of Von Willebrand Factor and Inflammation in Critically Ill Patients

42. Additional file 1 of Clinical and biochemical endpoints and predictors of response to plasma exchange in septic shock: results from a randomized controlled trial

48. ADAMTS13 regulation of VWF multimer distribution in severe COVID‐19

49. Comprehensive analysis of glomerular mRNA expression of pro- and antithrombotic genes in atypical haemolytic-uremic syndrome (aHUS)

50. Report On Complete Endpoints and Predictors of Response to Plasma Exchange – Results From a Randomized Controlled Trial in Septic Shock Patients

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